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1  NPE cells was used to record changes in the intracellular calcium concentration.
2 letely antagonized E2-induced attenuation of intracellular calcium concentration.
3 type switching in response to alterations in intracellular calcium concentration.
4 ed the excitotoxic glutamate-induced rise in intracellular calcium concentration.
5 ential and corresponding oscillations of the intracellular calcium concentration.
6 inase (PI3K) or MAPK signaling or increasing intracellular calcium concentration.
7 termined by the magnitude of the increase in intracellular calcium concentration.
8 calcineurin, which are both dependent on the intracellular calcium concentration.
9 ertussis toxin-sensitive pathway to increase intracellular calcium concentration.
10 se in both inositol phosphate production and intracellular calcium concentration.
11 isomer, NPS S-467, was not active in raising intracellular calcium concentration.
12 sed F-actin content, and increased the basal intracellular calcium concentration.
13 ellular calcium concentration with increased intracellular calcium concentration.
14 he transformation of membrane potential into intracellular calcium concentration.
15 -activated calcium channels and increase the intracellular calcium concentration.
16 f depolarization were mediated by increasing intracellular calcium concentration.
17 phospholipids and is regulated by changes in intracellular calcium concentration.
18 ed rises in inositol-1,4,5-trisphosphate and intracellular calcium concentration.
19 of OHCs which has been reported to depend on intracellular calcium concentration.
20 g thin filaments triggered by an increase in intracellular calcium concentration.
21  calcium retention with Delta160E may affect intracellular calcium concentration.
22 thymocyte exhibited persistent elevations in intracellular calcium concentration.
23 e, there was a 65% increase in the mean free intracellular calcium concentration.
24 nogenesis is regulated in part by an optimal intracellular calcium concentration.
25 signalling networks responsive to changes in intracellular calcium concentration.
26 ctivation of T cells, and Akt, and increased intracellular calcium concentration.
27  activate phospholipase C beta2 and increase intracellular calcium concentration.
28 , in parallel with their ability to increase intracellular calcium concentration.
29 to monitor the effect of CB(1) activation on intracellular calcium concentration.
30 gargin treatment, which results in increased intracellular calcium concentrations.
31 t CNP attenuates AVP-dependent elevations in intracellular calcium concentrations.
32 tained, but moderate, elevations of the free intracellular calcium concentrations.
33 operoxides that coincided wih an increase in intracellular calcium concentrations.
34 bitors and is not mediated by an increase in intracellular calcium concentrations.
35  that are activated in response to increased intracellular calcium concentrations.
36 C terminus on 1) UTP-stimulated increases in intracellular calcium concentration, 2) homologous desen
37 cant and concentration-dependent increase in intracellular calcium concentration, an effect that was
38 om or less in diameter caused rapid rises in intracellular calcium concentration, an effect that was
39           Similarly, SP-induced increases in intracellular calcium concentration and actomyosin stres
40 t the level of transcription by increases in intracellular calcium concentration and by the calcium-a
41 study was performed to determine the role of intracellular calcium concentration and calpain activity
42 beta occur immediately, including changes in intracellular calcium concentration and changes in membr
43  blocked spontaneous correlated increases in intracellular calcium concentration and compound postsyn
44 t that fails to bind Raf, potently increases intracellular calcium concentration and cytokine product
45  that the sigmoidal relationship between the intracellular calcium concentration and force production
46 of extracellular calcium with an increase of intracellular calcium concentration and inositol trispho
47 ted that hemichannel activity depends on the intracellular calcium concentration and is associated wi
48 the myofilament and reducing the affinity to intracellular calcium concentration and overall prolongi
49 h recombinant core and NS3 protein increased intracellular calcium concentration and reactive oxygen
50 olene and BAPTA-AM prevented the increase in intracellular calcium concentration and reduced cell rou
51         TCR engagement led to an increase in intracellular calcium concentration and the dissociation
52 iting concentrations of glucose increase the intracellular calcium concentration and the frequency of
53 the absence of GTP, the relationship between intracellular calcium concentration and the maximum rate
54 e data support the hypothesis that increased intracellular calcium concentrations and a diminished ca
55 ond to mechanical stimulation with increased intracellular calcium concentrations and increased inwar
56  by thapsigargin and ionomycin (that elevate intracellular calcium concentrations) and mutations in P
57 ed with activation of Vav-1, increase of the intracellular calcium concentration, and nuclear translo
58 yte fractional shortening without increasing intracellular calcium concentrations, and the biological
59               In most cell types, changes in intracellular calcium concentrations are sensed by calmo
60 f interest--spike trains and/or time varying intracellular calcium concentrations--are hidden.
