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1 tly triggered mobilization of Ca(2+) from an intracellular store.
2 release of calcium from ryanodine-sensitive intracellular stores.
3 d and/or RyR1-independent Ca(2)(+) leak from intracellular stores.
4 tivating K(+) current by Ca(2+) release from intracellular stores.
5 ay associated with rapid Ca(2+) release from intracellular stores.
6 ium channels or on calcium mobilization from intracellular stores.
7 d to the PLC pathway and Ca(2+) release from intracellular stores.
8 ed signaling upstream of Ca(2+) release from intracellular stores.
9 oval of extracellular Ca(2+) or depletion of intracellular stores.
10 t pathway) that stimulates Ca2+ release from intracellular stores.
11 hat the source of the Ca(2+) is release from intracellular stores.
12 channels, or mobilizing Ca(2+) release from intracellular stores.
13 4,5-triphosphate-stimulated Ca2+ efflux from intracellular stores.
14 nodine receptor-induced calcium release from intracellular stores.
15 but enhances stimulated Ca(2+) release from intracellular stores.
16 associated transient release of Ca(2+) from intracellular stores.
17 ulating factor induced BLyS release from the intracellular stores.
18 h Ca2+ influx, rather than Ca2+ release from intracellular stores.
19 2Y1 receptor-mediated release of Ca(2+) from intracellular stores.
20 (CRAC) channels following Ca2+ release from intracellular stores.
21 neurons and can mediate Ca(2+) release from intracellular stores.
22 elevation of cAMP or release of Ca(2+) from intracellular stores.
23 4,5-trisphospate (IP3) and release Ca2+ from intracellular stores.
24 provide counter ions for Ca(2+) handling in intracellular stores.
25 Ca2+ into the cytosol from extracellular or intracellular stores.
26 the apical membrane from latent preexisting intracellular stores.
27 (2+) despite the limited available Ca(2+) in intracellular stores.
28 zyme secretion and is largely mobilized from intracellular stores.
29 trisphosphate and the release of Ca(2+) from intracellular stores.
30 il surface was rapidly replaced by CD93 from intracellular stores.
31 cium, showing that WIN releases calcium from intracellular stores.
32 es a rise in cytosolic Ca(2+) from undefined intracellular stores.
33 2+ transients by returning cytosolic Ca2+ to intracellular stores.
34 on through TRPM8 channels or by release from intracellular stores.
35 n to control the rate of ER Ca(2+) leak from intracellular stores.
36 conductance through release of calcium from intracellular stores.
37 ing the MAG source, due to Ca2+ release from intracellular stores.
38 -trisphosphate and mobilization of Ca2+ from intracellular stores.
39 plasma membrane often releases calcium from intracellular stores.
40 ysis of drugs that prevent Ca2+ release from intracellular stores.
41 usion via a PMCA and Ca2+ transport from the intracellular stores.
42 phosphate, which causes release of Ca2+ from intracellular stores.
43 LTX(N4C) involves mobilization of Ca2+ from intracellular stores.
44 sitol 1,4,5-trisphosphate receptor-sensitive intracellular stores.
45 at the initial increase in [Ca2+](i) is from intracellular stores.
46 ion via a PMCA and Ca(2+) transport from the intracellular stores.
47 vation of IP(3) mediated Ca(2+) release from intracellular stores.
48 ch is mediated by the release of Ca(2+) from intracellular stores.
49 he level of G17-stimulated Ca2+ release from intracellular stores.
50 are a family of calcium release channels on intracellular stores.
51 (i) increase involves release of Ca(2+) from intracellular stores.
52 an signal by activating calcium release from intracellular stores.
53 kainic acid or mobilization of calcium from intracellular stores.
54 flux to the cytosol from the apoplast and/or intracellular stores.
55 n previously, to trigger Ca(2+) release from intracellular stores.
56 ther dependent on the release of Ca(2+) from intracellular stores.
57 by phospholipase C and release of Ca 2+ from intracellular stores.
58 nduced by IP(3)-mediated Ca(2+) release from intracellular stores.
59 mGluRs), and involved release of Ca(2+) from intracellular stores.
60 ough voltage-gated channels, or release from intracellular stores.
61 e cells is due to the release of Ca(2+) from intracellular stores.
62 igargin, suggesting it involves release from intracellular stores.
63 ombin-stimulated release of [Ca(2+)](i) from intracellular stores.
