ライフサイエンスコーパス: intracranial aneurysm

1 associated with arterial diseases, including intracranial aneurysm.

2 he pathophysiology, diagnosis and therapy of intracranial aneurysm.

3 , as well as those with PKD1, are at risk of intracranial aneurysm.

4 ween 3.6 and 6% of the population harbour an intracranial aneurysm.

5 ars with biennial follow-up in patients with intracranial aneurysm.

6 ith FMD with intracranial imaging who had an intracranial aneurysm.

7 hese 86 patients, 25 (53.8%) had more than 1 intracranial aneurysm.

8 (12.9%; 95% CI, 10.3%-15.9%) had at least 1 intracranial aneurysm.

9 five putative risk loci are associated with intracranial aneurysm.

10 correlation between ruptured and unruptured intracranial aneurysms.

11 of surgical and endovascular treatments for intracranial aneurysms.

12 ascular and surgical treatments for ruptured intracranial aneurysms.

13 ed in management of patients with unruptured intracranial aneurysms.

14 the spindle-shaped cells of the wall of the intracranial aneurysms.

15 culature to study the molecular pathology of intracranial aneurysms.

16 ts in whom the coils were implanted to treat intracranial aneurysms.

17 in axis contributes to growth and rupture of intracranial aneurysms.

18 olinium is a potential biomarker of unstable intracranial aneurysms.

19 rs are a crucial element in the treatment of intracranial aneurysms.

20 endovascular technique for the treatment of intracranial aneurysms.

21 ice for treatment of ruptured and unruptured intracranial aneurysms.

22 nsion and liver cysts, and 9% to 14% develop intracranial aneurysms.

23 established in the treatment of broad-based intracranial aneurysms.

24 e amount of interest for use in treatment of intracranial aneurysms.

25 close association between disturbed flow and intracranial aneurysms.

26 l adults older than 65 years with unruptured intracranial aneurysms.

27 dividuals; 10%-11% of these individuals have intracranial aneurysms.

28 and safety after pCONus-assisted coiling of intracranial aneurysms.

29 rum iron values are associated with ruptured intracranial aneurysms.

30 cytes in the arterial wall of two unruptured intracranial aneurysms.

31 rior cerebral artery is a common location of intracranial aneurysms.

32 diction of the risk of rupture of incidental intracranial aneurysms.

33 reasonably safe and effective treatment for intracranial aneurysms.

34 in 32 of 74 patients (43.2%), and 24 of 128 intracranial aneurysms (18.8%) were in the posterior com

35 In 199 consecutive patients with 234 clipped intracranial aneurysms, 273 intraoperative angiographic

36 oking history vs 163 of 564 patients without intracranial aneurysm (28.9%; P < .001).

37 of smoking was significantly associated with intracranial aneurysm: 42 of 78 patients with intracrani

38 Intracranial aneurysms 5 mm or larger occurred in 32 of

39 ntracranial aneurysm: 42 of 78 patients with intracranial aneurysm (53.8%) had a smoking history vs 1

40 ion in PKD1 is predictive for development of intracranial aneurysms (59 mutations are more commonly a

41 h TAAD alone, along with families with TAAD, intracranial aneurysms, abdominal aortic and bilateral i

42 SAH, mostly due to rupture of an intracranial aneurysm, accounts for a quarter of cerebro

43 One cause of stroke, intracranial aneurysm, affects approximately 2% of the p

44 s from the International Study of Unruptured Intracranial Aneurysms aimed to assess the natural histo

45 to craniotomy and clipping for some ruptured intracranial aneurysms, although the relative benefits o

46 These findings identify a Mendelian form of intracranial aneurysm and map the location of the underl

47 About 3% of adults have an unruptured intracranial aneurysm and this prevalence can increase t

48 g-target enrichment shows pleiotropy between intracranial aneurysms and antiepileptic and sex hormone

49 of ADPKD, most significant of which include intracranial aneurysms and cystic liver diseases.

50 ed data on the natural history of unruptured intracranial aneurysms and on the morbidity and mortalit

51 rive most of the genetic correlation between intracranial aneurysms and other cerebrovascular traits.

