ライフサイエンスコーパス: internal carotid artery

1 y in the M1-/proximal M2-segment or terminal internal carotid artery.

2 sensor of arterial blood located next to the internal carotid artery.

3 (20 x 18 mm) wide neck aneurysm of the right internal carotid artery.

4 t via the external carotid artery or via the internal carotid artery.

5 asymptomatic high-grade stenosis of the left internal carotid artery.

6 CA) with an intra-luminal suture through the internal carotid artery.

7 re acquired through atheroma in the proximal internal carotid artery.

8 -old man who experienced a dissection of the internal carotid artery.

9 ssociated with the intradural segment of the internal carotid artery.

10 ht and left middle cerebral artery and right internal carotid artery.

11 carotid cavernous fistulas, and 1 transected internal carotid artery.

12 VO and stenosis or occlusion of the cervical internal carotid artery.

13 carotid artery and in only 7.7% for the left internal carotid artery.

14 serted in the femoral artery and through the internal carotid artery.

15 ophthalmic artery is the first branch of the internal carotid artery.

16 D devices were implanted unilaterally in the internal carotid artery.

17 giography revealed unilateral aplasia of the internal carotid artery.

18 h the highest prevalence in the intracranial internal carotid artery.

19 ric NF-kappaB activity observed in the human internal carotid artery.

20 ent of uncoilable or failed aneurysms of the internal carotid artery.

21 form aneurysm in supraclinoid segment of the internal carotid artery.

22 that is segment-specific for the common and internal carotid arteries.

23 carotid artery bifurcation, and the proximal internal carotid arteries.

24 aortic arch, extracranial, and intracranial internal carotid arteries.

25 osclerotic calcification in the intracranial internal carotid arteries.

26 ior and posterior communicating arteries and internal carotid arteries.

27 ween the cavernous sinus and the external or internal carotid arteries.

28 ned intima-media thickness of the common and internal carotid arteries, -0.155 vs. 0.007; P=0.02) aft

29 etected in the middle cerebral artery (23%), internal carotid artery (13%), and vertebrobasilar arter

30 narrowing of diameter) stenosis of the left internal carotid artery (32 patients) was associated wit

31 n the middle cerebral arteries (36%) and the internal carotid arteries (34%).

32 er addition of intima-media thickness of the internal carotid artery (7.6%, P<0.001) but not intima-m

33 ; anterior cerebral artery, 45% and 84%; and internal carotid artery, 80% and 77%, respectively.

34 The UCAs were mostly located in the internal carotid artery (81%), and 90.2% had a maximum d

35 Internal carotid artery agenesis is a rare anomaly that

36 arotid artery, occlusive disease of terminal internal carotid artery, an abnormally straight course o

37 poventilation, vascular malformations of the internal carotid arteries and cardiac outflow tract, men

38 sive stenosis of the terminal portion of the internal carotid arteries and compensatory capillary col

39 elocity (V(max)) was evaluated in the distal internal carotid arteries and middle cerebral arteries.

40 tery vasculopathy affecting the intracranial internal carotid arteries and proximal middle cerebral a

41 rized by progressive occlusion of the distal internal carotid arteries and the formation of collatera

42 The left internal carotid artery and both internal jugular veins

43 successful in 71% of the cases for the right internal carotid artery and in only 7.7% for the left in

44 ly segmented perivascular regions: region 1 (internal carotid artery and M1 branch of middle cerebral

45 through the external carotid artery into the internal carotid artery and MCA.

46 d high-performance detection of intracranial internal carotid artery and middle cerebral artery M1 oc

47 a defect in contrast filling within the left internal carotid artery and right vertebral artery.

48 f occlusion of the middle cerebral artery or internal carotid artery and salvageable tissue as determ

49 ch as branching of the internal intracranial internal carotid artery and the basilar artery.

50 cerebral blood flow (CBF) regulation in the internal carotid artery and vertebral artery.

