ライフサイエンスコーパス: interferon-stimulated gene

1 IV infection, consistent with its role as an interferon-stimulated gene.

2 eral other PML-NB proteins, is encoded by an interferon-stimulated gene.

3 nd initiation of mRNA translation of type II interferon-stimulated genes.

4 ed synthesis of IFN-beta and upregulation of interferon-stimulated genes.

5 tiviral mechanism, without expression of the interferon-stimulated genes.

6 downregulates mRNA levels of type I IFN and interferon-stimulated genes.

7 cted mice revealed the induction of multiple interferon-stimulated genes.

8 the expression of type I IFNs and antiviral interferon-stimulated genes.

9 ing to enhance the expression of a subset of interferon-stimulated genes.

10 l antiviral cytokine, IFNB1, and a subset of interferon-stimulated genes.

11 n from highly regulated genes, including the interferon-stimulated genes.

12 1 phosphorylation and expression of numerous interferon-stimulated genes.

13 STAT2 phosphorylation and the expression of interferon-stimulated genes.

14 RABV and facilitates the production of some interferon-stimulated genes.

15 local antiviral states through expression of interferon-stimulated genes.

16 The 30 upregulated genes included 25 interferon-stimulated genes.

17 shown that ribavirin increases induction of interferon-stimulated genes.

18 ted genes, and block antiviral activities of interferon-stimulated genes.

19 pathway, and to interfere with induction of interferon-stimulated genes.

20 regulatory factors (IRFs), which upregulate interferon-stimulated genes.

21 ctors (IRFs) 3 and 7, type I interferon, and interferon-stimulated genes.

22 ation while simultaneously activating type I interferon-stimulated genes.

23 We report that the protein encoded by interferon stimulated gene 12 (ISG12), is a novel intera

24 Interferon stimulated gene 15 (ISG15) is a ubiquitin-lik

25 The signature's genes are: Interferon Stimulated Gene 15 (ISG15), Interleukin 16 (I

26 eport that vIRF1 interacts with the cellular interferon-stimulated gene 15 (ISG15) E3 ligase, HERC5,

27 This report reveals that interferon-stimulated gene 15 (ISG15) mitigates MNV-1 re

28 on of the oncogenic Kirsten-Ras (Ki-Ras) and interferon-stimulated gene 15 (ISG15) pathways is common

29 Accumulation of the interferon-stimulated gene 15 (ISG15) protein product, w

30 ce deficient in free and conjugated forms of interferon-stimulated gene 15 (ISG15) revealed that ISG1

31 virus replication through expression of the interferon-stimulated gene 15 (ISG15), a dimer homologue

32 We investigated interferon-stimulated gene 15 (ISG15), a poorly understo

33 virus (CCHFV), to cleave the Ub-like protein interferon-stimulated gene 15 (ISG15), a protein involve

34 s to coexpress PLpro and a substrate, murine interferon-stimulated gene 15 (ISG15), and found that PL

35 Cellular activation induced expression of interferon-stimulated gene 15 (ISG15), tripartite motif

36 FN-alpha/beta]) stimulates the expression of interferon-stimulated gene 15 (ISG15), which encodes a u

37 We report three genes (ISG15 [interferon-stimulated gene 15 kd], DSP [Desmoplakin], an

38 The ubiquitin-like modifier ISG15 (interferon-stimulated gene 15) is induced by interferons

39 The mRNA of interferon-stimulated gene-15 (ISG-15), also known as IS

40 cluding computational docking studies and an interferon-stimulated gene 54 (ISG54)-luciferase reporte

41 The interferon-stimulated gene 56 (ISG56) family is induced

42 upregulation of IRF3 primary response genes (interferon-stimulated gene 56 [ISG56], beta IFN [IFN-bet

43 7 days post infection, with no induction of interferon-stimulated genes after 3 days.

