ライフサイエンスコーパス: intestinal epithelial cell

1 ances LGG-stimulated protective responses in intestinal epithelial cells.

2 22, which promoted barrier function of human intestinal epithelial cells.

3 e a widespread source of genotoxic stress in intestinal epithelial cells.

4 soform-1 (RALDH1) is dominantly expressed in intestinal epithelial cells.

5 red response to TNFR1-mediated cell death in intestinal epithelial cells.

6 tion of the inflammasome in both myeloid and intestinal epithelial cells.

7 he exosomes released from C. parvum-infected intestinal epithelial cells.

8 ents of the innate immune system but also in intestinal epithelial cells.

9 infection in cell lines and in primary human intestinal epithelial cells.

10 ragweed did not affect the integrity of the intestinal epithelial cells.

11 n polymerization and caspase-3 activation in intestinal epithelial cells.

12 pression at the posttranscriptional level in intestinal epithelial cells.

13 tor was explored using CB1-knockdown (CB1Kd) intestinal epithelial cells.

14 trol mice or mice with deletion of EGFR from intestinal epithelial cells.

15 ling drives catenin-related transcription in intestinal epithelial cells.

16 and regulates lipid absorption and export in intestinal epithelial cells.

17 nsing of intraepithelial lymphocytes to kill intestinal epithelial cells.

18 cum gliadin-derived peptides in human Caco-2 intestinal epithelial cells.

19 was performed in monocytes, lymphocytes, and intestinal epithelial cells.

20 and restitution were reduced in the isolated intestinal epithelial cells.

21 vel and reduced glutathione concentration in intestinal epithelial cells.

22 mited TNF-dependent apoptosis in transformed intestinal epithelial cells.

23 e Heligmosomoides bakeri that get into mouse intestinal epithelial cells.

24 tion, and transcriptional activity of YAP in intestinal epithelial cells.

25 pendent binding to sulfated proteoglycans on intestinal epithelial cells.

26 stress and the unfolded protein response in intestinal epithelial cells.

27 ell death by regulating ERK1/2 MAP kinase in intestinal epithelial cells.

28 lity and the translocation efficiency across intestinal epithelial cells.

29 turnover and caspase-dependent apoptosis of intestinal epithelial cells.

30 act of oncogenic KRAS on the cell surface of intestinal epithelial cells.

31 ut activation of the Akt survival pathway in intestinal epithelial cells.

32 n of WDR26 in FPR1-mediated wound healing in intestinal epithelial cells.

33 CD59 uptake in both HeLa and polarized Caco2 intestinal epithelial cells.

34 e highly expressed in mouse and human normal intestinal epithelial cells.

35 ely regulates FPR1-mediated wound healing in intestinal epithelial cells.

36 to dysregulated renewal and replenishment of intestinal epithelial cells.

37 Fpn altered the adhesive properties of HT29 intestinal epithelial cells.

38 um secretome (STS)-induced outcomes in human intestinal epithelial cells.

39 indirectly regulate tolerogenic responses in intestinal epithelial cells.

40 secretion of inflammatory cytokines by HT-29 intestinal epithelial cells.

41 dulators of global translational capacity in intestinal epithelial cells.

42 roteins (SAA1 and SAA2) secreted by adjacent intestinal epithelial cells.

43 a dynamic network of proteins that encircle intestinal epithelial cells.

44 uced expression of antimicrobial peptides in intestinal epithelial cells.

45 ACSIN2 KD and endocytosis inhibition on live intestinal epithelial cells.

46 as unabated in mice with IKKbeta deletion in intestinal epithelial cells.

47 of all 9 membrane-bound AC isoforms in mouse intestinal epithelial cells.