51 roximal occlusion after stroke (intracranial internal carotid artery and/or middle cerebral artery M1

52 ng between November 2016 and April 2019 with internal carotid artery and/or proximal middle cerebral

53 Adults with occlusion of the intracranial internal carotid artery and/or the proximal middle cereb

54 urements of common carotid, bifurcation, and internal carotid arteries, and composite IMT variables c

55 were age, >50% stenosis of the contralateral internal carotid artery, and an aortic arch type II, wit

56 ements in the middle cerebral artery, distal internal carotid artery, and anterior cerebral artery di

57 ls or K-1735M2 cells were implanted into the internal carotid artery, and on day 10, the s.c. tumors

58 .1-3.1); and rupture of anterior cerebral or internal carotid artery aneurysm (OR, 1.9; 95% CI, 1.0-3

59 ients (2.04%), one patient (1.02%) had right internal carotid artery aneurysm and one patient (1.02%)

60 ular risk factors with a small-sized (<7 mm) internal carotid artery aneurysm, to more than 15% in pa

61 nd MR angiography was lower for detection of internal carotid artery aneurysms compared with that at

62 ive treatment of large or giant intracranial internal carotid artery aneurysms, demonstrated by high

63 tion for large or giant wide-necked proximal internal carotid artery aneurysms.

64 Two had basilar artery aneurysms, 7 had internal carotid artery anomalies, and another had a mid

65 One of the individuals with an internal carotid artery anomaly also had renal artery st

66 the distal internal carotid arteries, distal internal carotid arteries, anterior cerebral arteries, p

67 In this article we will discuss variants of internal carotid artery, anterior cerebral artery, anter

68 chnoid haemorrhage with aneurysms located in internal carotid artery, anterior communicating or anter

69 ld of view of conventional PET scanners, the internal carotid arteries are commonly used to obtain an

70 Tortuous course of internal carotid arteries at the neck level was visualiz

71 Alternatively, transection of the internal carotid artery at the bulb with eversion endart

72 Extracranial internal carotid artery atherosclerotic occlusive diseas

73 We used the setting of clinically indicated internal carotid artery balloon test occlusions in 44 pa

74 e and passive heat stress, with no change in internal carotid artery blood flow Neurovascular couplin

75 Baseline internal carotid artery blood flow was similar and uncha

76 -like lesion along the posterior wall of the internal carotid artery bulb and an underrecognized caus

77 disease (MMD)-like occlusions of the distal internal carotid arteries, but the mechanisms of pathoge

78 care for symptomatic 70%-99% stenosis of the internal carotid artery, but stenting might be an option

79 lectrocardiographic abnormality, stenosis of internal carotid artery (by ultrasound), congestive hear

80 oled in a water bath, and infused through an internal carotid artery catheter, which was positioned w

81 ic arterial calibre coincides with where the internal carotid artery changes from an elastic to muscu

82 Pseudo-occlusion (PO) of the cervical internal carotid artery (cICA) can be caused by distal I

83 Cervical internal carotid artery (cICA) occlusion is a recognized

84 For the internal carotid artery, cIMT measurements in both obese

85 the following: (a) stenosis delineation, (b) internal carotid artery delineation, (c) intravascular s

86 e-arm brachial index, atherosclerosis of the internal carotid artery, diabetes mellitus, and several

87 mellitus, coronary artery disease, smoking, internal carotid artery diameter, hyperlipidemia, hyperc

88 /mmHg; P = 0.49), nor did l-NMMA impact peak internal carotid artery dilatation during the steady-sta

89 /mmHg; P = 0.49), nor did l-NMMA impact peak internal carotid artery dilatation during the steady-sta

90 inct from Moyamoya disease, characterised by internal carotid artery dilatation, terminal segment ste

91 Internal carotid artery dissection (ICAD) is a frequent

92 MRI of the head with MR angiography showed internal carotid artery dissection on the left side and

93 effective for the treatment of extracranial internal carotid artery dissection, and whether it shoul

94 cular stenting in patients with extracranial internal carotid artery dissection.

95 Extracranial internal carotid artery dissections (EICAD) remain a rel

96 erebral arteries, bifurcations of the distal internal carotid arteries, distal internal carotid arter

97 tic vasospasm related to middle cerebral and internal carotid artery distributions than for anterior

98 months, 1,020 consecutive patients underwent internal carotid artery Doppler US.

