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1 egradation by enzymes such as neprilysin and insulin degrading enzyme.
2 mechanisms, including elevated expression of insulin-degrading enzyme.
3 ulin clearance due to down-regulation of the insulin-degrading enzyme.
4  of two endopeptidases, neprilysin (NEP) and insulin-degrading enzyme.
5 ecursor protein, presenilin 1, neprylisin or insulin-degrading enzyme.
6 retion and by inhibiting its degradation via insulin-degrading enzyme.
7 onstrated this activity can be attributed to insulin degrading enzyme, a previously described member
8 olytic degradation of monomeric Abeta by the insulin-degrading enzyme, a major Abeta-degrading enzyme
9 lar ADDLs applied to cells via seemingly the insulin-degrading enzyme activity.
10 TNF-alpha directly reduced the expression of insulin degrading enzyme and chaperone molecules (heat s
11 activities of other Abeta-degrading enzymes, insulin degrading enzyme and neprilysin, were unchanged.
12 e zinc-dependent insulin-degrading proteases insulin-degrading enzyme and cathepsin D were impaired;
13 minal fragment, and Abeta-degrading enzymes (insulin-degrading enzyme and neprilysin) in APP/CCL2 and
14 transition metal is an essential cofactor in insulin-degrading enzyme and several key Zn2+ finger tra
15 with Cav-1 and caveolae-associated proteins, insulin-degrading enzyme and the tight junction protein
16 AD) markers, synaptophysin, APP, neprilysin, insulin-degrading enzyme and transthyretin in MCAT, Abet
17 nt of mRNA/protein changes in neprilysin and insulin-degrading enzyme and, instead, may involve neuro
18 ned, including endothelin-converting enzyme, insulin-degrading enzyme, and neprilysin.
19 e endothelin-converting enzymes, neprilysin, insulin-degrading enzyme, and plasmin.
20     Insulin resistance and enzymes including insulin degrading enzymes are implicated in AD where bre
21                                   A role for insulin-degrading enzyme as both a beta-endorphin-proces
22 e-2 diabetes, and the identification of IDE (insulin-degrading enzyme) as a diabetes susceptibility g
23                   The steady-state levels of insulin-degrading enzyme did not change significantly, w
24 metabolism and alternate processing, such as insulin degrading enzyme, endothelin-converting enzyme-1
25                         Cysteine residues in insulin degrading enzyme have been reported as non-criti
26 sed gamma-secretase activities and decreased insulin degrading enzyme (IDE) activities.
27 0 and Abeta42) with the fully active form of insulin degrading enzyme (IDE) through unrestrained, all
28 nverting the twelve cysteine residues in rat insulin degrading enzyme (IDE) to serines resulted in a
29                                              Insulin degrading enzyme (IDE) utilizes a large catalyti
30      Reduction of cell surface expression of insulin degrading enzyme (IDE), which cleaves the C-term
31 es PTEN through lysosome pathway mediated by insulin degrading enzyme (IDE).
32 bled vimentin/nestin complexes interact with insulin degrading enzyme (IDE).
33 ed clearance and degradation of amyloid-B by insulin degrading enzyme (IDE).
34                                              Insulin-degrading enzyme (IDE) (insulysin) is a zinc met
35 increase in NO results in S-nitrosylation of insulin-degrading enzyme (IDE) and dynamin-related prote
36 Previously, we showed that gE interacts with insulin-degrading enzyme (IDE) and facilitates VZV infec
37 hat they result in altered expression of the insulin-degrading enzyme (Ide) and fibroblast growth fac
38                  Recent reports suggest that insulin-degrading enzyme (IDE) and neutral endopeptidase
39                                              Insulin-degrading enzyme (IDE) can degrade insulin and a
40                                    Mammalian insulin-degrading enzyme (IDE) cleaves insulin, among ot
41                                              Insulin-degrading enzyme (IDE) exists primarily as a dim
42 cently found that homozygous deletion of the insulin-degrading enzyme (IDE) gene in mice results in a
43                                              Insulin-degrading enzyme (IDE) has been implicated as a
44                                              Insulin-degrading enzyme (IDE) hydrolyzes bioactive pept
45                  We investigated the role of insulin-degrading enzyme (IDE) in amylin degradation, am
46                                  Deletion of insulin-degrading enzyme (IDE) in mice causes accumulati
47                  Recent studies suggest that insulin-degrading enzyme (IDE) in neurons and microglia
48                                We found that insulin-degrading enzyme (IDE) interacts with gE through
49                                              Insulin-degrading enzyme (IDE) is a component of a cytos
50                                              Insulin-degrading enzyme (IDE) is a highly conserved zin
51                                Inhibition of insulin-degrading enzyme (IDE) is a possible target for
52                                              Insulin-degrading enzyme (IDE) is a protease that cleave
53                                              Insulin-degrading enzyme (IDE) is a ubiquitous zinc-meta
54                                              Insulin-degrading enzyme (IDE) is a zinc metalloprotease
55                                              Insulin-degrading enzyme (IDE) is a zinc metalloprotease
56                                              Insulin-degrading enzyme (IDE) is an atypical zinc-metal
57                                              Insulin-degrading enzyme (IDE) is identified as such a p
58                                              Insulin-degrading enzyme (IDE) is involved in the cleara
59                                              Insulin-degrading enzyme (IDE) is one of the proteins th
60  involved in mitAbeta catabolism is the long insulin-degrading enzyme (IDE) isoform (IDE-Met(1)).
