戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 fficking that contributes to invasion is the integrin alpha6beta4.
2 wn to form a complex with the hemidesmosomal integrin alpha6beta4.
3  Rac1 activation upon laminin-332 binding to integrin alpha6beta4.
4                                          The integrin alpha6beta4, a laminin receptor that stabilizes
5                              In normal mice, integrin alpha6beta4, a receptor for laminin alpha5, is
6                            Expression of the integrin alpha6beta4, a receptor for the laminin family
7                 Knockdown of laminin-binding integrins (alpha6beta4, alpha3beta1) or associated tetra
8 ponse of the actin cytoskeleton and specific integrins (alpha6beta4, alpha6beta1, and alpha3beta1) to
9                        Our results show that integrin alpha6beta4 altered the expression of 538 genes
10 diated by transmembrane proteins such as the integrin alpha6beta4 and bullous pemphigoid antigen 2 wi
11 t that the interaction between laminin-10/11-integrin alpha6beta4 and the phosphatidylinositol 3-kina
12                                 Keratinocyte integrins alpha6beta4 and alpha3beta1 bind laminin-5, a
13 age and motility of epithelial cells through integrins alpha6beta4 and alpha3beta1, respectively.
14 n expression patterns, in which the exosomal integrins alpha6beta4 and alpha6beta1 were associated wi
15                                Targeting the integrins alpha6beta4 and alphavbeta5 decreased exosome
16 e effects by forming a complex with HER2 and integrin alpha6beta4 at the cell surface that disrupts d
17 en established, this study demonstrates that integrin alpha6beta4 can dramatically impact the epigeno
18            In summary, our data suggest that integrin alpha6beta4 confers a motile and invasive pheno
19  Likewise, we provide the novel finding that integrin alpha6beta4 confers an enhanced ability on cell
20 utant BM could not induce stable adhesion by integrin alpha6beta4, consistent with the presence of ju
21          Based on previous observations that integrin alpha6beta4 cooperates with c-Met in pancreatic
22                            We determine that integrin alpha6beta4-dependent overexpression of autotax
23 ingle cells having similar expression of the integrin alpha6beta4 dimer, single cells demonstrated hi
24 ow that keratinocytes lacking hemidesmosomal integrin alpha6beta4 exhibit increased focal adhesion fo
25 rate that increased autotaxin secretion from integrin alpha6beta4 expressing cells acts to enhance ch
26 er strongly and specifically bind NFAT1 from integrin alpha6beta4 expressing cells.
27 tment of lung disease by identifying a novel integrin alpha6beta4-expressing alveolar epithelial cell
28       S100A4 expression correlated well with integrin alpha6beta4 expression in established cell line
29 ethylation were up-regulated dramatically by integrin alpha6beta4 expression, including S100A4, FST,
30 n abrogates strictly polarized expression of integrin alpha6beta4 in basal keratinocytes and negative
31 tor (EGF-R) combines with the hemidesmosomal integrin alpha6beta4 in both normal and neoplastic kerat
32 tic cancer cells showed increased loading of integrin alpha6beta4 into sEVs-a process that required C
33                                          The integrin alpha6beta4 is an essential component of hemide
34                                              Integrin alpha6beta4 is an essential, dynamic adhesion r
35                                          The integrin alpha6beta4 is associated with carcinoma progre
36 r of the epithelium, whereas in mutant mice, integrin alpha6beta4 is expressed around the cell surfac
37                                              Integrin alpha6beta4 is highly expressed in pancreatic c
38                            Like alphavbeta3, integrin alpha6beta4 is overexpressed in many cancers an
39                                              Integrin alpha6beta4 is up-regulated in pancreatic adeno
40 iles of MDA-MB-435 cells that stably express integrin alpha6beta4 (MDA/beta4) and vector-only-transfe
41                  We further demonstrate that integrin alpha6beta4 regulates the activity of the mecha
42 ing to residual expression of laminin 332 or integrin alpha6beta4, respectively.
43 cytes and negatively impacts the laminin-332/integrin alpha6beta4 signaling axis guiding keratinocyte
44 y bisulfate sequencing, thus suggesting that integrin alpha6beta4 signaling can lead to the demethyla
45                                              Integrin alpha6beta4 signaling proceeds through Src fami
46 me bisulfite sequencing (WGBS) revealed that integrin alpha6beta4 signaling promotes an overall hypom
47 eover, matrix metalloproteinase activity and integrin alpha6beta4 signaling were required for AREG se
48  invasive and motile phenotype downstream of integrin alpha6beta4 signaling.
49   Taken together, these data illustrate that integrin alpha6beta4 stimulates invasion by promoting au
50  laminin 5, collagen, or an antibody against integrin alpha6beta4, suggesting that signaling through
51 ed, in part, by JNK-suppressed expression of integrin alpha6beta4 that binds HER2 and amplifies HER2
52 e many known signaling functions mediated by integrin alpha6beta4 that promote invasive properties ha
53                 In this study, we found that integrin alpha6beta4 up-regulates several genes in the e
54          The correlation of these genes with integrin alpha6beta4 was confirmed in The Cancer Genome
55 ich mediates rapid tumor cell migration, and integrin alpha6beta4, which often mediates stable cell a