64 ch enhanced release of calcium from parasite intracellular stores.
65 c reticulum that allow efflux of Ca(2+) from intracellular stores.
66 pathway, and reduce the Ca(2+)content of the intracellular stores.
67 IP3 receptor-regulated calcium release from intracellular stores.
68 the release and sequestration of Ca(2+) from intracellular stores.
69 l in mouse microglia was due to release from intracellular stores.
70 onists through augmented Ca(2+) release from intracellular stores.
71 here they mediate the release of Ca(2+) from intracellular stores.
72 P3 receptors, and release of Ca(2+) from the intracellular stores.
73 flux and Ca(2+) -induced Ca(2+) release from intracellular stores.
74 llular Ca(2+) influx and Ca(2+) release from intracellular stores.
75 by Ca(2+)-induced Ca(2+) release (CICR) from intracellular stores.
76 1) receptors and Ca(2+) recruitment from the intracellular stores.
77 type VDCCs, or block of calcium release from intracellular stores.
78 it apoptosis and promote Ca(2+) release from intracellular stores.
79 ic channels that mediate Ca(2+) release from intracellular stores.
80 cium influx rather than calcium release from intracellular stores.
81 spholipase C and release of calcium from the intracellular stores.
82 ositol 1,4,5-trisphosphate (IP(3))-sensitive intracellular stores.
83 hose activation is dependent on depletion of intracellular stores.
84 lular Ca(2+) and trigger Ca(2+) release from intracellular stores.
85 +), but at a lower dose released Ca(2+) from intracellular stores.
86 ), critical signals for calcium release from intracellular stores.
87 ignaling mobilize peptide:MHC-II export from intracellular stores.
88 hrombin-induced release of calcium ions from intracellular stores.
89 s probably triggered by the depletion of the intracellular stores, a mechanism known as store-operate
90 schaemia: Ca(2+) influx, Ca(2+) release from intracellular stores, actin filament depolymerization, a
91 te hypoxia, by causing Ca2+ release from the intracellular stores, activates CCE in isolated canine P
92 CR to determine whether calcium release from intracellular stores affected action potential waveform,
93 ntaneous calcium (Ca(2+)) release (SCR) from intracellular stores after the end of a preceding action
94 2 activation and that release of Ca(2+) from intracellular stores alone, in the absence of extracellu
95 pulating calcium uptake into or release from intracellular stores also modulated the level of reverbe
96 phate production and release of calcium from intracellular stores also were augmented after myo-inosi
97 hat is shaped by the Ca(2+) release from the intracellular store and extracellular Ca(2+) influx.
99 TIM1, 2) are acting as sensors for Ca(2+) in intracellular stores and activate Orai channels at the p
100 phate (IP3)-mediated release of calcium from intracellular stores and activation of protein kinase C
101 ion, as cADPR regulates calcium release from intracellular stores and ADPR controls cation entry thro
102 s (and/or glia) to evoke Ca(2+) release from intracellular stores and an increase in ventilation that
104 activation-induced Ca(2+) mobilization from intracellular stores and Ca(2+) influx from the extracel
105 tor, which causes the release of Ca(2+) from intracellular stores and Ca(2+)-dependent activation of
106 induced Ca2+ transients involve release from intracellular stores and Ca2+ signaling is essential for
107 sible and were mediated by Ca2+ release from intracellular stores and calcineurin-mediated Kv2.1 deph
108 es release of calcium from the IP3-sensitive intracellular stores and calcium-calmodulin formation.
109 lifies the CRTH2-induced Ca(2+) release from intracellular stores and coincidentally forfeits its own
110 d the Ca(2+) content of ionomycin-releasable intracellular stores and decreased endoplasmic reticulum
111 l types are triggered by Ca(2+) release from intracellular stores and driven by store-operated Ca(2+)
112 cal agents that alter Ca2+ mobilization from intracellular stores and experiments involving injection
114 -entry process activated by the depletion of intracellular stores and has an important role in many c
115 estingly, BMP-2 induced calcium release from intracellular stores and increased calcineurin phosphata
116 pecific channels mediating Ca2+ release from intracellular stores and influx across the plasma membra
117 riant responsible for Ca2+ mobilization from intracellular stores and influx through voltage-gated Ca
119 is related to more efficient FV release from intracellular stores and microparticle production driven
120 tween the physiological release of Ca2+ from intracellular stores and opening of Ca2+ influx channels
121 esses essential for LTD-calcium release from intracellular stores and phosphatase activation-were abn
122 phate, leading to the release of Ca(2+) from intracellular stores and protein kinase C (PKC) activati
123 required to induce depletion of Ca(2+) from intracellular stores and provide insights into the abili
124 receptors leads both to calcium release from intracellular stores and to dendritic protein synthesis.
