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1 tokines (transforming growth factor beta and interleukin 10).
2 -1, B-cell activating factor, interleukin-6, interleukin-10).
3 d inflammation (tumor necrosis factor-alpha, interleukin-10).
4 m-3; indoleamine 2, 3-dioxygenase (IDO); and interleukin 10.
5 osis factor alpha, surfactant protein D, and interleukin-10.
6 FNgamma and TLR4 signaling, and secretion of interleukin-10.
7 derline significant inverse correlation with interleukin-10.
8 erleukin-6, tumor necrosis factor-alpha, and interleukin-10.
9 retion of protective immunomodulatory factor interleukin-10.
10 air levels of the immunosuppressive cytokine interleukin-10.
12 tor-alpha (+66%) and decreased production of interleukin-10 (-23%) by monocytes of sepsis patients.
15 hydrocortisone decreased the blood level of interleukin-10, a cytokine centrally involved in the reg
19 anti-SLAMF4 and anti-CD3 increased levels of interleukin 10 and interferon gamma secretion by IEL, co
20 (Tregs) and the immunosuppressive cytokines interleukin 10 and transforming growth factor beta is st
21 produce the anti-inflammatory mediators IL (interleukin)-10 and TGF-beta1, and the angiogenic growth
23 ry human tissues, activate anti-inflammatory interleukin-10 and improve colitis symptoms in a colitis
24 od-stage specific CD4(+) T cells coproducing interleukin-10 and interferon gamma (P = .001), which we
25 d set of traits, including a triad of IP-10, interleukin-10 and interleukin-6, anticipate subsequent
26 for inducible protein-10 in driving systemic interleukin-10 and morbidity and highlighting the potent
27 tly increased the anti-inflammatory cytokine interleukin-10 and reduced proinflammatory cytokines, ty
28 l suppression was attributed to a release of interleukin-10 and S100A12 and increased PD-L1 expressio
29 e is a turtle herpesvirus, (iii) it contains interleukin-10 and semaphorin genes (the first time thes
30 also revealed viral counterparts of cellular interleukin-10 and semaphorin, which have not been descr
32 necrosis factor alpha, interleukin 6 (IL-6), interleukin 10, and lipopolysaccharide binding protein w
33 w defective production of interleukin 1beta, interleukin 10, and monocyte chemoattractant protein 1 i
35 interferon gamma (IFN-gamma), interleukin 2, interleukin 10, and tumor necrosis factor alpha (TNF-alp
36 rleukin 1beta, interleukin 6, interleukin 8, interleukin 10, and tumor necrosis factor alpha release
37 xpress programmed death ligand 1 (PD-L1) and interleukin-10, and directly suppress liver cytotoxic CD
38 consisting of interleukin-6, interleukin-8, interleukin-10, and fractalkine was identified to be the
39 ly loaded with interleukin-6, interleukin-8, interleukin-10, and fractalkine, was significantly highe
40 interleukin-6, tumor necrosis factor alpha, interleukin-10, and interferon-gamma in acute disease.
41 ignificant increase in plasma interleukin-8, interleukin-10, and interleukin-1 receptor antagonist po
42 rculatory suppressive factors arginase-1 and interleukin-10, and lower total antioxidant capacity.
43 ion of pericentrin attenuated interleukin-6, interleukin-10, and MCP1 secretion, suggesting that the
44 related with decreased expression of CTLA-4, interleukin-10, and transforming growth factor-beta.
