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1 content in biological tissues (i.e., porcine intervertebral disc).
2 yme regulating GAG synthesis in cells of the intervertebral disc.
3 uctures, such as the articular cartilage and intervertebral disc.
4  cornea, and successive lamellar rings in an intervertebral disc.
5 en a result of a degenerative process in the intervertebral disc.
6 agnosed with large calcifications within the intervertebral disc.
7 y neuron changes to a single affected lumbar intervertebral disc.
8 ogically hyperosmotic environment within the intervertebral disc.
9 adaptation to diurnal osmotic loading of the intervertebral disc.
10 ten the direct result of degeneration of the intervertebral disc.
11 TS activity in nucleus pulposus cells of the intervertebral disc.
12 lcium phosphate-like whitlockite crystals in intervertebral discs.
13  perichondrium, and mesenchymal cells of the intervertebral discs.
14  directly by forming the nucleus pulposus of intervertebral discs.
15 re necessary for the resorption of herniated intervertebral discs.
16 us fibrosus in 48 (60%) of the 80 samples of intervertebral discs.
17 ome, which later comprises the vertebrae and intervertebral discs.
18 nges in achieving functional regeneration of intervertebral discs.
19 ing rise to the cells that form and maintain intervertebral discs.
20  premature degeneration and calcification of intervertebral discs.
21 ignaling mechanisms control this response in intervertebral discs.
22 and water content in the nucleus pulposus of intervertebral discs.
23 g signaling is required for formation of the intervertebral discs.
24 cular mechanisms responsible for forming the intervertebral discs.
25    CCN3 was expressed in embryonic and adult intervertebral discs.
26 of alternating vertebral bodies (centra) and intervertebral discs.(1) Recent studies in zebrafish hav
27  worsening destruction of vertebral body and intervertebral disc, abnormal vertebral marrow signal ch
28 f this study was that Chd synthesized in the intervertebral disc accumulates in the vertebral body.
29 Sensory afferent nerve fiber growth into the intervertebral disc after injury-induced inflammation ma
30 y has correlated macroscopic and microscopic intervertebral disc alterations starting in the second d
31 how that the loss of residual strains in the intervertebral disc alters the microenvironment and inst
32                  The physiologically hypoxic intervertebral disc and cartilage rely on the hypoxia-in
33            It is characterized by asymmetric intervertebral disc and facet joint degeneration, leadin
34 hich only revealed a protrusion of the L5-S1 intervertebral disc and no apparent cause for the patien
35 low back pain is degenerative disease of the intervertebral disc and other structures of the lumbar s
36 e a novel mechanism of NGF regulation in the intervertebral disc and potentially other pathogenic con
37 th circumferentially aligned fibers, such as intervertebral discs and arteries, are abundant in natur
38 ithin the developing annulus fibrosis of the intervertebral discs and increased apoptosis of chondroc
39 es were taken at lumbar vertebrae L1-L5 plus intervertebral discs and the thigh (midthigh, 10 cm dist
40 hat express substance P deep within diseased intervertebral discs and their association with pain sug
41 of vertebral body height, abnormal signal in intervertebral disc, and loss of disc height.
42 nt of anterior elements, relative sparing of intervertebral discs, and cold abscesses.
43 ading of the lower back with degeneration of intervertebral discs, and experiments on cadaver spines
44 tebrae, zygapophyseal and sacroiliac joints, intervertebral discs, and neurovascular structures) are
45           Pathologic processes affecting the intervertebral disc are affected by genetic factors and
46           Nucleus pulposus (NP) cells of the intervertebral disc are essential for synthesizing extra
47                 Articular cartilage (AC) and intervertebral discs are cartilaginous tissues with a si
48                                              Intervertebral discs are important structural components
49 f a metameric series of vertebral bodies and intervertebral discs, as well as adjoining ribs and ster
50                          Degeneration of the intervertebral discs, bulging, and facet joint arthropat
51  pain is associated with degeneration of the intervertebral disc, but specific mechanisms of pain gen
52                                              Intervertebral disc calcification is a rare condition in
53 n our study, we present a case of idiopathic intervertebral disc calcification within the cervical se
54 tochord gives rise to the middle part of the intervertebral disc, called the nucleus pulposus.