125 s of the BCM model, calcium as released from intracellular stores and triggered by M1 muscarinic acet
126 volved the release of Ca2+ from IP-sensitive intracellular stores and was expressed via the internali
127 clic ADP-ribose evoked a slow Ca2+ leak from intracellular stores and were able to increase the Ca2+-
128 bilization from caffeine/ryanodine-sensitive intracellular stores and were not due to inositol 1,4,5-
129 different Ca(2+) signatures from the Ca(2+) intracellular stores and whether the amplitude of Ca(2+)
130 he route of Ca(2+) entry (i.e., release from intracellular stores and/or influx across the plasma mem
131 ed by PLC activation, release of Ca(2+) from intracellular stores, and activation of store-operated C
132 and ryanodine receptors) release Ca(2+) from intracellular stores, and by depleting the stores trigge
134 el were [Ca(2+)] in the cytosol, [Ca(2+)] in intracellular stores, and smooth muscle membrane potenti
135 ase, PKA, CaMKII, and release of Ca(2+) from intracellular stores are necessary for modulation of GAB
136 problem concerns the mechanism by which the intracellular stores are refilled subsequent to depletio
138 ligand-gated channels that release Ca2+ from intracellular stores--are emerging as key sites for regu
139 rough gap junctions and calcium release from intracellular stores as mediators of collective gradient
140 sphosphate-mediated Ca(2+) mobilization from intracellular stores as well as decreased beta-cell Ca(2
141 affected the initial release of Ca(2+) from intracellular stores as well as sustained Ca(2+) levels.
142 +) influx for triggering Ca(2+) release from intracellular stores at presynaptic terminals during in
144 e lipo-oxygenase blockers released Ca2+ from intracellular stores but this was not associated with su
145 the release from or re-uptake of Ca(2+) into intracellular stores but, through protein kinase G, both
146 a(2+) transients were caused by release from intracellular stores, but depended strongly on store-ope
147 ateaus are not mediated by Ca2+ release from intracellular stores, but rather by an NMDA-dependent sm
148 ors (IP3Rs) release calcium ions, Ca2+, from intracellular stores, but their roles in mediating Ca2+
149 LR2 ligands stimulate release of Ca(2+) from intracellular stores by activating TLR2 phosphorylation
152 ) influx by ionomycin or Ca(2+) release from intracellular stores by thapsigargin alone failed to ind
154 nce of depolarization or Ca(2+) release from intracellular stores can drive neurotransmitter release
155 ed calcium channels (VGCCs) and release from intracellular stores, causing an increase in cAMP-respon
156 hydrolase (CEH) catalyzes the hydrolysis of intracellular stored cholesteryl esters (CEs) and thereb
157 They also suggest that Ca2+ release from intracellular stores contributes significantly to increa
158 PC5) and Na+/Ca2+ exchanger, indicating that intracellular store deficiency was compensated for by Ca
159 oplasmic reticulum Ca(2+) sensor that senses intracellular store depletion and migrates to plasma mem
160 osphate (NAADP)-mediated Ca(2+) release from intracellular stores during stimulus-secretion coupling
162 lux from extracellular space or release from intracellular stores, eliminates fast inactivation induc
163 cascades, resulting in calcium release from intracellular stores, ERK1/2 activation, and long term c
164 harmacologically induced Ca(2+) release from intracellular stores evoked a significant extracellular
165 s respond is through release of calcium from intracellular stores followed by store refilling from ex
166 ntracellular Ca2+ caused by its release from intracellular stores, followed by Ca2+ influx, possibly
167 cess is the excessive release of Ca(2+) from intracellular stores, followed by excessive entry of Ca(
168 calcium increase-the release of calcium from intracellular stores following activation of an IP3-depe
170 ar molecule, which is rapidly mobilized from intracellular stores following treatment with interferon
171 endocytic recycling compartment are critical intracellular stores for the rapid recycling of internal
172 eutrophils, both on the cell membrane and in intracellular stores; however, BLyS release from each of
173 h TZDs increased calcium concentrations from intracellular stores; however, only ciglitazone produced
174 However, the concentration of Ca(2+) in the intracellular stores (i.e., mitochondria and granules) w
175 in induces a modest increase in calcium from intracellular stores, IBTU was unable to block the respo
176 ation; (ii) BCR-mediated Ca(2+) release from intracellular stores; (iii) Ca(2+) entry from the extrac
177 mediated via the liberation of calcium from intracellular stores, implicating functional P2Y recepto
178 nist caffeine evoked Ca(2+) release from the intracellular store in activated T cells but not in rest
181 i) and the calcium that can be released from intracellular stores in HEp-2 or VAX-3 cells overexpress
183 a role for the release of calcium ions from intracellular stores in mediating spatially compartmenta
184 t demonstration of mobilization of Ca2+ from intracellular stores in neurons by depolarization withou
185 es: (i) both CPA and Thg release Ca(2+) from intracellular stores in P. falciparum parasites; (ii) Th
186 ld enhancement in the release of Ca(2+) from intracellular stores in response to agents that release
188 rkA mobilization to the plasma membrane from intracellular stores in response to proton challenge.
