ライフサイエンスコーパス: interstitial fibrosis

1 cued angiotensin II-induced perivascular and interstitial fibrosis.

2 tion slowing in an in vitro model of cardiac interstitial fibrosis.

3 ressive chronic kidney disease and pulmonary interstitial fibrosis.

4 ular cells is involved in the development of interstitial fibrosis.

5 chronic Banff components tubular atrophy and interstitial fibrosis.

6 ion of the kidney parenchyma and attenuating interstitial fibrosis.

7 uolization in myocytes and in another patchy interstitial fibrosis.

8 of different rhubarb extract against tubulo-interstitial fibrosis.

9 glomerulosclerosis, and more tubular atrophy/interstitial fibrosis.

10 bal glomerulosclerosis, tubular atrophy, and interstitial fibrosis.

11 characterized by tubular cell apoptosis and interstitial fibrosis.

12 ctive oxygen species, and manifested cardiac interstitial fibrosis.

13 rstitial changes, and histologic findings of interstitial fibrosis.

14 ular hypertrophy, disarray of myofibers, and interstitial fibrosis.

15 re protected against cardiac dysfunction and interstitial fibrosis.

16 ant decreases in myocardial apoptosis and LV interstitial fibrosis.

17 g novel disease-modifying therapy, targeting interstitial fibrosis.

18 dysfunction by promoting tubular atrophy and interstitial fibrosis.

19 ilateral ureteral obstruction model of renal interstitial fibrosis.

20 in recovered more rapidly and exhibited less interstitial fibrosis.

21 ment of extracellular volume as a measure of interstitial fibrosis.

22 n content of the heart increases, leading to interstitial fibrosis.

23 developed concentration- and time-dependent interstitial fibrosis.

24 tracellular matrix, and development of renal interstitial fibrosis.

25 n quantify the burden of global and regional interstitial fibrosis.

26 in association with myocyte hypertrophy and interstitial fibrosis.

27 re taken from lung cancer resections without interstitial fibrosis.

28 There was no increase in interstitial fibrosis.

29 t RGC-32 is essential for the onset of renal interstitial fibrosis.

30 ch TIN, with most showing diffuse, expansile interstitial fibrosis.

31 ition important for the development of renal interstitial fibrosis.

32 cted kidney, with marked tubular atrophy and interstitial fibrosis.

33 thers to be responsible for nephron loss and interstitial fibrosis.

34 t and correlated directly with the degree of interstitial fibrosis.

35 d seven showed > or =40% tubular atrophy and interstitial fibrosis.

36 ly contributing to the pathogenesis of renal interstitial fibrosis.

37 inhibition of ATX partially attenuated renal interstitial fibrosis.

38 erstitial inflammation, tubular atrophy, and interstitial fibrosis.

39 a novel therapeutic strategy to combat renal interstitial fibrosis.

40 although only sacubitril/valsartan prevented interstitial fibrosis.

41 l inflammation, minimal tubular atrophy, and interstitial fibrosis.

42 myocyte hypertrophy and disarray, as well as interstitial fibrosis.

43 p53/miR-199a-3p/SOCS7/STAT3 pathway in renal interstitial fibrosis.

44 allografts showed less glomerular injury and interstitial fibrosis.

45 aused tubular obstruction, inflammation, and interstitial fibrosis.

46 tially improved cardiac function and reduced interstitial fibrosis.

47 essened bladder mucosa damage, and decreased interstitial fibrosis.

48 s glomerular sclerosis, tubular atrophy, and interstitial fibrosis.

49 ructure, and arrested glomerulosclerosis and interstitial fibrosis.

50 tubule cell (RPTC) injury, inflammation, and interstitial fibrosis.

51 d promoted macrophage persistence and severe interstitial fibrosis.

52 lar hypertrophy, left atrial dilatation, and interstitial fibrosis.

53 tractile dysfunction, and heart failure with interstitial fibrosis 1 week after pressure overload.

54 ve stress (1 day post-I/R), hypertrophy, and interstitial fibrosis (1 week).

