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1 asal ganglia, occipital WM, thickness of the internal capsule).
2 1 (brainstem and cerebrum) to 0.74 +/- 0.06 (internal capsule).
3 r the hippocampus and posterior limbs of the internal capsule.
4 orpus callosum (CC), optic chiasm (Och), and internal capsule.
5 alamic radiations, and posterior limb of the internal capsule.
6 ially the frontal parts of the brain and the internal capsule.
7 for thalamic axons that failed to enter the internal capsule.
8 or commissure, subcortical fiber tracts, and internal capsule.
9 ductions in the frontal lobe, cerebellum and internal capsule.
10 neurons during electrical stimulation of the internal capsule.
11 the corticospinal tracts at the level of the internal capsule.
12 ar connections, all of which are part of the internal capsule.
13 01 to <.03) except the posterior limb of the internal capsule.
14 lly medially across the inferior edge of the internal capsule.
15 ventromedial putamen, on either side of the internal capsule.
16 e zone, and, by P5, they reach the primitive internal capsule.
17 me fraction within the posterior limb of the internal capsule.
18 mporal lobe, and right posterior limb of the internal capsule.
19 es in the putamen, thalamus, insula, and the internal capsule.
20 erally implanted in the anterior limb of the internal capsule.
21 of the striatum and the anterior limb of the internal capsule.
22 homolog of the primate anterior limb of the internal capsule.
23 ed white matter volumes in pyramids and left internal capsule.
24 and inferior fronto-occipital fasciculi, and internal capsule.
25 wing the fornix and the anterior limb of the internal capsule.
26 e ventral border of the anterior limb of the internal capsule.
27 m, and to subcortical structures through the internal capsule.
28 te cells with an axon projecting through the internal capsule.
30 the corpus callosum and anterior limb of the internal capsule (-0.02; p =.01) at hospital discharge.
31 e, 130 x 10(-5) mm(2)/sec; posterior limb of internal capsule, 109 x 10(-5) mm(2)/sec) and three gray
32 10(-5) mm(2)/sec at birth; anterior limb of internal capsule, 130 x 10(-5) mm(2)/sec; posterior limb
33 th irregular tails, often extending into the internal capsule; 22 of these patients experienced at le
34 , associations with the anterior limb of the internal capsule, a subcortical white matter region, rem
36 sed atrophy in the anterior limb of the left internal capsule adjacent to the head of the left caudat
38 cle, the anterior and posterior limbs of the internal capsule (ALIC and PLIC), the external capsule,
39 Pe), nucleus accumbens, anterior limb of the internal capsule (ALIC) and anterior lateral anterior co
40 rocedures targeting the anterior limb of the internal capsule (aLIC) can be effective in patients wit
41 fibers that are part of the anterior limb of internal capsule (ALIC) in MDD and diabetic subjects usi
42 Disconnection of the anterior limb of the internal capsule (ALIC) was assessed via individual and
43 at underwent DBS to the anterior limb of the internal capsule (ALIC), the nucleus accumbens or the su
44 the diencephalon and growing out through the internal capsule among groups of back-labelled cells tha
45 ter effect size in the posterior limb of the internal capsule and a tendency for decreased fractional
46 kB-immunostained fibers were observed in the internal capsule and as two distinct fascicles within th
47 of fractional anisotropy in the genu of the internal capsule and bilateral increase of overall water
48 We show that striatonigral axons pioneer the internal capsule and cerebral peduncle and are temporall
49 atonigral (direct) pathway in pioneering the internal capsule and cerebral peduncle, and in guiding a
50 for corticofugal outputs passing through the internal capsule and cerebral peduncle, there is accumul
52 ending and descending axon tracts within the internal capsule and cerebral peduncle.SIGNIFICANCE STAT
53 ed increases in fractional anisotropy in the internal capsule and cingulum and decreases in the poste
54 and myelin content in the right genu of the internal capsule and clusters of mineral depositions, co
55 se an updated model of tinnitus, wherein the internal capsule and corpus callosum play important role
56 w images for differentiation of GPi from the internal capsule and external globus pallidus, respectiv
57 e normally situated in the ventral thalamus, internal capsule and hypothalamus, were more dispersed i
58 e effects of DBS in the anterior limb of the internal capsule and nucleus accumbens region (ALIC-NAcc
61 t on thalamic axons as they grow through the internal capsule and subplate but is not present in the
62 hich attracts initial axon growth toward the internal capsule and that this activity may be due to Ne
63 ns of white matter, particularly in the left internal capsule and the left frontal lobe (P = .02-.05)
66 the ventral part of the anterior limb of the internal capsule and were followed for at least 3 years
67 the ventral part of the anterior limb of the internal capsule and were followed for at least 3 years
68 imulated thalamic axons coursing through the internal capsule and, as a control, the basolateral (BL)
70 tter (anterior and posterior subcortical and internal capsule) and four of gray matter (cortex, thala
71 y low within areas of white matter (fimbria, internal capsule) and select neuronal fields (hippocampa
72 h frontal lobes, posterior limb of the right internal capsule, and both cerebellar hemispheres in pat
76 ing confirmed that striatum but also insula, internal capsule, and external capsule were associated w
78 trum semiovale, in the posterior limb of the internal capsule, and in the cerebral peduncle; the thal
79 lly located in the cerebral white matter and internal capsule, and infrequently in the brain stem.