44 ol 25-hydroxylase (CH25H) is expressed as an interferon-stimulated gene and mediates antiviral activi

45 virus blocked the transcription of multiple interferon-stimulated genes and also downregulated a net

46 eron genes (STING; N153S) upregulates type I interferon-stimulated genes and causes perivascular infl

47 Trisomy 21 cells show increased induction of interferon-stimulated genes and decreased expression of

48 that the transcription factor PLZF activates interferon-stimulated genes and facilitates natural kill

49 including the induction of a large number of interferon-stimulated genes and histological evidence of

50 ranscripts in expression of TNF-alpha, other interferon-stimulated genes and induction of apoptosis.

51 pigenomic changes and augments expression of interferon-stimulated genes and ligands for multiple T c

52 The expression of interferon-stimulated genes and selected mediators were

53 ed by virus than are several other classical interferon-stimulated genes and that viral induction of

54 mice exhibited a defect in the expression of interferon-stimulated genes and were unable to clear the

55 ability to activate STAT1, the expression of interferon stimulated genes, and ultimately to the anti-

56 rt Jak-STAT signaling to limit expression of interferon-stimulated genes, and block antiviral activit

57 downregulating the translation of antiviral interferon-stimulated genes, and by co-opting SG protein

58 r signaling, control of viral replication by interferon-stimulated genes, and clearance of virus-tran

59 In MDMs, viperin was the most up-regulated interferon-stimulated genes, and it significantly inhibi

60 y be involved in transcription repression of interferon-stimulated genes, and its association with se

61 ic cells, to distinct expression patterns of interferon-stimulated genes, and to the expression of th

62 Vitamin D related and interferon stimulated genes are good candidates as they

63 NAi components and homologs of antiviral and interferon-stimulated genes are also dsRNA-activated gen

64 or the proper shutdown of a subset of type I interferon-stimulated genes as inflammation subsides, pl

65 First, the pretreatment level of interferon-stimulated genes as well as genetic determina

66 Further, LUCAT1 limits transcription of interferon stimulated genes by interacting with STAT1 in

67 G37S mutation led to increased expression of interferon-stimulated genes dependent on the cGAS-STING

68 ' ability to express type I IFN and activate interferon-stimulated genes during subsequent infection

69 cases identified the limited upregulation of interferon-stimulated genes early in infection in severe

70 Consistently, transcription of interferon-stimulated genes (eg, OAS1, ISG15, Mx1; each

71 e that acted on host histone H2BJ to promote interferon-stimulated gene expression and on viral 3C pr

72 edia from HPAIV-infected cells, p38 controls interferon-stimulated gene expression by coregulating ST

73 A striking absence of interferon-stimulated gene expression in patients with K

74 ngle cell transcriptomics, we establish that interferon-stimulated gene expression is induced in micr

75 Interferon-stimulated gene expression was upregulated in

76 herapeutic interventions and associations of interferon-stimulated gene expression with patient progn

77 aramyxovirus infection to activate IRF-3 and interferon-stimulated gene expression, but they failed t

78 d the following pharmacodynamics parameters: interferon-stimulated gene expression, cytokine and chem

79 proach revealed a link between: 1) increased interferon-stimulated gene expression, IFNalpha levels,

80 MEM173)-IRF3-dependent signalling to elevate interferon-stimulated gene expression, potentiate type I

81 ctors in vivo is to closely monitor signs of interferon-stimulated gene expression, since a narrow wi

82 d CXCL10, as well as type I interferons, and interferon-stimulated gene expression.

83 IFN synthesis followed by JAK/STAT-dependent interferon-stimulated gene expression.

84 Although IFN-I signal via the interferon-stimulated gene factor 3 (ISGF3) complex cons

85 ngs imply that type I IFN signaling [through interferon-stimulated gene factor 3 (ISGF3)] is surprisi

86 (STAT1) and STAT2, which form heterotrimers (interferon-stimulated gene factor 3 [ISGF3]) with interf

87 ted in the absence of IKKepsilon because the interferon-stimulated gene factor 3 complex (ISGF3) does

88 e I infection also inhibits IFN-beta-induced interferon-stimulated gene factor 3-mediated gene expres

89 ittle is known about the mechanisms by which interferon-stimulated genes function to inhibit viral re

90 MMR-related febrile seizures, harboring the interferon-stimulated gene IFI44L (rs273259: P = 5.9 x 1

91 ivate the IRF7 antiviral program and type II interferon-stimulated genes implicated in antigen-presen

92 myeloid cells increases expression of type I interferon stimulated genes in response to LPS.

