ライフサイエンスコーパス: interlobular

1 perfused juxtamedullary nephron preparation, interlobular and afferent arteriolar diameter responses

2 12-DIHETE, had no effect on diameters of the interlobular and afferent arterioles at concentrations u

3 In contrast, 5,6-EET constricted the interlobular and afferent arterioles by 16 +/- 3% (N = 6

4 nM, 11,12-EET increased the diameters of the interlobular and afferent arterioles by 18 +/- 2% (N = 1

5 Diameters of interlobular and afferent arterioles preconstricted with

6 1,12(S,R)-EET increased the diameters of the interlobular and afferent arterioles.

7 caused graded increases in diameters of the interlobular and afferent arterioles.

8 e control diameter, whereas the diameters of interlobular and arcuate arteries declined to 50%+/-12%

9 here a significant difference between small (interlobular and bile ductules) and large (intrahepatic

10 gradients of development (i.e. early to late interlobular and posterior to anterior, respectively).

11 reshly isolated from afferent arterioles and interlobular arteries averaging between 10 to 40 microns

12 usside (SNP) increased the diameter of renal interlobular arteries preconstricted with phenylephrine

13 ETHODS AND Sprague-Dawley rat interlobar and interlobular arteries were examined in terms of superoxi

14 ithelial cells and endothelial cells of some interlobular arteries, in parallel with SODD, during acu

15 Interlobular arteriolar diameter and flow, afferent and

16 Interlobular arteriolar injury was not increased in the

17 forming arborizing networks connected to the interlobular bile duct by single tributaries.

18 e three-dimensional (3D) architecture of the interlobular bile duct during cholestasis, we used 3D co

19 e each of a portal vein, hepatic artery, and interlobular bile duct numbered 11 +/- 6 per biopsy (ran

20 The diameter of the interlobular bile duct remains constant after BDL, a res

21 In contrast, interlobular bile ducts and septal bile ducts had detect

22 were primarily localized around the damaged interlobular bile ducts in PBC.

23 ly by chronic non-suppurative destruction of interlobular bile ducts leading to advanced fibrosis, ci

24 ffusion in individual bile canaliculi and in interlobular bile ducts of intact livers in living mice,

25 Notably, on average there were 2.3 +/- 2.2 interlobular bile ducts per portal tract, compared to 2.

26 PDC-E2 monoclonal antibody (mAb) C355.1, the interlobular bile ducts showed typical aberrant apical s

27 The average minimum external diameter of interlobular bile ducts was 13 +/- 4 microm, of hepatic

28 disease characterized by the destruction of interlobular bile ducts, leading to cholestasis and live

29 Interlobular bile ducts, septal bile ducts, and prolifer

30 On average, there are two interlobular bile ducts, two hepatic arteries, and one p

31 t was detected and quantified in the mesh of interlobular bile ducts.

32 disease characterized by the destruction of interlobular biliary ductules, which progressively leads

33 or protein was detected in interalveolar and interlobular connective tissue cells adjacent to glandul

34 We show a highly reproducible sequence of interlobular duct remodeling, where cholangiocyte prolif

35 Short segments of interlobular duct were microdissected from guinea-pig pa

36 was expressed in the glandular alveolar and interlobular ductal cells in the lacrimal gland and all

37 toring the luminal pH of isolated guinea-pig interlobular ducts after microinjection of an extracellu

38 yte growth factor likely collects within the interlobular ducts and becomes a component in normal tea

39 e cells and in the ductal cells of small and interlobular ducts in CP.

40 Only in the interlobular ducts is diffusion augmented by regulatable

41 Interlobular ducts were isolated from the rat pancreas a

42 erminal ductal lobular units, rather than in interlobular ducts.

43 sists of acini connected by intercalated and interlobular ducts.

44 lls and associated with the cells lining the interlobular ducts.

45 canalicular network and the sink function of interlobular ducts.

46 1a-negative palisading granulomas in widened interlobular fibrous septa were detected.

47 acteristics: the intercalated, intralobular, interlobular, intralobar, interlobar, and main excretory

48 significantly more intralobular (P < 0.001), interlobular (P < 0.05), and total pancreatic fat (P < 0

49 Larger vessels concentrated in interlobular regions lacked CD36 and were instead marked

50 s of peripheral, dilated airways (n = 3) and interlobular septa (n = 3).

51 A network of interlobular septa acted as a barrier or pathway to the

52 , reticular or granular opacities (thickened interlobular septa and ground-glass opacities at CT), cy

53 increased attenuation located in or near the interlobular septa can be seen on micro-CT studies.

54 es enlarge and approach each other along the interlobular septa, causing a fine reticular pattern on

55 d ASIR-HD images for small anatomic details (interlobular septa, centrilobular region, and small bron

56 revealed lymphadenopathy, nodules, thickened interlobular septa, focal consolidation, reticular opaci

57 solidation or reticular) patterns, thickened interlobular septa, vascular enlargement, air bronchogra

58 aeruginosa accumulation at the edges of the interlobular septa.

59 facilitated along the renal vessels and the interlobular septa.

60 ocytic infiltration of the alveolar wall and interlobular septa.

61 e-in-bud pattern (accuracy, 93% vs 78%-80%), interlobular septal thickening (accuracy, 78%-83% vs 61%

62 The predominant HRCT presentation of PAP was interlobular septal thickening (ILST;100%) and ground gl

63 omy visible on thin-section CT scans include interlobular septal thickening and diseases with periphe

64 nant HRCT presentation of idiopathic PAP was interlobular septal thickening and ground glass opacitie

65 Interlobular septal thickening and ground-glass attenuat

66 ; interquartile range, 23-38 years) and more interlobular septal thickening and mediastinal lymphaden

67 00%), consolidation in nine of 14 (64%), and interlobular septal thickening in two of 14 (14%).

68 nd-glass opacity or airspace consolidation), interlobular septal thickening, and gastric distention a

69 consisting of multifocal opacity and smooth interlobular septal thickening, possibly with small effu

70 and left lower lobe abnormalities, GGOs, and interlobular septal thickening.

71 nd-glass opacities (GGO), consolidation, and interlobular septal thickening.

72 bubble sign, peribronchovascular thickening, interlobular thickening, and number of involved lobes 4

73 n associated with autism, and the cerebellar interlobular variation in Spry3 expression coincident wi