ライフサイエンスコーパス: intimal

1 search on atherosclerosis has focused on the intimal accumulation of lipids and inflammatory cells, t

2 IL-6 on endothelial cell integrity or on the intimal accumulation of smooth muscle cells, macrophages

3 Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a

4 r BMP activity, atherosclerotic lesion size, intimal and medial calcification, and inflammation.

5 as a profound influence on the population of intimal and medial endothelial, macrophage, and smooth m

6 Intimal and medial hyperplasia correlated significantly

7 Intimal and medial hyperplasia was significantly inhibit

8 p67phox, immunoreactive NoxA1 is present in intimal and medial SMCs of human early carotid atheroscl

9 The importance of CyPA for intimal and medial thickening was shown by strong correl

10 Histology revealed abnormal intimal and medial thickening, disorganized and fragment

11 Both intimal and valvular calcifications are closely associat

12 on responses by immune cells resident in the intimal aortic wall.

13 ients who received ATG by changes in maximal intimal area (1.0 +/- 1.2 versus 2.3 +/- 2.6 mm(2); P =

14 odel (OA-NO(2) treatment resulted in reduced intimal area and intima to media ratio versus vehicle- o

15 acity to deliver drug more evenly within the intimal area rather than concentrating drug around the s

16 At 1 year, the SVG mean intimal area was 4.3 +/- 2.1 mm(2), and the occlusion ra

17 ne concentration to an evolution law for the intimal area, we reduce the problem to a single nonlinea

18 with intimal gathering without alteration of intimal area.

19 lear if the category of acute rejection with intimal arteritis (ARV) is relevant to short- and long-t

20 Intimal arteritis (Banff v-lesion) was an independent hi

21 Intimal arteritis (the presence of v-lesions) in kidney

22 teria of AMR: T cell-mediated rejection with intimal arteritis (v) lesion (TCMRV; n = 78), total anti

23 The Banff scores of intimal arteritis (v1, v2 and v3) represented low, moder

24 aft outcomes are affected by the severity of intimal arteritis.

25 l activation, a response resembling clinical intimal arteritis.

26 pathomechanisms of arterial calcifications: intimal associated with atherosclerosis and medial knows

27 nd other inflammatory cells are found in the intimal atherosclerotic lesions.

28 fic sclerosis, is notably different from the intimal atherosclerotic process commonly associated with

29 The spasm lesion more frequently showed an intimal bump at baseline and intimal gathering during sp

30 ic state and facilitates the formation of an intimal bump in patients with vasospastic angina.

31 An intimal bump was defined as 1 or more intimal projection

32 of chondrocyte-like cells in atherosclerotic intimal calcification of C57BL/6 LDLr(-/-) mice using bo

33 that this effect could be due to a defect in intimal cell differentiation from endothelial to mesench

34 cpep1-null mice show decreases in medial and intimal cell proliferation as well as vessel remodeling

35 ed how well vascular elastography can detect intimal changes in a mouse model of carotid remodeling.

36 d elastography method for early detection of intimal changes in small animals.

37 mic stresses have normalized without obvious intimal changes.

38 Increased accumulation of less organized intimal collagen, laminin, alpha-smooth muscle actin, an

39 ll pathology contributes to AAD with typical intimal damage at sites of mechanical trauma.

40 solving inflammatory response that can cause intimal destruction, arterial thrombosis, and end-organ

41 Focal intimal disruption (FID) has been described in >20% of t

42 FID was defined as an intimal disruption with contrast material-filled outpouc

43 Nearly all the intimal dissection tears were located at sites of previo

44 Thrombi because of superficial intimal erosion characteristically complicate matrix-ric

45 y until day 42, at which point the degree of intimal expansion (IE) of vessels was assessed by histol

46 y until day 42, at which point the degree of intimal expansion (IE) of vessels was assessed by histol

47 peratively results in T cell infiltrates and intimal expansion 4 wk later.

48 eic arterial interposition grafts, producing intimal expansion and calcification in the absence of T

49 ation of human IL-6 reduces the magnitude of intimal expansion and total T cell infiltration but incr

50 Intimal expansion can give rise to dangerous events such

51 enhances allogeneic T cell infiltration and intimal expansion in a model of human allograft rejectio

52 odels characterized by VSMC accumulation and intimal expansion to determine the function of AIP1.