189 modulin (CaM) regulates calcium release from intracellular stores in skeletal muscle through its asso
191 The nicotine-induced release of Ca(2+) from intracellular stores in T cells did not require extracel
194 enerating metabolites that release Ca2+ from intracellular stores, including nicotinic acid adenine d
195 B.T1 signaling triggers calcium release from intracellular stores, increasing glucose-induced insulin
197 ncentration ([Ca(2+)]i) mainly released from intracellular stores, indicating that alpha7 nAChR is fu
198 ryotes, GAAPs regulate the Ca(2+) content of intracellular stores, inhibit apoptosis, and promote cel
199 presented that constitutive Ca(2+) flux from intracellular stores into mitochondria is required for b
200 e the rapid release of calcium (Ca(2+)) from intracellular stores into the cytosol, which is essentia
201 he hippocampus release calcium (Ca(2+)) from intracellular stores intrinsically and in response to ac
202 (V(m)) and the release of calcium (Ca) from intracellular stores is a crucial ingredient of heart fu
203 a2+-ATPase (SERCA)-mediated Ca2+ uptake into intracellular stores is also accelerated by PKC activati
206 a2+]i caused by the release from presynaptic intracellular stores is coincident with an enhancement o
207 rough voltage-gated calcium channels or from intracellular stores is critical for proper AChE inserti
208 n: an initial transient release of Ca2+ from intracellular stores is followed by a sustained phase of
212 little or opposite effects, but release from intracellular stores is required for maximal acceleratio
214 cells, receptor-induced Ca(2+) release from intracellular stores is usually accompanied by sustained
215 h Ca2+ influx, rather than Ca2+ release from intracellular stores, is essential for growth factor-ind
216 y frequency involves Ca(2+) recruitment from intracellular stores leading to increases in intracellul
217 ic receptor and stimulates Ca2+ release from intracellular stores, leading to the enhancement of syna
218 , in addition to gating calcium release from intracellular stores, mAChR activation facilitates volta
222 ets that could be manipulated to enhance the intracellular store of A3 and potentially enhance A3 ant
223 l array implicates mitochondria as the major intracellular store of Ca2+ involved in IR-evoked respon
224 e conditions, wild-type cells accumulated an intracellular store of Mg that supported growth under de
225 ells not only express NKp80 but also contain intracellular stores of AICL colocalizing with the Golgi
226 t, thapsigargin, is dependent on emptying of intracellular stores of Ca(2+) and the ensuing influx of
228 haemic sIPSCs were not affected by depleting intracellular stores of calcium or by blocking the neuro
229 icromol/L) caused profound Ca2+ release from intracellular stores of intact or permeabilized cells.
230 has been shown to trigger Ca2+ release from intracellular stores of invertebrate eggs and mammalian
233 human airway cell Duox activity by depleting intracellular stores of NADPH, as it generates intracell
234 nge of abilities to mobilize Ca(2+) from the intracellular stores of permeabilized hepatocytes and ar
235 ed an enhanced capacity to produce acid from intracellular stores of polysaccharides, could grow fast
239 ated in a convertase-independent manner from intracellular stores of the complement component C3.