61 ling to 0 degrees C evoked a 40% increase in intracellular calcium concentration as determined by liv
62 osphorylation of TCRzeta, ZAP70, and LAT and intracellular calcium concentration, as well as IL-2 gen
63  mechanotransduction is often an increase in intracellular calcium concentration associated with intr
64 uscle cell membrane, a transient increase of intracellular calcium concentration, binding of calcium
65   Naive thymocytes were highly motile at low intracellular calcium concentrations, but during positiv
66 filament, but inhibits contractility at high intracellular calcium concentration by disrupting the ac
67           This suggests that the increase in intracellular calcium concentration by these agents is d
68 rocess was partially reversed by raising the intracellular calcium concentration by use of the ionoph
69  fluorescent spectroscopic determinations of intracellular calcium concentrations (Ca(i)) and the rat
70 sue suggest that membrane voltage (V(m)) and intracellular calcium concentrations (Ca) become dissoci
71 POINTS: For the heart to function as a pump, intracellular calcium concentration ([Ca(2+) ]i ) must i
72  of AE that directly evaluate alterations in intracellular calcium concentration ([Ca(2+)](i)) and Ca
73           The relationship between increased intracellular calcium concentration ([Ca(2+)](i)) and ch
74 ulmonary arterial smooth muscle cell (PASMC) intracellular calcium concentration ([Ca(2+)](i)) and pH
75 c Ca(2+) confirmed the transient rise in the intracellular calcium concentration ([Ca(2+)](i)) during
76 ng this activation results in an increase in intracellular calcium concentration ([Ca(2+)](i)) follow
77                                          The intracellular calcium concentration ([Ca(2+)](i)) has be
78 hat in rodents, neuronal activity raised the intracellular calcium concentration ([Ca(2+)](i)) in ast
79 nvestigated the role of a Ca(2+) channel and intracellular calcium concentration ([Ca(2+)](i)) in osm
80 neuron axon we found that activity-dependent intracellular calcium concentration ([Ca(2+)](i)) in the
81 ates from cytosol to membrane in response to intracellular calcium concentration ([Ca(2+)](i)) increa
82 trocytes display excitability in the form of intracellular calcium concentration ([Ca(2+)](i)) increa
83                                              Intracellular calcium concentration ([Ca(2+)](i)) is a k
84 ases, which were synchronized with a rise in intracellular calcium concentration ([Ca(2+)](i)) near t
85  changes in ciliary beat frequency (CBF) and intracellular calcium concentration ([Ca(2+)](i)) of rab
86 PC6-5 cascade causes a prolonged increase in intracellular calcium concentration ([Ca(2+)](i)) that i
87 po) stimulates a significant increase in the intracellular calcium concentration ([Ca(2+)](i)) throug
88 trophysiological methods and measurements of intracellular calcium concentration ([Ca(2+)](i)) to sho
89                                              Intracellular calcium concentration ([Ca(2+)](i)) was ex
90                                              Intracellular calcium concentration ([Ca(2+)](i)) was me
91                                              Intracellular calcium concentration ([Ca(2+)](i)) was me
92 ellular-dependent early increase (30 min) in intracellular calcium concentration ([Ca(2+)](i)), follo
93                       Small increases in the intracellular calcium concentration ([Ca(2+)](i)), incre
94 s trigger an early and transient increase of intracellular calcium concentration ([Ca(2+)](i)), requi
95 em is associated with stereotypic changes in intracellular calcium concentration ([Ca(2+)](i)), yet t
96 ive oxygen species (ROS)-evoked and pericyte intracellular calcium concentration ([Ca(2+)](i))-mediat
97 with TNF induces an abnormal increase in the intracellular calcium concentration ([Ca(2+)](i)).
