ライフサイエンスコーパス: intestinal lumen

1 helium and their subsequent release into the intestinal lumen.

2 ecause of the lack of cognate antigen in the intestinal lumen.

3 recognize ligands that co-localize along the intestinal lumen.

4 to promote transmucosal fluid effux into the intestinal lumen.

5 ainst parasitic helminthes that dwell in the intestinal lumen.

6 derived resistin-like molecule beta into the intestinal lumen.

7 ytes to deploy catalytic activities into the intestinal lumen.

8 on, with concomitant water movement into the intestinal lumen.

9 s limited to the apical membranes facing the intestinal lumen.

10 and subsequent accumulation of fluid in the intestinal lumen.

11 trophic feedings and the microecology of the intestinal lumen.

12 the release of BB membrane vesicles into the intestinal lumen.

13 can participate in antigen sampling from the intestinal lumen.

14 did not affect the expression of pmrH in the intestinal lumen.

15 d signal-induced release to be active in the intestinal lumen.

16 is involved in dietary zinc uptake from the intestinal lumen.

17 led code for the presence of a threat in the intestinal lumen.

18 express and release leptin apically into the intestinal lumen.

19 generate microbicidal concentrations in the intestinal lumen.

20 ating takes place extracellularly within the intestinal lumen.

21 polarized secretion of interleukin-6 to the intestinal lumen.

22 (PMN) across the epithelial monolayer to the intestinal lumen.

23 ctivity of neutrophils as they arrive in the intestinal lumen.

24 ely across the epithelial monolayer into the intestinal lumen.

25 ation across the epithelial monolayer to the intestinal lumen.

26 sive inflammation to the normal flora in the intestinal lumen.

27 FTR), should reduce fluid secretion into the intestinal lumen.

28 y infects the host by translocating from the intestinal lumen.

29 usually considered a digestive enzyme in the intestinal lumen.

30 um, while 2570.00 microg IgE/day entered the intestinal lumen.

31 ation of NaCl from interstitial fluid to the intestinal lumen.

32 dically exposed to pancreatic trypsin in the intestinal lumen.

33 ered by the presence of bacterial LPS in the intestinal lumen.

34 rusion of infected epithelial cells into the intestinal lumen.

35 pouchings (diverticula) originating from the intestinal lumen.

36 in spatially extended environments like the intestinal lumen.

37 neural circuits based on the content of the intestinal lumen.

38 g on the signal intensity they induce in the intestinal lumen.

39 a perennial carbon and energy source in the intestinal lumen.

40 highly vascularized mucosa and the anaerobic intestinal lumen.

41 dified to deliver hepcidin directly into the intestinal lumen.

42 y, and impaired protein digestion within the intestinal lumen.

43 Simian immunodeficiency virus (SIV)-inflamed intestinal lumen.

44 esumably caused by excess fatty acids in the intestinal lumen.

45 ese mice, hREG3A travels via the bile to the intestinal lumen.

46 ere translocated into tissues outside of the intestinal lumen.

47 hosts actively sequester bacteria within the intestinal lumen.

48 esent at nearly 50 mum concentrations in the intestinal lumen.

49 th commensal microbes that reside within the intestinal lumen.

50 l grams of IgA are secreted every day in the intestinal lumen.

51 the commensal microbes during growth in the intestinal lumen.

52 and abrogated mucus secretion into the large intestinal lumen.

53 estine, to be detached and released into the intestinal lumen.

54 he presence of viable tachyzoites within the intestinal lumen.

55 tips of IEC microvilli and accumulate in the intestinal lumen.

56 r reflects the low-oxygen environment of the intestinal lumen.

57 macrophage-derived [(3)H]cholesterol in the intestinal lumen.

58 a lamprey secreted taurocholic acid into its intestinal lumen.

59 tivation and immune cell infiltration in the intestinal lumen.

60 les and thus are secreted to function in the intestinal lumen.