61                  Recent reports suggest that insulin-degrading enzyme (IDE) may have specificity for
62 re we show that transgenic overexpression of insulin-degrading enzyme (IDE) or neprilysin (NEP) in ne
63 a hormone vital for glucose homeostasis, and insulin-degrading enzyme (IDE) plays a key role in its c
64                                              Insulin-degrading enzyme (IDE) selectively degrades the
65 ves gamma-secretase activation and decreased insulin-degrading enzyme (IDE) steady-state levels in a
66                                              Insulin-degrading enzyme (IDE) was found to rapidly clea
67  and the active site histidine (His(112)) of insulin-degrading enzyme (IDE) were mutated.
68                                              Insulin-degrading enzyme (IDE), a 110-kDa metalloendopep
69 e and find that it is indistinguishable from insulin-degrading enzyme (IDE), a thiol metalloendopepti
70                                              Insulin-degrading enzyme (IDE), a Zn2+-metalloprotease,
71                            Two substrates of insulin-degrading enzyme (IDE), amyloid beta-protein (Ab
72  key finding was the decreased expression of insulin-degrading enzyme (IDE), an enzyme that regulates
73 reviously showed that the amyloid scavenger, insulin-degrading enzyme (IDE), generates T40-derived am
74 ), insulin receptor substrate 2 (IRS-2), and insulin-degrading enzyme (IDE), key regulators of insuli
75 main that interacts with a putative receptor insulin-degrading enzyme (IDE), replicated as extensivel
76 cated in the non-coding region (intron 1) of insulin-degrading enzyme (IDE), was the most strongly as
77 genetic system for functional studies of the insulin-degrading enzyme (IDE), which cleaves and inacti
78 terminal region also mediates binding to the insulin-degrading enzyme (IDE), which is proposed to be
79     One candidate gene in this region is the insulin-degrading enzyme (IDE), which, in the GK rat mod
80                              The rate of the insulin-degrading enzyme (IDE)-catalyzed hydrolysis of t
81 grading enzymes such as neprilysin (NEP) and insulin-degrading enzyme (IDE).
82 fter purification, is indistinguishable from insulin-degrading enzyme (IDE).
83                                              Insulin-degrading enzyme (IDE, insulysin) is the best ch
84 lities to alter the substrate-selectivity of insulin-degrading enzymes (IDE).
85                             It is known that insulin-degrading-enzyme (IDE) plays a crucial role in t
86 adation in MDM is sensitive to lysosomal and insulin degrading enzyme inhibitors but insensitive to p
87 panning the 5' untranslated region region of insulin-degrading enzyme is associated with serum cotini
88 ilarly, Abeta degradation extracellularly by insulin-degrading enzyme is facilitated by ApoE.
89          Each of the ICDs are degraded by an insulin degrading enzyme-like activity, but they can be
90 ing enzyme 1, but positively correlated with insulin-degrading enzyme, neprilysin and P-glycoprotein.
91 l new biomarkers, a P. falciparum homolog of insulin-degrading enzyme (PfIDEh) met our search criteri
92 r sites as wild-type Abeta by neprilysin and insulin-degrading enzyme, the two most widely studied Ab
93 o a 1-h incubation with either neprilysin or insulin degrading enzyme, whereas the monomer is rapidly
94              Pretreatment of the medium with insulin-degrading enzyme, which degrades Abeta monomers
95 rotease Ste23 in yeast, a homologue of human insulin-degrading enzyme, which is required for efficien