98 ypes, these parameters are controlled by the intracellular calcium concentration ([Ca(2+)](i)).
99                    FSS rapidly increases the intracellular calcium concentration ([Ca(2+)]) and nitri
100 tes to skeletal muscle atrophy by increasing intracellular calcium concentration ([Ca(2+)]i) and enha
101 n above 0.03 mM leads to a rapid increase in intracellular calcium concentration ([Ca(2+)]i) and inos
102                      Transient elevations in intracellular calcium concentration ([Ca(2+)]i) and migr
103                       Sustained increases in intracellular calcium concentration ([Ca(2+)]i) are pres
104 , a specific alpha7 nAChR agonist, increases intracellular calcium concentration ([Ca(2+)]i) mainly r
105 ated influences of transmembrane voltage and intracellular calcium concentration ([Ca(2+)]i) that gat
106   In resistance arteries, coupling a rise of intracellular calcium concentration ([Ca(2+)]i) to endot
107 ing combined with indo-1 measurement of free intracellular calcium concentration ([Ca(2+)]i) was used
108 his was followed by a sustained elevation of intracellular calcium concentration ([Ca(2+)]i) which co
109 blasts were used for microinjection and free intracellular calcium concentration ([Ca(i)]) was measur
110          This study investigated the role of intracellular calcium concentration ([Ca]i) as a possibl
111 tes, as indicated by increases in astrocytic intracellular calcium concentrations ([Ca(2)(+)](i)).
112 ating is dictated by membrane voltage (Vm ), intracellular calcium concentrations ([Ca(2+) ]i ) and e
113 tamate receptors induced a rapid increase in intracellular calcium concentrations ([Ca(2+)](i)) and a
114  analyze spontaneous dynamic fluctuations in intracellular calcium concentrations ([Ca(2+)](i)) in sm
115 es the cell membrane and blunts increases in intracellular calcium concentrations ([Ca(2+)](i)) simil
116 n neurons enabled simultaneous monitoring of intracellular calcium concentrations ([Ca(2+)]i) in mult
117 in filaments are activated by an increase in intracellular calcium concentration [Ca(2+)](i) and by m
118               TARC, MDC, and SDF-1 increased intracellular calcium concentrations [Ca(2+)](i) when co
119 lowed, within 205+/-34 seconds, by increased intracellular calcium concentration, [Ca(2+)](i).
120                                         High intracellular calcium concentration, [Ca]i, lowered prop
121 implicated its activity in the regulation of intracellular calcium concentration ([Ca2+](i)) and secr
122 n analog iloprost alone had no effect on the intracellular calcium concentration ([Ca2+](i)), it did
123 ngs showed that BDNF elicited an increase in intracellular calcium concentration ([Ca2+]c).
