ライフサイエンスコーパス: intra-abdominal abscess

1 ns for diagnostic imaging of suspected acute intra-abdominal abscess.

2 x inflammation did LA significantly increase intra-abdominal abscesses.

3 ed with their ability to induce experimental intra-abdominal abscesses.

4 ortant virulence factors in the formation of intra-abdominal abscesses.

5 and are both able to induce the formation of intra-abdominal abscesses.

6 s the anaerobe most frequently isolated from intra-abdominal abscesses.

7 s included three anastomotic leaks and three intra-abdominal abscesses.

8 roparesis, intra-abdominal fluid collection, intra-abdominal abscess (10% vs 25%, P = 0.027), severe

9 icture was 10.3%, wound dehiscence 4.4%, and intra-abdominal abscess 5.8%, and did not significantly

10 The third complication was an intra-abdominal abscess after a TV appendectomy.

11 ving the enteric anastomosis site, including intra-abdominal abscess and enterocutaneous fistula form

12 Intra-abdominal abscess and higher blood loss decreased

13 linical symptoms and signs of peritonitis or intra-abdominal abscess and isolation of Candida species

14 s, and postoperative complications including intra-abdominal abscess and pancreatic fistula (all P <

15 E ADVICE 6: In patients with IBD who have an intra-abdominal abscess and/or phlegmonous inflammation

16 e risk of penetrating complications (such as intra-abdominal abscesses and fistulae), although more t

17 ll response necessary for the development of intra-abdominal abscesses and underscore the role of IL-

18 sible hole, diffuse fibrinopurulent exudate, intra-abdominal abscess, and extraluminal fecalith were

19 Pneumonia, intra-abdominal abscess, and sepsis were the most common

20 RIM101 contributes to persistence within intra-abdominal abscesses, at least in part through acti

21 OR], 0.93; 95% CI, 0.38-2.32; P = .88) or on intra-abdominal abscess development (OR, 0.89; 95% CI, 0

22 .093), postoperative hemorrhage (P = 0.174), intra-abdominal abscess formation (P = 0.199), biliary l

23 Intra-abdominal abscess formation was more common follow

24 stoperative pyrexia, ileus, wound infection, intra-abdominal abscess formation, operative time, and p

25 , and secondary outcome was the incidence of intra-abdominal abscess formation.

26 sus no irrigation (NI) reduces postoperative intra-abdominal abscess (IAA) in children with perforate

27 interval [UI] 34.8-36.6) of 10 175 patients, intra-abdominal abscess in 1619 (18.3%, 17.5-19.1) of 88

28 management included CT-guided drainage of an intra-abdominal abscess in 23 patients, and total parent

29 ppendicitis (PA) is the most common cause of intra-abdominal abscess in children and is associated wi

30 There were no intra-abdominal abscess in either groups.

31 vitro and in vivo they were attenuated in an intra-abdominal abscess infection model.

32 s necessary for maximal virulence in a mouse intra-abdominal abscess model.

33 after 90 days included wound infections and intra-abdominal abscess (n = 75) and failure to thrive (

34 ted (ie, percutaneous drainage of associated intra-abdominal abscess or colon resection).

35 There were no intra-abdominal abscesses or other major complications a

36 r IBD recurrence (n = 66, 12%) or because of intra-abdominal-abscess or fistula (n = 93, 16%).

37 t colonic procedures, OR = 1.5), presence of intra-abdominal abscess (OR = 5.0) or enteric fistula (O

38 (OR, 4.90; 95% CI, 1.41-17.06; P = .01) and intra-abdominal abscess (OR, 7.46; 95% CI, 1.65-33.66; P

39 ileostomy closure (6.5%), trauma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).

40 Terminal ileal chronic perforation with intra-abdominal abscess, peritoneal space to rectal and

41 With CPG-directed care, intra-abdominal abscess rate decreased from 0.24 to 0.10

42 inal COS (12): mortality, bowel obstruction, intra-abdominal abscess, recurrent appendicitis, complic

43 ndpoints were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital

44 f surgery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.

45 nly isolated organism from clinical cases of intra-abdominal abscesses, suggesting its potential to i

46 c fluid collections, one pseudocyst, and one intra-abdominal abscess; two donors underwent reoperatio

47 rapeutic antibiotics, and the development of intra-abdominal abscess was significantly lower in patie

48 Patients who had an anastomotic leak or intra-abdominal abscess were included in the infection g

49 ith Bacteroides fragilis is the formation of intra-abdominal abscesses, which are induced by the caps

50 are mortality and postoperative morbidities (intra-abdominal abscess, wound dehiscence, and intestina