55 ia length 4.4+/-0.4 versus 5.2+/-0.6 mg/mm), interstitial fibrosis (11.2+/-3.1% versus 18.5+/-3.5%),

56 phritis [23 of 25 (92%)], and glomerular and interstitial fibrosis [12 of 25 (48%)] were predominant

57 th minimal tubular atrophy (20%) and/or <20% interstitial fibrosis (24%).

58 Other features included interstitial fibrosis (5), poorly-formed granulomas (4),

59 Interstitial fibrosis, a common pathological feature of

60 is associated with a dramatic inhibition of interstitial fibrosis, a fivefold reduction in alpha-smo

61 s specialized macrophage population enhanced interstitial fibrosis after ischemic injury.

62 ce were protected against the development of interstitial fibrosis after obstructive injury.

63 recovery and late graft function and reduced interstitial fibrosis after transplant.

64 autophagy in the CryABR120G hearts decreased interstitial fibrosis, ameliorated ventricular dysfuncti

65 f cardiac disease, leading to an increase in interstitial fibrosis and a decrease in ventricular func

66 ignificantly attenuated cardiac hypertrophy, interstitial fibrosis and apoptosis, and better cardiac

67 nterstitial inflammation and subsequently to interstitial fibrosis and atrophy of the renal tubules i

68 ) has demonstrated utility to detect diffuse interstitial fibrosis and changes in regional myocardial

69 severe AKI, characterized by development of interstitial fibrosis and CKD.

70 uroursodeoxycholic acid or BGP-15, decreased interstitial fibrosis and collagen deposition in ovaries

71 s at week 6 could predict the development of interstitial fibrosis and de novo donor specific anti-HL

72 infiltration of inflammatory cells, enhanced interstitial fibrosis and elevated serum creatine kinase

73 activation of sodium transporters, and cause interstitial fibrosis and glomerular injury.

74 sed urinary albumin/creatinine ratio, tubulo-interstitial fibrosis and glycosuria without changes in

75 rine model of aristolochic acid (AA)-induced interstitial fibrosis and human AA nephropathy, we obser

76 the thickness of interventricular septum and interstitial fibrosis and increases anterior wall thickn

77 they develop a cardiomyopathy that includes interstitial fibrosis and infiltration of the myocardium

78 study assessed the development of allograft interstitial fibrosis and inflammation (GIF+"i"), a hist

79 eas patients with graft loss frequently have interstitial fibrosis and inflammation (IF+i=ci>0+i>0).

80 he periostin gene showed less injury-induced interstitial fibrosis and inflammation and were protecte

81 nd histopathological abnormalities including interstitial fibrosis and inflammation, and either fully

82 Renal interstitial fibrosis and interstitial active inflammati

83 All biopsies showed less than 30% interstitial fibrosis and less than 30% to 40% glomerulo

84 ysyl oxidase-like 2 (Loxl2)-is essential for interstitial fibrosis and mechanical dysfunction of path

85 Kidney histology demonstrated interstitial fibrosis and nephrocalcinosis in addition t

86 ypertrophic remodeling, pulmonary edema, and interstitial fibrosis and prevents cardiac dysfunction a

87 ells before the onset of proteinuria delayed interstitial fibrosis and progression of glomerular scle

88 Chronic kidney disease is characterized by interstitial fibrosis and proliferation of scar-secretin

89 Biomarkers and new methods to assess interstitial fibrosis and regional myocardial function h

90 Kidney transplants with both interstitial fibrosis and subclinical inflammation but n

91 obstruction model, ICG-001 ameliorated renal interstitial fibrosis and suppressed renal expression of

92 egardless of age, pulmonary hypertension, or interstitial fibrosis and that this relation may be chan

93 ype mice, NOX4-deficient mice exhibited more interstitial fibrosis and tubular apoptosis after obstru

94 rom acute and subclinical rejection (67.4%); interstitial fibrosis and tubular atrophy (14.4%); BK vi