124 n of Ca2+-signaling pathways, the effects on intracellular calcium concentration ([Ca2+]i) after expo
125  current, which is induced by an increase in intracellular calcium concentration ([Ca2+]i) and is its
126                                              Intracellular calcium concentration ([Ca2+]i) and Na(+)-
127  showed spontaneous rhythmic oscillations in intracellular calcium concentration ([Ca2+]i) and sponta
128            Tyrosine kinases indirectly raise intracellular calcium concentration ([Ca2+]i) by activat
129                                 Elevation of intracellular calcium concentration ([Ca2+]i) by applica
130 was measured using micropuncture techniques, intracellular calcium concentration ([Ca2+]i) by fura-2
131 toring the effects of NGF on the increase in intracellular calcium concentration ([Ca2+]i) following
132 ed the relations between isometric force and intracellular calcium concentration ([Ca2+]i) in fibrobl
133         The relationship between tension and intracellular calcium concentration ([Ca2+]i) in intact
134 record changes in the membrane potential and intracellular calcium concentration ([Ca2+]i) in SCN neu
135                     The effect of albumin on intracellular calcium concentration ([Ca2+]i) in single
136                            In response to an intracellular calcium concentration ([Ca2+]i) increase,
137                                              Intracellular calcium concentration ([Ca2+]i) measuremen
138                                          The intracellular calcium concentration ([Ca2+]i) of growth
139  changes in ciliary beat frequency (CBF) and intracellular calcium concentration ([Ca2+]i) of rabbit
140 microg/ml) dramatically enhanced BCR-induced intracellular calcium concentration ([Ca2+]i) responses
141 e expressed in brain endothelium and mediate intracellular calcium concentration ([Ca2+]i) signaling,
142 sed to determine the sources for the rise in intracellular calcium concentration ([Ca2+]i) that occur
143 T lymphocytes results in a rapid increase in intracellular calcium concentration ([Ca2+]i) that paral
144                                  We measured intracellular calcium concentration ([Ca2+]i) transients
145 troprusside (SNP) on intracellular pH (pHi), intracellular calcium concentration ([Ca2+]i) transients
146  contractility is mediated by a reduction in intracellular calcium concentration ([Ca2+]i) via inhibi
147 activate any membrane current in cells where intracellular calcium concentration ([Ca2+]i) was buffer
148                                              Intracellular calcium concentration ([Ca2+]i) was determ
149                                          The intracellular calcium concentration ([Ca2+]i) was measur
150  After addition of lipopolysaccharide (LPS), intracellular calcium concentration ([Ca2+]i) was measur
151                                          The intracellular calcium concentration ([Ca2+]i) was monito
152                                              Intracellular calcium concentration ([Ca2+]i) was monito
153                                     Decay in intracellular calcium concentration ([Ca2+]i) was record
154 rane repolarisation, even though the average intracellular calcium concentration ([Ca2+]i) was still
155 ing combined with indo-l measurement of free intracellular calcium concentration ([Ca2+]i) was used t
156  (PLCgamma1) and corresponding elevations in intracellular calcium concentration ([Ca2+]i) were intac
157 ssing of bursts during up-states, changes in intracellular calcium concentration ([Ca2+]i) were measu
158  of nontransgenic recipients, and changes in intracellular calcium concentration ([Ca2+]i) were monit
159        Our results indicate that the rise in intracellular calcium concentration ([Ca2+]i) which acco
160 f ECs inhibited agonist-induced increases in intracellular calcium concentration ([Ca2+]i), in both E
161 e inhibition of oxidative phosphorylation on intracellular calcium concentration ([Ca2+]i), phosphory
162 chanical stimuli with transient increases in intracellular calcium concentration ([Ca2+]i).
163 could result from a progressive elevation of intracellular calcium concentration ([Ca2+]i).
164                         Quantitation of free intracellular calcium concentrations ([Ca2+]i) after mit
165                                  The minimum intracellular calcium concentrations ([Ca2+]i) for trigg
166 - current (I(Cl(Ca))) evoked by adding fixed intracellular calcium concentrations ([Ca2+]i) to the pi
167                                         High intracellular calcium concentrations ([Ca2+]i; 10-1000 m
168 the fluorescent dye fluo-3 AM and changes in intracellular calcium concentration [Ca2+]i were analyze
169 a2+ exchanger, I(NaCa), which depends on the intracellular calcium concentration [Ca2+]i.