53 -J(2), ciglitazone, and pioglitazone reduced intimal expansion, intimal infiltration of CD45RO(+) mem

54 effector T cells into the artery intima and intimal expansion.

55 It is pathologically defined as intimal fibromuscular dysplasia.

56 of the mesenchymal cells responsible for the intimal fibrosis in systemic sclerosis (SSc) has not bee

57 ment membrane or arteriopathy manifesting as intimal fibrosis.

58 appearance, concentric intramural haematoma, intimal flap (the most definite sign), and double lumen

59 However, dissections with intimal flap extension into the aortic arch between the

60 racteristics were comparable, but the mobile intimal flap group showed a lower re-intervention rate (

61 We analysed whether a high intimal flap mobility (IFM) of the dissection membrane h

62 ocedure time, number of fenestrations of the intimal flap per patient, necessity of additional aortic

63 In four patients, fenestration of the intimal flap was performed twice.

64 However, only 3 patients presented an intimal "flap" on angiography.

65 as a negative regulator in IFN-gamma-induced intimal formation, in part by downregulating IFN-gamma-J

66 ently showed an intimal bump at baseline and intimal gathering during spasm compared with the nonspas

67 Intimal gathering was defined as a folding/gathering of

68 al narrowing during spasm is associated with intimal gathering without alteration of intimal area.

69 . 22.3%; p = 0.006) and tendency for greater intimal growth (percent intimal volume: 9.2 +/- 9.3% vs.

70 aths produced pronounced (85%) inhibition of intimal hyperplasia (0.15+/-0.05 vs 1.01+/-0.16), withou

71 cold ischemia time developed severe TV with intimal hyperplasia (alpha-smooth muscle actin positive

72 sue factor (TF) and thrombin are involved in intimal hyperplasia (IH) and remodelling following vascu

73 This leads to intimal hyperplasia (IH) formation that induces inflamma

74 Intimal hyperplasia (IH) limits the patency of all cardi

75 Intimal hyperplasia (IH) remains a major cause of poor p

76 r processes, constrictive vessel remodeling, intimal hyperplasia (IH), and retarded re-endothelializa

77 e major pro-restenotic pathologies including intimal hyperplasia (IH), endothelium impairment, and ve

78 emodeling and the formation of flow-limiting intimal hyperplasia (IH).

79 vs. 0.77+/-0.10 micromol/L; P<0.01) and less intimal hyperplasia (risk ratio [95% confidence interval

80 cent VSMCs; however, CD98hc deletion reduced intimal hyperplasia after arterial injury.

81 n shown to be involved in the development of intimal hyperplasia after vascular injury.

82 by the development of vascular inflammation, intimal hyperplasia and accelerated atherosclerosis.

83 igating mechanisms of hemodynamically driven intimal hyperplasia and arterial wall remodeling.

84 nt vascular functions in disease, especially intimal hyperplasia and atherosclerosis.

85 ID(-/-) recipients with further reduction of intimal hyperplasia and fibrosis scores.

86 lly challenging and can lead to failure from intimal hyperplasia and foreign body reaction.

87 and protects against injury-induced arterial intimal hyperplasia and inflammation, providing protecti

88 Intimal hyperplasia and lesion size were measured histol

89 Frequently, this leads to intimal hyperplasia and lumen narrowing, with significan

90 d induce ischemic organ damage through rapid intimal hyperplasia and luminal occlusion.

91 perone function is an important regulator of intimal hyperplasia and may have implications for molecu

92 ors, and medications are associated with SVG intimal hyperplasia and occlusion after CABG.

93 We assessed the following correlates for intimal hyperplasia and occlusion: patient characteristi

94 /-) mice, which displayed significantly more intimal hyperplasia and perivascular fibrosis compared t

95 rotid arteries attenuated the development of intimal hyperplasia and reduced arterial wall thickening

96 namic manipulations can reproducibly augment intimal hyperplasia and remodeling end points in mouse v

97 clinically relevant mouse model of arterial intimal hyperplasia and remodeling.

98 id focal stenosis yields reproducible murine intimal hyperplasia and substantial differentials in art

99 ew mechanism for the development of vascular intimal hyperplasia and suggest a novel therapeutic stra

100 istic insight into the formation of vascular intimal hyperplasia and suggest a novel therapeutic stra

101 SMCs, but not in endothelial cells, reduced intimal hyperplasia and suppressed the SMC synthetic phe

102 werful tool for unraveling the mechanisms of intimal hyperplasia and vascular remodeling, although th

103 Two of our recipients developed severe intimal hyperplasia and vasculopathy early post-transpla

104 ft failure includes thrombotic occlusion and intimal hyperplasia at the site of vascular anastomosis.