46 and 3) selective secretion of interleukin-6, interleukin-10, and vascular endothelial growth factor t
49 ation markers, such as YM-1, arginase-1, and interleukin-10 by activation of mer receptor tyrosine ki
50 erleukin-6, tumor necrosis factor-alpha, and interleukin-10 by lipopolysaccharide-stimulated leukocyt
51 egulatory B-cell-associated surface markers, interleukin-10, chemokine receptors, and immunoglobulin
52 ue of the cellular immunomodulatory cytokine interleukin 10 (cIL-10), which, due to alternative splic
55 UL111A encodes cytomegalovirus-encoded human interleukin-10 (cmvIL-10), a homolog of the potent immun
56 solvers" had high levels of G-CSF, CCL2, and interleukin-10 compared with the "hypothermic" group whe
57 repeatedly measured plasma interleukin-6 and interleukin-10 concentrations using cytometric bead arra
60 application, secreting human proinsulin and interleukin-10, cured 66% of mice with new-onset diabete
61 ctive: To determine the mechanisms of IL-10 (interleukin-10) deficient-EPC-derived exosome dysfunctio
62 um induces pancolitis in colitis-susceptible interleukin-10-deficient mice and this phenotype require
63 lonized germfree wild-type and colitis-prone interleukin-10-deficient mice with defined bacterial con
64 accelerated onset of experimental colitis in interleukin-10-deficient mice, altered bacterial composi
65 ion, splenic natural killer cells induced an interleukin-10-dependent elimination of splenic dendriti
66 ammatory interleukin-6 and anti-inflammatory interleukin-10 during three distinct time periods after
67 ecrosis factor gene (TNF) G308A), rs1800890 (interleukin-10 gene (IL10) T3575A), rs6457327 (human leu
68 nin also triggered anti-inflammatory factors interleukin-10, heme oxygenase-1, and Hsp70 in macrophag
71 Individually, log10 mean concentrations of interleukin 10 (IL-10) and CXCL10 were significantly hig
72 ated by fluorescence-activated cell sorting; interleukin 10 (IL-10) and IL-18 and soluble FAS ligand
73 ded early increases in Th2 cytokines such as interleukin 10 (IL-10) and IL-5 and late-stage increases
74 rus-specific T cells, elevated production of interleukin 10 (IL-10) and programmed death-1 (PD-1) the
75 killed spherules [FKS]) and for secretion of interleukin 10 (IL-10) and proinflammatory cytokines in
76 ced smaller amounts of the immunosuppressive interleukin 10 (IL-10) and transforming growth factor be
79 harmacologic or genetic approaches inhibited interleukin 10 (IL-10) expression and increased expressi
80 o be a potent inducer of human and murine DC interleukin 10 (IL-10) in vitro, a cellular event that w
84 l coma score was associated with an elevated interleukin 10 (IL-10) level in serum specimens from HSV
85 erleukin 8, tumor necrosis factor alpha, and interleukin 10 (IL-10) production in 20 HIV-infected pat
87 n gamma, interleukin 1beta, CCL5/RANTES, and interleukin 10 (IL-10) were elevated in RSV+ bronchiolit
88 ly, levels of the anti-inflammatory cytokine interleukin 10 (IL-10) were markedly elevated in monocyt
89 eukocytes, gamma interferon (IFN-gamma), and interleukin 10 (IL-10) were significantly reduced in inf
90 sulting from S1PR1 inhibition were driven by interleukin 10 (IL-10), a potent neuroprotective and ant
92 expression of the anti-inflammatory cytokine interleukin 10 (IL-10), and decreasing that of the pro-i
94 ess interferon gamma, more neutrophils, more interleukin 10 (IL-10), and increased M. tuberculosis nu
95 erleukin1beta, interleukin 2, interleukin 6, interleukin 10 (IL-10), and soluble CD14 compared with H
96 roduce anti-inflammatory cytokines including interleukin 10 (IL-10), IL-35 and transforming growth fa
97 gland sonicate showed elevated production of interleukin 10 (IL-10), interleukin 13, interferon gamma
98 interleukin 5 (IL-5), interleukin 6 (IL-6), interleukin 10 (IL-10), interleukin 22 (IL-22), and tumo
99 ted individuals, induced protein 10 (IP-10), interleukin 10 (IL-10), macrophage inflammatory protein
100 (n = 96), but CXCL10, interleukin 6 (IL-6), interleukin 10 (IL-10), tumor necrosis factor alpha, and
101 st the hypothesis that constitutive level of interleukin 10 (IL-10), which may be high in elderly sub
102 The frequencies of FoxP3+ CD4+ T cells and interleukin 10 (IL-10)-expressing CD4+ T cells were incr
114 tozoan parasite Leishmania mexicana requires interleukin-10 (IL-10) and FcgammaRIII (an activating Ig
115 gnant compared to nonpregnant women with CL, interleukin-10 (IL-10) and IL-4 expression were approxim
116 ther immunosuppressive pathways, such as the interleukin-10 (IL-10) and programmed death 1 (PD-1) pat
118 cal source of the anti-inflammatory cytokine interleukin-10 (IL-10) and the metalloproteinase MMP9.