80 le, bilateral retrolenticular region part of internal capsule, and left posterior thalamic radiation.
81 ped myelination of the posterior limb of the internal capsule, and more immature gyral folding than t
83 ed increased volume in the basal ganglia and internal capsule, and otherwise normal brain volumes.
84 diffusivity measures of the corpus callosum, internal capsule, and posterior thalamic radiation contr
85 um bundle, superior longitudinal fasciculus, internal capsule, and splenium of the corpus callosum.
86 c radiation, the retrolenticular part of the internal capsule, and the sagittal stratum (p<.05, corre
88 of the frontal lobes, posterior limbs of the internal capsules, and cerebellar hemispheres in patient
91 (subcortical white matter, corpus callosum, internal capsule, anterior commissure), gray matter (glo
93 motor system through the corona radiata and internal capsule are well described in non-human primate
94 ein (MBP) at the fimbria hippocampus and the internal capsule areas in the 7-day-old BACO rat brain w
95 matter in the corpus callosum, subcortex and internal capsule areas while largely spared cortical neu
98 xons show increased fasciculation within the internal capsule, as well as abnormal turning and branch
99 ractional anisotropy in the anterior limb of internal capsule at discharge and in genu of corpus call
102 m, bilateral anterior and posterior limbs of internal capsule, bilateral retrolenticular region part
103 e we demonstrate that a set of neurons in an internal-capsule bordering regions of the primate dorsal
104 fected 293T cells, as it had been toward the internal capsule, but not toward control-transfected 293
105 tical axon outgrowth was directed toward the internal capsule, but outgrowth was nondirected and supp
106 tions, superior fronto-occipital fasciculus, internal capsule, callosal isthmus, and the corona radia
107 alamo-cortical loop, brainstem, external and internal capsules, callosal and cerebellar structures.
108 igma)) were measured in the corpus callosum, internal capsule, caudate nucleus, lentiform nucleus, an
109 ontrol subjects in the posterior limb of the internal capsule, corona radiata, posterior frontal whit
110 st in the centrum semiovale, corona radiata, internal capsule, corpus callosum, and subcortical white
112 addition, reduced FA in anterior limb of the internal capsule correlated significantly with an increa
113 ctional anisotropy with age was found in the internal capsule, corticospinal tract, left arcuate fasc
114 f basal ganglia circuits can drive secondary internal capsule defects and thereby may contribute to t
116 ed FA values were located bilaterally in the internal capsule extending into the globus pallidus and
117 nferior longitudinal fasciculi, limbs of the internal capsule, external capsule and cerebellum (p < 0
118 ngs in the putamen into the rostral putamen, internal capsule, external capsule, caudate nucleus, and
119 beta = -0.01 (P = .04)] and retrolenticular internal capsule [FA, beta = -0.01 (P = .002); RD, beta
123 te matter for HT+M versus HT (p = 0.036) and internal capsule for HT+M compared to HT (p = 0.001) and
124 mulation of the ventral anterior limb of the internal capsule for obsessive-compulsive disorder and p
125 for Isl1 in diencephalic cells bordering the internal capsule for the normal development of the stria
126 ntal contributions of striatonigral axons to internal capsule formation, we have made use of Sox8-EGF
129 m, cortical plate, hemispheric white matter, internal capsule, ganglionic eminence, ventricular zone,
130 out the striatal neuropil, as well as in the internal capsule, globus pallidus, and substantia nigra.