105 afts are prone to thrombosis, infection, and intimal hyperplasia at the venous anastomosis.

106 ockout (Nlrc5(-/-)) mice exhibit more severe intimal hyperplasia compared with wild-type mice after c

107 Stenosis due to intimal hyperplasia development often occurs after place

108 pression in A20 heterozygote mice aggravated intimal hyperplasia following partial carotid artery lig

109 Although intimal hyperplasia has been reported in human hand tran

110 Drugs that inhibit intimal hyperplasia have been developed, however there i

111 The effect of NO-ELIP delivery to attenuate intimal hyperplasia in a balloon-injured artery was dete

112 ivo lentiviral delivery of miR-128 prevented intimal hyperplasia in a mouse model of carotid restenos

113 Average intimal hyperplasia in all arteries was 2+/-4%.

114 hat miRNA-223 inhibits dedifferentiation and intimal hyperplasia in diabetic mice by decreasing PDGFR

115 ing EC repair and the ensuing development of intimal hyperplasia in eNOS(-/-) mice.

116 TP activity is associated with inhibition of intimal hyperplasia in grafted veins, reduced inflammato

117 We noted significantly greater intimal hyperplasia in HET versus WT allografts, indicat

118 Viral delivery of circ_Lrp6 shRNA prevented intimal hyperplasia in mouse carotids.

119 Pioglitazone significantly rescues excessive intimal hyperplasia in Nlrc5(-/-) mice and attenuates th

120 lasma HDL-C levels by 93 +/- 26% and reduced intimal hyperplasia in the grafted vein by 38 +/- 6.2%.

121 external stent to reduce the development of intimal hyperplasia in vein grafts.

122 ide a novel clinical approach for decreasing intimal hyperplasia in vein grafts.

123 ctile gene expression and greatly attenuates intimal hyperplasia in vivo.

124 e for some of the smooth muscle cells within intimal hyperplasia lesions of vein grafts.

125 Within intimal hyperplasia lesions, smooth muscle cells are a m

126 Intimal hyperplasia of saphenous vein grafts (SVGs) can

127 No intimal hyperplasia of the coronary arteries was observe

128 rvival, myocardial lesion size and degree of intimal hyperplasia of the coronary arteries were analyz

129 Intimal hyperplasia produces restenosis (re-narrowing) o

130 Pcsk6(-/-) mice showed reduced intimal hyperplasia response upon carotid ligation in vi

131 echnically flexible method for enhancing the intimal hyperplasia response.

132 atelet Treatment After Grading Stent-Induced Intimal Hyperplasia Study (PRODIGY) trial assessed unsel

133 erplasia, hampering efforts to identify anti-intimal hyperplasia therapies.

134 heath that is highly effective in preventing intimal hyperplasia through perivascular delivery of rap

135 Intimal hyperplasia with luminal obliteration developed

136 atelet Treatment After Grading Stent-Induced Intimal Hyperplasia) trial and randomized to short (6 mo

137 re small, whereas wild-type mice had diffuse intimal hyperplasia, a high intima/media ratio, and infl

138 om the development of vascular inflammation, intimal hyperplasia, and accelerated atherosclerosis.

139 , fracturing of the internal elastic lamina, intimal hyperplasia, and eyelid vessel damage.

140 as cerebral and coronary arterial vasospasm, intimal hyperplasia, and hypertension.

141 jury, leading to intramural neoangiogenesis, intimal hyperplasia, and luminal occlusion.

142 preventing smooth muscle cell proliferation, intimal hyperplasia, and proteoglycan deposition.

143 GF-beta) has profound stimulatory effects on intimal hyperplasia, but it is unknown whether these eff

144 However, in wild-type mice, there is limited intimal hyperplasia, hampering efforts to identify anti-

145 ype recipient mice which had severe vascular intimal hyperplasia, interstitial fibrosis, and inflamma

146 In human, rat, and mouse intimal hyperplasia, PCSK6 was expressed by proliferatin

147 ke decreasing atherosclerosis, angiogenesis, intimal hyperplasia, pulmonary arterial hypertension, an

148 s upon initiation of conditions that promote intimal hyperplasia, suggesting a mechanism by which the

149 he response to vascular injury that leads to intimal hyperplasia, this study investigated the in vivo

150 The former is pathophysiologic resulting in intimal hyperplasia, whereas the latter is physiological

151 ial role in the formation and development of intimal hyperplasia, which is the main cause of graft fa

152 orphologic adaptations included proximal LCA intimal hyperplasia, which was exacerbated by diet-induc

153 his mechanism has important implications for intimal hyperplasia, wound healing, and fibrotic disease

154 e characterized histologically by concentric intimal hyperplasia.

