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1 g proteins, insulin receptor substrate 1 and insulin receptor substrate 2.
2 th Y497F were not capable of phosphorylating insulin receptor substrate 2.
3  binding to insulin receptor substrate-1 and insulin receptor substrate-2, activation of PI3K and pro
4 results in enhanced IR signaling through the insulin receptor substrate 2-AKT pathway in beta-cells a
5     Moreover, miR-33a and -b also target the insulin receptor substrate 2, an essential component of
6                           Insulin-stimulated insulin receptor substrate 2 and Akt-2 phosphorylation w
7 alues, whereas only VV-TPN increased hepatic insulin receptor substrate 2 and maintained normal hepat
8                  Insulin signaling proteins, insulin receptor substrate 2 and phosphatidylinositol 3
9 , no significant tyrosine phosphorylation of insulin receptor substrate-2 and modulation of the immed
10   IL-10 increased tyrosyl phosphorylation of insulin receptor substrate-2 and stimulated the enzymati
11 ted tyrosine phosphorylation of JAK kinases, insulin receptor substrate-2, and signal transducer and
12 uced cell growth, activation of JAK kinases, insulin receptor substrate-2, and STAT3 and expression o
13 n key insulin-signaling molecules, including insulin receptor substrate-2, and substrate metabolism t
14 ximately 40% reduction in insulin-stimulated insulin receptor substrate-2-associated phosphatidylinos
15 ciated with defects in insulin activation of insulin receptor substrate-2-associated phosphatidylinos
16 cking either insulin receptors (betaIRKO) or insulin receptor substrate 2 (betaIRS2KO).
17 cy appeared necessary for phosphorylation of insulin receptor substrate-2 but not for IGFR1 activatio
18 e-inactive Fes blocks the IL-4 activation of insulin receptor substrate-2, but not STAT6.
19                                              Insulin receptor substrate-2-deficient (IRS2(-/-)) mice
20  kinase 1, insulin receptor substrate 1, and insulin receptor substrate 2, factors with molecular mas
21  observed at D13S285 (LOD = 1.86), where the insulin receptor substrate 2 gene resides.
22 es IGF-I-induced tyrosine phosphorylation of insulin receptor substrate 2 (IRS-2) and inhibits IRS-2-
23                    Appropriate regulation of insulin receptor substrate 2 (IRS-2) expression in pancr
24                                              Insulin receptor substrate 2 (IRS-2) has recently been s
25 oved insulin-stimulated insulin receptor and insulin receptor substrate 2 (IRS-2) phosphorylation, IR
26                                              Insulin receptor substrate 2 (IRS-2) plays a critical ro
27 ubsequent 52% decrease in insulin-stimulated insulin receptor substrate 2 (IRS-2) tyrosine phosphoryl
28                            The gene encoding insulin receptor substrate 2 (IRS-2), a mediator of insu
29 cAMP response element-binding protein (CREB)-insulin receptor substrate 2 (Irs-2), and increased beta
30  the brain, targets insulin receptor (INSR), insulin receptor substrate 2 (IRS-2), and insulin-degrad
31 that promotes beta-cell growth and survival, insulin receptor substrate 2 (IRS-2), is a member of a f
32  insulin-induced tyrosine phosphorylation of insulin receptor substrate 2 (IRS-2), whereas it had min
33                                              Insulin receptor substrate-2 (IRS-2) belongs to the IRS
34 wild-type (WT) and mutant hIL-4Ralpha in 32D/insulin receptor substrate-2 (IRS-2) cells.
35 related with tyrosine phosphorylation of the insulin receptor substrate-2 (IRS-2) in macrophages.
36                                              Insulin receptor substrate-2 (IRS-2) is a multisite dock
37                                              Insulin receptor substrate-2 (IRS-2) is phosphorylated o
38          In this report, we demonstrate that insulin receptor substrate-2 (IRS-2) is tyrosyl-phosphor
39                                We found that insulin receptor substrate-2 (IRS-2) levels were markedl
40                                              Insulin receptor substrate-2 (IRS-2) plays a critical ro
41                                              Insulin receptor substrate-2 (IRS-2) plays an essential
42                Here we show that deletion of insulin receptor substrate-2 (IRS-2), a component of the
43  to abused drugs decreases the expression of insulin receptor substrate-2 (IRS-2; a protein involved
44  GM-CSF) induced tyrosine phosphorylation of insulin-receptor substrate-2 (IRS-2) and its association
45 ptor (Igf1r(+/-)) are bred with mice lacking insulin receptor substrate 2 (Irs2(-/-)), the resulting
46 teins involved in insulin signaling, such as insulin receptor substrate 2 (IRS2) and glucose transpor
47                 The recent identification of insulin receptor substrate 2 (IRS2) as a central player
48 3-CDK4 complex, which in turn phosphorylates insulin receptor substrate 2 (IRS2) at serine 388, there
49  component of all types of diabetes, and the insulin receptor substrate 2 (IRS2) branch of signaling
50                                          The insulin receptor substrate 2 (Irs2) branch of the insuli
51 a/cAMP mediates these actions by stimulating insulin receptor substrate 2 (IRS2) expression.