240 clopiazonic acid, which mobilize Ca(2+) from intracellular stores of the endoplasmic reticulum, evoke
241 We found that dead cells not only released intracellular stores of uric acid but also produced it i
243 to bFGF without affecting Ca2+ release from intracellular stores or 1-oleoyl-2-acetyl-sn-glycerol-in
244 eatments that prevented release of Ca2+ from intracellular stores or activation of PKA blocked ATPgam
245 cal neurons by sustained Ca(2+) release from intracellular stores or by a brief episode of oxygen and
247 ly that cAMP potentiates Ca(2+) release from intracellular stores, primarily because of a protein kin
249 obably reflects mobilization of [Ca2+]i from intracellular stores rather than entry via voltage-gated
250 rocessing of caspases-3 and -7 occurred when intracellular store release was the sole Ca(2+) perturba
253 d transient rise in [Ca(2+)](i), mostly from intracellular stores, simultaneously with a drop in pH(i
255 mission-evoked calcium (Ca(2+)) release from intracellular stores stabilizes dendrites during the per
256 PLC-gamma2, and of Ca(2+) mobilization from intracellular stores, stimulated by muH chain crosslinki
257 lcium channels and calcium mobilization from intracellular stores strongly implicates a role for calc
259 in response to agents that release Ca2+ from intracellular stores (such as arachidonic acid, C2-ceram
261 enesis; and stimulate release of Ca(2+) from intracellular stores that appears independent of IP(3) a
262 and dGal-1 independently release Ca(2+) from intracellular stores that cooperate to induce optimal re
263 oth forms of LTD involve Ca(2+) release from intracellular stores, the Ca(2+) sensors involved are di
264 renoceptors and mGluRs mobilize calcium from intracellular stores, the mechanisms and pools of calciu
265 te receptors and the release of calcium from intracellular stores, the NPY-IR puncta fuse with the ce
266 nstitutively between the plasma membrane and intracellular stores, thereby providing a protected rece
267 ymphocytes triggers the release of Ca2+ from intracellular stores; this release of Ca2+ results in th
268 l Ca2+ release, the graded Ca2+ release from intracellular stores through inositol 1,4,5-trisphosphat
269 r responses by inducing calcium release from intracellular stores through its preferred neurokinin 1
271 y on the controlled release of Ca(2)(+) from intracellular stores to activate stage-specific Ca(2)(+)
272 ) channels that regulate Ca(2+) release from intracellular stores to control a diverse array of cellu
273 calcium channels of the plasma membrane and intracellular stores to determine what sources of calciu
274 mic reticulum to promote Ca(2+) release from intracellular stores to increase the concentration of cy
275 cose transporter-4 (GLUT4) redistribute from intracellular stores to the cell periphery in response t
276 BRs rapidly mobilizes additional TGFBRs from intracellular stores to the cell surface, increasing the
277 te receptor, AMPAR, can rapidly traffic from intracellular stores to the plasma membrane, altering ne
281 Mobilization of this increased CXCL1 from intracellular stores to the venular surface triggered be
282 , a protein that "gates" Ca(2+) release from intracellular stores, triggers Ca(2+) cell influx and th
283 receptor type 1 (RyR1) releases Ca(2+) from intracellular stores upon nerve impulse to trigger skele
284 ion leads to robust Ca(2+) mobilization from intracellular stores via activation of phospholipase C a
285 lified by Ca(2+)-induced Ca(2+) release from intracellular stores via activation of ryanodine recepto
286 lly, ouabain induced Ca(2+) release from the intracellular stores via the activation of IP3 receptors
287 hate (PIP2) hydrolysis and Ca2+ release from intracellular stores via the phospholipase C (PLC)-inosi
288 (3)R] and the ability to release Ca(2+) from intracellular stores via type 1 Ins(1,4,5)P(3)Rs were in
290 Carbachol-mediated release of Ca(2+) from intracellular stores was significantly higher in Bax tra
292 a2+]i by releasing Ca2+ from buffer sites or intracellular stores, we examined in detail the effect o
293 ents (Ca2+ syntillas) caused by release from intracellular stores were found in isolated nerve termin
294 secretion dependent upon Ca(2+) release from intracellular stores, whereas sustained secretion requir
295 , arising from both extracellular medium and intracellular stores, which induces the activation of ad
296 in B lymphocytes causes Ca(2+) release from intracellular stores, which, in turn, activates ion chan
297 tors (GPCRs) that cause calcium release from intracellular stores while other stimuli depolarize tast
300 ary for the release of free fatty acids from intracellular stores within neutrophil precursor cells.