119 tive transfer into mice; they also increased interleukin-10 (IL-10) and transforming growth factor-be
121 hibition of MIP-2 and KC expression involved interleukin-10 (IL-10) and, to a lesser extent, IL-4 and
124 s (twice in relation to control animals) and interleukin-10 (IL-10) concentrations in tissue in the i
126 ved suppressor cells, the immune-suppressive interleukin-10 (IL-10) cytokine, and the down-modulatory
127 bitory signals, including increased PD-1 and interleukin-10 (IL-10) expression by cytotoxic T cells i
129 ibitor demonstrate a significant increase of interleukin-10 (IL-10) gene expression, which indicates
131 Although gamma interferon (IFN-gamma) and interleukin-10 (IL-10) have been shown to be critically
132 sts a role for the immunomodulatory cytokine interleukin-10 (IL-10) in hepatitis C virus (HCV)-specif
133 wing that B1a cells secreted a high level of interleukin-10 (IL-10) in response to C. burnetii infect
134 producers of the anti-inflammatory cytokine interleukin-10 (IL-10) in the intestine, thus playing an
140 that rhesus macaques immunized against viral interleukin-10 (IL-10) manifest delayed rhesus cytomegal
141 lls producing the anti-inflammatory cytokine interleukin-10 (IL-10) modulates the clearance of CKRP-S
142 ion, while alternative activation of MDMs by interleukin-10 (IL-10) or LPS-plus-IL-1beta treatment si
143 es inhibitory networks, such as the PD-1 and interleukin-10 (IL-10) pathways, that impair immunity an
145 stemic reinfection is associated with robust interleukin-10 (IL-10) production and impaired protectiv
147 t triggered higher inflammatory cytokine and interleukin-10 (IL-10) production, a delayed gammadelta
148 differentiation to plasma cells, as well as interleukin-10 (IL-10) production, both of which are dep
151 creened the gamma interferon (IFN-gamma) and interleukin-10 (IL-10) responses to 6 HCMV peptide pools
152 significant neutrophil recruitment and early interleukin-10 (IL-10) responses, suggesting that the re
154 ascular endothelial growth factor (VEGF) and interleukin-10 (IL-10) than MSCs homing to non-pFUS-trea
155 001) and significantly lower serum levels of interleukin-10 (IL-10) than patients with MRSA bacteremi
156 patients induces a rapid increase in plasma interleukin-10 (IL-10) to levels that are significantly
157 ound that adipose tissue macrophages produce interleukin-10 (IL-10) upon feeding, which suppresses he
158 production of the anti-inflammatory cytokine interleukin-10 (IL-10) via the activation of the transcr
159 the levels of regulatory T cells (Tregs) and interleukin-10 (IL-10) were elevated at the end of the e
160 ates inflammation, the beneficial effects of interleukin-10 (IL-10) were further examined in leptin-d
161 We have previously shown that expression of Interleukin-10 (IL-10), a classical anti-inflammatory cy
162 inhibits the induction of anti-inflammatory interleukin-10 (IL-10), a phenotype effectively reversed
164 igh concentrations of lung tissue-associated interleukin-10 (IL-10), an anti-inflammatory and immunos
166 nterferon (IFN-gamma), interleukin-6 (IL-6), interleukin-10 (IL-10), and complement factor H was unaf
167 e secretion of interferon-gamma (IFN-gamma), interleukin-10 (IL-10), and transforming growth factor-b
168 okines, such as tumor necrosis factor alpha, interleukin-10 (IL-10), IL-12, IL-1alpha, IL-6, and IL-1
171 ines including interferon-gamma (IFN-gamma), interleukin-10 (IL-10), IL-6, and IL-1 are produced in r
173 tumor necrosis factor alpha (TNF-alpha) and interleukin-10 (IL-10), two cytokines strongly implicate
174 demand hematopoiesis in part by induction of interleukin-10 (IL-10)- and IL-27-mediated mechanisms.
