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1  study that scid cells make higher levels of interlocus and hybrid joints than their normal counterpa
2 determine the number of AA mismatches (after interlocus and intralocus subtraction) and their hydroph
3 lymorphisms can be assessed jointly with the interlocus associations.
4 ompatibility is determined by intralocus and interlocus comparisons of triplets in polymorphic sequen
5 tum agree with a previous observation of the interlocus concerted evolution (sequences corresponding
6 NA tandem arrays is influenced by intra- and interlocus concerted evolution and their expression is c
7 polyploid formation, thereby indicating that interlocus concerted evolution has not been an important
8 ward one homeologue, possibly resulting from interlocus concerted evolution.
9 but that multilocus effects and variation in interlocus correlations contribute to this flip-flop phe
10 ackground selection are not likely causes of interlocus differences.
11 th greater than 1000:1 odds and with average interlocus distance of 1.46 cM.
12 0 yeast artificial chromosomes, with average interlocus distance of approximately equal to 100 kb.
13                             The precision of interlocus distance of genomically-proximate loci was be
14  The measured dependence of the precision of interlocus distance on genomic distance singles out intr
15                   Maps can be built assuming interlocus distances are independent or proportional bet
16 ative information concerning locus order and interlocus distances in humans and rhesus monkeys.
17 pitopes exclusive to the C-locus antigens or interlocus epitopes among A, B, and C antigens.
18 that at least 23% of this increase is due to interlocus gene conversion (IGC) with up to 4.3 megabase
19  paralogous sequences, a phenomenon known as interlocus gene conversion (IGC).
20                                              Interlocus gene conversion can also explain high sequenc
21 les by locus, implying that large amounts of interlocus gene conversion have not occurred since these
22                We found little evidence that interlocus gene conversion plays an important role in th
23  findings reveal the extent of the impact of interlocus gene conversion upon the spectrum of human in
24 nged in tandem and show evidence of repeated interlocus gene conversion.
25 of unequal crossover and discover regions of interlocus gene conversion.
26 e the statistical properties of a measure of interlocus genetic associations under the assumption tha
27 or sterility) evolves by the accumulation of interlocus incompatibilities between diverging populatio
28 itional analyses under a range of models for interlocus interactions.
29                       We find no significant interlocus interactions.
30                We observed a great degree of interlocus, intertissue, and interindividual epigenetic
31 loci could potentially be joined to generate interlocus joints.
32 s, however, adjacent genes had low levels of interlocus LD and loosely linked genes had high levels o
33 loy a resampling method to determine whether interlocus LD exceeds expectations.
34 at 15% of two-locus combinations; almost all interlocus LD involves loci with significant geographic
35                                       Excess interlocus LD is observed at 15% of two-locus combinatio
36  and loosely linked genes had high levels of interlocus LD, suggesting strong epistatic selection.
37 s of relative trajectories, we find that the interlocus motion is distance-dependent with a varying f
38  including cell-type specificity, intra- and interlocus ordering, and allelic exclusion.
39              Dilution analyses revealed that interlocus rearrangements occur about 1,000 times less f
40  leukemia virus and PCR was used to identify interlocus recombinants.
41  "illegitimate" T-cell receptor Vgamma-Jbeta interlocus recombination events.
42 ty in a explicit form, expressed in terms of interlocus recombination fractions.
43                                          The interlocus recombination process may contribute to the t
44                                         This interlocus sexual conflict (IRSC) has been proposed as a
45 us theory by suggesting that when loci under interlocus sexual conflict are located on the sex chromo
46                                              Interlocus sexual conflict is generally described as a c
47                                 In addition, interlocus V(D)J rearrangements occur, giving rise to so
48 ct, exclusion of the anomalous locus reduces interlocus variability of statistics summarizing populat
49 ing revealed substantial interindividual and interlocus variability, with HLA-DQ the most variable.
50 at such historical division can increase the interlocus variance in Fst, but neither a historical nor
51 y to monitor mutation efficiency, strain and interlocus variation in mutation induction, and extensiv