48 pacity of both toxins to induce apoptosis in intestinal epithelial cells-a hallmark feature of AAHC-b

49 Intestinal epithelial cells absorb nutrients, respond to

50 The current study examines survivin in intestinal epithelial cells after ileocecal resection.

51 LGG-derived secretory protein that protects intestinal epithelial cells against inflammation.

52 RNA-mediated beta-catenin knockdown in human intestinal epithelial cells all result in significant lo

53 Rotavirus (RV) encounters intestinal epithelial cells amidst diverse microbiota, o

54 terization of previously unknown subtypes of intestinal epithelial cell and their gene signatures.

55 Specifically, rat intestinal epithelial cells and a human intestinal epith

56 M2 mediates the caspase-1-dependent death of intestinal epithelial cells and bone marrow cells in res

57 strate that dietary adjustments affect small intestinal epithelial cells and can be used to modulate

58 ule relative to other signalling pathways in intestinal epithelial cells and colorectal cancer (CRC)

59 es revealed exclusive expression of Clr-a by intestinal epithelial cells and crypt cells throughout t

60 ation of STAT3 and expression of survivin in intestinal epithelial cells and expression of IL6 in col

61 rms live within a specialised tunnel of host intestinal epithelial cells and have anterior-ventral pr

62 nhibition of miR-23a and miR-155 in cultured intestinal epithelial cells and in acutely injured mucos

63 ound that exogenous IAP induced autophagy in intestinal epithelial cells and in macrophages.

64 immune signalling pathway that functions in intestinal epithelial cells and may present useful targe

65 ing HIF-1alpha [HIF-1alpha-knockout (KO)] in intestinal epithelial cells and mice lacking NTR1 [NTR1-

66 In analyses of intestinal epithelial cells and mice, we identified uc.1

67 tion, where it alters nutrient metabolism in intestinal epithelial cells and microbiome, leading to i

68 xpression of a transgene increased growth of intestinal epithelial cells and organoids.

69 The H2b(DeltaIEC) mice had DNA damage to intestinal epithelial cells and proliferative exhaustion

70 , which in turn promote pSTAT3 signalling in intestinal epithelial cells and protection from intestin

71 NLR Nlrp9b that is specifically expressed in intestinal epithelial cells and restricts rotavirus infe

72 to many environmental factors that influence intestinal epithelial cells and the underlying mucosal i

73 -inflammatory effect of LGG is reinforced by intestinal epithelial cells and thereby maintains intest

74 ed cAMP generation in acutely isolated small intestinal epithelial cells, and significantly impaired

75 Nfil3 transcription oscillates diurnally in intestinal epithelial cells, and the amplitude of the ci

76 est, Sgpp2 deficiency suppressed DSS-induced intestinal epithelial cell apoptosis and improved mucosa

77 ve colonic mucosal inflammation by promoting intestinal epithelial cell apoptosis and mucosal TH17 re

78 Intestinal epithelial cells are constantly exposed to pa

79 xual development occurs when cultured feline intestinal epithelial cells are supplemented with linole

80 nable us to conditionally express Neurod1 in intestinal epithelial cells at different stages of diffe

81 suppress larval diapause, is changed in the intestinal epithelial cells at larval diapause.

82 of cytoprotective IL-18 from IKKalpha-mutant intestinal epithelial cells because of elevated caspase

83 membrane-bound guanylyl cyclase expressed in intestinal epithelial cells, binds the paracrine hormone

84 y reduces adhesion of C. difficile to Caco-2 intestinal epithelial cells but does not affect activati

85 nflammatory and cytoprotective properties in intestinal epithelial cells, but has not been previously

86 Intestinal epithelial cells (Caco-2 and IEC-6 lines) wer

87 l pattern recognition receptor through which intestinal epithelial cells can recognize and control fu

88 not TCRgammadelta(+) IEL, TCRbeta(+) IEL, or intestinal epithelial cells, can promote survival of dif

89 ulators of innate immune signaling, in HT-29 intestinal epithelial cells challenged with TNF-alpha or

90 fluid flow application initiates changes in intestinal epithelial cell characteristics relative to t

91 hat the expression levels of RALDH1 in small intestinal epithelial cells correlated with the activity

92 to be required for regulation of TNF-induced intestinal epithelial cell death and survival.

