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1 encoded by herpesvirus Saimiri and to human interleukin-8 receptors.
4 oid cells transfected with formyl peptide or interleukin-8 receptors, agonist stimulation activated n
5 ine transfected with human formyl peptide or interleukin-8 receptors and normal human neutrophils as
6 ncise syntheses of (+/-)-frondosin B (1), an interleukin-8 receptor antagonist, have been achieved fr
7 ously that chemokine receptors including the interleukin-8 receptor B (CXCR2) and the Duffy blood gro
9 une activation: alkaline phosphatase (ALPL), interleukin-8 receptor-beta (IL8RB), and defensin-1 (DEF
10 2, angiotensin receptors types 1 and 2, and interleukin-8 receptor could not activate SRF in the pre
11 ng the top of the lamellipodium, whereas the interleukin 8 receptors CXCR1 and CXCR2 and the integrin
14 functional domains of the two CXC chemokine (interleukin-8) receptors, CXCR1 (formally IL-8RA) and CX
15 ocrine tumor cells expressed another type of interleukin-8 receptor, CXCR2, suggesting an autocrine m
17 Gialpha subfamily members and it attenuates interleukin-8 receptor-mediated mitogen-activated protei
18 ctase, bcl-2, interferon regulatory factors, interleukin 8 receptor, neural cell adhesion molecule-li
20 of intracellular loops between the MCP-1 and interleukin-8 receptors to create chimeric receptors rev
21 leukin-8 (IL-8) that allow high affinity and interleukin-8 receptor type 1 (IL8R1)-specific binding o
22 ins from IL-8, which binds to both IL8R1 and interleukin-8 receptor type 2 (IL8R2) with high affinity