99 No dilatation was observed in the left internal carotid artery during the cluster attack.

100 acute anemic events (AAE), and extracranial internal carotid artery (eICA) stenoses, detectable via

101 fraction of microemboli infused through the internal carotid artery failed to be lysed or washed out

102 score of 5 or less) and LVO (basilar artery, internal carotid artery, first [M1] or second [M2] segme

103 Similar results were obtained in the internal carotid artery for compliance (10.1, 11.0, and

104 BackgroundStenosis of the internal carotid artery has a higher risk for stroke.

105 RA measured flow in the basilar artery (BA), internal carotid arteries (ICA), and the ascending aorta

106 fused at a rate of 0.1 ml min(-1) through an internal carotid artery (ICA) - the major vascular suppl

107 th common carotid arteries (CCAs) and in one internal carotid artery (ICA) 2 mm above the flow divide

108 dy sought to compare VIFs extracted from the internal carotid artery (ICA) and its branches with an a

109 erebral blood flow (CBF) was measured at the internal carotid artery (ICA) and vertebral artery (VA)

110 ckground Symptomatic acute occlusions of the internal carotid artery (ICA) below the circle of Willis

111 the statement include the following: (a) All internal carotid artery (ICA) examinations should be per

112 del to achieve reperfusion (REP) through the internal carotid artery (ICA) following small clot embol

113 Evidence for significant linkage to internal carotid artery (ICA) IMT (two-point log odds [L

114 intake with common carotid artery (CCA) and internal carotid artery (ICA) IMT and IMT progression.

115 secting aneurysm in the cavernous segment of internal carotid artery (ICA) is a relatively rare entit

116 CT) angiography to distinguish true cervical internal carotid artery (ICA) occlusion from pseudo-occl

117 from different institutions for intracranial internal carotid artery (ICA) or middle cerebral artery

118 d treatment for acute ischemic stroke due to internal carotid artery (ICA) or middle cerebral artery

119 acute anemic events (AAEs) and extracranial internal carotid artery (ICA) stenosis as risk factors f

120 o carotid plaque thickness and the degree of internal carotid artery (ICA) stenosis.

121 k-systolic velocity greater than 1.25 m/sec, internal carotid artery (ICA) to common carotid artery (

122 section beginning at the cervical segment of internal carotid artery (ICA) together with a dissecting

123 measures of the common carotid artery (CCA), internal carotid artery (ICA), and bulb segments of the

124 ents aged>18 years with vasospasm>50% of the internal carotid artery (ICA), anterior cerebral artery

125 ts aged >18 years with vasospasm >50% of the internal carotid artery (ICA), anterior cerebral artery

126 representative extracerebral blood vessels [internal carotid artery (ICA), basilar artery (BA), midd

127 tutes of Health Stroke Scale (NIHSS) < 6 and internal carotid artery (ICA), M1, or M2 occlusions from

128 Patients with baseline intracranial internal carotid artery (ICA), M1-middle cerebral artery

129 1)) or Ang II (150 ng) was injected into the internal carotid artery (ICA).

130 of the ECA, common carotid artery (CCA), and internal carotid artery (ICA).

131 teral complete occlusion of the intracranial internal carotid artery (ICA; 26 patients: median Nation

132 Carotid T- or L-type occlusion (terminal internal carotid artery [ICA] with M1 middle cerebral ar

133 rta and the large extracranial arteries--the internal carotid arteries (ICAs) and the vertebral arter

134 ) of the common carotid artery (CCA) and the internal carotid artery (ICAs) and with incident or prog

135 nd the maximum intima-media thickness of the internal carotid artery in 2965 members of the Framingha

136 was 3 (1-4), and the occlusion site was the internal carotid artery in 97 patients (13.3%), M1 in 20

137 selective hypothermic perfusion of a single internal carotid artery in four baboons.

138 ations (common carotid artery, carotid bulb, internal carotid artery) in both the left and right caro

139 diffuse microinfarcts induced by unilateral internal carotid artery injection of cholesterol crystal

140 associated with higher risk of stroke due to internal carotid artery injuries, but monitoring was not

141 Among those with isolated internal carotid artery injuries, five of nine with dela

142 adjusting for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk rati

143 Among patients who sustained an internal carotid artery injury with or without additiona

144 coronary artery calcium (CAC) and common and internal carotid artery intima media thickness (IMT) and

145 in men with normal ABI, significantly higher internal carotid artery intima-media thickness was obser

146 r abdominal aortic calcium score, common and internal carotid artery intima-media thickness, and ankl

147 of this trial was to determine the safety of internal carotid artery, intra-arterially delivered auto

148 tima-media thickness (IMT) of the common and internal carotid arteries is an established surrogate fo

149 Because the proximal internal carotid artery is anastomosed to the common car

150 Atherosclerosis leading to stenosis of the internal carotid artery is the underlying cause of 8-15%

151 Internal carotid artery lumen and IA diameters are both

152 f the walls of the common carotid artery and internal carotid artery may add to the Framingham risk s

153 ble clinical response than those with tandem internal carotid artery-MCA occlusion and early recanali

154 The percentage diameter stenosis of the internal carotid artery measured at US angiography stron

155 n the middle cerebral artery to extracranial internal carotid artery more than or equal to 3.