93 d externally desynchronized WT mice to study intestinal epithelial cell death.

94 used to compare the role of myeloid- versus intestinal epithelial cell-derived IL-33 during dextran

95 Surprisingly, the lack of intestinal epithelial cell-derived IL-33 had no impact o

96 ctionally restrains colitic disease, whereas intestinal epithelial cell-derived IL-33 is dispensable.

97 and prevents the unlimited proliferation of intestinal epithelial cells despite constitutive beta-ca

98 active G protein-coupled receptor (US28) in intestinal epithelial cells develop serrated polyps in t

99 Deletion of EGFR from intestinal epithelial cells did not affect tumor growth.

100 t, analysis of genomic architecture in mouse intestinal epithelial cells disclosed that microbiota co

101 orders, and loss of Myo7b in differentiating intestinal epithelial cells disrupts intermicrovillar ad

102 In contrast, the targeted deletion of HuR in intestinal epithelial cells enhanced miR-675 production

103 s innate immune evasion enables infection of intestinal epithelial cells, escape from adaptive immuni

104 er Ikkepsilon phosphorylation in transformed intestinal epithelial cells, establishing a positive fee

105 Intestinal epithelial cells exert front-line responses t

106 alpha and TP53, which increases survivin and intestinal epithelial cell expansion during therapeutic

107 Here, we show that intestinal epithelial cells expressing IRE1beta have an

108 *Intestinal epithelial cell expression of Nod1 receptors

109 Interestingly, exosomes released from intestinal epithelial cells following C. parvum infectio

110 lk1 expressing tuft cells regulate the whole intestinal epithelial cells following injury through par

111 tabolic and immunological responses in human intestinal epithelial cells following their co-culture w

112 Here, we elucidate a mechanism of action on intestinal epithelial cells for extracellular CDNs.

113 mensal microbes in contact with living human intestinal epithelial cells for more than a week in vitr

114 Intestinal epithelial cells form a barrier that is criti

115 The apical brush border membrane (BBM) of intestinal epithelial cells forms a highly structured an

116 commensal-specific CD4(+) T cells on primary intestinal epithelial cells from these samples.

117 At baseline, p85alpha-deficient intestinal epithelial cells had less Trp53 and more surv

118 F and EC co-culture on patient-derived human intestinal epithelial cells (HIECs), and incorporate per

119 (also called IL28A or interferon lambda2) in intestinal epithelial cell (IEC) activation, studying it

120 The mechanisms by which macrophages regulate intestinal epithelial cell (IEC) barrier properties are

121 pends on a tightly regulated balance between intestinal epithelial cell (IEC) death and proliferation

122 Intestinal epithelial cell (IEC) junctions constitute a

123 The role of intestinal epithelial cell (IEC) polarization in the mai

124 nd how this affects macroautophagy-regulated intestinal epithelial cell (IEC) processes essential for

125 that DUSP10 knockout (KO) mice had increased intestinal epithelial cell (IEC) proliferation and migra

126 ng mice had shortened intestines, diminished intestinal epithelial cell (IEC) proliferation, and decr

127 signaling plays an important role in driving intestinal epithelial cell (IEC) responses to bacterial

128 esized that TNF exerts beneficial effects on intestinal epithelial cell (IEC) responses to injury.

