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1 iders (91.3%) and 86 bicyclists (69.4%) were intoxicated.
2 milarly intoxicated than if tested while not intoxicated.
3 nly apparent if the animals are tested while intoxicated.
4 grade memory task of alcohol impairment when intoxicated.
5 lls are not able to take up TpeL and are not intoxicated.
6 tic brain-injured (TBI) patients are alcohol intoxicated.
14 e drinking, physical fighting, driving while intoxicated, and alcohol-related health problems and wer
15 or driver age, sex, history of driving while intoxicated, and survival status, implementation of the
16 fects and clinical presentation if abused or intoxicated, and to know what anesthetic options would b
17 clists, did not use helmets, were more often intoxicated, and were more often injured during nighttim
23 ects on vulnerable neurons or mediated by GA-intoxicated astrocytes that fail to support neuron funct
26 0 adults who had blood alcohol measured were intoxicated (blood alcohol content >22 mmol/L [100 mg/dL
38 ating that BFT did not injure HT29/C1 cells, intoxicated cells exhibited regulatory volume decrease,
39 capacity of Stxs to alter gene expression in intoxicated cells have been limited to individual genes.
40 p1-induced mitochondrial fission within VacA-intoxicated cells inhibited the activation of the proapo
42 e the mammalian cell, ExoU rapidly lyses the intoxicated cells via its phospholipase A(2) (PLA(2)) ac
44 sing depolarisation of the inner membrane in intoxicated cells, together with increased outer membran
50 , which inhibits the accumulation of cAMP in intoxicated cultured cells, significantly decreases the
51 1-556) plus anthrax toxin protective antigen intoxicated cultured mammalian cells and caused actin re
52 re significantly reduced or abolished in Stx-intoxicated D-THP-1 cells in which the expression of NLR
53 involvement in risky driving, riding with an intoxicated driver and being taken advantage of sexually
54 to determine probable cause and to arrest an intoxicated driver at the scene, the results are prelimi
56 astrocytes in the hippocampus of 26 lethally intoxicated drug addicts and 35 matched controls are des
58 s, and improved locomotor activities in MPTP-intoxicated Gfaf(cre) mice, but not Gdnf( astro) mice la
64 ifetime ounces of alcohol consumed and times intoxicated in lifetime estimated at visits 1 week or mo
65 2 are activated in vivo in the colon of Stx2-intoxicated infant rabbits, a model in which Stx2 induce
66 SDL), information learned while an animal is intoxicated is recalled more effectively when the subjec
67 abrogates IL-1beta and IL-18 secretion by DT-intoxicated keratinocytes, while ZAKalpha deletion or in
68 CBD treatment significantly improved ethanol-intoxicated larval survival rate by 40% and also improve
69 ght percent of trespassers were killed while intoxicated (median alcohol level, 56 mmol/L [260 mg/dL]
70 ary bacterial burden was observed in alcohol-intoxicated mice at 16 and 24 h and was associated with
72 etected Stx2a in kidney sections from orally intoxicated mice in the same region as the epithelial ce
75 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated mice where it inhibits parkin through tyrosi
76 istopathology, and full protection of orally intoxicated mice with monoclonal antibody (MAb) 11E10 di
78 s and protected dopaminergic neurons in MPTP-intoxicated mice, but at levels less than simvastatin.
99 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-intoxicated monkey model to further elucidate the nature
100 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated monkeys (when motor symptoms are less appare
101 rior olive neurons obtained from chronically intoxicated monkeys revealed a significant up-regulation
102 only used antimalarial agent, endows anthrax-intoxicated murine peritoneal macrophages with a 50% and
106 enzimidazoles, those who were acutely ill or intoxicated, or those reporting treatment within the pre
107 chemical and histological parameters in mice intoxicated orally versus intraperitoneally with Stx2a.
112 ne clearance based on a normal CT scan among intoxicated patients with no gross motor deficits appear
113 en; mean age, 40.3 years), 13 age-matched CO-intoxicated patients without parkinsonism, and 13 age-ma
114 the cervical spine should not be cleared in intoxicated patients, resulting in prolonged immobilizat
118 ompare the unadjusted mortality of the total intoxicated population and for specific intoxication sub
124 3-beta-glucanase mRNA was up-regulated in Al-intoxicated roots, with highest expression after 12 h.
125 ists that rescue neurotransmission in BoNT/A-intoxicated synapses provides compelling evidence for po
127 gans undergoes olfactory learning (OL) while intoxicated, the learning becomes state dependent such t
128 ocations of the individual subunits in cells intoxicated, under various conditions, with hybrid heter
129 ith toxins) and endogeneous (serum from mice intoxicated via oral, intranasal, and intravenous routes
132 nfers significant survival benefits for mice intoxicated with alpha-toxin or wSP in both therapeutic
134 odulate enterotoxin expression because cells intoxicated with heat-labile toxin overproduce and relea
136 We observed similar results in aged monkeys intoxicated with MPTP: they developed severe DOPA-respon
138 ted with Stx1a behave differently than those intoxicated with Stx2 subtypes: cells challenged with St
139 bunit delayed the mean time to death of mice intoxicated with Stx2a and reduced the cytotoxic effect