156 occlusions of the middle cerebral artery or internal carotid artery, most of whom had undergone endo

157 raluminal 4-0 monofilament nylon suture into internal carotid artery (n=195).

158 e disequilibrium on chromosome 20p11 for the internal carotid artery near wall, next to the gene PAX1

159 Patients with symptomatic atherosclerotic internal carotid artery occlusion (AICAO) and hemodynami

160 ted to the present pooled analysis (120 with internal carotid artery occlusion and 360 with isolated

161 ings were obtained in a patient with a right internal carotid artery occlusion and an infarct in the

162 al hypoperfusion was achieved with bilateral internal carotid artery occlusion and pharmacologically

163 MR angiograms in four patients showed internal carotid artery occlusion in one.

164 ents with middle cerebral artery or terminal internal carotid artery occlusion using computed tomogra

165 of angiographically proven asymptomatic left internal carotid artery occlusion with normal CT after a

166 giographically confirmed Moyamoya (n = 8) or internal carotid artery occlusions (n = 10).

167 ment middle cerebral artery +/- intracranial internal carotid artery occlusions on baseline computed

168 ascular features were dilatation of proximal internal carotid artery, occlusive disease of terminal i

169 T) using B-mode ultrasound in the common and internal carotid arteries of 141 CAD case patients and 1

170 d 25-mm thrombus was injected into the right internal carotid artery of a rat.

171 When injected into the internal carotid artery of mice, K-1735 melanoma cells p

172 ue, defined as intima-media thickness of the internal carotid artery of more than 1.5 mm, the net rec

173 1735M2 melanoma cells were injected into the internal carotid artery of naive or treated mice.

174 visceral metastasis) were injected into the internal carotid artery of nude mice.

175 Injection of human tumour cells into the internal carotid artery of syngeneic or nude mice produc

176 patients with acute stroke with intracranial internal carotid artery or first segment of middle cereb

177 Eligible patients had occlusions in the internal carotid artery or middle cerebral artery (M1 or

178 cortical vein opacification in patients with internal carotid artery or middle cerebral artery (MCA)

179 All consecutive patients with internal carotid artery or middle cerebral artery occlus

180 -reperfusion time in large-artery occlusion (internal carotid artery or middle cerebral artery) on ou

181 emic stroke due to proximal occlusion of the internal carotid artery or of the first segment of the m

182 patients with occlusion of the intracranial internal carotid artery or proximal middle cerebral arte

183 artery can arise from different parts of the internal carotid artery or the distal branches of the ex

184 patients with stroke due to occlusion of the internal carotid artery or the first segment of the midd

185 In 24 patients with terminal internal carotid artery or the proximal middle cerebral

186 (large hypophyseal opening accommodating the internal carotid arteries) or osteichthyans (facial nerv

187 r disease, atherosclerosis of the common and internal carotid arteries, or diabetes mellitus was incr

188 r segments and the extracircular part of the internal carotid artery (p < 0.05).

189 Internal carotid artery patency was 100%, while the over

190 The angle-corrected internal carotid artery peak systolic and end-diastolic

191 Internal carotid artery peak systolic velocities decreas

192 Internal carotid artery peak systolic velocity was used

193 jection/external organ (IV/EO) method and an internal carotid artery perfusion (ICAP) technique in pa

194 gotomized decerebrated rats, ligation of the internal carotid arteries preserved peripheral chemorece

195 (0-0.31 mg/kg) infused unilaterally via the internal carotid artery produced stable hemiparkinsonism

196 MPTP (0.4 mg/kg) was infused into one internal carotid artery, producing a contralateral hemi-

197 gnificant reduction in antegrade flow in the internal carotid artery proximal to the filter device, t

198 lood pressure, and total cholesterol for the internal carotid artery (r2=0.21).