131 of myelination in the posterior limb of the internal capsule, gyral maturation, signal intensity abn
132 fiber tracts within the anterior limb of the internal capsule have a significantly higher (P < 0.01)
134 n began at postnatal day 5 (P5) (E36) in the internal capsule (IC) and at P11 in the medial corpus ca
135 tor regions through the corona radiata (CR), internal capsule (IC) and crus cerebri of the cerebral p
137 nd inversely varied with FA in the bilateral internal capsule (IC) at 2-weeks (p = 0.0294, FDR correc
138 al projection in the corona radiata (CR) and internal capsule (IC) can assist in evaluating a patient
140 upon DA application or by stimulation of the internal capsule (IC), which also supports self-stimulat
142 ly and orthodromically by stimulation of the internal capsule (IC; conduction velocity, 2.4 +/- 0.2 m
143 ite matter volume around the optic tract and internal capsule in anophthalmic subjects showed a large
144 including the corpus callosum, fimbria, and internal capsule in the brain, and pyramidal tracts and
145 y increased ODI in the posterior limb of the internal capsule in unmedicated patients (242 voxels; x
146 major CNS axon tracts including those of the internal capsule, in part via cell-nonautonomous mechani
147 the ventral part of the anterior limb of the internal capsule is effective and tolerable for treatmen
149 ns pioneer the pathway from neocortex to the internal capsule, leading to the proposal that they are
150 1.17 x10(3)mum(2)/sec), anterior limb of the internal capsule (left, 1.11 x10(3)mum(2)/sec; right, 1.
151 1.17 x10(3)um(2)/sec), anterior limb of the internal capsule (left, 1.11 x10(3)um(2)/sec; right, 1.0
153 putamen, corpus callosum, posterior limb of internal capsule), level of brainstem, grey- white matte
156 onal anisotropy in the posterior limb of the internal capsule, measured with magnetic resonance spect
157 y of the deep gray matter nuclei, brainstem, internal capsule, motor cortex and corticospinal pathway
158 =47), nucleus accumbens/anterior limb of the internal capsule (n=4) or a combination of targets (n=8)
159 timulation of the human anterior limb of the internal capsule/NAcc region for treatment-resistant obs
160 timulation (DBS) of the anterior limb of the internal capsule/NAcc region for treatment-resistant obs
161 anatomy, except for larger basal ganglia and internal capsule, not explained by antipsychotic dose.
163 tently lower diffusivity in the thalamus and internal capsule of all tobacco-exposed infants suggests
165 ion restriction in the posterior limb of the internal capsule, often also optic radiation, brainstem
166 In contrast, bilateral involvement of the internal capsule on brain MRI was associated with poorer
167 ury was prominent in the corpus callosum and internal capsule on day 3 and then partially recovered o
169 oring, closed-loop direct stimulation of the internal capsule or striatum, especially the dorsal site
170 = 50) receiving DBS to anterior limb of the internal capsule or subthalamic nucleus zones, optimal f
171 gets of cortical efferent axons, the nascent internal capsule, or the medial wall of the dorsal telen
172 bocyanine dye tracing from the callosum, the internal capsule, or the primary somatosensory cortex.
173 ventral portion of the anterior limb of the internal capsule over a 20-year period using the Leksell
175 ells) throughout the whole brain, and in the internal capsule, periventricular white matter and senso
176 and the contralateral posterior limb of the internal capsule (PLIC) after age correction and were co
177 and abnormal signal in the posterior limb of internal capsule (PLIC) had an AUC of 0.82 (0.76-0.87).