93 Furthermore, our work identifies ACE2 as an interferon-stimulated gene in lung cells, suggesting tha

94 gulated expression of type I interferons and interferon-stimulated genes in both the duodenum and lun

95 ments provoked lower levels of expression of interferon-stimulated genes in cells than wild type-deri

96 HIV-1(BaL) induced a subset of interferon-stimulated genes in monocyte-derived macropha

97 tance protein A in the skin and induction of interferon-stimulated genes in peripheral blood cells de

98 N-beta) response by increasing expression of interferon-stimulated genes in the lung.

99 pe STAT2 in mediating the expression of many interferon-stimulated genes, in protecting cells against

100 ase state is associated with upregulation of interferon-stimulated genes, indicating a possible role

101 rent MOIs are correlated with differences in interferon-stimulated gene induction, indicating that th

102 rus concentrations also resulted in distinct interferon-stimulated gene induction.

103 ection that can also be enhanced by blocking interferon-stimulated gene induction.

104 Here, we examine the influence of the interferon-stimulated gene IRAV (FLJ11286) on dengue vir

105 Whether the expression of interferon-stimulated genes is a marker of ongoing virem

106 compound 54 effectively induced a transient interferon stimulated gene (ISG) response in mice and cy

107 S-CoV with aberrant cytokine, chemokine, and Interferon Stimulated Gene (ISG) responses in patients p

108 in humanized mice and modelled intrahepatic interferon stimulated gene (ISG) responses.

109 One pattern shows high local expression of interferon stimulated genes (ISG(high)) and cytokines, h

110 Interferon-stimulated gene (ISG) 15 mediates antiviral r

111 Human myxovirus resistance 2 (MX2/MXB) is an interferon-stimulated gene (ISG) and was recently identi

112 antiviral signaling protein (MAVS)/TBK1/IRF3/interferon-stimulated gene (ISG) axis, causing either an

113 The bromodomain inhibitor JQ1 suppressed interferon-stimulated gene (ISG) expression and in combi

114 type I IFN receptor IFNAR exhibited impaired interferon-stimulated gene (ISG) expression and, in the

115 heral blood mononuclear cells and quantified interferon-stimulated gene (ISG) expression in CD4 T cel

116 Viral kinetics and interferon-stimulated gene (ISG) expression in periphera

117 -infected chimpanzees in whose livers type I interferon-stimulated gene (ISG) expression is strongly

118 served in females are associated with higher interferon-stimulated gene (ISG) expression levels in T

119 nhanced interferon signaling, as measured by interferon-stimulated gene (ISG) expression, and decreas

120 w constitutive up-regulation of inflammatory interferon-stimulated gene (ISG) expression, which is me

121 directs alpha/beta interferon production and interferon-stimulated gene (ISG) expression, which limit

122 encountered is predominantly mediated by the interferon-stimulated gene (ISG) family.

123 Cholesterol 25-hydroxylase (CH25H) as an interferon-stimulated gene (ISG) has recently been shown

124 ce, we defined the antiviral function of the interferon-stimulated gene (ISG) Ifit2 in limiting infec

125 rikingly, we discovered that ACE2 is a human interferon-stimulated gene (ISG) in vitro using airway e

126 resent evidence that MHV infection can delay interferon-stimulated gene (ISG) induction mediated by b

127 III IFNs differed greatly in their level of interferon-stimulated gene (ISG) induction with a clearl

128 Interferon-stimulated gene (ISG) products take on a numb

129 esses peripheral type I interferon (IFN) and interferon-stimulated gene (ISG) responses, including th

130 alian target of rapamycin (mTOR) to suppress interferon-stimulated gene (ISG) responses.