129 Intestinal epithelial cell (IEC) shedding is a fundament

130 Using mice with inducible intestinal epithelial cell (IEC)-specific deletion of Ph

131 Furthermore, intestinal epithelial cell (IEC)-specific loss of CD47 d

132 la pathogenesis, we instead demonstrate that intestinal epithelial cell (IEC)-specific NAIP-NLRC4 act

133 e miRNA-processing enzyme, Dicer, identified intestinal epithelial cells (IEC) and Hopx-positive cell

134 Using HT-29 intestinal epithelial cells (IEC) as a model we have dem

135 Here, we integrated intestinal epithelial cells (IEC) derived from human int

136 rylated beta-catenin (pbeta-Cat(Ser-552)) in intestinal epithelial cells (IEC) during colitis and col

137 Intestinal epithelial cells (IEC) exclusively express th

138 Intestinal epithelial cells (IEC) have important functio

139 ation patterns and transcriptomes of primary intestinal epithelial cells (IEC) of children newly diag

140 unctions as an intrinsic tumor suppressor in intestinal epithelial cells (IEC), by regulating their r

141 Human intestinal epithelial cells (IECs) (HCT-116) were infect

142 Intestinal epithelial cells (IECs) act as a physical bar

143 We deleted Cosmc in mouse intestinal epithelial cells (IECs) and found marked redu

144 xpressed abundantly on the apical surface of intestinal epithelial cells (IECs) and functions as the

145 directly affects expression of DRA in human intestinal epithelial cells (IECs) and in the intestines

146 mily 6 member 8 (SLC6A8, also called CRT) in intestinal epithelial cells (IECs) and mice, and we meas

147 lambda (IFNL) is expressed at high levels by intestinal epithelial cells (IECs) and mucosal immune ce

148 de methylome- and transcriptome-profiling of intestinal epithelial cells (IECs) and sperm cells of ma

149 Vibrio cholerae colonizes intestinal epithelial cells (IECs) and uses a MARTX toxi

150 ways driving disease-specific alterations of intestinal epithelial cells (IECs) are largely unknown.

151 Among barrier cells, intestinal epithelial cells (IECs) are particularly depe

152 Myeloid cells interact with intestinal epithelial cells (IECs) by producing various

153 mbrane-derived microparticles (PMN-MPs) onto intestinal epithelial cells (IECs) during TEM leads to l

154 Intestinal epithelial cells (IECs) exist in a metabolic

155 A similar phenotype occurs in mice whose intestinal epithelial cells (IECs) fail to express the t

156 Intestinal epithelial cells (IECs) form a critical barri

157 Intestinal epithelial cells (IECs) from Hdac3(IKO) mice

158 iota dysregulation promotes LIF secretion by intestinal epithelial cells (IECs) in a mouse colitis mo

159 We report that small intestinal epithelial cells (IECs) in Crohn's disease (C

160 Ifnlr1 We found that expression of IFNLR1 on intestinal epithelial cells (IECs) in the small intestin

161 RNA and accessible chromatin data from adult intestinal epithelial cells (IECs) in zebrafish, stickle

162 bundantly in the intestines, its function in intestinal epithelial cells (IECs) is unknown.

163 clustering on the viability of EPEC-infected intestinal epithelial cells (IECs) is unknown.

164 dent protein kinase II gamma (CAMK2gamma) in intestinal epithelial cells (IECs) modulates inflammator

165 ction of an IGF2BP1 knockout specifically in intestinal epithelial cells (IECs) of adult mice.

166 induced phosphorylation of STAT1-Y701 within intestinal epithelial cells (IECs) of suckling mice.

167 Intestinal epithelial cells (IECs) play an indispensable

168 Intestinal epithelial cells (IECs) provide a barrier tha

169 Intestinal epithelial cells (IECs) regulate gut immune h

170 (MNoV), we determine that a small number of intestinal epithelial cells (IECs) serve as the reservoi

171 We found that targeted deletion of HuR in intestinal epithelial cells (IECs) specifically decrease

172 on of lymphoid cells that reside between the intestinal epithelial cells (IECs) that form the intesti

173 amentous bacteria (SFB) are transferred into intestinal epithelial cells (IECs) through adhesion-dire

174 oteins villin 1 (VIL1) and gelsolin (GSN) in intestinal epithelial cells (IECs) to determine whether

175 cts type III secretion system effectors into intestinal epithelial cells (IECs) to target inflammator

176 mouse ISCs, progenitors, and differentiated intestinal epithelial cells (IECs) using Villin-Cre.