55 we investigated LPP action in rabbit primary intervertebral disc cells cultured ex vivo in a three-di
56  CHST3 mRNA was significantly reduced in the intervertebral disc cells of human subjects carrying the
57                                           In intervertebral disc cells, hypoxia promotes expression o
58        Low-dose NVP-BGJ398 treatment reduced intervertebral disc defects of lumbar vertebrae, loss of
59 enitors also caused severe short stature and intervertebral disc defects.
60                                              Intervertebral disc degeneration (IDD) causes chronic ba
61                                              Intervertebral disc degeneration (IDD) contributes to ba
62  pulposus (NP) has been observed in cases of intervertebral disc degeneration (IDD).
63 wn, studies on the effects and mechanisms of intervertebral disc degeneration (IVDD) are still lackin
64                      Osteoarthritis (OA) and intervertebral disc degeneration (IVDD) as major cause o
65                                              Intervertebral disc degeneration (IVDD) is a primary con
66                                              Intervertebral disc degeneration (IVDD) is linked to low
67                                              Intervertebral disc degeneration (IVDD) is mainly caused
68                                          The intervertebral disc degeneration (IVDD)-related diseases
69 or for several chronic conditions, including intervertebral disc degeneration and associated back pai
70 ales, and thickening of the skin, as well as intervertebral disc degeneration and extensive synovial
71 e therapeutic strategy for the prevention of intervertebral disc degeneration and its associated morb
72 ative connective tissue pathologies, such as intervertebral disc degeneration and osteoarthritis.
73 ation (Col9a1(-/-)), osteoarthritis (OA) and intervertebral disc degeneration develop prematurely.
74 was to assess the prevalence and timeline of intervertebral disc degeneration in mice homozygous for
75            The finding of premature onset of intervertebral disc degeneration in this mouse model may
76                                              Intervertebral disc degeneration is a leading cause of c
77                                              Intervertebral disc degeneration is highly prevalent wit
78                                              Intervertebral disc degeneration is linked to loss of ex
79                                              Intervertebral disc degeneration is the leading cause of
80                                              Intervertebral disc degeneration was also detected, with
81 ioral phenotype in a rodent model of chronic intervertebral disc degeneration which provides a means
82  (12.5 mo old) showed increased incidence of intervertebral disc degeneration with a concomitant decr
83  a future therapeutic intervention to retard intervertebral disc degeneration, partial inhibition of
84 ges consistent with anatomic signs of OA and intervertebral disc degeneration.
85 e to the changes occurring in the ECM during intervertebral disc degeneration.
86 on of innervating sensory neurons in chronic intervertebral disc degeneration.
87 ic mechanisms of nociceptive pain in chronic intervertebral disc degeneration.
88 represents a promising strategy for treating intervertebral disc degeneration.
89 osis for several spinal disorders, including intervertebral disc degenerative changes in T1w and T2w
90  defects in caudal vertebrae due to abnormal intervertebral disc development, although with higher pe
91             The nucleus pulposus (NP) of the intervertebral disc develops from the notochord.
92 arrow signal changes, and abnormal signal in intervertebral disc did not necessarily indicate worseni
93 thin a single breed (PBonferroni = 0.01) and intervertebral disc disease (IVDD) across breeds (PBonfe
94 ents for diseases such as osteoarthritis and intervertebral disc disease.
95  is a promising avenue for treating advanced intervertebral disc disease.
96 nking sulfate homeostasis with back pain and intervertebral disc disorder, our study identifies SLC26
97  [1.01-1.06]), arthrosis (1.15 [1.09-1.21]), intervertebral disc disorders (1.13 [1.09-1.17]), and sp
98                                   In injured intervertebral discs, disruptions in fibre organization
99 pression of Sca1 in mesenchymal cells of the intervertebral discs during development of the spinal co
100 n in vivo mechanical responses of the lumbar intervertebral discs during functional tasks.
101 Interestingly, lubricin was prominent in the intervertebral disc, especially in the nucleus pulposus.
102 IX collagen is an important component of the intervertebral disc extracellular matrix.
103 d ADAMTS-4 messenger RNA expression in human intervertebral disc fibrochondrocytes.
