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1 /STAT signaling, can attenuate granulomatous interstitial nephritis.
2 aving renal dysfunction that suggested acute interstitial nephritis.
3 ocyte proliferation, lymphoid depletion, and interstitial nephritis.
4 had little effect on T cell infiltration and interstitial nephritis.
5  a rise in creatinine revealed polyoma virus interstitial nephritis.
6 fection/pyelonephritis, viral nephritis, and interstitial nephritis.
7  basic protein, and renal tubular Ag-induced interstitial nephritis.
8 ients with the idiopathic variety of chronic interstitial nephritis.
9 the induction and persistence of leptospiral interstitial nephritis.
10 r epithelium, and varying degrees of chronic interstitial nephritis.
11 ng urinary tract infection (UTI) and chronic interstitial nephritis.
12 hocyte injury during allograft rejection and interstitial nephritis.
13 immune checkpoint inhibitor-associated acute interstitial nephritis.
14 CXCL9) is upregulated in patients with acute interstitial nephritis.
15 immune checkpoint inhibitor-associated acute interstitial nephritis.
16 creas grafts and no evidence of recurrent PV interstitial nephritis 22 and 37 months after retranspla
17  were acute tubular necrosis (ATN) and acute interstitial nephritis (AIN) in both adults and children
18 ry (AKI), most commonly manifesting as acute interstitial nephritis (AIN), although glomerular diseas
19 odies, glomerular IgG and C3 deposition, and interstitial nephritis all developed by 3-5 wk of age.
20  differentiating acute tubular necrosis from interstitial nephritis and as an alternative for indicat
21 idney transplant recipient who developed BKV interstitial nephritis and carcinoma of the bladder with
22 ncope, arthritic disorders, nephrolithiasis, interstitial nephritis and drug-induced pancreatitis ass
23                            Risk for allergic interstitial nephritis and immunosuppression at the time
24  convoluted tubules of the kidney by causing interstitial nephritis and inducing the same cytokines i
25                 Polyoma virus (PV) can cause interstitial nephritis and lead to graft failure in rena
26 can be seen in conjunction with SV40-induced interstitial nephritis and pneumonitis.
27 ansplant patients has been reported to cause interstitial nephritis and subsequent graft loss.
28             This was followed by progressive interstitial nephritis and tubulointerstitial renal fibr
29         Polyoma virus infection causes acute interstitial nephritis and ureteral stenosis in humans b
30 mice treated with ICIs exhibited significant interstitial nephritis and vasculitis/periarteritis, con
31  important in macrophage accumulation during interstitial nephritis and wound healing.
32 ous adverse event during deferred treatment (interstitial nephritis) and one during the placebo phase
33 -immune crescentic glomerulonephritis, acute interstitial nephritis, and amyloid nephropathy.
34  has been associated with ureteral stenosis, interstitial nephritis, and hemorrhagic cystitis.
35                       Peripheral neuropathy, interstitial nephritis, and vasculitis were more common
36                                       Tubulo-interstitial nephritis antigen (TINag) is an extracellul
37 the parent strain caused severe cystitis and interstitial nephritis as determined by histopathologica
38       Prior history of glomerulonephritis or interstitial nephritis, as cause of renal failure, repre
39                         Polyoma virus tubulo-interstitial nephritis-associated graft dysfunction usua
40 s who lost their primary kidney grafts to PV interstitial nephritis becomes a difficult one.
41             Novel features of VEXAS included interstitial nephritis, cardiac involvement, stroke, and
42                                              Interstitial nephritis caused by BK polyomavirus is a re
43 hich the most common renal lesion is chronic interstitial nephritis (CIN).