199 thickness of (and presence of plaque in) the internal carotid artery significantly (albeit modestly)

200 rtonic NaCl (1.5 Osm/L, 100 mul) through the internal carotid artery significantly (P<0.01) increased

201 039 (95% CI, -0.0009 to 0.0088) mm/y for the internal carotid artery sites (P = .02).

202 the common carotid artery, carotid bulb, and internal carotid artery sites and in mean CIMT of the co

203 nial occlusion, and presence of extracranial internal carotid artery stenosis (>50%) demonstrated on

204 nial carotid disease was defined as cervical internal carotid artery stenosis (>50%) or occlusion.

205 he systolic velocity ratio for assessment of internal carotid artery stenosis and decrease some of th

206 Internal carotid artery stenosis and intima-media thickn

207 impairment and decline associated with left internal carotid artery stenosis and intima-media thickn

208 Twenty patients with > or =50% internal carotid artery stenosis and with > or =3 emboli

209 and investigated the impact of contralateral internal carotid artery stenosis on carotid artery stump

210 Thirty-one patients with 50%-99% internal carotid artery stenosis underwent dynamic contr

211 lasty and stenting for symptomatic > or =70% internal carotid artery stenosis were randomized in a do

212 ing CAS with cerebral embolic protection for internal carotid artery stenosis were randomly assigned

213 ransient monocular blindness associated with internal-carotid-artery stenosis is a risk factor for st

214 ia the Circle of Willis upon stenosis of the internal carotid arteries, supply blood to the anterior

215 lus elicits shear-mediated dilatation of the internal carotid artery, termed cerebral shear-mediated

216 mpared with the IVT and NRT groups combined: internal carotid artery terminus (75 vs 190 cm3; P < .00

217 between SI and the IMT was stronger for the internal carotid artery than for the common carotid arte

218 s a progressive stenosis of the supraclinoid internal carotid artery that causes stroke (especially i

219 ting centre with a confirmed stenosis of the internal carotid artery that was deemed equally suitable

220 the carotid artery (carotid sinus and distal internal carotid artery) that are typically "susceptible

221 stantially for the bifurcation of the distal internal carotid artery, the posterior cerebral artery,

222 ive [60.0%] studies), and bifurcation of the internal carotid artery (three of nine [33.3%] aneurysms

223 ll patients had at least 80% stenosis in one internal carotid artery, three of them also had contrala

224 -0 surgical suture through the left cervical internal carotid artery to obstruct the blood flow into

225 c 3D-generic model of a graft connecting the internal carotid artery to the jugular vein.

226 continuous wave ultrasound directed onto the internal carotid artery triggered Xe release from circul

227 ffness index were assessed in the common and internal carotid arteries using duplex ultrasound equipp

228 rosclerotic plaque on carotid bifurcation or internal carotid artery using the Mannheim consensus def

229 low velocity in the ipsilateral extracranial internal carotid artery (V(ICA)) was calculated.

230 ured vessels, vessel grade, and vessel type (internal carotid artery, vertebral artery) with BCVI-ass

231 alth Stroke Scale score), site of occlusion (internal carotid artery vs M1 segment of middle cerebral

232 topotecan; P < 0.05), catheterization route (internal carotid artery vs. external carotid or posterio

233 lar (LV) mass, ECG ST-T segment abnormality, internal carotid artery wall thickness and decreased LV

234 pite this, patients with CGD had a 22% lower internal carotid artery wall volume compared with contro

235 IMT for the common and internal carotid arteries was determined through B-mode

236 or the maximum intima-media thickness of the internal carotid artery was 1.21 (95% CI, 1.13 to 1.29),

237 Saccular aneurysm of left internal carotid artery was diagnosed in 2 patients (2.0

238 Test balloon occlusion of the internal carotid artery was performed in 44 patients wit

239 Extradural aneurysms in the internal carotid artery were included; fusiform aneurysm

240 ts of the intima and media of the common and internal carotid artery were made with high-resolution u

241 deling index, lipid core, and calcium in the internal carotid artery were significant predictors of e

242 ted to an endovascular SAH through the right internal carotid artery while under anesthesia.

243 led a high-grade stenosis of the ipsilateral internal carotid artery with a 4-cm intraluminal thrombu

244 rague-Dawley rats were infused via the right internal carotid artery with glucose (4 mg/kg/min) or eq

245 arises from the supraclinoid segment of the internal carotid artery within the subarachnoid space an

246 tion into the lumen of the proximal cervical internal carotid artery without evidence of calcificatio