155 ascular graft that resists both clotting and intimal hyperplasia.

156 ay in the TEV's resistance to occlusion from intimal hyperplasia.

157 roliferate is essential for the formation of intimal hyperplasia.

158 ay provide a novel approach to inhibition of intimal hyperplasia.

159 s (SMCs) is essential for the development of intimal hyperplasia.

160 oangiogenesis and results in lumen-occlusive intimal hyperplasia.

161 et proteins (HSP27 and CREB) associated with intimal hyperplasia.

162 es suggest that LMO7 is upregulated in human intimal hyperplastic arteriovenous fistula and cardiac a

163 coefficient was 0.80 (P<0.001) and with mean intimal index was 0.92 (P<0.001).

164 Maximal intimal thickness and mean intimal index were 0.73+/-0.50 mm and 20.9+/-10.6%, resp

165 Maximal intimal thickness and mean intimal index were recorded.

166 Intimal infiltration and modification of plasma-derived

167 and pioglitazone reduced intimal expansion, intimal infiltration of CD45RO(+) memory T cells, and pl

168 specific sites of the arterial tree through intimal inflammation, necrosis, fibrosis, and calcificat

169 ar traps probably perpetuates and propagates intimal injury and potentiates thrombosis due to superfi

170 Toll-like receptor 2 ligation in superficial intimal injury, a process implicated in superficial eros

171 n maintaining the health and function of the intimal layer in vasculature.

172 NTN1 protein was highly expressed in the intimal layer of the control vessels.

173 h an increased accumulation of apo(a) in the intimal layer of the vessel wall.

174 f medial vascular smooth muscle cells to the intimal layer, and ultimately acute thrombosis.

175 eoangiogenesis as well as hyperplasia of the intimal layer, implicating T cells in the maladaptive be

176 endothelial progenitors able to assemble the intimal layers.

177 entiated into smooth muscle cells within the intimal lesion and may provide a novel clinical approach

178 ring inflammatory cells do not contribute to intimal lesion formation.

179 blockade by TAK-779 reduced the severity of intimal lesions (53+/-10% vs. 16+/-2%; P<0.05) and decre

180 tantly, the donor hearts did not display any intimal lesions characteristic of chronic rejection.

181 After wire-injury, intimal lesions in AGER1-tg mice were small, whereas wil

182 els with medial hypertrophy or initial-stage intimal lesions in lung sections from VM macaques demons

183 iple lineages, and potentially contribute to intimal lesions in vivo.

184 expression of Id1 was observed in concentric intimal lesions of heritable PAH cases.

185 D In mouse carotid arteries with established intimal lesions tailored to resemble the substrate of hu

186 severe and irreversible PAH, associated with intimal lesions, colocalizing with proliferating ECs and

187 bone marrow transplantation did not develop intimal lesions, which indicates that CCR1-bearing infla

188 contribution of host cell precursors to the intimal lesions.

189 Axl-dependent survival is very important for intimal leukocytes; however, Axl expression in BM cells

190 last-like synoviocytes (FLS) in the synovial intimal lining of the joint are key mediators of inflamm

191 ia reflected the alterations observed in the intimal lipids, yet being more subtle.

192 Intimal lymphatics in the atherosclerotic lesions exhibi

193 aging of atherosclerotic lesions in terms of intimal macrophage accumulation and distribution and dis

194 of maladapted endothelial cells and triggers intimal macrophage accumulation.

195 Intimal macrophage infiltration promotes atherosclerotic

196 Intimal macrophages play a critical role in atherosclero

197 assess the distribution and accumulation of intimal macrophages using CD36-targeted nanovesicles.

198 argeted delivery of therapeutic compounds to intimal macrophages.

199 make ligand-coated Enano (L-Enano) to target intimal macrophages.

200 -mediated recognition and uptake of oxLDL by intimal macrophages.

201 targeted delivery of bioactive compounds to intimal macrophages.