177 Intestinal epithelial cells (IECs) were isolated and pur

178 ed C2BBe1 cells as a model, we observed that intestinal epithelial cells (IECs) were permissive to EV

179 MHC class II was expressed on intestinal epithelial cells (IECs) within the ileum at s

180 Here we report that, unlike non-metastatic intestinal epithelial cells (IECs), metastatic IECs expr

181 tinal epithelial cell line and mouse primary intestinal epithelial cells (IECs), we demonstrated that

182 Using human intestinal epithelial cells (IECs), we discovered that S

183 Here we show that mouse apoptotic intestinal epithelial cells (IECs), which undergo contin

184 verall metabolic activity in macrophages and intestinal epithelial cells (IECs).

185 es the expression and production of LCN-2 in intestinal epithelial cells (IECs).

186 and excludes luminal factors from contacting intestinal epithelial cells (IECs).

187 h PMN-MPs upon PMN activation and binding to intestinal epithelial cells (IECs).

188 ing small extracellular vesicles (sEVs) from intestinal epithelial cells (IECs).

189 t this is due to activation of C3 within the intestinal epithelial cells in a cathepsin-dependent man

190 s microvillus membrane expression of DMT1 in intestinal epithelial cells in a PKCalpha-dependent mann

191 y, targeted deletion of insulin receptors in intestinal epithelial cells in Apc(Min/+) mice did not c

192 RAGE was abundant in the intestinal epithelial cells in both suckling pups and ad

193 nd down-regulation of 196 lincRNAs in murine intestinal epithelial cells in culture.

194 also decreased secretion of ApoB-48 from rat intestinal epithelial cells in response to oleic acid st

195 rogramming of the gene expression profile in intestinal epithelial cells in response to TNF-alpha sti

196 ole of the chromatin remodeling machinery in intestinal epithelial cells in the colitis response and

197 Rotavirus specifically infects the intestinal epithelial cells in the host small intestine

198 virulence genes and increases attachment to intestinal epithelial cells in vitro in a QseC-dependent

199 of the vitamin A-converting enzyme RALDH1 in intestinal epithelial cells in vivo and in vitro, respec

200 production of d-amino acid oxidase (DAO) by intestinal epithelial cells, including goblet cells, whi

201 eals that TFEB is critical for resistance to intestinal epithelial cell injury, potentially mediated

202 How intestinal epithelial cells interact with the microbiota

203 intraepithelial lymphocytes with respect to intestinal epithelial cells; intestinal histological sco

204 arget activation of an IKKbeta/NCoR1 loop in intestinal epithelial cells lead to derepression of gene

205 ian iron exporter ferroportin exclusively in intestinal epithelial cells leads to loss of intestinal

206 Here, using a human intestinal epithelial cell line (HCT116), we show that l

207 Using a human intestinal epithelial cell line and mouse primary intest

208 We further show that the mouse intestinal epithelial cell line MODE-K and RAW264.7 macr

209 rat intestinal epithelial cells and a human intestinal epithelial cell line were infected with C. sa

210 thylamine and tryptamine toxicity on a human intestinal epithelial cell line.

211 there is deregulation in differentiation of intestinal epithelial cell lineages that may influence t

212 Previous studies in intestinal epithelial cell lines suggested a role for th

213 Two intestinal epithelial cell lines were used to study the

214 We infected macrophage and intestinal epithelial cell lines with the E. faecalis st

215 The intestinal epithelial cell lining provides the first lin

216 he licensing of cytotoxic T cells to mediate intestinal epithelial cell lysis.