170                     The relationship between intracellular calcium concentration, [Ca2+]i, and fixed-
171                                   Changes in intracellular calcium concentration, [Ca2+]i, modulate t
172 virus for cell fusion induced an increase in intracellular calcium concentration, causing premature o
173 vents in eosinophils, including increases in intracellular calcium concentration, cell surface expres
174  determine that a large biphasic increase in intracellular calcium concentration, characterized by re
175 dary to reduced ROS levels and reduced basal intracellular calcium concentration compared with mock c
176                                   Changes in intracellular calcium concentration (DeltaCa(i)2+) induc
177 2 gene expression was dependent on a rise in intracellular calcium concentration derived from extrace
178 e propensity to produce a global rise in the intracellular calcium concentration differed among the v
179 lasma membrane in response to DAG at a basal intracellular calcium concentration due to the inaccessi
180 shing the SR calcium store, the evolution of intracellular calcium concentration during a train of lo
181  changes in caldesmon phosphorylation and/or intracellular calcium concentrations during signal trans
182 and are instrumental in generating sustained intracellular calcium concentration elevations that are
183 ccur with essentially equal magnitude at all intracellular calcium concentrations examined (range, 0-
184      Cold preservation caused an increase in intracellular calcium concentration first detected at 1
185 s that exhibit activity-related increases in intracellular calcium concentration (FLiCRE).
186  1990s that astrocytes undergo elevations in intracellular calcium concentration following activation
187 mmature B cells display greater increases in intracellular calcium concentrations following Ag stimul
188 was reduced dose dependently by increases in intracellular calcium concentration from 0.1 to 0.5 micr
189 echanical flow stimulation by changing their intracellular calcium concentration in a manner similar
190 ke activity which leads to a greater rise in intracellular calcium concentration in aging than that i
191  the hyperpolarization-activated current and intracellular calcium concentration in both normal contr
192  fura-2 fluorescence revealed an increase of intracellular calcium concentration in cells when challe
193         Both SPC and LPC induce increases in intracellular calcium concentration in GPR4-, but not ve
194 S1P (10 microM) also stimulated increases in intracellular calcium concentration in HTM cells.
195 strated that as in eukaryotic organisms, the intracellular calcium concentration in prokaryotes is ti
196              Eosinophils showed an increased intracellular calcium concentration in response to Alter
197                                        Basal intracellular calcium concentration in single DCT cells
198  since it failed to stimulate an increase in intracellular calcium concentration in the CCR5 transfec
199 thick filament stress but are independent of intracellular calcium concentration in the physiological
200 phin, contributing to abnormal regulation of intracellular calcium concentrations in dystrophic muscl
201                      Fluorescence imaging of intracellular calcium concentrations in live RA FLS stim
202 ted in a 0.56 micromol/L shift toward higher intracellular calcium concentrations in nonfailing myoca
203 h glutamate, NMDA, or kainate (KA) increased intracellular calcium concentrations in RA FLS, demonstr
204 ined the role of nucleus isthmi in enhancing intracellular calcium concentrations in retinotectal fib
205  integrin receptors initiates an increase in intracellular calcium concentrations in T cells, potenti
206 al slices showed increased baseline and peak intracellular calcium concentrations in the zona glomeru
207 dicators Fura-2 and Fluo-3 we show that root intracellular calcium concentrations increase in respons
208                                          The intracellular calcium concentration increased significan
209 SEC to cold results sequentially in elevated intracellular calcium concentration, increased calpain a
210                  Subsequent to elevations in intracellular calcium concentrations induced by ionomyci
211           Activation of G2A by LPC increased intracellular calcium concentration, induced receptor in
212                   The subsequent increase in intracellular calcium concentration induces proteolytic
213 lcium sensors that transduce fluctuations in intracellular calcium concentrations into changes in mem
214 n is mediated by PKA and that an increase in intracellular calcium concentration is required for maxi
215 bit aberrant T cell expansion by maintaining intracellular calcium concentration levels low enough to
216 r-alpha, changes in osmolarity, elevation in intracellular calcium concentration, lysophosphatidic ac
217 cardial mechanisms: calcitropes, which alter intracellular calcium concentrations; myotropes, which a
218 ing large, transient, localized increases in intracellular calcium concentration near the calcium-con
219 ever, hVPLA2 induced neither the increase in intracellular calcium concentration nor cPLA2 phosphoryl
220  arginine deiminase activity depends on high intracellular calcium concentrations occurring in dying
221                             Furthermore, the intracellular calcium concentration of isolated neuroepi
222 chemia was insensitive to maneuvers altering intracellular calcium concentration or myofilament calci
223 n of mitochondrial complex I, an increase in intracellular calcium concentration, or formation of rea
224                  SignificanceOscillations in intracellular calcium concentration play an essential ro
225 nium, quinine and dextromethorphan increased intracellular calcium concentration preferentially in is
226  channels in the plasma membrane, and if the intracellular calcium concentration reaches a threshold,
227                                              Intracellular calcium concentrations regulate diverse ce
228  PIP2 and, in response to local increases in intracellular calcium concentration, release it to inter
229  forms of protein kinase C (PKC) and elevate intracellular calcium concentrations, respectively.