61 for example, vha-6), which are essential for intestinal lumen acidification in Caenorhabditis elegans

62 s designed to rapidly disassemble within the intestinal lumen after use, reliably degrading while mai

63 Bloating of the intestinal lumen also activates a broad innate immune re

64 atory responses and fluid secretion into the intestinal lumen and also can enhance the immunogenicity

65 population, reside in close proximity to the intestinal lumen and are conserved throughout vertebrate

66 hat the [(3)H]AS-binding site resides in the intestinal lumen and are consistent with preclinical dat

67 s mislocalize birefringent material into the intestinal lumen and are lacking in acidified intestinal

68 sequestrants (BAS) complex bile acids in the intestinal lumen and decrease intestinal FXR activity.

69 In contrast, they are secreted into the intestinal lumen and degraded during dauer arrest, only

70 y the fibers in the anatomically challenging intestinal lumen and demonstrate wireless control of sen

71 to successfully establish replication in the intestinal lumen and does not result from ordered spread

72 s from wheat, rye, and barley persist in the intestinal lumen and elicit an immune response in geneti

73 gA production, which is also abundant in the intestinal lumen and has a crucial role as the first def

74 ce resulted in acute release of BAs into the intestinal lumen and increased proliferation of the inte

75 e gut microbiota is compartmentalized in the intestinal lumen and induces local immune responses, but

76 d toxin A receptor sequesters toxin A in the intestinal lumen and inhibits its effects of ileal mucos

77 A, can directly bind to such antigens in the intestinal lumen and interfere with epithelial attachmen

78 stant Klebsiella pneumoniae, emerge from the intestinal lumen and invade the bloodstream of vulnerabl

79 ), glycerophosphocholine, and choline in the intestinal lumen and is the primary source of systemic c

80 gA promoted V. cholerae agglutination in the intestinal lumen and limited the ability of the bacteria

81 measured the effects of BA release into the intestinal lumen and on cell renewal.

82 ed back from intestinal villi into the small intestinal lumen and reinfected the Peyer's patches.

83 erichia coli, and Proteus mirabilis from the intestinal lumen and represents a potential therapeutic

84 oncentrations of fructose and glucose in the intestinal lumen and serum, respectively, and the tumors

85 erial species can destroy antibiotics in the intestinal lumen and shield antibiotic-sensitive pathoge

86 eased from the intestinal epithelia into the intestinal lumen and systemic circulation and bind to th

87 distributing host defense machinery into the intestinal lumen and that microvillus-derived LVs modula

88 l barrier between the high Ag content of the intestinal lumen and the sterile environment beyond the

89 the epithelium at the interface between the intestinal lumen and the sterile environment of the lami

90 ypically avirulent pathogens remained in the intestinal lumen and were eventually outcompeted by the

91 cidal enteric alpha-defensins into the small intestinal lumen, and cryptdin-4 (Crp4) is the most bact

92 Bile salt synthesis, secretion into the intestinal lumen, and resorption in the ileum occur in a

93 organisms mainly infected the intestines and intestinal lumen, and we only sporadically observed few

94 on; increased leak of serum protein into the intestinal lumen; and altered tight junction structure.

95 lial MMCs, and levels in the bloodstream and intestinal lumen are maximal at the time of worm expulsi

96 HD5 peptides isolated from the intestinal lumen are proteolytically processed forms--HD

97 When alpha-defensin levels in the intestinal lumen are restored by intravenous administrat

98 es that express hREG3A, which travels to the intestinal lumen, are less sensitive to colitis than con

99 t accumulation of dietary cholesterol in the intestinal lumen at the end of 6-hour lipid infusion and

100 ion of processed forms of proguanylin in the intestinal lumen but not in plasma supports the notion t

101 mosomoides polygyrus, which both live in the intestinal lumen, but it does not contribute to immunity

102 tead relied on antibiotic degradation in the intestinal lumen by BLA.

103 is thought to promote uptake of Stx from the intestinal lumen by compromising the epithelial barrier.