217 Intestinal epithelial cells, macrophages, and dendritic

218 Using intestinal epithelial cells, macrophages, and primary sp

219 onstrate that constitutive AKT activation in intestinal epithelial cells markedly enhances tumor inva

220 ice and attenuated LPS inhibitory effects on intestinal epithelial cell migration along the crypt-vil

221 observations, a role for RvE1 in regulating intestinal epithelial cell migration and proliferation d

222 as a prorepair lipid mediator by increasing intestinal epithelial cell migration and proliferation,

223 Here, we showed that impairment of intestinal epithelial cell migration occurred in lipopol

224 sis of a complex miRNA regulatory program in intestinal epithelial cell models provides novel evidenc

225 Here, seeking to elucidate whether intestinal epithelial cells modulate Lactobacillus rhamn

226 t that forced expression of Neurod1 programs intestinal epithelial cells more towards an EE cell fate

227 that HDAC3 activity was sharply increased in intestinal epithelial cells of microbiota-replete mice c

228 ne profiling on manipulated cancer cells and intestinal epithelial cells of Prss8 mouse models, gene

229 d circulating glucose was trapped within the intestinal epithelial cells of rats and humans that unde

230 ibited no change in cell viability in normal intestinal epithelial cells or human matched-normal colo

231 the autophagy gene Atg7 specifically in all intestinal epithelial cells or in Lgr5(+)ISC, we show th

232 mice with tissue-specific deletion of VDR in intestinal epithelial cells or myeloid cells.

233 calization was followed during the course of intestinal epithelial cell polarization as cells progres

234 CLAMP distribution changes during intestinal epithelial cell polarization, regulates the f

235 Intestinal epithelial cell populations were purified; we

236 scripts in specific respiratory, corneal and intestinal epithelial cells, potentially explaining the

237 histocompatibility complex (MHC) class II on intestinal epithelial cells prevented CD4(+)CD8alphaalph

238 GI bacterial GUS activity in 1 d and reduces intestinal epithelial cell proliferation in 5 d, both bl

239 However, intestinal epithelial cell proliferation is not impeded,

240 tgr1(-/-) and Mtg16(-/-) mice have increased intestinal epithelial cell proliferation.

241 is of experimental colitis via inhibition of intestinal epithelial cell proliferation.

242 Deletion of EGFR from myeloid cells, but not intestinal epithelial cells, protects mice from colitis-

243 Intestinal epithelial cells provide an important coloniz

244 Unexpectedly, conditional host intestinal epithelial cells RALDH1 deletion failed to re

245 Boosting BAI1-mediated uptake by intestinal epithelial cells (rather than myeloid cells)

246 lated and characterized TCRs recognizing the intestinal epithelial cell receptor and colorectal cance

247 t determines the injury response and enables intestinal epithelial cell regenerative plasticity.

248 s, the role of these cytokines in regulating intestinal epithelial cell renewal is largely unknown.

249 enforcing specific expression of guanylin in intestinal epithelial cells restored GUCY2C signaling, e

250 Overexpression of Reg3g in intestinal epithelial cells restricts bacterial coloniza

251 (also known as Eif2ka4) in CD11c(+) APCs or intestinal epithelial cells resulted in enhanced intesti

252 ghout the body or restricted specifically to intestinal epithelial cells resulted in loss of AHR-depe

253 nism, direct silencing of BLM Na/K-ATPase in intestinal epithelial cells resulted in selective stimul

254 Overexpression of Cdg7_FLc_0990 in intestinal epithelial cells resulted in significant chan

255 Initial in vitro studies using cultured intestinal epithelial cells revealed that the neddylatio

256 P promoter region are differentially used in intestinal epithelial cell(s) (IEC).

257 Thus, our data indicate that intestinal epithelial cells serve as gatekeepers for the

258 In vitro studies using Caco2 intestinal epithelial cells showed that in the presence

259 were studied in wild-type mice and mice with intestinal epithelial cell-specific (DeltaIEC) disruptio