104            We obtained 34 control samples of intervertebral disc from previously healthy individuals
105                   We collected 46 samples of intervertebral discs from 38 patients during spinal fusi
106 sures in the central nucleus pulposus of the intervertebral disc generate prestrain in the outer annu
107          Nerve ingrowth deeper into diseased intervertebral disc has been reported, but how common th
108                 Nerve growth into degenerate intervertebral discs has been linked with the developmen
109   Conventional microdiscectomy treatment for intervertebral disc herniation alleviates pain but does
110 painful neuroinflammation that can accompany intervertebral disc herniation, is associated with local
111  likely to have low back pain or symptoms of intervertebral disc herniation, with secondary problems
112 utic potential of TARPs for the treatment of intervertebral disc herniation.
113 g to increased susceptibility of spontaneous intervertebral disc herniations in a clinically relevant
114            We examined nerve growth into the intervertebral disc in the pathogenesis of chronic low b
115  an important role for nerve growth into the intervertebral disc in the pathogenesis of chronic low b
116 orphometry and deformation of lumbar (L2-S1) intervertebral discs in 10 healthy participants while pe
117                 From nucleus pulposus of the intervertebral disc (in which the bulk collagen monomer
118 from the dorsal root ganglia in animals with intervertebral disc injury demonstrated altered TRPV1 ac
119 e outer third of the annulus fibrosus of the intervertebral disc is innervated.
120  to hypoxia in nucleus pulposus cells of the intervertebral disc is regulated by the hypoxia-inducibl
121    Nucleus pulposus, the central zone of the intervertebral disc, is gel-like and has a similar colla
122 r understanding of functional changes in the intervertebral disc (IVD) and interaction with endplate
123 mplicated in the regulation of articular and intervertebral disc (IVD) cartilage homeostasis.
124 l loads are important for homeostasis of the intervertebral disc (IVD) cell matrix, with physiologic
125 ithout pro-inflammatory cytokine IL-1beta in intervertebral disc (IVD) cells such as human annulus fi
126 dies have demonstrated biologic responses of intervertebral disc (IVD) cells to loading, although the
127                                              Intervertebral disc (IVD) degeneration activates the dif
128                                              Intervertebral disc (IVD) degeneration and associated sp
129                                              Intervertebral disc (IVD) degeneration and consequent lo
130      Cellular senescence is a contributor to intervertebral disc (IVD) degeneration and low back pain
131                                              Intervertebral Disc (IVD) degeneration has been associat
132 of significant socio-economic importance and intervertebral disc (IVD) degeneration has been implicat
133 ommon cause of low back pain, the cascade of intervertebral disc (IVD) degeneration is initiated by t
134                                              Intervertebral disc (IVD) degeneration is often associat
135                             The aetiology of intervertebral disc (IVD) degeneration remains poorly un
136 on of senescent cells is closely linked with intervertebral disc (IVD) degeneration, a prevalent age-
137                       Back pain is linked to intervertebral disc (IVD) degeneration, but clinical stu
138 ted the role of HTRA1 in the pathogenesis of intervertebral disc (IVD) degeneration.
139 ain is extremely high and is often linked to intervertebral disc (IVD) degeneration.
140             The nucleus pulposus (NP) of the intervertebral disc (IVD) demonstrates substantial chang
141           Role of cilia and IFT80 protein in intervertebral disc (IVD) development, maintenance, and
142                                              Intervertebral disc (IVD) disease (IDD) is a complex, mu
143                                              Intervertebral disc (IVD) disorder and age-related degen
144  embedded within the nucleus pulposus of the intervertebral disc (IVD) during maturation.
145          Its incidence is closely related to intervertebral disc (IVD) failure, which is likely cause
146 sues also require interactions for postnatal intervertebral disc (IVD) growth and maintenance is less
147                                          The intervertebral disc (IVD) has long been considered uniqu
148                     The main pathogenesis of intervertebral disc (IVD) herniation involves disruption
149                                Recurrence of intervertebral disc (IVD) herniation is the most importa
150 anical signals can either promote or disrupt intervertebral disc (IVD) homeostasis, the molecular mec
151            The biomechanical function of the intervertebral disc (IVD) is a critical indicator of tis
152               A targeted injury to the mouse intervertebral disc (IVD) is often used to recapitulate
153                    The central region of the intervertebral disc (IVD) is rich in proteoglycans, lead
154 anabolic and anti-catabolic growth factor on intervertebral disc (IVD) matrix and cell homeostasis.
155                          Degeneration of the intervertebral disc (IVD) results in a range of symptoma
156                                     Narrowed intervertebral disc (IVD) space is a characteristic of I
157 -derived shear stiffness measurements of the intervertebral disc (IVD) taken throughout the day and t
158 flammatory cytokine expression in pathologic intervertebral disc (IVD) tissues.