95 Subclinical rejection and interstitial fibrosis and tubular atrophy (IF/TA) in pro

96 ome of doubling of interstitium or ESRD from interstitial fibrosis and tubular atrophy (IF/TA) in the

97 Chronic allograft damage, defined by interstitial fibrosis and tubular atrophy (IF/TA), is a

98 Interstitial fibrosis and tubular atrophy (IFTA) associa

99 MicroRNA (miRNA) alterations accompanying interstitial fibrosis and tubular atrophy (IFTA) in kidn

100 t 6-month urinary CCL2: Cr is a predictor of interstitial fibrosis and tubular atrophy (IFTA) on 24-m

101 The biopsy diagnosis of interstitial fibrosis and tubular atrophy (IFTA) was bas

102 abnormal glomerulosclerosis, 23.9% abnormal interstitial fibrosis and tubular atrophy (IFTA), 4.8% a

103 ntation and if production is associated with interstitial fibrosis and tubular atrophy (IFTA).

104 py is a contributing factor to the origin of interstitial fibrosis and tubular atrophy (IFTA).

105 gression of chronic kidney disease (CKD) are interstitial fibrosis and tubular atrophy (IFTA).

106 poorly characterized histopathologic entity interstitial fibrosis and tubular atrophy (IFTA).

107 lant patients with normal histology (n = 5), interstitial fibrosis and tubular atrophy (n = 6), subcl

108 emptive therapy group had moderate to severe interstitial fibrosis and tubular atrophy (odds ratio, 2

109 R at 12 months (P=0.001) and higher risk for interstitial fibrosis and tubular atrophy (P=0.01).

110 INK4a(-/-) mice developed significantly less interstitial fibrosis and tubular atrophy after ischemia

111 -dependent senescence, significantly reduced interstitial fibrosis and tubular atrophy and associated

112 P = 0.009), viral nephropathies (P = 0.002), interstitial fibrosis and tubular atrophy and inflammati

113 al cellular senescence in the development of interstitial fibrosis and tubular atrophy and kidney gra

114 lganciclovir therapy may lead to less severe interstitial fibrosis and tubular atrophy and to signifi

115 sult in chronic histological damage of which interstitial fibrosis and tubular atrophy are dominant f

116 Interstitial fibrosis and tubular atrophy are major cont

117 question in the human kidney allografts with interstitial fibrosis and tubular atrophy not otherwise

118 hose with normal histology, while those with interstitial fibrosis and tubular atrophy plus inflammat

119 sease process can be identified, what drives interstitial fibrosis and tubular atrophy progression in

120 After 1 year, interstitial fibrosis and tubular atrophy score was sign

121 Interstitial fibrosis and tubular atrophy score, interst

122 Extensive interstitial fibrosis and tubular atrophy without a clea

123 cimens for the degree of glomerulosclerosis, interstitial fibrosis and tubular atrophy, and vascular

124 ic signs of AKI to CKD transition, including interstitial fibrosis and tubular atrophy, and with an a

125 nic allograft injury (CAI), characterized by interstitial fibrosis and tubular atrophy, leads to a pr

126 flamed histology (1.4 [0.4, 4.2], normal and interstitial fibrosis and tubular atrophy, n = 52), and

127 that together were associated with increased interstitial fibrosis and tubular atrophy.

128 ith interstitial inflammation, and extensive interstitial fibrosis and tubular atrophy.

129 een observed in renal transplants developing interstitial fibrosis and tubular atrophy; however, whet

130 le cardiac remodelling, such as hypertrophy, interstitial fibrosis, and abnormal activity of the card

131 renal injury, including glomerulosclerosis, interstitial fibrosis, and albuminuria.

132 global glomerulosclerosis, tubular atrophy, interstitial fibrosis, and arteriosclerosis.

133 te foot process effacement, tubular atrophy, interstitial fibrosis, and casts, were observed.

134 y associated with cardiomyocyte hypertrophy, interstitial fibrosis, and contractile dysfunction.

135 development of left ventricular hypertrophy, interstitial fibrosis, and diastolic dysfunction.