52 of kidney disease; polycystic liver disease; intracranial aneurysms and other extrarenal manifestatio

53 manifestations include an increased level of intracranial aneurysms and polycystic liver disease (PLD

54 We enrolled 2143 patients with ruptured intracranial aneurysms and randomly assigned them to neu

55 The International Study of Unruptured Intracranial Aneurysms and the International Subarachnoi

56 Morphological factors of intracranial aneurysms and the surrounding vasculature c

57 e could be affected by geometric features of intracranial aneurysms and the surrounding vasculature i

58 to assess the natural history of unruptured intracranial aneurysms and to measure the risk associate

59 ecause they included individuals with either intracranial aneurysms and/or very-early-onset disease.

60 al MR angiography follow-up in patients with intracranial aneurysm, and (e) MR angiography screening

61 d traits including blood pressure, migraine, intracranial aneurysm, and coronary artery disease.

62 ptimal in patients with detected, incidental intracranial aneurysm, and treatment may be considered i

63 arenal complications, including liver cysts, intracranial aneurysms, and cardiac valvular disease, sh

64 h genetic or other disorders associated with intracranial aneurysms, and people who smoke and have hy

65 ey disease (ADPKD) are at risk of developing intracranial aneurysms, and subarachnoid haemorrhage is

66 Embolization of intracranial aneurysms, angioplasty and stenting of extr

67 In specimens of intracranial aneurysms, aortic dissections, and dolichoe

68 Intracranial aneurysms are associated with disturbed vel

69 Saccular intracranial aneurysms are balloon-like dilations of the

70 idely available, many tissue samples such as intracranial aneurysms are both fibrous and minute, and

71 brain arteriovenous malformations (AVM) and intracranial aneurysms are both intracranial vascular di

72 associated with the management of unruptured intracranial aneurysms are complex.

73 rrhage and surgical obliteration of ruptured intracranial aneurysms are frequently associated with ne

74 Unruptured intracranial aneurysms are increasingly being detected a

75 c stroke, carotid and intracranial stenosis, intracranial aneurysms, arteriovenous malformations, mal

76 a large kindred that segregates nonsyndromic intracranial aneurysm as a dominant trait with high pene

77 arachnoid hemorrhage secondary to a ruptured intracranial aneurysm, at our institution.

78 ategies were evaluated: (a) no screening for intracranial aneurysm, (b) one-time screening with annua

79 al MR angiography follow-up in patients with intracranial aneurysm, (c) MR angiographic screening eve

80 ent's knowledge that they have an unruptured intracranial aneurysm can lead to substantial stress and

81 Intracranial aneurysms can be treated with endovascular

82 ch and Japanese cohorts including over 2,100 intracranial aneurysm cases and 8,000 controls.

83 noses of subarachnoid hemorrhage, unruptured intracranial aneurysm, cerebral arteriovenous malformati

84 sign of subarachnoid haemorrhage, unruptured intracranial aneurysm, cerebral venous sinus thrombosis,

85 ies affecting the cerebrovasculature include intracranial aneurysms, cervical artery dissection, fibr

86 To assess ferromagnetic properties of intracranial aneurysm clips reported to be nonferromagne

87 -dominant polycystic kidney disease, such as intracranial aneurysms, cluster in families.

88 he most frequent neurologic complications of intracranial aneurysm coiling.

89 ears with endovascular treatment in detected intracranial aneurysm, (d) MR angiography screening ever

90 The annual incidence of unruptured intracranial aneurysm detection was normalized to the in

91 y outcomes were 14-day and 90-day unruptured intracranial aneurysm detection, respectively.

92 The presence of intracranial aneurysm did not vary with location of extr

93 vational studies of patients with unruptured intracranial aneurysms, discharge outcomes were better a

94 reatment are the most important drawbacks of intracranial aneurysm embolization.

95 nt are being used for patients with ruptured intracranial aneurysms: endovascular detachable-coil tre

96 They play a vital role in the assessment of intracranial aneurysms, especially in evaluation of the

97 In patients with a ruptured intracranial aneurysm, for which endovascular coiling an

98 atic genetic variants in the pathogenesis of intracranial-aneurysm formation is unknown.