159 al bodies were only mildly affected, but the intervertebral disc (IVD) was reduced or missing.
160 breakdown of the extracellular matrix of the intervertebral disc (IVD), disc height loss, and inflamm
161 commonly implicated in this condition is the intervertebral disc (IVD), which frequently herniates, r
162 s (NP), the central gelatinous region of the intervertebral disc (IVD).
163  commonly associated with pathologies of the intervertebral disc (IVD).
164 atic activity elevated in degenerative human intervertebral disc (IVD). Here, we examined the discs i
165                                              Intervertebral discs (IVD) are essential components of t
166 ures of nucleus pulposus (NP) cells from the intervertebral discs (IVD) of bovine tails were transfec
167 reporter genes and injected in isolated goat intervertebral discs (IVD).
168 o-vanillin and RG-7112, remove SnCs in human intervertebral discs (IVDs) and reduce SASP release, but
169 uloskeletal (MSK) tissues like cartilage and intervertebral discs (IVDs) but require long acquisition
170 al bodies were only moderately affected, the intervertebral discs (IVDs) were either missing or incom
171 en associated with the degeneration of human intervertebral discs (IVDs).
172 hondrocytes in knee joint and in NP cells in intervertebral disc led to the decrease in CTGF expressi
173 ures including cortical and cancellous bone, intervertebral discs, ligaments, and cartilage directly
174 obtained from healthy and pathological human intervertebral disc matrices.
175  study to assess the therapeutic benefits of intervertebral disc matrix repair and regeneration by ev
176 ned peptide therapy with LfcinB and BMP7 for intervertebral disc matrix repair and to understand cell
177                               Indeed, entire intervertebral discs, normally composed by nucleus pulpo
178 f senescent cells taken from the degenerated intervertebral discs of aged humans.
179              Immunohistochemical staining of intervertebral discs of Ccn2 null embryos shows a decrea
180 e commonly used to diagnose prolapsed lumbar intervertebral disc (PLID).
181 he alterations in the range of motion (ROM), intervertebral disc pressure (IDP), articular cartilage
182  patients: dyspnoea, headache, hypertension, intervertebral disc protrusion, and malignant lung neopl
183 ting potential for future studies related to intervertebral disc replacement therapy.
184 sforming growth factor can be transferred to intervertebral discs, resulting in increased proteogylca
185       Unilateral disc puncture of one lumbar intervertebral disc revealed a bilateral behavioral phen
186  proteins in articular cartilages, meniscus, intervertebral disc, rib, and tracheal cartilages on sam
187           Thirty-six formalin-fixed embedded intervertebral disc samples of varying grades of degener
188 h varying anatomic corridors to approach the intervertebral disc space and implanted materials have e
189           CT images at the level of the L4-5 intervertebral disc space were extracted from the medica
190 ry responses associated with degeneration of intervertebral discs that cause chronic back pain, and w
191 n nucleus pulposus (NP) cells of the healthy intervertebral disc, the mechanisms that control express
192  expression of Notch signaling components in intervertebral disc tissue from mature rats and from hum
193  the PKC pathways, specifically PKCdelta, in intervertebral disc tissue homeostasis.
194 anases other than ADAMTS-8 was identified in intervertebral disc tissue, as was mRNA for TIMP-3.
195 aggrecanase activities were also detected in intervertebral disc tissue.
196 r of metalloproteinases 3 (TIMP-3), in human intervertebral disc tissue.
197 ell type than BM-MSCs for use in engineering intervertebral disc tissue.
198 n clock phase and amplitude in cartilage and intervertebral disc tissues in vivo and in tissue explan
199  biosynthesis towards maintenance of healthy intervertebral disc tissues.
200 grecan-rich hydrated tissue that permits the intervertebral disc to resist compressive loads.
201 a CV2-dependent flow of Chd protein from the intervertebral disc to the vertebral body.
202 (alpha1) XI protein both localize within the intervertebral disc-vertebral junction region encompassi
203         Aggrecan extracted from human lumbar intervertebral discs was incorporated into tissue cultur
204 a murine organ culture model in which intact intervertebral discs were cocultured with peritoneal mac
205  of the spine and hips, and deterioration of intervertebral discs with characteristic radiographic ch

 
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