104 ay gain a specific survival advantage in the intestinal lumen by decreasing the expression of microbi

105 ins, termed cryptdins, are secreted into the intestinal lumen by mouse Paneth cells in response to mi

106 onmental dsRNAs are imported from the acidic intestinal lumen by SID-2 via endocytosis and are releas

107 e chloride secretion by enterocytes into the intestinal lumen by the cystic fibrosis transmembrane co

108 uggested that histones are released into the intestinal lumen by the high turnover of the intestinal

109 ins in the basolateral membranes, and in the intestinal lumen by weaker interactions with apical memb

110 that commensal microorganisms present in the intestinal lumen can affect the efficiency of serovar Ty

111 in and that movement of bacteria through the intestinal lumen can occur very rapidly in the absence o

112 nced by exogenous factors present within the intestinal lumen, CD1d expression was analyzed in severa

113 himurium bacteria harvested from the chicken intestinal lumen (cecum) was compared with that of a lat

114 Starting from the intestinal lumen, conjugated bile salts cross the otherw

115 ocalization of gut granule contents into the intestinal lumen, consistent with a defect in intracellu

116 ransfer of CX3CR1(+) cells directly into the intestinal lumen, consistent with intraluminal CX3CR1(+)

117 ransfer of CX3CR1(+) cells directly into the intestinal lumen, consistent with intraluminal CX3CR1(+)

118 sting that a reduced ability to colonize the intestinal lumen contributed to its attenuation.

119 he availability of electron acceptors in the intestinal lumen, creating a favorable niche for pathoge

120 resist gastric and bile acids, colonize the intestinal lumen, cross the intestinal barrier, survive

121 s in a net efflux of salt and water into the intestinal lumen, culminating in watery diarrhea.

122 icochemical environment of the center of the intestinal lumen differs from that of the epithelial sur

123 depletion of IFB-2, IFC-2 and IFD-2 leads to intestinal lumen dilation although depletion of IFC-1, I

124 iously shown that adenosine is formed in the intestinal lumen during active inflammation from neutrop

125 Adenosine is formed in the intestinal lumen during active inflammation from neutrop

126 We conclude that adenosine released in the intestinal lumen during active inflammation may induce I

127 Shiga toxins expressed in the intestinal lumen during infection with Shiga-toxigenic E

128 We show that IL-4 and IL-13 protect against intestinal lumen-dwelling worms primarily by inducing in

129 dicated that under conditions similar to the intestinal lumen environment, the genes identified are u

130 ical origin but BB is more accessible in the intestinal lumen, especially regarding protein.

131 profile similar to that of bacteria from the intestinal lumen, except that levels of transcription, t

132 up 1B PLA2 digestion of phospholipids in the intestinal lumen facilitates postprandial lysophospholip

133 oteins involved in their absorption from the intestinal lumen fluctuate in a circadian manner.

134 are minimally absorbed and washed out of the intestinal lumen for application as antisecretory agents

135 and that its presence is not required in the intestinal lumen for dietary iron absorption.

136 plant sterols from enterocytes back into the intestinal lumen for excretion.

137 of S. typhimurium persists in the C. elegans intestinal lumen for the rest of the worms' life.

138 is driven by fluid transport, recapitulating intestinal lumen formation.

139 rier limiting micro-organisms present in the intestinal lumen from colonizing enterocytes.

140 intestine forms a barrier that separates the intestinal lumen from the host's internal milieu and is

141 mucus functions to segregate contents of the intestinal lumen from the intestinal epithelium.

142 ed the apical compartment that simulates the intestinal lumen, from the BC which represented the bloo

143 mic circulation via the enterocytes into the intestinal lumen has traditionally been considered a min

144 sms that underlie protease regulation in the intestinal lumen have remained unclear.

145 ens in microbe-rich environments such as the intestinal lumen; however, the mechanisms are unclear.

146 bulin isotypes that are transported into the intestinal lumen, IgA and IgM, for antigiardial host def

147 Achieving a "black" intestinal lumen in 3D T1-weighted sequences with intrav

148 d fermentation acids, factors present in the intestinal lumen in patients with active IBDs.

149 y to secrete the bioactive peptides into the intestinal lumen in response to dietary factors; release

150 asolaterally, and is hence found in both the intestinal lumen in the mucosal layer as well as in the

151 ysis and metabolic parameters from the small intestinal lumen indicate onset of ischemia earlier than

152 and that psyllium, through its action in the intestinal lumen, indirectly affected the intravascular

153 Dietary sugars are transported from the intestinal lumen into absorptive enterocytes by the sodi

154 te the absorption of dietary copper from the intestinal lumen into blood as well as utilizing copper

155 Although these mice take up iron from the intestinal lumen into mature epithelial cells normally,

156 nsient transference of microspheres from the intestinal lumen into mesenteric lymph that was not obse

157 ts in the translocation of microbes from the intestinal lumen into the circulation.