136 kidney function, more glomerulosclerosis and interstitial fibrosis, and greater propensity to progres

137 ent, mitochondrial damage, apoptosis, and LV interstitial fibrosis, and hence contributes to both sys

138 iting pathological myofibroblast activation, interstitial fibrosis, and HF progression.

139 expression, attenuated tubular apoptosis and interstitial fibrosis, and improved renal function and t

140 es positively correlated with adipose tissue interstitial fibrosis, and in vitro experiments indicate

141 tion, hypoperfusion, increased deposition of interstitial fibrosis, and increased apoptosis of endoth

142 very, reduced postinjury tubular atrophy and interstitial fibrosis, and increased the regenerative ca

143 ich had severe vascular intimal hyperplasia, interstitial fibrosis, and inflammation.

144 omyocyte necrosis with replacement fibrosis, interstitial fibrosis, and intercalated disc dissociatio

145 representing a surrogate marker of reactive interstitial fibrosis, and late gadolinium enhancement (

146 loss), accumulation of perinuclear glycogen, interstitial fibrosis, and myocardial gap junction distr

147 well as reduced levels of oxidative stress, interstitial fibrosis, and myocyte apoptosis.

148 in the thickness of interventricular septum, interstitial fibrosis, and phosphorylated p38 mitogen-ac

149 iron deposition, blocked the development of interstitial fibrosis, and prevented immune cell infiltr

150 tivity, cell proliferation, cyst growth, and interstitial fibrosis, and significantly improved renal

151 gnificantly less tubular senescence, reduced interstitial fibrosis, and superior renal function 30 da

152 in promotes cell proliferation, cyst growth, interstitial fibrosis, and the decline in renal function

153 hrough macrophage infiltration, resulting in interstitial fibrosis, and through altered paracrine sig

154 function through mitochondrial apoptosis, LV interstitial fibrosis, and to some extent to myocardial

155 teriolosclerosis, global glomerulosclerosis, interstitial fibrosis, and tubular atrophy) all increase

156 on, biopsy-proved acute rejection, >=grade 2 interstitial fibrosis, and tubular atrophy), as well as

157 with increased fractional interstitial area, interstitial fibrosis, and tubular atrophy.

158 ormal mitochondria; extensive subendocardial interstitial fibrosis; and marked hypertrophy of arterio

159 and genetic blockade of p53 attenuated renal interstitial fibrosis, apoptosis, and inflammation in mi

160 In contrast, chronic Banff scores (interstitial fibrosis, arteriolar hyalinosis, mesangial

161 EDP-305 also dose-dependently reduced interstitial fibrosis as assessed by morphometric quanti

162 ed with myocardial segmental dysfunction and interstitial fibrosis, as assessed by ECV, in HCM patien

163 In addition, these hearts develop interstitial fibrosis associated with diffuse N-cadherin

164 Hypertension, lower eGFR, and interstitial fibrosis associated with glomerulosclerosis

165 inated the difference in obstruction-induced interstitial fibrosis between wild-type and VDR-null mic

166 ntly preserves cardiac function, and reduces interstitial fibrosis but does not alter the numbers of

167 centage of glomerulosclerosis, and degree of interstitial fibrosis but not immunomodulatory treatment

168 Hypothyroidism increased atrial interstitial fibrosis, but connexin 43 was not affected.

169 us casts, with marked glomerulosclerosis and interstitial fibrosis by 6 weeks of age.

170 aracterized by deteriorating graft function, interstitial fibrosis, cardiac hypertrophy, and occlusiv

171 Furthermore, fingolimod decreased interstitial fibrosis, cardiomyocyte hypertrophy, and ch

172 The extent of atrial interstitial fibrosis, cardiomyocyte myocytolysis, cardi

173 ntrarenal polyomavirus load levels and Banff interstitial fibrosis ci scores.