131 Human Schlafen 11 (SLFN11) is an interferon-stimulated gene (ISG) that we previously have

132 al model of disease, identifying significant interferon-stimulated gene (ISG) transcript upregulation

133 UL46-expressing cell lines did not activate interferon-stimulated gene (ISG) transcription following

134 with tetratricopeptide repeats 2 (Ifit2), an interferon-stimulated gene (ISG) with possible RNA-bindi

135 ACE2 has been proposed as an interferon-stimulated gene (ISG).

136 ruses did not evoke a detectable IFN-beta or interferon-stimulated gene (ISG; MX1, oligoadenylate syn

137 used a bioinformatic screen to identify two interferon-stimulated genes (ISG) with poorly characteri

138 n in hBMECs and inhibit the transcription of interferon-stimulated genes (ISG).

139 in-activating enzyme (UBE1L) associates with interferon-stimulated gene ISG15 that binds and represse

140 emonstrates that expression of a single host interferon-stimulated gene, ISG15, restricts HCMV replic

141 basal levels of type I interferon (IFN) and interferon stimulated genes (ISGs) and causes blunted in

142 hepatitis C (CHC) have ongoing expression of interferon stimulated genes (ISGs) in the liver.

143 The induced IFNs activate many interferon stimulated genes (ISGs) that have direct anti

144 feron alpha (IFN-alpha) and the induction of interferon stimulated genes (ISGs), as well as the expre

145 4 hours before each IFN injection to measure interferon stimulated genes (ISGs).

146 between IFN-alpha and IL28B stimulation for interferon stimulated genes (ISGs).

147 Hs being impaired in their ability to induce interferon stimulated genes (ISGs).

148 Interferon-stimulated genes (ISGs) act in concert to pro

149 Here, we screen a library of nearly 400 interferon-stimulated genes (ISGs) against a biologicall

150 lture system to inducibly express individual interferon-stimulated genes (ISGs) and determined their

151 ction of type I interferons, which activates interferon-stimulated genes (ISGs) and directs a multifa

152 nstant includes the coordinate activation of interferon-stimulated genes (ISGs) and immune effector f

153 type specific expression patterns of hepatic interferon-stimulated genes (ISGs) and single nucleotide

154 st response to viral infections, hundreds of interferon-stimulated genes (ISGs) are induced.

155 bsence of virus infection, microRNAs repress interferon-stimulated genes (ISGs) associated with cell

156 The expression of hundreds of interferon-stimulated genes (ISGs) causes the cellular "

157 Increased expression of interferon-stimulated genes (ISGs) distinguished cells f

158 called 'interferon signatures', of canonical interferon-stimulated genes (ISGs) encoding molecules im

159 Interestingly, interferon-stimulated genes (ISGs) encoding proteins suc

160 horylation and impaired induction of several interferon-stimulated genes (ISGs) following WNV infecti

161 Interferon-stimulated genes (ISGs) form the backbone of

162 tetratricopeptide repeats (IFIT), which are interferon-stimulated genes (ISGs) implicated in regulat

163 le inhibiting tumor IFNG signaling decreases interferon-stimulated genes (ISGs) in cancer cells, it i

164 tion of interferons (both type I and II) and interferon-stimulated genes (ISGs) in CH10273.

165 IFN-lambda can induce antiviral interferon-stimulated genes (ISGs) in epithelia, while t

166 sts that ribavirin augments the induction of interferon-stimulated genes (ISGs) in patients treated f

167 Cells express a plethora of interferon-stimulated genes (ISGs) in response to viral

168 ctivated JAK/STAT pathways and expression of interferon-stimulated genes (ISGs) is upregulated.