202 s were associated with common carotid artery intimal media thickness (CC IMT) in European-Americans [

203 unction was assessed by using carotid-artery intimal media thickness (cIMT) and flow-mediated dilatio

204 onary artery calcification (CAC) and carotid intimal media thickness at baseline in 2000-2002 among p

205 nic variation in sex differences for carotid intimal media thickness was less pronounced.

206 ions also included body composition, carotid intimal-media thickness (c-IMT), ambulatory blood pressu

207 subclinical atherosclerosis (common carotid intimal-media thickness) and 2) associated risk factors

208 onographic investigations of fatty liver and intimal medial thickness of the common carotid arteries,

209 y calcium score, common and internal carotid intimal medial thickness, and major electrocardiographic

210 linical cardiovascular disease (CAC, carotid intimal medial thickness, stenosis, and ankle brachial i

211 ncluding multiple areas of patchy or diffuse intimal, medial or adventitial abnormalities with thicke

212 accumulation of fatty cells in the arterial intimal-medial layers.

213 The estimated mean differences in carotid intimal-medial thickness between groups of men with vari

214 Intimal-medial thickness was weakly, positively associat

215 al disease (coronary calcium, common carotid intimal-medial thickness, and ankle-brachial index) in 5

216 res, including ankle-brachial index, carotid intimal-medial thickness, and echocardiographic left ven

217 Type IV collagen was strictly intimal/medial.

218 e B(4)-dependent VSMC phenotypic modulation, intimal migration, and proliferation.

219 their expression of ABCA1 in comparison with intimal monocyte-derived macrophages, however, are unkno

220 ectively, 5 and 2 cases), 5 scaffolds showed intimal neovessels or marked peristrut low-intensity are

221 ncy of this innate immune receptor decreased intimal neutrophil adherence in regions of local flow di

222 These data redefine the lineage of intimal phagocytes and suggest that proliferation is ins

223 An intimal bump was defined as 1 or more intimal projections into the lumen that disappeared afte

224 monocytes/macrophages significantly enhances intimal proliferation after arterial injury.

225 s from atherogenic damage by reducing vessel intimal proliferation and inhibiting adhesion molecule e

226 with standard-dose cyclosporine and reduces intimal proliferation at 12 months in de novo heart tran

227 injury, Nf1(+/-) mice demonstrated increased intimal proliferation compared with wild-type mice.

228 hypothesis that Ras-Erk signaling regulates intimal proliferation in a murine model of NF1 arterial

229 ion molecule expression, and enhanced aortic intimal proliferation.

230 phospholipids, increasing their affinity for intimal proteoglycans; contribute to cholesterol loading

231 o luminal region, their proliferation in the intimal region, and neointima formation.

232 artery characterized by an expansion of the intimal region.

233 a synthetic phenotype has been implicated in intimal remodeling during vascular injury.

234 on stabilization of PECAM-1, which reverses intimal remodeling in vivo.

235 f cytokines and chemokines in human synovial intimal resident fibroblast-like synoviocytes (FLS).

236 OCT readily identified the intimal rupture site (n = 7), the thickness (348 +/- 84

237 rsus WT allografts correlated with increased intimal SMC proliferation, and a higher number of infilt

238 decreased expression of PTEN was observed in intimal SMCs of human atherosclerotic lesions underlying

239 ABCA1 expression by intimal SMCs was significantly reduced between early and

240 The intimal SMLCs in mouse GAD lesions differ from medial sm

241 Although intimal SMLCs may originate by migration and modulation

242 Intimal SMLCs, but not medial smooth muscle cells, expre

243 Intimal smooth muscle cells (SMCs) contribute to the foa

244 erosclerosis, where monocyte egress into the intimal space is central to formation of fatty plaques.

245 These data suggest that targeting intimal stiffening and/or the EC response to intima stif

246 Intimal surface areas of ascending aortas were measured

247 at the critical interface of blood with the intimal surface of diseased arteries and propagate and a

248 round dilation at the region affected by the intimal tear.

