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1 DL) cells have been shown to express several integrins (alphav, alpha5, beta1, beta3) that use RGD (a
2 negative IkappaBalpha perturbed ET-1-induced integrin alphaV and integrin beta1 expression.
3 ed with lymph node metastasis, expression of integrin alphaV and KRAS mutation status.
4 rtic root aneurysm tissue confirmed elevated integrin alphaV and reduced MRC2 in clinical disease spe
5                                              Integrin-alphaV and -beta1, fibronectin, and versican co
6 ug, as well as metastasis-related integrins (integrin-alphav and integrin-beta1).
7 ed a direct physical interaction of MBD with integrins alphav and beta1, caveolin-1, and transferrin
8                   The level of expression of integrins alphav and beta3 and the amount of assembled F
9   In addition, a cross-talk between EMP2 and integrins alphaV and beta3 was shown in the regulation o
10              In contrast, expression of Mer, integrin alphaV, and NPC1 was required for efficient GP-
11  a tumor vasculature-specific antiangiogenic integrin alphav antagonist and tumor-specific antibody-i
12 urthermore, simultaneous treatments with the integrin alphav antagonist and tumor-specific antibody-I
13  monotherapies with either an antiangiogenic integrin alphav antagonist or antibody-IL-2 fusion prote
14 Systemic administration of neutralizing anti-integrin alphaV antibody or a genetic deficiency of inte
15         Our study demonstrates that the sOPN/integrin alphav axis, which induces T cell chemotaxis to
16 st, is inhibited by blocking the other beta1 integrins, alphav beta1 and alpha5 beta1.
17 of mouse mAb LM609 that is directed to human integrin alphav beta3 and has potential applicability in
18    We have shown that osteopontin binding to integrin alphav beta3 in osteoclasts stimulates gelsolin
19 NA plasmids encoding the individual complete integrin alphaV, beta3, and beta6 subunits were used to
20 fibronectin, and gelatin, and mRNA levels of integrin alphav/beta3 were determined.
21 ine osteoclast precursors bind matrix is the integrin alphav beta5 and that granulocyte-macrophage co
22                                      Soluble integrin alphav beta5 reacted with seven different Ad se
23     We report here the expression of soluble integrin alphav beta5, which retains the ability to reco
24                        Finally, we show that integrin alphaV/beta5 acts as the irisin receptor on ast
25  conclude that deletion or inhibition of the integrin alphav beta6 did not protect animals from P. ae
26                                  Deletion of integrin alphav beta6 has been associated with significa
27                                              Integrin alphaV can form heterodimers with several beta
28 ne and C1q as well as the "eat-me" receptors integrin-alphav (CD51) and CD36 in cellular uptake.
29                                         More integrin alphaV + cells are observed in immunotherapy ex
30 s revealed that PKCalpha was up-regulated by integrin alphav in a three-dimensional microenvironment-
31                             In mice, lack of integrin alphav in the immune system resulted in loss of
32 (2) regulates the amount and distribution of integrin alphaV in the plasma membrane.
33 on and blocked the signaling transduction by integrin alphaV, inhibited migration signaling pathways
34 ed that milk OPN increased the expression of integrin alphav, integrin beta3, and CD44 in jejunum of
35 ression of the cell surface molecules alpha6-integrin, alphav-integrin, and the c-kit receptor.
36              These findings suggest that the integrin alphaV is developmentally regulated, has a dist
37                         Src interaction with integrin alphaV is required for integrin alphaV-mediated
38  that Periostin (Postn) via interaction with Integrin-alphav (Itgav) regulates HSC proliferation.
39 alpha constitutes a crucial component of the integrin alphav-mediated pathway(s) that promote p53 rel
40 s stable knockdown of PKCalpha inhibited the integrin alphav-mediated relocalization of p53.
41 raction with integrin alphaV is required for integrin alphaV-mediated Src activation, and the subsequ
42       A pathway was identified that involved integrin alphav-mediated up-regulation of PKCalpha and P
43  functionally linked to higher expression of integrin alphaV on effector CD4(+) T cells.
44 ectin and vitronectin, by down-regulation of integrin alphav, or by a peptide corresponding to 13 aa
45 to macrophages and, considering that Mer and integrin alphaV promote phagocytosis of apoptotic cells,
46 Mechanistically, the reduction of functional Integrin alphaV resulted in the dysregulation of several
47 zation of sOPN and blockade of its receptor, integrin alphav, significantly inhibited CD4(+) T cell m
48                      Our results showed that integrin alphav-specific MAb reduced infectivity by 85%.
49 neration of Tregs required expression of the integrin alphav subunit by DCs; mice that lacked alphav
50                            Expression of the integrin alphav subunit was measured in purified subpopu
51 TSRI265) selected for its ability to bind to integrin alphav(v)beta3 and block alpha(v)beta3 interact
52                                              Integrin alphav was observed to promote a relocalization
53 ntegrin alpha5 whereas surface expression of integrin alphaV was unaffected.
54 microenvironments requires the expression of integrin alphav, which acts to suppress p53 activity.