44 immune checkpoint inhibitor-associated acute interstitial nephritis compared to several control group
45       Kidneys from rats immunized to produce interstitial nephritis display a net generation of nitri
46                                              Interstitial nephritis due to BK polyomavirus occurred i
47  hemodynamics, proteinuria, tubular biology, interstitial nephritis, fibroblast formation, and fibros
48 c ESRD (ESRD caused by glomerulonephritis or interstitial nephritis) for which the SIR was 4.22 (95%
49                                      Chronic interstitial nephritis frequently accompanies renal dise
50 tiating 38 patients with biopsy-proven acute interstitial nephritis from other forms of acute kidney
51 igh discrimination for differentiating acute interstitial nephritis from other forms of acute kidney
52 gnesium), rebound acid hypersecretion, acute interstitial nephritis, gastric carcinoid tumor, cardiov
53               We conclude that in autoimmune interstitial nephritis, generation of nitric oxide throu
54  pump inhibitors (PPI) use and risk of acute interstitial nephritis has been described.
55 ly-onset nephropathy with features of tubulo-interstitial nephritis, hypertension and tendency for hy
56 une-related adverse event manifests as acute interstitial nephritis (ICI-AIN).
57 on the course of T cell-dependent autoimmune interstitial nephritis in Brown Norway rats.
58 nsfer of CSF-1 or GM-CSF incited more severe interstitial nephritis in IFN-gammaR-deficient than in I
59  retroviral gene transfer approach to incite interstitial nephritis in MRL-Fas(lpr) mice that is rapi
60  primate polyomavirus SV40 is known to cause interstitial nephritis in primary infections and progres
61                                   CPV caused interstitial nephritis in six renal allografts, a xenogr
62 ent neuropathy, granulomatous glomerular and interstitial nephritis, interstitial lung disease, arter
63 uld indicate that graft loss secondary to PV interstitial nephritis is not an absolute contraindicati
64 wn, but the clinical course of biopsy-proven interstitial nephritis is not well defined.
65 enetic changes occur in human BK virus (BKV) interstitial nephritis (ISN).
66 r, FAN1 mutations predispose to karyomegalic interstitial nephritis (KIN) and cancer rather than to F
67                                 Karyomegalic interstitial nephritis (KIN) is a chronic interstitial n
68                                 Karyomegalic interstitial nephritis (KIN) is a rare renal interstitia
69 mutations in FAN1 as a cause of karyomegalic interstitial nephritis (KIN), a disorder that serves as
70 ase 1 (FAN1) in humans leads to karyomegalic interstitial nephritis (KIN), a rare hereditary kidney d
71   Mutations in human Fan1 cause karyomegalic interstitial nephritis (KIN), but it is unclear whether
72  the human DNA damage syndrome, karyomegalic interstitial nephritis (KIN).
73 -Schiff D staining of infected kidney showed interstitial nephritis, mononuclear cell infiltrates, an
74 increased, chondrocalcinosis, synovitis, and interstitial nephritis (n=1 each).
75 neumonia is inconsistent, and although acute interstitial nephritis, nutritional deficiencies (includ
76 -AKI most commonly manifests as acute tubulo-interstitial nephritis on kidney biopsy and generally sh
77 including crystals and tubular injury, acute interstitial nephritis, or mitochondrial toxicity.
78 ith CKD due to nephronophthisis, chronic GN, interstitial nephritis, or unknown etiology.
79 male sex, age >/=50 years, diabetes, chronic interstitial nephritis, polycystic kidney disease, and 1
80 Immune checkpoint inhibitor-associated acute interstitial nephritis presents significant clinical cha
81 iation between SIGEI and risk for iatrogenic interstitial nephritis: SIGEI was seen in only 4 of 15 p
82    Using the MRL/MpJ fas<lpr> model of lupus interstitial nephritis, we found that KTC did not expres
83 ystemic complications of SS (ie, vasculitis, interstitial nephritis) were also more common in men (64
84     Far less common is the entity of primary interstitial nephritis wherein the glomerular and vascul
85 immune checkpoint inhibitor-associated acute interstitial nephritis, with a receiver operating charac