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1 on to generate motility patterns linked with intestinal secretion.
2  ligands guanylin and uroguanylin, regulates intestinal secretion.
3  (CFTR)-related chloride channel, triggering intestinal secretion.
4 enteroid lumens, recapitulating ETEC-induced intestinal secretion.
5 tion routes to induce a response in serum or intestinal secretion.
6 associated chloride (Cl-) flux that underlie intestinal secretion.
7 , possibly representing a novel mechanism of intestinal secretion.
8 l toxin and second-messenger agonist-induced intestinal secretion.
9  permeabilizing effect of the toxin leads to intestinal secretion.
10 vation of a CCK-releasing peptide present in intestinal secretion.
11 A) and IgG in the serum and increased IgA in intestinal secretions.
12  and one third of this derived from recycled intestinal secretions.
13  (135-fold increase), in large but not small intestinal secretions.
14 1-6) abolished the CCK releasing activity of intestinal secretions.
15 ctive at induction of specific antibodies in intestinal secretions.
16  II) has been described in the regulation of intestinal secretion and absorption via angiotensin subt
17                   Cholera toxin (CT)-induced intestinal secretion and Chinese hamster ovary cell (CHO
18 previously reported inhibition of CT-induced intestinal secretion and CHO elongation by platelet-acti
19 caspase 8 activation and reduced TxA-induced intestinal secretion and disruption.
20 R dsRNA pretreatment, TxA-induced changes in intestinal secretion and histopathologic inflammation we
21 ay a proinflammatory role in toxin A-induced intestinal secretion and inflammation and that CRH(1) re
22 ulating the severity of enterotoxin-mediated intestinal secretion and inflammation by activating both
23  partially protected against toxin A-induced intestinal secretion and inflammation, and this effect w
24 f cGMP concentration is necessary for proper intestinal secretion and intestinal epithelial cell prol
25 l enteric neural control and coordination of intestinal secretion and motility suggests that either t
26 l enteric neural control and coordination of intestinal secretion and motility, and acts as a common
27 he action of S. boulardii on toxin A-induced intestinal secretion and mucosal permeability to [3H]man
28 ence of secretory phospholipase A2 in tears, intestinal secretions, and leukocytes, this enzyme may p
29 ltifunctional peptides that affect appetite, intestinal secretions, and motility whereas others regul
30 lin and uroguanylin, are local regulators of intestinal secretion by activation of receptor-guanylate
31 n for the reported suppression of CT-induced intestinal secretion by inhibitors of protein synthesis.
32 ic CL have a greater propensity for a higher intestinal secretion clearance (CL(IE)).
33                                              Intestinal secretions from immunized mice completely inh
34 le molecular mechanisms mediating ST-induced intestinal secretion have been defined, advancements in
35               DSS treatment increased PGE(2) intestinal secretion in all groups except COX-2(-/-) mic
36                                          The intestinal secretion induced by zonula occludens toxin f
37        The role of oxidants in promoting net intestinal secretion is important, but the cellular mech
38 and that the presence of these antibodies in intestinal secretions is sufficient for protection again
39  advantageous, as intraluminal nutrients and intestinal secretions might modulate the intestinal immu
40  ileal loops were used for the evaluation of intestinal secretion, mucosal disruption, and apoptosis.
41 n-CLR dsRNA pretreatment, TxA induced robust intestinal secretion, myeloperoxidase activity, and hist
42 t INT-767 on hepatic bile acid synthesis and intestinal secretion of glucagon-like peptide-1 (GLP-1)
43       Bariatric surgery results in increased intestinal secretion of hormones GLP-1 and anorexigenic
44                                              Intestinal secretion of ions and fluid is increased by F
45 cretory diarrhea that was mediated by excess intestinal secretion of potassium.
46                   This resulted from reduced intestinal secretion of triglycerides following ingestio
47                 Cholera toxin (CT) increases intestinal secretion of water and electrolytes and modul
48                                          The intestinal secretions of IDO-deficient mice had elevated
49 f IgG in sera and IgA in tracheal, lung, and intestinal secretions, significantly higher (4-6 fold) t
50 utative luminal CCK-releasing factor (LCRF), intestinal secretions were collected by perfusing a modi
51 ited high levels of anti-SEB IgA in lung and intestinal secretions, whereas the toxoid without proteo