176 2s in this process and instead discovered an interleukin-10 (IL-10)-dependent cellular crosstalk in t
177 enous replacement therapy induced a complex, interleukin-10 (IL-10)-dependent, antigen-specific syste
179 We hypothesized that naturally occurring, interleukin-10 (IL-10)-producing Bregs maintain toleranc
181 sorders has identified a functional group of interleukin-10 (IL-10)-producing regulatory B cells (Bre
189 ered via the local and sustained delivery of interleukin-10 (IL-10; anti-inflammatory) and anti-trans
190 and chemokine (gamma interferon [IFN-gamma], interleukin 10 [IL-10], IL-13, IL-6, granulocyte-macroph
191 (tumor necrosis factor alpha [TNF-alpha] and interleukin-10 [IL-10]), BMMs were transfected with sele
192 nical malaria (gamma interferon [IFN-gamma], interleukin-10 [IL-10], and tumor necrosis factor alpha
193 cell (Treg) percentages; and interleukin 6, interleukin 10, IL-17A, interleukin 22, interleukin 23,
200 his work, we showed that the main sources of interleukin 10 in peripheral blood mononuclear cells (PB
201 ministered lentiviral vector encoding murine interleukin-10 in altering the onset and relapse of dext
203 ature" comprising interferon (IFN)-gamma and interleukin-10 in T1D patients and IFN-gamma in siblings
205 2.24 reduced the production of TNF-alpha and interleukin-10, inhibited phagocytic activity and stimul
206 response as modelled by an interleukin-6 and interleukin-10 interaction term was not (relative risk,
207 her in ZIKV-infected patients, and levels of interleukin 10, interferon gamma-induced protein 10 (IP-
208 otential control of HIV infection, including interleukin 10, interleukin 13, and interleukin 22.
209 interleukin-5, interleukin-6, interleukin-9, interleukin-10, interleukin-12, interleukin-13, tumor ne
210 (tumor necrosis factor-alpha, interleukin-6, interleukin-10, interleukin-12, interleukin-17) at day 1
211 controls n = 16) displayed reduced levels of interleukin-10, interleukin-12p40, interferon-gamma and
212 leukin-1alpha, interleukin-6, interleukin-8, interleukin-10, interleukin-15, eotaxin-3, interferon ga
213 leukin-8/CCL8) and cytokines (interleukin-6, interleukin-10, interleukin-17, granulocyte-macrophage c
214 rleukin-1beta, interleukin-6, interleukin-8, interleukin-10, interleukin-18, and tumor necrosis facto
216 twork suggested that post-spinal cord injury interleukin-10 is driven by inducible protein-10, wherea
217 expression of the anti-inflammatory cytokine interleukin 10, it did not strongly contribute to the ab
218 L-10) and Latency-associated cytomegalovirus interleukin 10 (LAcmvIL-10) (collectively vIL-10) are ex
219 beta and interferon gamma levels, and higher interleukin 10 levels than PFCE-air-treated HbSS mice.
220 rrelated positively with Leishmania-specific interleukin 10 levels, negatively with Leishmania-specif
221 ; increased serum interleukin 6, CXCL10, and interleukin 10 levels; increased neutrophil counts; and
228 al lesion, whereas a robust monocyte-derived interleukin 10-mediated profile is observed in children
231 mainly produced by CD4(+)CD25(-) cells, and interleukin 10 messenger RNA expression was associated w
232 rs with human AAT resulted in an increase in interleukin-10 messenger RNA and CD8(+)CD11c(+)CD205(+)
234 ed positive for tumor necrosis factor alpha, interleukin 10, or interleukin 6 production by HBV surfa
235 ntercellular adhesion molecule-1 (P < .001), interleukin-10 (P = .041), monocyte chemotactic protein-
236 eron-gamma elevation (P < .001) and possibly interleukin-10 (P = .07) were associated with toxicity.