52  Here we show that a conditional knockout of insulin receptor substrate 2 (Irs2) in mouse pancreas be
53                                              Insulin Receptor Substrate 2 (IRS2) is a signaling adapt
54 s, proliferation, and survival by increasing insulin receptor substrate 2 (IRS2) levels and identify
55                                              Insulin receptor substrate 2 (IRS2) suppression induced
56 s is sufficient to enhance the expression of insulin receptor substrate 2 (IRS2) to levels observed i
57 MR-409 induces Akt signaling by induction of insulin receptor substrate 2 (IRS2), a master regulator
58 2)-associated binding proteins 1-3 (GAB1-3), insulin receptor substrate 2 (IRS2), docking protein 1 (
59  mice lacking insulin signaling intermediate insulin receptor substrate 2 (IRS2), we confirmed that h
60 The study also provides strong evidence that insulin receptor substrate 2 (Irs2), which is known to h
61 ronic morphine-induced downregulation of the insulin receptor substrate 2 (IRS2)-thymoma viral proto-
62  of sarcomas and found a marked induction of insulin receptor substrate-2 (IRS2) and phosphorylated A
63                                          The insulin receptor substrate-2 (Irs2) branch of the insuli
64                                 Mice lacking insulin receptor substrate-2 (Irs2) develop beta cell fa
65 hat mice heterozygous for a null mutation in insulin receptor substrate-2 (Irs2) display a 17% increa
66 3K signaling leads to feedback inhibition of insulin receptor substrate-2 (IRS2) expression, an upstr
67                                              Insulin receptor substrate-2 (Irs2) is a critical mediat
68            Here, we show that, in mice, less insulin receptor substrate-2 (Irs2) signaling throughout
69                                  We identify insulin receptor substrate-2 (Irs2), a known cAMP-respon
70 ing through direct and indirect induction of insulin receptor substrate-2 (Irs2), an essential insuli
71 s (week 15); liver and adipose AhR and IRS2 (insulin receptor substrate 2) mRNA abundance, and PCB-77
72 ysis occurs normally in B cells deficient in insulin receptor substrate-2 or the p85alpha subunit of
73 d the downstream signaling pathway involving insulin receptor substrate 2, phosphatidylinositol 3'-ki
74                                          The insulin receptor substrate 2/phosphatidylinositol 3'-kin
75 s with inhibition of NF-kappaB showed normal insulin receptor substrate-2 phosphorylation and only a
76 reduction of p85 beta specifically increases insulin receptor substrate-2 phosphorylation.
77 vity is totally due to IL-10 stimulating the insulin receptor substrate-2/PI 3-kinase/Akt pathway, wh
78         In a separate cohort, phosphorylated insulin receptor substrate-2 (pIRS-2) and insulin growth
79                  In addition, an increase in insulin receptor substrate-2 protein and a differential
80 eas insulin receptor autophosphorylation and insulin receptor substrate 2 tyrosine phosphorylation we
81 pression of glucose transporters 1 and 4 and insulin receptor substrate 2 was increased and that of p
82 sducer and activator of transcription 6, and insulin receptor substrate 2 was strikingly but transien
83 ruitment of phosphatidylinositol 3-kinase to insulin receptor substrate-2 was also detected.
84 nsulin-dependent tyrosine phosphorylation of insulin receptor substrate-2 was enhanced in the p85beta
85   The hepatic mRNA level of the HIF-2 target insulin receptor substrate-2 was higher, whereas that of
86 ic screen of HD, we found that activation of insulin receptor substrate-2, which mediates the signali
87 lts in decreased tyrosine phosphorylation of insulin receptor substrate-2 with impeded insulin signal