260 We generated intestinal epithelial cell-specific adenylyl cyclase 6 (

261 We also identified that intestinal epithelial cell-specific deletion of Nod1 (Vi

262 Using mice with an intestinal epithelial cell-specific deletion of PPARdelt

263 From an investigation of mice with intestinal epithelial cell-specific deletion of the p38a

264 ng colonic homeostasis by characterizing the intestinal epithelial cell-specific EP4 knockout (EP4 cK

265 Intestinal epithelial cell-specific Hif1a loss reduced a

266 ctivates p38gamma in mouse colon tissues and intestinal epithelial cell-specific p38gamma knockout (K

267 lonic wounds in a process involving FPR1 and intestinal epithelial-cell-specific NOX1-dependent redox

268 microbiota compared with miRNAs in any other intestinal epithelial cell subtype.

269 regulate miRNA expression in IESCs and other intestinal epithelial cell subtypes will elucidate a cri

270 (LPA) receptor 1 regulates proliferation of intestinal epithelial cells, such that the absence of LP

271 pression of C3/C3 fragments primarily in the intestinal epithelial cells, suggesting local involvemen

272 hree-dimensional structures of primary small intestinal epithelial cells that contain all differentia

273 ated a gut-restricted expression of Clr-f on intestinal epithelial cells that is spatially matched by

274 rom patients with CD were applied to healthy intestinal epithelial cells, the epithelial cells increa

275 stimulation of the proliferative response of intestinal epithelial cells to GPCR agonists that act vi

276 We further show that IFN-I acts on intestinal epithelial cells to increase the proportion o

277 Exposure of intestinal epithelial cells to RvE1 promoted wound repai

278 helial cells that contain all differentiated intestinal epithelial cell types.

279 as observed in confluent monolayers of human intestinal epithelial cells under basal conditions, expo

280 Unlike other cells and tissues, intestinal epithelial cells undergo rapid cell death upo

281 ur data indicate that exosomes released from intestinal epithelial cells upon C. parvum infection can

282 nduces inflammatory diarrhea and uptake into intestinal epithelial cells using the Salmonella pathoge

283 enerated transgenic mice expressing IL-33 in intestinal epithelial cells (V33 mice).

284 erformed experiments in mice lacking EGFR in intestinal epithelial cells (Villin-Cre; Egfr(f/f) and V

285 In infected mice, proliferation of small intestinal epithelial cells was compromised in an SseF/S

286 ange in the proliferation of normal or tumor intestinal epithelial cells was observed upon genetic in

287 uanylin, and uroguanylin mRNA and protein by intestinal epithelial cells was preserved following leth

288 Loss of Bcl-3 in intestinal epithelial cells was sufficient to increase t

289 ogical and genetic perturbations in cultured intestinal epithelial cells, we found that NM2C controls

290 owever, as LT also promotes ETEC adhesion to intestinal epithelial cells, we postulated that increase

291 hepatocytes, pancreatic endocrine cells, and intestinal epithelial cells when treated with defined so

292 demonstrated that VgpA is translocated into intestinal epithelial cells, where it interacts with EBP

293 e intracellular niche at the brush border of intestinal epithelial cells, where it undergoes a comple

294 colon of UC patients, miR-24 is localized to intestinal epithelial cells, which prompted an investiga

295 that ERK5 provides a common bypass route in intestinal epithelial cells, which rescues cell prolifer

296 s regardless of an increase in the number of intestinal epithelial cells with nuclear beta-catenin an

297 Here, a genome-scale CRISPR/Cas9 screen in intestinal epithelial cells with the prototypical intrac

298 Thereafter, T. trichiura larvae moult within intestinal epithelial cells, with adult worms embedded i

299 viral replication, is found predominantly in intestinal epithelial cells, with chromogranin A-positiv

300 p in the translocation of cholera toxin into intestinal epithelial cells without exerting measurable