ライフサイエンスコーパス: intracarotid

1 ere randomized to receive bolus injection of intracarotid (3-5 mg) or intravenous (10-20 mg) 1% propo

2 s, activation of carotid chemoreceptors with intracarotid administration of 100 nmol sodium cyanide r

3 -1 (HO-1) was studied in the rat brain after intracarotid administration of hyperosmolar mannitol.

4 rent oHSV variants (MSC-oHSV) and found that intracarotid administration of MSC-oHSV, but not of puri

5 Intracarotid administration of NAC resulted in high deli

6 reduce cellular glutathione levels prior to intracarotid alkylator administration.

7 out interfering with the CNS cytotoxicity of intracarotid alkylators, even after BSO depletion of CNS

8 al confrontational naming activation with an intracarotid amobarbital (Amytal) injection procedure (I

9 Language lateralization testing by intracarotid amobarbital injection showed no evidence of

10 aging can become a viable alternative to the intracarotid amobarbital procedure (IAP), the current go

11 ion and BRS before and during left and right intracarotid amobarbital procedure (IAP).

12 e focal epilepsies against the results of an intracarotid amobarbital procedure (IAP).

13 Functional MRI (fMRI) and the intracarotid amobarbital test (IAT) are generally congru

14 had bilateral language representation by the intracarotid amobarbital test.

15 Intracarotid amobarbital testing (IAT) is currently the

16 Memory profiles were obtained from intracarotid amobarbital testing and non-invasive verbal

17 significantly with memory lateralization by intracarotid amobarbital testing.

18 antly reduced declarative memory capacities (intracarotid amobarbital testing: P < 0.001; verbal memo

19 ing form intracranial electrodes, use of the intracarotid amobarbitol perfusion test, and the role of

20 xplored as alternatives to the more invasive intracarotid amytal procedure.

21 Many studies have compared fMRI with the intracarotid amytal test for establishing language domin

22 itive mapping and may be able to replace the intracarotid amytal test for language and memory lateral

23 Intracarotid arterial hyperosmolar mannitol (ICAHM) bloo

24 tes of administration, such as craniotomy or intracarotid arterial infusion of noxious agents causing

25 ments were undertaken to investigate whether intracarotid arterial injection of CsA reduces brain dam

26 A recent study showed that a single intracarotid arterial injection of cyclosporin A (CsA) c

27 al FMT signal was disrupted after unilateral intracarotid artery (ICA) MPTP infusion which reduced ip

28 C6 tumors were significantly increased after intracarotid bradykinin infusion (2.8- and 2.2-fold, res

29 sed permeability across the BTB, compared to intracarotid bradykinin infusion alone.

30 in tumors and normal brain, with or without intracarotid bradykinin infusion, using RIA.

31 Intracarotid cold saline infusion (ICSI) is potentially

32 ing responses of the carotid sinus nerves to intracarotid cyanide.

33 Intracarotid fluorescein isothiocyanate-BSA infusion was

34 vate their sympathetic nervous system during intracarotid glucose infusion but their brains are gener

35 Here, 1 h intracarotid glucose infusions (4 mg/kg/min) selectively

36 lar infusions of mannitol, 60 min unilateral intracarotid glucose infusions at 4 mg/kg/min in awake r

37 Intracarotid glucose infusions stimulated c-fos expressi

38 lucose and the insulin secretory response to intracarotid glucose injection are blunted in TRPC3-defi

39 out 10% of PVN neurons with FLI activated by intracarotid glucose.

40 PA administration were used: i.p. injection, intracarotid (i.c.) injection [with and without blood-br

41 apture therapy could be enhanced by means of intracarotid (i.c.) injection of sodium borocaptate (BSH

42 However, when given by the intracarotid (i.c.) route after only 5 min of recirculat

43 changes in tissue NADH concentrations, after intracarotid (IC) infusion of equal volumes of normal sa

44 A single unilateral intracarotid infusion of 1-methyl-4-phenyl-1,2,3,6-tetra

45 Intracarotid infusion of ALD-401 does not lead to clinic

46 7 days after stroke followed 2 days later by intracarotid infusion of ALD-401 versus sham harvest and

47 n brain tumor microvessel permeability after intracarotid infusion of bradykinin in the RG2 rat gliom

48 Intracarotid infusion of bradykinin selectively increase

49 Intracarotid infusion of bradykinin selectively increase

50 Intracarotid infusion of bradykinin selectively increase

51 The coupled intracarotid infusion of mannitol and CsA (10 mg/kg) was

52 mg/kg i.v.), administered following rostral intracarotid infusion of mannitol to disrupt the blood-b

53 ded before and for 4 hours after a 20-minute intracarotid infusion of PACAP-38 (1 ug/ml/kg, 10 ug/ml/

54 ucose were more variable and widespread than intracarotid infusions.

55 neurons displayed an increased discharge to intracarotid injection of both hypertonic NaCl and AngII

56 in vivo single-unit recordings revealed that intracarotid injection of hypertonic NaCl produced a con

57 a nigra of monkeys 4 days after a unilateral intracarotid injection of N-methyl-4-phenyl-1,2,3,6-tetr

58 mma-MSH (1.25 nmol) are not inhibited by the intracarotid injection of SHU9119 (1.25-12.5 nmol) or th

59 ent-derived brain metastases via an improved intracarotid injection protocol that permits tumor cell

60 in unlesioned and MPTP-lesioned (unilateral intracarotid injection) rhesus monkeys following an intr

61 In contrast, intracarotid injections of fibroblasts or U87 glioma cel

62 yes of isoflurane-anesthetized piglets using intracarotid injections of sodium fluorescein.

63 Intracarotid low dose bradykinin infusion can selectivel

64 avenous CsA dose, or by osmotic opening with intracarotid mannitol.

65 ce bearing intracranial xenografts and in an intracarotid mouse model of multiple brain metastases.

66 Intracarotid or intracerebroventricular infusion of hype

67 However, intracarotid perfusion of insulin induced the phosphoryl

68 Using brain cerebral intracarotid perfusion, we found that the transport rate

69 f 13 computed tomography angiography derived intracarotid plaque subcomponents (eg, lipid, intraplaqu

70 inistered at the time of reperfusion via the intracarotid route to maximize their localization in the

71 ffects, which are much more pronounced after intracarotid than after intravenous administration.