230 nce of 0.03 mM Ca2+, NPS R-467 increased the intracellular calcium concentration response in a concen
231 nor endothelin-1, both of which elevated the intracellular calcium concentration, restored insulin-st
232 ells in reduced calcium medium, and lowering intracellular calcium concentration, results in the loss
233                           In some cells, the intracellular calcium concentration rise produced by 50
234 nnel) underlies the sustained or oscillatory intracellular calcium concentration signal required for
235           DMS also inhibited the increase in intracellular calcium concentration stimulated by platel
236 ide to L929 cells caused rapid elevations in intracellular calcium concentration that were independen
237                    Apoptotic stimuli augment intracellular calcium concentration through inositol 1,4
238 NMDA receptor is modulated by changes in the intracellular calcium concentration, through activation
239 lex is a molecular switch that ties shifting intracellular calcium concentration to association and d
240 alcium channels, and ultimately elevates the intracellular calcium concentration to increase the rele
241 ts that neurons monitor their activity using intracellular calcium concentrations to regulate their i
242 AChRs), and our data suggest that changes in intracellular calcium concentrations triggered by nAChR
243                         Notably, the rise of intracellular calcium concentration upon immunoglobulin
244 embrane proteins is to measure the change in intracellular calcium concentration upon receptor stimul
245 ure for histamine release and for changes in intracellular calcium concentration using video imaging.
246 lular calcium release because no increase in intracellular calcium concentration was detected in resp
247         Also, the lysoPC-induced increase in intracellular calcium concentration was inhibited in ECs
248                                              Intracellular calcium concentration was measured by micr
249                                   Changes of intracellular calcium concentration were involved not on
250         These receptor-mediated increases in intracellular calcium concentration were measured by bot
251 e to 4-CMC or caffeine, similar increases in intracellular calcium concentration were observed in Sta
252                                   Changes in intracellular calcium concentration were quantitated by
253                                     Parasite intracellular calcium concentrations were altered by cha
254  was not affected but systolic and diastolic intracellular calcium concentrations were decreased and
255                   In the present study, free intracellular calcium concentrations were examined in re
256                                              Intracellular calcium concentrations were measured in is
257 such as tyrosine protein phosphorylation and intracellular calcium concentrations, were found to be i
258 andamide acting on CB(1) receptors increases intracellular calcium concentration when administered in
259 predicts that hyperexcited states cause high intracellular calcium concentrations, which could trigge
260 ory about reading neuronal information using intracellular calcium concentrations, which includes the
261  MIP-1beta induced a significant increase in intracellular calcium concentrations, which was blocked
262 n enhanced IL-6 secretion and an increase in intracellular calcium concentrations, which were depende
263 tin filaments with latrunculin A or reducing intracellular calcium concentration with BAPTA-AM.
264 ucagon-mediated increases in cAMP levels and intracellular calcium concentrations, with EC50 values n
265 al signals are transduced through changes in intracellular calcium concentrations, yet only a few cal

 
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