174 Moderate/severe interstitial fibrosis (ci>/=2) at 1 year was observed in

175 The total Banff score equaled the sum of interstitial fibrosis (CI), tubular atrophy, arteriolar

176 glomerular hypertrophy, podocyte injury, and interstitial fibrosis compared with diabetic controls fe

177 remodeling, smaller LV cavity size, and more interstitial fibrosis compared with high gradient (HG) n

178 titin; and 5) both stiff cardiomyocytes and interstitial fibrosis contribute to high diastolic left

179 ths were associated with moderate and severe interstitial fibrosis developed at 24 months after trans

180 ey pathology, including hyperplasia of PTCs, interstitial fibrosis, development of glomerular cysts,

181 uding cardiac magnetic resonance measures of interstitial fibrosis (extracellular volume fraction), a

182 e breaks conditioned by strands of increased interstitial fibrosis (fibrous tissue content in heart f

183 eases in cardiomyocyte cell surface area and interstitial fibrosis following pressure overload.

184 ed this model to examine renal apoptosis and interstitial fibrosis following unilateral urethral obst

185 In CatA-TG mice, LV interstitial fibrosis formation was enhanced by 19%, and

186 The presence of glomerulitis or chronic interstitial fibrosis (g and ci scores by Banff, respect

187 dences of acute mild arteriolopathy, striped interstitial fibrosis, glomerular congestion, and tubula

188 han 40% global glomerular sclerosis, or with interstitial fibrosis greater than 30% or for patients o

189 iratory diseases, including airway diseases; interstitial fibrosis; hypersensitivity pneumonitis; oth

190 Loss of kidney graft function due to interstitial fibrosis (IF) and tubular atrophy (TA) is t

191 Interstitial fibrosis (IF) and tubular atrophy (TA) was

192 The incidence of (interstitial fibrosis) IF/(tubular atrophy) TA at month

193 of pro-fibrotic cytokines, thereby promoting interstitial fibrosis in a paracrine manner.

194 Ang-(1-7) concomitantly reduced interstitial fibrosis in association with a significant

195 crease in both myofibroblast recruitment and interstitial fibrosis in bleomycin-treated syndecan-4-nu

196 ities, suggesting that imaging biomarkers of interstitial fibrosis in cancer survivors are related to

197 liorate hypertension, renal hypertrophy, and interstitial fibrosis in diabetes.

198 ificantly to the tubular apoptosis and renal interstitial fibrosis in kidney diseases.

199 model can be used to monitor treatments for interstitial fibrosis in LN with drugs currently being d

200 e was significantly less lung congestion and interstitial fibrosis in MAFbx KO than in WT mice.

201 d blunts expression of profibrotic genes and interstitial fibrosis in mice subjected to sustained pre

202 ific knockdown of Shroom3 markedly abrogated interstitial fibrosis in mice with unilateral ureteric o

203 oad-induced heart failure despite inhibiting interstitial fibrosis in mice.

204 r expansion (ECE), which has been related to interstitial fibrosis in previous clinical and subclinic

205 independently associated with the degree of interstitial fibrosis in protocol biopsies, scored accor

206 several inflammatory molecules and increased interstitial fibrosis in regenerating muscle.

207 ant alterations in microvascular density and interstitial fibrosis in remodeling myocardial segments.

208 diseases characterized by tubular damage and interstitial fibrosis in the absence of glomerular lesio

209 tant to aldosterone-dependent progression of interstitial fibrosis in the kidney.

210 ammation and tubular injury with progressive interstitial fibrosis in the late stages of U-IRI.

211 with extensive fibrosis in the LAI group and interstitial fibrosis in the LCx group.

212 tubular cell apoptosis and suppressed renal interstitial fibrosis in UUO.

213 howed preserved cardiac function and reduced interstitial fibrosis in vivo and reduced fibroblast pro

214 rowth factor beta1 (TGF-beta) promotes renal interstitial fibrosis in vivo and the expression of mese

215 and we have shown that HA is a correlate of interstitial fibrosis in vivo.