169 The interferon-stimulated genes (ISGs) ISG56 and ISG54 are s

170 duction of type I IFN, type III IFN, and the interferon-stimulated genes (ISGs) Mx and ISG56 by infec

171 Transcription of these interferon-stimulated genes (ISGs) occurs upon activatio

172 highly ranking hits included a short list of interferon-stimulated genes (ISGs) previously reported t

173 d the resulting up-regulation of hundreds of interferon-stimulated genes (ISGs) provide an immediate

174 a1 (IL-29), IFN-lambda2/3 (IL-28A/B) and the interferon-stimulated genes (ISGs) such as myxovirus res

175 ssion of interferon-alpha/beta and antiviral/interferon-stimulated genes (ISGs) that limit infection.

176 s C virus (HCV) infection, yet the component interferon-stimulated genes (ISGs) that mediate the anti

177 nd primary fibroblasts leads to induction of interferon-stimulated genes (ISGs) through JAK/STAT sign

178 lement (ISRE) activity and the expression of interferon-stimulated genes (ISGs) upon alpha interferon

179 HCMV is known to repurpose the interferon-stimulated genes (ISGs) viperin and tetherin

180 The role of IMPDH and interferon-stimulated genes (ISGs) was investigated in t

181 The expression of six interferon-stimulated genes (ISGs) was measured by quant

182 amma interferon (IFN-gamma) and 16 antiviral interferon-stimulated genes (ISGs) were upregulated 3.1-

183 in the robust expression of type I IFNs and interferon-stimulated genes (ISGs), a strong activation

184 lpha induces the expression of more than 300 interferon-stimulated genes (ISGs), and this blunts infl

185 Here, we report that one of the interferon-stimulated genes (ISGs), cholesterol 25-hydro

186 restriction factors against CSFV, including interferon-stimulated genes (ISGs), have been characteri

187 he expression of type I interferon (IFN) and interferon-stimulated genes (ISGs), including a variety

188 he expression of type I interferon (IFN) and interferon-stimulated genes (ISGs), including a variety

189 e initial transcription of multiple critical interferon-stimulated genes (ISGs), including IFITM3 and

190 ponses and with the up-regulation of several interferon-stimulated genes (ISGs), including those invo

191 the utility of this system, we show that two interferon-stimulated genes (ISGs), ISG20 and tetherin,

192 nterferon response factor 3 (IRF3)-dependent interferon-stimulated genes (ISGs), ISG54 and ISG56, in

193 s associated with intrahepatic expression of interferon-stimulated genes (ISGs), known to influence t

194 Interferons (IFNs) induce the expression of interferon-stimulated genes (ISGs), many of which are re

195 al state through upregulation of hundreds of interferon-stimulated genes (ISGs), most of which have u

196 As expected, expression of Rb1 and interferon-stimulated genes (ISGs), plasma soluble CD163

197 ntiviral response is mediated by a family of interferon-stimulated genes (ISGs), providing cell-intri

198 from viral infection by inducing hundreds of interferon-stimulated genes (ISGs), some of which encode

199 include NF-kappaB-regulated genes as well as interferon-stimulated genes (ISGs), such as ISG15 and bo

200 ants induce moderate levels of expression of interferon-stimulated genes (ISGs), suggesting either th

201 romotes the upregulation of antiviral type I interferon-stimulated genes (ISGs), we hypothesized that

202 nus can antagonize the antiviral activity of interferon-stimulated genes (ISGs), we pretreated cells

203 , resulting in the production of hundreds of interferon-stimulated genes (ISGs), which can inhibit vi

204 esulting in the transcription of hundreds of interferon-stimulated genes (ISGs), which define the ant

205 uced expression of interferon (IFN)-beta and interferon-stimulated genes (ISGs), while knockdown of T

206 which induces the expression of hundreds of interferon-stimulated genes (ISGs).

207 to suppress the mRNA expression of selected interferon-stimulated genes (ISGs).

208 the expression of IFN-gamma and 16 antiviral interferon-stimulated genes (ISGs).

209 ctions by inducing expression of hundreds of interferon-stimulated genes (ISGs).

210 ne responses through the concerted action of interferon-stimulated genes (ISGs).

211 TAT1 activation, and diminished induction of interferon-stimulated genes (ISGs).

212 induction of beta interferon (IFN-beta) and interferon-stimulated genes (ISGs).