158 at microbial molecules translocated from the intestinal lumen into the host's internal environment ma

159 Microbial molecules translocated from the intestinal lumen into the host's internal environment pl

160 n is induced when S. Typhi transits from the intestinal lumen into the ileal mucosa.

161 C) expression when bacteria transit from the intestinal lumen into the intestinal mucosa.

162 6 antigen were tested for movement from the intestinal lumen into the subepithelium.

163 translocation of microbial products from the intestinal lumen into the systemic circulation occurs du

164 lear leukocytes (PMNs; neutrophils) into the intestinal lumen is a classical phenomenon associated wi

165 that although phospholipid digestion in the intestinal lumen is a prerequisite for efficient cholest

166 e shedding of apoptotic enterocytes into the intestinal lumen is observed in inflammatory bowel disea

167 In conclusion, pro-HD-5 in the intestinal lumen is processed by trypsin in a complex in

168 etic stimulation of vagal afferents from the intestinal lumen is sufficient to evoke a reward phenoty

169 rget genes. Release of endogenous BAs in the intestinal lumen is sufficient to promote ISC renewal an

170 e immunity in the hostile environment of the intestinal lumen, it should be of interest to define bio

171 butyrate-producing Clostridia from the mouse intestinal lumen, leading to decreased butyrate levels,

172 aim-1 animals are improperly oriented in the intestinal lumen, leading to reduced levels of parasite

173 ctors and bound virulent bacteria within the intestinal lumen, leading to their engulfment by neutrop

174 ority of commensal bacteria are found in the intestinal lumen, many species have also adapted to colo

175 t study tests the hypothesis that CEL in the intestinal lumen may influence the type of lipoproteins

176 Here, we show that bloating of the intestinal lumen of Caenorhabditis elegans caused by mic

177 al bacterial communities residing within the intestinal lumen of mammals have evolved to flourish in

178 step in the translocation of prions from the intestinal lumen of mammals in vivo, which is a precurso

179 peptidoglycan or Pam(3)Cys injected into the intestinal lumen of mice resulted in receptor redistribu

180 o colonize the mucus that accumulates in the intestinal lumen of mice with CF.

181 cherichia coli strains can take place in the intestinal lumen of N2 wild-type worms at a rate of 10(-

182 The half-life of intact IgE in the intestinal lumen of rats 10 days after infection was 3.2

183 IgM, transferred through breast milk to the intestinal lumen of suckling offspring, coats the pathog

184 the delivery of compounds directly into the intestinal lumen or the aspiration of intestinal fluids

185 esent, they increase water movement into the intestinal lumen over and above the levels observed with

186 cillus plantarum freely circulate within the intestinal lumen, pathogenic strains such as Erwinia car

187 cell alpha-defensins secreted into the small intestinal lumen persist as intact and functional forms

188 Long chain triglyceride (>C12) in the intestinal lumen potently inhibits gastric emptying and

189 Colonization of the intestinal lumen precedes invasive infection for a wide

190 ella pneumoniae, and Escherichia coli in the intestinal lumen, predisposing patients to bloodstream i

191 y, reduced growth, emaciated body, distended intestinal lumen, rectal swelling, and prolific infectio

192 thelia induces neutrophil recruitment to the intestinal lumen, resulting in neutrophil extravasation

193 Notably, neutrophil emigration to the intestinal lumen results in the generation of organized

194 ntestine: first, they are eliminated via the intestinal lumen; second, pro-inflammatory T(H)17 cells

195 hen Salmonella typhimurium is present in the intestinal lumen, several environmental and regulatory c

196 The biocomplexity of the intestinal lumen suggests that interactions between the

197 lamine produced from dietary fats within the intestinal lumen that can modulate lipid metabolism, ins

198 smotic imbalance, creating conditions in the intestinal lumen that favor xanthine stone accumulation.