216 erity of tubular atrophy, tubular casts, and interstitial fibrosis increased significantly in Nr4a1(-

217 deletion of miR-214 significantly attenuated interstitial fibrosis induced by unilateral ureteral obs

218 In a mouse model of renal interstitial fibrosis induced by unilateral ureteral obs

219 ressive acinar cell vacuolization and death, interstitial fibrosis, inflammation, and circulatory rel

220 Treatment with 5-MTP ameliorates renal interstitial fibrosis, inhibits IkappaB/NF-kappaB signal

221 Interstitial fibrosis is an important component of diast

222 Interstitial fibrosis is an outcome measure of increasin

223 ate that the severity and structure of local interstitial fibrosis is associated with atrial conducti

224 Six weeks post-reperfusion, interstitial fibrosis is greater in aged kidneys based o

225 Diffuse interstitial fibrosis is present in diverse cardiomyopat

226 ularization (at 28 days, post-I-R) and lower interstitial fibrosis, leading to improved left ventricu

227 myocardial apoptosis and the development of interstitial fibrosis, left atrial hypertrophy, and pulm

228 Existing data suggest that increased interstitial fibrosis may occur abnormally in renal tran

229 [95% CI, -0.029% to -0.00012%]; P=0.048 for interstitial fibrosis; mean difference, -1.3 [95% CI, -2

230 This study sought to evaluate the diffuse interstitial fibrosis measured by T1 mapping cardiac mag

231 Myocardial interstitial fibrosis (MIF) is a histological hallmark o

232 sinus node dysfunction, cardiac hypertrophy, interstitial fibrosis, multi-focal myocardial fiber deat

233 P significantly decreased volume fraction of interstitial fibrosis, normalized sarcoplasmic reticulum

234 We studied the effect of interstitial fibrosis on conduction velocity (CV) in the

235 s as well as the percent tubular atrophy and interstitial fibrosis on renal biopsy were assessed.

236 kidneys into C57BL6 mice was associated with interstitial fibrosis (P<0.0001), tubular atrophy (P<0.0

237 reservation of tubules (P=0.008) and reduced interstitial fibrosis (P=0.01), with decreased myofibrob

238 vealed a significant decrease in replacement/interstitial fibrosis, p-SMAD2/3 and p-ERK1/2.

239 Interstitial fibrosis plays a key role in the developmen

240 losclerosis, and greater tubular atrophy and interstitial fibrosis predicted ESRD.

241 plantation is an independent risk factor for interstitial fibrosis progression and is associated with

242 Pepstatin A treatment slowed interstitial fibrosis progression following IRI induced

243 , these data suggest that obesity-associated interstitial fibrosis promotes a macrophage phenotype si

244 al-derived Wnt ligand is sufficient to drive interstitial fibrosis provides strong support for the ma

245 on of EMT-related gene expression with early interstitial fibrosis (r<0.45) for size-mismatched allog

246 reversed cardiac and myocyte hypertrophy and interstitial fibrosis, reduced the propensity for ventri

247 P proteins in enhancing or suppressing renal interstitial fibrosis, respectively, we examined whether

248 Renal interstitial fibrosis results from activation and prolif

249 l dysfunction, nitric oxide disarrays, renal interstitial fibrosis, sarcopenia, and worsening protein

250 t post was used to assess volume fraction of interstitial fibrosis, sarcoplasmic reticulum calcium AT

251 re observed for g, C4d, tubular atrophy, and interstitial fibrosis scores in early AMR patients and t

252 ous intimal thickening, tubular atrophy, and interstitial fibrosis scores were associated with graft

253 igh proximal tubular proliferation rates and interstitial fibrosis, similar to known polycystic kidne

254 ongitudinal conduction blocks coincided with interstitial fibrosis strands that were exaggerated in t

255 nd CSCs similarly inhibited infarct size and interstitial fibrosis, SVPs were superior in inducing an

256 histologic scores for glomerulosclerosis and interstitial fibrosis than untreated controls.