181 orpus callosum (SCC), posterior limbs of the internal capsules (PLIC), superior longitudinal fascicul
182 d white matter, increased myelination of the internal capsule, poorer motor performance, and profound
183 und mainly in the left posterior limb of the internal capsule, posterior corona radiata, and partly i
184 ttern beginning in the cerebellum, pons, and internal capsule; proceeding caudocranially from the spl
185 ared with hypothermia alone in the thalamus, internal capsule, putamen and caudate, and there was red
186 mid, and anterior cingulate, as well as the internal capsule, putamen, and globus pallidus and basal
187 nd vessel walls of the posterior limb of the internal capsule, putamen, globus pallidus and cerebellu
188 mulation of the ventral anterior limb of the internal capsule rapidly improves mood and anxiety with
189 hen cocultured with MDT, suggesting that the internal capsule releases a chemoattractant for cortical
190 icothalamic and thalamocortical axons in the internal capsule requires Fz3 expression in the ventral
194 ion of thalamic/brainstem fibers through the internal capsule, results in a complex mingling of thala
195 ial corticospinal tracts at the level of the internal capsule (right Z score 5.21, p=0.0081; left Z s
196 us callosum, the right posterior limb of the internal capsule, right external capsule, and the right
197 nal capsule, the retrolenticular part of the internal capsule (RLIC), the body and splenium of the co
198 to Broca's area (RR, 2.5; 95% CI, 1.3-5.0), internal capsule (RR, 2.2; 95% CI, 1.1-4.4), Wernicke's
201 fic brain regions (including corona radiata, internal capsule, superior and inferior longitudinal fas
202 sterior limb and the retrolenticular part of internal capsule, superior and posterior corona radiata,
203 ut adolescence (in the posterior limb of the internal capsule, superior corona radiata, and cerebella
204 d to pain processing (e.g., corpus callosum, internal capsule, superior longitudinal fasciculus and s
205 WM loss was concentrated in the brainstem, internal capsule, temporal and frontal regions and the m
206 rticospinal tract), corpus callosum, fornix, internal capsule (thalamocortical and corticothalamic tr
207 f the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal forebrain, occipital,
208 tures (corpus callosum, anterior commissure, internal capsule, thalamus, caudoputamen, and cortex).
209 sociated with increased FA of regions of the internal capsule that contain auditory thalamocortical a
210 lsive Scale, one in the anterior limb of the internal capsule that overlapped with a previously ident
211 -compared with controls-in the pyramids, the internal capsule, the cerebral peduncle and the hand are
212 ntricular regions, the posterior limb of the internal capsule, the external capsule, and the pyramida
213 defined and topographic anterior limb of the internal capsule, the specific locations and organizatio
215 th abnormalities in the anterior limb of the internal capsule, the white matter (WM) bundle carrying
216 ither through the ventrolaterally positioned internal capsule to subcortical targets or through the d
217 eral DBS of the ventral anterior limb of the internal capsule (vALIC) between April 2005 and October
218 targeted at the ventral anterior limb of the internal capsule (vALIC) in 25 patients with TRD during
219 echanisms may lie in the efficacy of ventral internal capsule/ventral striatum (VCVS) DBS in both maj
220 Deep brain stimulation (DBS) of the ventral internal capsule/ventral striatum (VCVS) is an emerging
222 ng the ventral striatum/anterior limb of the internal capsule (VS/ALIC) in 10 patients with poststrok
224 Myelination of the posterior limb of the internal capsule was less developed, and gyral maturatio
226 nisotropy (FA) in the posterior limbs of the internal capsules was determined with diffusion-weighted
227 Automated GWR_si (putamen/posterior limb of internal capsule) was performed with an area under the c
228 chemoattractant Netrin-1 is expressed in the internal capsule, we cocultured cortical explants with E
229 us pallidus and the adjacent optic tract and internal capsule were identified with microelectrode rec
230 The basal ganglia, cortex and lobar, and internal capsule were the frequently involved areas of t
232 the corpus callosum, the frontal WM, and the internal capsule; were compared with those of five age-m
233 substantia innominata, globus pallidus, and internal capsule, where PHA-L-labeled terminals abutted
234 l axon growth is directed toward the nascent internal capsule, which could account for the timing dif
235 Converging evidence suggests the ventral internal capsule white matter tracts traversing the rost
236 actional anisotropy and lower diffusivity in internal capsule white matter; lower regional blood flow
237 nderwent DBS to the ventral anterior limb of internal capsule with subsequent programming uninformed
239 l capsule (Z score 4.30, p=0.0021), the left internal capsule (Z score 4.27, p=0.0278), and left cere
240 rticospinal tracts at the level of the right internal capsule (Z score 4.30, p=0.0021), the left inte
241 d growth of dorsal thalamic axons toward the internal capsule zone of ventral telencephalic explants