199 that regulates the secretion of IgA into the intestinal lumen, the polymeric immunoglobulin receptor,

200 ite displaying wild-type colonization of the intestinal lumen, the straight-rod Deltapgp1 and Deltapg

201 ree fatty acids and monoacylglycerols in the intestinal lumen, the uptake of these products into ente

202 ood accumulation in the anterior part of the intestinal lumen, thereby triggering rhythmical movement

203 ovilli release unilamellar vesicles into the intestinal lumen; these vesicles retain the right side o

204 on-absorbable disaccharide, which alters the intestinal lumen through multiple mechanisms that lead t

205 Iron uptake from the intestinal lumen through the apical surface of polarized

206 very early after infection, migrate into the intestinal lumen to capture bacteria.

207 During late gestation, villi extend into the intestinal lumen to dramatically increase the surface ar

208 quently linked to its translocation from the intestinal lumen to host tissues.

209 , allow dendritic cells direct access to the intestinal lumen to obtain information about commensal a

210 gest that the oligomers can diffuse from the intestinal lumen to other tissues, resulting in a diseas

211 cid transporter reclaims bile acids from the intestinal lumen to preserve their enterohepatic recircu

212 ctively targets virulent C. rodentium in the intestinal lumen to promote pathogen eradication and hos

213 e progresses through its life cycle from the intestinal lumen to submucosa and back again.

214 Glucose amplifies its own transport from the intestinal lumen to the bloodstream by increasing lumina

215 t constitutes a signaling mechanism from the intestinal lumen to the body's defenses.

216 data suggest that a transport nexus from the intestinal lumen to the eggs introduces bottlenecks that

217 lesterol and triglyceride transport from the intestinal lumen to the lymph.

218 ase inhibition on cholesterol transport from intestinal lumen to the lymphatics was evaluated in lymp

219 its information about the composition of the intestinal lumen to the mucosal immune system.

220 w molecular weight soluble antigens from the intestinal lumen to underlying CD103(+) LP-DCs.

221 e transepithelial transport of SIgA from the intestinal lumen to underlying gut-associated organized

222 ncrease in bile acid availability within the intestinal lumen triggers C. difficile germination.

223 transitional junction seals the pit from the intestinal lumen until differentiation-driven, basal-to-

224 ce, sodium ions and water also flow into the intestinal lumen via a paracellular route.

225 stages, and that pancreatic AFGPs enter the intestinal lumen via the pancreatic duct to prevent inge

226 ted robust secretion of cholesterol into the intestinal lumen via the sterol-exporting heterodimer ad

227 oximately 3-4% of the dose injected into the intestinal lumen was absorbed, relative to a subcutaneou

228 Recovery of dietary FA in the intestinal lumen was comparable between WT and KO, consi

229 eptor (pIgR)-mediated IgA transport into the intestinal lumen was interrogated.

230 icial roles of nonpathogenic bacteria in the intestinal lumen were described.

231 bed, they were expected to accumulate in the intestinal lumen where a potential inhibition capacity o

232 rbed, they are expected to accumulate in the intestinal lumen where a potential inhibition capacity o

233 ich led to relocation of the pathogen to the intestinal lumen where it was outcompeted by commensals.

234 nteric alpha-defensins are secreted into the intestinal lumen where they have potent microbicidal act

235 ce, the precise role of secretory IgA in the intestinal lumen, where it coats a diverse array of comm

236 porulating C. perfringens cells in the small intestinal lumen, where it then causes epithelial cell d

237 upon inflammation they are secreted into the intestinal lumen, where they promote prostaglandin synth

238 e hormone that induces bile release into the intestinal lumen which in turn aids in fat digestion and

239 V. cholerae caused more clouds of DNA in the intestinal lumen, which appeared to be neutrophil extrac

240 ocated at the critical interface between the intestinal lumen, which is chronically exposed to food a

241 cations in volume, bulk and viscosity in the intestinal lumen, which will alter metabolic pathways of

242 l passage of ions and water to lubricate the intestinal lumen while restricting fluid loss.

243 irculation, as well as the vaginal vault and intestinal lumen, with CCL20 playing a central role.