237 r-alpha, p = 0.003; interleukin-6, p = 0.01; interleukin-10, p = 0.005), and acute kidney injury when
238 matory (interleukin-8) to anti-inflammatory (interleukin-10) plasma cytokine levels was greater in pa
239 evels of specific gamma interferon-positive, interleukin-10-positive T cells, which protect animals f
240 oantibody-positive) and partially regulated (interleukin-10-positive, pauci-autoantibody-positive) re
241 CL had increased frequencies of circulating interleukin 10-producing CD4(+)CD25(-)CD127(-/low) cells
242 of interferon gamma, reduced the numbers of interleukin 10-producing T cells, and increased neutroph
243 on, patients with AAH had greater numbers of interleukin 10-producing T cells, and reduced levels of
244 sion of naturally occurring Foxp3(+) CD25(+) interleukin-10-producing antigen-specific regulatory T c
245 ed lymphocytes were generated by coculturing interleukin-10-producing dendritic cells obtained from h
246 arrying the TLR1 80R-allele showed increased interleukin 10 production with C. burnetii exposure.
249 ion, which was associated with a decrease in interleukin-10 production by hepatic T cells and a more
250 C resulted in higher interleukin-6 and lower interleukin-10 production by lipopolysaccharide-stimulat
251 cutaneous inflammatory responses induces of interleukin-10 production in dendritic cells and priming
252 d interleukin-17 production and decreases in interleukin-10 production that were not evident among sp
256 eline [interleukin-6] and [interleukin-6] x [interleukin-10] profiles, whereas patients with the lowe
257 and interferon gamma (r = 0.562, P = .005), interleukin 10 (r = 0.453, P = .03), and sCD27 secretion
258 C-reactive protein (r = -0.70, P = 0.0006), interleukin-10 (r = -0.59, P = 0.007), and interleukin-6
260 ange complexes and a lower interleukin-1beta/interleukin-10 ratio than the control group (P < 0.05).
262 SELEX experiment developing aptamers against Interleukin 10 receptor alpha chain (IL-10RA) and experi
264 ncluding transforming growth factor beta and interleukin 10 receptor, were suppressed in low-ENL excr
267 ges, stimulated M1 macrophage activation and interleukin 10 release, and decreased tumor necrosis fac
269 6 hours but had normalized by day 2, whereas interleukin-10 remained persistently elevated and high-d
271 T-cell proliferative, interferon gamma, and interleukin 10 responses to HBV, with increased frequenc
272 ntiviral vector-mediated local expression of interleukin-10 resulted in significantly increased level
273 s (CD4+, CD25+, and FoxP3+) and its cytokine interleukin 10, resulting in downregulation of T effecto
274 ically-delivered lentiviral vectors encoding interleukin-10 safely penetrated local mucosal tissue an
276 We found that levels of interleukin-6 and interleukin-10, specific markers of cardiac remodeling (
277 perfusion benefits, and increased levels of interleukin-10, suggestive of an anti-inflammatory effec
278 ediated boosting of antibody titers to viral interleukin-10, there was modest evidence for increased
279 founding variables by multivariate analysis, interleukin-10/tissue necrosis factor ratio at 72 hours
281 ation and production of interferon gamma and interleukin 10) to overlapping hepatitis B virus (HBV) p
282 ed expression of mannose receptor-1 (CD206), interleukin-10, transforming growth factor-beta, arginas
284 omarkers (C-reactive protein, interleukin 6, interleukin 10, tumor necrosis factor alpha), and the me
285 here is accumulating evidence that the viral interleukin-10 (vIL-10) ortholog of both human and rhesu
287 that the parasite-driven regulatory cytokine interleukin-10 was exclusively coming from the intermedi
292 eukin-6, interleukin-8, interleukin-17A, and interleukin-10 were measured by enzyme-linked immunosorb
293 or necrosis factor-alpha, interleukin-6, and interleukin-10 were observed 5 hours after cecal ligatio
296 s, as well as an anti-inflammatory cytokine (interleukin-10), were reduced in the lungs of vaccinated
297 tokines (C-X-C motif chemokine ligand 10 and interleukin 10), which correlated with populations of in
298 uced expression of c-Jun, interleukin-6, and interleukin-10, which were identified as NFAT target gen
299 production of the anti-inflammatory cytokine interleukin 10 while inhibiting B-cell expression of pro
300 specific CD4(+) T-cells in children produced interleukin 10, while responses in adults were dominated