249 tality associated with blunt thoracic aortic intimal tears (IT) within the American College of Surgeo

250 CAV was defined as an intimal thickening >/= 0.5 mm in the follow-up with rega

251 nd specificity of 81% and 75% to detect CAV (intimal thickening >0.5 mm), whereas the PPV and NPV wer

252 ophy, arteriolar hyaline thickening, fibrous intimal thickening (CV), and fraction of sclerotized glo

253 ities were present in all children including intimal thickening (intima/lumen cross-sectional area ra

254 recipients, angiography was normal; however, intimal thickening (intima/media cross-sectional area ra

255 LITA showed a significant intimal thickening (P=0.05) and a nonsignificant medial

256 Lesions were classified into pathological intimal thickening (PIT), VH-IVUS-derived thin-capped fi

257 ly, restenosis after angioplasty, vein graft intimal thickening and atherogenesis.

258 day 24 posttransplantation and analyzed for intimal thickening and cellular infiltration.

259 rol allografts, which also developed greater intimal thickening and stenosis than hGSTA4-4 transduced

260 evealed patent ductus arteriosus with normal intimal thickening but dilated cardiac chambers.

261 , whereas arteriolar hyalinosis and vascular intimal thickening did not.

262 Intimal thickening is driven by damage to the endotheliu

263 Intimal thickening is usually attributed to the migratio

264 e, changes in right ventricular pressure and intimal thickening of pulmonary small artery are a littl

265 Cs together with fibrosis provoke concentric intimal thickening of the coronary arteries, which is th

266 (plaque erosion), most often on pathological intimal thickening or fibroatheromas.

267 typically characterized by diffuse coronary intimal thickening with pathological vessel remodeling,

268 CCR1-deficient recipients, it increased GAD intimal thickening with SMLC proliferation in only the w

269 cimens comprised an undiseased portion (thin intimal thickening with the underlying media) and a dise

270 olar hyalinosis, and 32.9% abnormal vascular intimal thickening).

271 patterns are observed: (1) shear stabilized intimal thickening, (2) tension induced wall thinning an

272 o the coronary circulation, LITA grafts show intimal thickening, increased intima/media ratio, and ma

273 ially drive the various vascular phenotypes (intimal thickening, media hypertrophy, adventitial thick

274 we propose and study a mathematical model of intimal thickening, posed as a free boundary problem.

275 lates most strongly with venous and small IV intimal thickening, similar to the pattern observed in P

276 tion ensues within 4 weeks, characterized by intimal thickening, T-cell infiltrates, and vascular cel

277 , v score, allograft glomerulopathy, fibrous intimal thickening, tubular atrophy, and interstitial fi

278 ity, as indicated by the loss of Eph-B4, and intimal thickening.

279 SMC) proliferation and migration and thereby intimal thickening.

280 cent medial thickness (arteries) and percent intimal thickness (%IT) (arteries, veins, and IV) relati

281 7.9 versus 12.8 +/- 12.3%; = 0.003), maximal intimal thickness (0.2 +/- 0.2 versus 0.3 +/- 0.3 mm; P

282 e demonstrated no difference in mean maximal intimal thickness (0.35+/-0.36 versus 0.36+/-0.26 mm; P=

283 On the basis of maximum intimal thickness (MIT) at BL, recipients were divided i

284 Maximal intimal thickness and mean intimal index were 0.73+/-0.5

285 Maximal intimal thickness and mean intimal index were recorded.

286 revealed significant differences of arterial intimal thickness between rejecting and AD-MSC-treated a

287 ng's modulus was most associated with higher intimal thickness but not medial or adventitial thicknes

288 point included change in first-year maximal intimal thickness by intravascular ultrasound, a recogni

289 In contrast, maximum intimal thickness did not predict long-term mortality.

290 Morphometry showed that intimal thickness increased by 26% via carotid branch li

291 Correlation of LGE with maximal intimal thickness using the Pearson coefficient was 0.80

292 The mean (SD) 12-month increase in maximal intimal thickness was 0.03 (0.05) mm with everolimus 1.5

293 protein-coupled receptor 180, also known as intimal thickness-related receptor (ITR).

294 ES coating is distributed in the surrounding intimal tissue over the course of several weeks.

295 Exposure of intimal tissue to drug revealed differential distributio

296 aortic segments revealed the distribution of intimal TLR2 expression and the atheroprotective outcome

297 l-known inflammatory reactions consequent to intimal trapping and oxidation of LDL.

298 Change in intimal volume greater than the median and vascular remo

299 tendency for greater intimal growth (percent intimal volume: 9.2 +/- 9.3% vs. 4.4 +/- 5.3%; p = 0.07)

300 EBP50 is up-regulated in intimal VSMC following endoluminal injury and promotes n