257 ATX contributes to the development of renal interstitial fibrosis through LPA-mediated effects on fi

258 oss and lesions of focal glomerulosclerosis, interstitial fibrosis, tubular atrophy and arterioloscle

259 bly, donor age correlated significantly with interstitial fibrosis, tubular atrophy, and glomeruloscl

260 Interstitial fibrosis, tubular atrophy, and glomeruloscl

261 disease, among other factors, the result is interstitial fibrosis, tubular atrophy, and graft failur

262 al end points, including glomerulosclerosis, interstitial fibrosis, tubular injury, and inflammation.

263 Interstitial fibrosis/tubular atrophy (IF/TA) contribute

264 d percentages of artery luminal stenosis and interstitial fibrosis/tubular atrophy (IF/TA) of the cor

265 KCC2, E-cadherin, and 18S rRNA diagnostic of interstitial fibrosis/tubular atrophy (IF/TA).

266 Interstitial fibrosis/tubular atrophy (IFTA) is an impor

267 l casts per square millimeter, and degree of interstitial fibrosis/tubular atrophy (IFTA) were indepe

268 model included GFR (P<0.001) and presence of interstitial fibrosis/tubular atrophy (P=0.003) at diagn

269 VAN = 10, normal functioning graft = 73, and interstitial fibrosis/tubular atrophy = 59) from 168 uni

270 with increased allograft glomerulopathy and interstitial fibrosis/tubular atrophy lesions (P<0.001 f

271 intended primary end point was the change in interstitial fibrosis/tubular atrophy score between impl

272 ejection and better renal function with less interstitial fibrosis/tubular atrophy versus calcineurin

273 l blood pressure, change in body mass index, interstitial fibrosis/tubular atrophy, and change in ren

274 We used global glomerulosclerosis, interstitial fibrosis/tubular atrophy, artery luminal st

275 Banff-2 antibody-mediated rejection, Banff-5 interstitial fibrosis/tubular atrophy, in samples from s

276 recipient clinical characteristics included interstitial fibrosis/tubular atrophy, larger cortical n

277 ibodies), in three different models of renal interstitial fibrosis, unilateral ureteral obstruction,

278 (type p, q, or r) opacities, whereas grade 3 interstitial fibrosis was associated (P = 0.02) with the

279 Finally, interstitial fibrosis was evident 20 weeks after radiati

280 Interstitial fibrosis was markedly increased in C4KO hea

281 Interstitial fibrosis was present in segments both with

282 Interstitial fibrosis was significantly increased within

283 ated apoptotic pathway and its role in renal interstitial fibrosis, we established a mouse model in w

284 Glomerulosclerosis and interstitial fibrosis were also significantly ameliorate

285 icited increases in hypertrophic markers and interstitial fibrosis were blunted in the knockout anima

286 Cardiomyocyte size and interstitial fibrosis were increased equally in both gen

287 FLC response >= VGPR and absence of severe interstitial fibrosis were independent predictors of ren

288 ar hyalinosis, mesangiolysis, and focal mild interstitial fibrosis were present.

289 Agt gene expression, renal hypertrophy, and interstitial fibrosis were studied.

290 d extracellular volume fraction, a marker of interstitial fibrosis, were determined with a model for

291 Ketamine treatment also enhanced bladder interstitial fibrosis, whereas ketamine + Cox-2 inhibito

292 ar cells significantly reduced the extent of interstitial fibrosis, which correlated with reduced fib

293 ar cells may be involved in the induction of interstitial fibrosis, which in turn, leads to loss of r

294 cludes induction of renal cyst formation and interstitial fibrosis while the latter entails urine-con

295 tion within the allograft and development of interstitial fibrosis, whilst type III collagen depositi

296 Histology indicated papillary and interstitial fibrosis with ageing, which was higher in t

297 Four had diffuse interstitial fibrosis with chronic inflammation, and two

298 Interstitial fibrosis with cross-linked collagen is prom

299 ing, and fatal lung disease characterized by interstitial fibrosis with decreasing lung volumes and h

300 characterized by myocardial remodeling with interstitial fibrosis, with reduced diastolic function a