ライフサイエンスコーパス: intervillous space

1 he uterus, expelling maternal blood from the intervillous space.

2 placenta are localized predominantly in the intervillous space.

3 lated with monocyte density in the placental intervillous space.

4 te to the accumulation of macrophages in the intervillous space.

5 leukin (IL)-10, in plasma collected from the intervillous space.

6 al endometrium to initiate blood flow to the intervillous space.

7 microm over the villous tips throughout the intervillous space.

8 of transport rates via ease of access to the intervillous space.

9 on where maternal macrophages infiltrate the intervillous space.

10 tin sulfate A (CS) abundantly present in the intervillous space.

11 recruit B cells to further produce Ig in the intervillous spaces.

12 depositions of IgG3 and IgM were observed in intervillous spaces.

13 Epithelial leaks and intervillous spaces also increased significantly in CLE+

14 it was found that IRBCs adhere mainly in the intervillous space and also at significant levels to the

15 to an accumulation of macrophages within the intervillous space and increased production of tumor nec

16 Although CSPG is present both in the intervillous space and on the syncytiotrophoblast surfac

17 antigens caused immediate breaks, increased intervillous spaces, and increased IELs in the intestina

18 Epithelial breaks, intervillous spaces, and the number of intraepithelial l

19 ous CSPGs, we conclude that the CSPGs of the intervillous spaces are the receptors for placental IRBC

20 ction showed parasitized erythrocytes in the intervillous space but no hemozoin in macrophages nor in

21 in sulfate A (CSA) to sequester in placental intervillous spaces, causing severe sequelae for mother

22 However, the structures of the intervillous-space CSPGs changed considerably during the

23 ly, monocyte infiltration into the placental intervillous space has been identified as a key risk fac

24 this model, the depth of diffusion into the intervillous space is inversely proportional to the effi

25 infected erythrocytes (IEs) sequester in the intervillous space (IVS) of the placenta causing placent

26 sulfate proteoglycans (CSPGs) present in the intervillous spaces mediate the adherence of IRBCs in th

27 lfated, extracellular CSPGs localized in the intervillous spaces, minor amounts of two cell-associate

28 the placenta, the low sulfated CSPGs of the intervillous spaces most efficiently bind IRBCs.

29 -normal maternal blood velocity entering the intervillous space of the placenta, distortion of the co

30 eoglycan (CSPG) present predominantly in the intervillous space of the placenta.

31 cells (IRBCs) selectively accumulate in the intervillous spaces of placenta, leading to poor fetal o

32 droitin sulfate proteoglycans (CSPGs) in the intervillous spaces of the placenta are the receptors fo

33 infected erythrocytes (IE) accumulate in the intervillous spaces of the placenta by binding to chondr

34 an mediate the sequestration of IRBCs in the intervillous spaces of the placenta during the entire se

35 droitin sulfate proteoglycans (CSPGs) in the intervillous spaces of the placenta mediate the IRBC adh

36 arum-infected red blood cells (IRBCs) in the intervillous spaces of the placenta.

37 rythrocytes and maternal inflammation in the intervillous spaces of the placenta.

38 tration of late-stage parasites in placental intervillous spaces that express VAR2CSA and bind specif

39 emonstrate that the CSPGs are present in the intervillous spaces throughout the second and third trim

40 a significant correlate of most components (intervillous space, villous, trophoblast, and capillary

41 placental weight (r = 0.41, P = 0.0009), and intervillous space volume (r = 0.38, P = 0.0024).

42 Intervillous space volume was increased in OT1DM (OT1DM

43 uterus are suspended as cell columns in the intervillous space, where they experience significant am

44 increased, epithelial leaks/gaps formed, and intervillous spaces widened.