ライフサイエンスコーパス: intestinal ischemia

1 other hemodynamic imbalances (heart failure, intestinal ischemia).

2 blood flow may result in clinically relevant intestinal ischemia.

3 (n = 24) and then subjected to 30 minutes of intestinal ischemia.

4 olved in the generation of activators during intestinal ischemia.

5 our after a 15-minute period of normothermic intestinal ischemia.

6 angiography scan, which was compatible with intestinal ischemia.

7 , to even more severe symptoms suggestive of intestinal ischemia.

8 of recipients following SITx after prolonged intestinal ischemia (6 hours).

9 Intestinal ischemia after hemorrhagic shock results in g

10 te adhesion, and maintained blood flow after intestinal ischemia and may therefore be of value in red

11 gression, in patients with cirrhosis without intestinal ischemia and recent (<6 months) thrombosis in

12 le of T lymphocytes and neutrophils (PMN) in intestinal ischemia and reperfusion injury (IRI) using e

13 emote (lung) complement activation following intestinal ischemia and reperfusion injury and that CR2-

14 otal role in the burn- and endotoxin-induced intestinal ischemia and reperfusion injury, with subsequ

15 ce in sterile inflammatory injury induced by intestinal ischemia and reperfusion, as well as in a mod

16 talloproteinase-8 as a mediator of injury in intestinal ischemia and reperfusion.

17 nificantly increased in C3aR(-/-) mice after intestinal ischemia, and C3aR(-/-) mice also mobilized m

18 ed on patients who were diagnosed with acute intestinal ischemia between May 1993 and July 2000.

19 Intestinal ischemia followed by reperfusion leads to loc

20 C57Bl/6 mice were subjected to 90 minutes of intestinal ischemia followed or not by reperfusion.

21 Intestinal ischemia has a wide variety of causes, includ

22 restore adequate intestinal blood flow, and intestinal ischemia has been implicated in the activatio

23 of mucosal injury, yet the reasons for which intestinal ischemia in NEC occurs in the first place rem

24 Acute intestinal ischemia is reported to have a poor prognosis

25 e that the prognosis for patients with acute intestinal ischemia is substantially better than previou

26 Etiologies were intestinal ischemia (n = 30; 59%) and hemorrhage (n = 21

27 , 2 OR-TAA(A)] developed fatal postoperative intestinal ischemia on day 2 or 3.

28 to determine whether isoflurane, given after intestinal ischemia, protects against intestinal IRI and

29 ; 95% confidence interval [CI], 1.7 to 2.3), intestinal ischemia (relative risk, 6.0; 95% CI, 4.5 to

30 educe intestinal injury in mice subjected to intestinal ischemia-reperfusion (I/R) injury.

31 ti-inflammatory pathway in a murine model of intestinal ischemia-reperfusion (I/R) injury.

32 Intestinal ischemia-reperfusion (IIR) often occurs durin

33 Sex differences in responses to intestinal ischemia-reperfusion (IR) have been recognize

34 nt-rich preservation solution in alleviating intestinal ischemia-reperfusion (IR) injury in a large a

35 Intestinal ischemia-reperfusion (IR) injury is initiated

36 Intestinal ischemia-reperfusion (IR) injury is initiated

37 e used a neutrophil-dependent mouse model of intestinal ischemia-reperfusion (IR) injury to explore t

38 e used a neutrophil-dependent mouse model of intestinal ischemia-reperfusion (IR) injury to investiga

39 endent adenosine production in modulation of intestinal ischemia-reperfusion (IR) injury.

40 Intestinal ischemia-reperfusion (IR)-induced damage requ

41 Intestinal ischemia-reperfusion after severe shock state

42 he role of neutrophil-derived MMP-9 in acute intestinal ischemia-reperfusion and its interaction with

43 urane anesthesia, the mice were subjected to intestinal ischemia-reperfusion by occlusion (clamping)

44 Intestinal ischemia-reperfusion caused bacterial translo

45 Intestinal ischemia-reperfusion injury (IRI) can occur i

46 atile anesthetic-mediated protection against intestinal ischemia-reperfusion injury (IRI).

47 Intestinal ischemia-reperfusion injury was induced by te

48 growth factor significantly protects against intestinal ischemia-reperfusion injury.

49 There was up to 3-fold more tissue MDA after intestinal ischemia-reperfusion than after sham laparoto

50 iNOS mediates bacterial translocation after intestinal ischemia-reperfusion, using iNOS knockout mic

51 iNOS knockout mice were more resistant to intestinal ischemia-reperfusion-induced bacterial transl

52 e, suggesting that iNOS might play a role in intestinal ischemia-reperfusion-induced loss of gut barr

53 ion generally has a milder course than small intestinal ischemia-reperfusion.

54 ng an in vivo isolated jejunal loop model of intestinal ischemia-reperfusion.

55 Intestinal ischemia/reperfusion (I/R) injury is a relati

56 role of OLFM4-positive neutrophils in murine intestinal ischemia/reperfusion (IR) injury.

57 As beta2-GPI is critical for intestinal ischemia/reperfusion (IR)-induced tissue dama

58 at PAR(2) modulates GIT and tissue damage in intestinal ischemia/reperfusion by a mechanism dependent

59 ndence for local and remote injury following intestinal ischemia/reperfusion in a clinically relevant

60 lmonary vascular resistance in both sham and intestinal ischemia/reperfusion injured animals compared

61 Pathophysiological states that produce intestinal ischemia/reperfusion injury (I/R) initiate an

62 re, we report on the use of a mouse model of intestinal ischemia/reperfusion injury to investigate th

63 Following intestinal ischemia/reperfusion injury, PLV H-130 treate

64 when administered in a therapeutic manner in intestinal ischemia/reperfusion injury.

65 n-4 positive neutrophil has a role in murine intestinal ischemia/reperfusion injury.

66 Our data provide primary evidence that intestinal ischemia/reperfusion is a leading pathophysio

67 re activated in a microenvironment shaped by intestinal ischemia/reperfusion, and investigated local

68 Using a mouse model of intestinal ischemia/reperfusion, we administered peptide

69 (Rv)D5n-3 DPA protected against colitis and intestinal ischemia/reperfusion-induced inflammation in

70 PLV PP-5 and a significantly lower (p <.05) intestinal ischemia/reperfusion-mediated increase in mic

71 g system in the neuroimmune communication in intestinal ischemia/reperfusion.

72 ents with cirrhosis and PVT with evidence of intestinal ischemia require urgent anticoagulation to mi

73 so in cardiac tamponade-induced nonocclusive intestinal ischemia, the superior mesenteric artery flow

74 Acute intestinal ischemia was diagnosed in 170 patients.

75 Intestinal ischemia was induced in vivo for 60 min, foll

76 Mesenteric encasement with intestinal ischemia was successfully relieved in 10 of 1

77 ck, myocardial ischemia, ischemic stroke, or intestinal ischemia were similar in the two groups (P =

78 onsidered in patients with cirrhosis without intestinal ischemia who develop recent (<6 months) PVT t

79 PVG rats were subjected to 30-min intestinal ischemia with a subgroup of animals receiving

80 as opposed to IP microdialysis detects small intestinal ischemia with higher sensitivity and specific

81 Adult rats were subjected to intestinal ischemia, with histologic and biochemical dam