213 antiviral pathways through the induction of interferon-stimulated genes (ISGs).

214 on of which is required for transcription of interferon-stimulated genes (ISGs).

215 hat mediate this defence are the products of interferon-stimulated genes (ISGs).

216 r, RelA directly regulates a small subset of interferon-stimulated genes (ISGs).

217 is revealed a heightened basal expression of interferon-stimulated genes (ISGs).

218 ables the virus to circumvent the effects of interferon-stimulated genes (ISGs).

219 cytokines, type I interferons, and numerous interferon-stimulated genes (ISGs).

220 se consists predominantly of upregulation of interferon-stimulated genes (ISGs).

221 ING) expression and impair the production of interferon-stimulated genes (ISGs).

222 by stimulating the expression of hundreds of interferon-stimulated genes (ISGs).

223 cade leading to the induction of hundreds of interferon-stimulated genes (ISGs).

224 acellular actions of the proteins encoded by interferon-stimulated genes (ISGs); among these are the

225 in granule cell neurons, we identified three interferon-stimulated genes (ISGs; Ifi27, Irg1 and Rsad2

226 led to a relative decrease in expression of interferon-stimulated genes (ISGs; MX1 and IFIT5) and in

227 eron (IFN) family cytokines stimulate genes (interferon-stimulated genes [ISGs]) that are integral to

228 specific CD8(+) T cells correlated with both interferon-stimulated gene levels in T cells and hepatic

229 Increased expression of interferon-stimulated genes may favor the persistence of

230 The interferon-stimulated gene myxovirus resistance 2 has be

231 ther cytokines and chemokines, and activated interferon-stimulated genes, natural killer cells, and l

232 robust interferon response and induction of interferon-stimulated genes observed during later stages

233 ated the role of the ubiquitin-like modifier interferon-stimulated gene of 15 kDa (ISG15) in the path

234 not regulate IFN-inducible transcription of interferon-stimulated genes or generation of antiviral r

235 of Toll-like receptor 7 and upregulation of interferon-stimulated genes play a central role.

236 Upregulation of interferon stimulated genes precedes these defects, whic

237 on of phosphorylated STAT1 and the classical interferon-stimulated gene produces MxA and OAS.

238 result, the interface of MERS-CoV and human interferon-stimulated gene product 15 (hISG15) was probe

239 athways through the removal of ubiquitin and interferon-stimulated gene product 15 (ISG15) from targe

240 Interferon-stimulated gene product 15 (ISG15) is a key c

241 l modification by ubiquitin (Ub) and Ub-like interferon-stimulated gene product 15 (ISG15) to stabili

242 dification of host proteins by ubiquitin and interferon-stimulated gene product 15 (ISG15), subsequen

243 ugated with either ubiquitin (Ub) or Ub-like interferon-stimulated gene product 15 (ISG15).

244 f alpha and gamma interferons, the antiviral interferon-stimulated gene product 2'-5' oligoadenylate

245 Here, we report that the interferon-stimulated gene product C19orf66 (herein name

246 were more sensitive to the expression of the interferon-stimulated gene product IFIT2 than the parent

247 were more sensitive to the expression of the interferon-stimulated gene product IFIT2 than the parent

248 Protein kinase R (PKR), a classical interferon-stimulated gene product, phosphorylates the e

249 E The results of this study demonstrate that interferon-stimulated gene products PARP7, PARP10, and P

250 However, interferon-stimulated gene products that inhibit CoVs ar

251 ses green fluorescent protein (GFP) under an interferon-stimulated gene promoter, we repeatedly obser

252 ich prevents the induction of interferon and interferon-stimulated gene promoters.

253 sults suggest that SAMHD1 is a non-classical interferon-stimulated gene regulated through cell type-d

254 anscription decreased in MDA5(-/-) mice, the interferon-stimulated gene response remained intact.

255 n-specific elements were a time shift of the interferon-stimulated gene response, selective induction

256 protein 1 were quantified as measures of the interferon-stimulated genes response.

257 n of IRF7, a transcription factor regulating interferon-stimulated genes, revealing a previously unre

258 In search of the interferon-stimulated gene(s) that is responsible for th

259 a high-throughput genetic screen of a human interferon-stimulated gene short-hairpin RNA library, we

260 n CHB patients, peg-IFN treatment induced an interferon-stimulated gene signature in monocytes and in

261 otype in COVID-19, including a heterogeneous interferon-stimulated gene signature, HLA class II downr

262 ects HCV clearance, IFN-lambda3 up-regulates interferon-stimulated genes, similar to IFN-alpha and IF

263 The induced levels of known interferon-stimulated genes such as the 2'5'-oligoadenyl

264 tiviral response measured by upregulation of interferon-stimulated genes, such as CXCL10 and DAI.

265 In a previous screen of putative interferon-stimulated genes, SUN2 was shown to inhibit H

266 xpression of types I and III interferons and interferon-stimulated genes than do cells from children

267 Taking into account the wide range of interferon stimulated genes that may be induced by the S

268 is a processing body (P-body) protein and an interferon-stimulated gene that can affect replication o

269 Schlafen 11 (Slfn11) is an interferon-stimulated gene that controls the synthesis o

270 Cholesterol 25-hydroxylase (CH25H) is an interferon-stimulated gene that converts cholesterol to

271 Viperin is an interferon-stimulated gene that exerts antiviral effects

272 transmembrane protein 3 (IFITM3) gene is an interferon-stimulated gene that inhibits the replication

273 he zinc finger antiviral protein (ZAP) is an interferon-stimulated gene that restricts the replicatio

274 nate antiviral pathways comprised of several interferon-stimulated genes that are active against EBV.

275 ignaling pathways, inducing a vast number of interferon-stimulated genes that are overlapping but dis

276 ted cells, we sought to characterize further interferon-stimulated genes that target the pre-integrat

277 mutant demonstrated increased expression of interferon-stimulated genes, the SCID phenotype was not

278 aling pathways that induce the expression of interferon-stimulated genes; the proteins encoded by the

279 TLR4 can induce both IFNbeta and interferon-stimulated genes through its adapter molecule

280 roducts correlated with the amplification of interferon-stimulated genes to up to 10 times above norm

281 at is associated with enhanced expression of interferon-stimulated gene transcripts.

282 ponding MxA and USP18 mRNAs, suggesting that interferon-stimulated gene translation is inhibited in s

283 As also repressed the induction of antiviral interferon-stimulated genes upon IFNalpha- treatment.

284 preciated individual- and cell-type-specific interferon-stimulated gene upregulation, we describe tem

285 IL28B acts on interferon-stimulated genes via the JAK-STAT pathway, wh

286 In the liver and brain, expression of interferon-stimulated genes was detected by 1 to 3 h fol

287 I interferon receptor, strong expression of interferon-stimulated genes was observed in the lungs of

288 were normal; however, the early induction of interferon-stimulated genes was selectively and severely

289 By systematic screening using a panel of interferon-stimulated genes we identify two siblings and

290 n effects of each virus on the expression of interferon-stimulated genes were also investigated in A5

291 Many interferon-stimulated genes were detected in the bronchi

292 (IFITM3) messenger RNA (the transcript of an interferon-stimulated gene) were measured in the same he

293 dritic cells, and the expression patterns of interferon-stimulated genes, were most closely linked to

294 city of TRIF/IRF3 signaling and suggest that interferon-stimulated genes, whether induced by IFNbeta

295 ally and significantly enriched for a set of interferon-stimulated genes which on further in silico a

296 These data serve to characterize an interferon-stimulated gene with antiviral activity again

297 One of these, IFIT2, is an interferon-stimulated gene with well-established antivir

298 eplication by upregulating the expression of interferon-stimulated genes with diverse antiviral prope

299 Nalpha induces the expression of an array of interferon-stimulated genes within minutes of receptor e

300 and spread in brain cells if not blocked by interferon-stimulated genes within the brain.IMPORTANCE