99 the recent International Study of Unruptured Intracranial Aneurysms found that the rupture rate of sm

100 Intracranial aneurysms from patients without ADPKD also

101 Intracranial aneurysms have been reported in PKD2, as we

102 The frequency and patterns of screening for intracranial aneurysms have not been defined.

103 The genetic risk of intracranial aneurysm (IA) development has been ascribed

104 The pathogenesis of intracranial aneurysm (IA) formation and rupture is comp

105 Intracranial aneurysm (IA) rupture leads to subarachnoid

106 As endovascular approaches to intracranial aneurysm (IA) treatment continue to evolve,

107 ation of the aorta have a high prevalence of intracranial aneurysms (IA) and suffer subarachnoid hemo

108 Intracranial aneurysms (IA) pose significant health risk

109 racranial aneurysms (M[U]IA), deciding which intracranial aneurysms (IA) should be treated and which

110 Intracranial aneurysms (IAs) affect about 3% of the popu

111 Intracranial aneurysms (IAs) are a high-risk factor for

112 Intracranial aneurysms (IAs) are a significant public he

113 yses in selection footprints and the risk of intracranial aneurysms (IAs) in Nunavik Inuit revealed a

114 omewide screen of 48 sib pairs affected with intracranial aneurysms (IAs) that revealed suggestive li

115 bdominal aortic aneurysms (AAAs) but not for intracranial aneurysms (IAs), and that hypertension is a

116 y disease (ADPKD) have a higher incidence of intracranial aneurysms (ICA) than the general population

117 lycystic kidney disease (ADPKD) and saccular intracranial aneurysms (ICA), the risk of MRA-defined gr

118 Intracranial aneurysms (ICAs) are focal dilatations that

119 The prevalence of intracranial aneurysm in patients with fibromuscular dys

120 though the clinical benefit of screening for intracranial aneurysm in patients with FMD has yet to be

121 or endovascular coiling after rupture of an intracranial aneurysm in the follow-up of the Internatio

122 The prevalence of intracranial aneurysm in women diagnosed with FMD is sig

123 To examine the prevalence of intracranial aneurysm in women diagnosed with FMD.

124 -silico trial, which models the treatment of intracranial aneurysms in 164 virtual patients with 82 d

125 erformed prospectively 10 years after EVT of intracranial aneurysms in a single institution.

126 g and surveillance strategies for unruptured intracranial aneurysms in patients with ADPKD.

127 showing strong evidence for association with intracranial aneurysms in the combined dataset, includin

128 4,701 patients with 6,411 intracranial aneurysms, including 1201 prospective patie

129 sensitivity and specificity for detection of intracranial aneurysms, including small aneurysms, in pa

130 technique are presented in the management of intracranial aneurysms, including: preoperative aneurysm

131 3-year-old man with progressive, right-sided intracranial aneurysms, ipsilateral to an impressive cut

132 Intracranial aneurysm is a common life-threatening disea

133 Intracranial aneurysm is a common neurological problem;

134 The rupture of an intracranial aneurysm is a sudden devastating event with

135 Once an unruptured intracranial aneurysm is detected, decisions regarding o

136 EVT of intracranial aneurysm is effective for prevention of lon

137 d annual follow-up in patients with detected intracranial aneurysm is the optimal strategy (cost, $19

138 The pathogenesis of intracranial aneurysm is unknown, and because catastroph

139 The management of unruptured intracranial aneurysms is controversial.

140 When the prevalence of intracranial aneurysms is greater than 10%, annual ruptu

141 Rupture of an intracranial aneurysm leads to subarachnoid hemorrhage,

142 In case of multiple (unruptured) intracranial aneurysms (M[U]IA), deciding which intracra

143 572); venous thromboembolism (n(e) = 4,607); intracranial aneurysm (n(e) = 1,328); CAD (n(e) = 12,716

144 normal elastic and intracranial arteries and intracranial aneurysms obtained at autopsy from patients

145 A 64-year-old woman was diagnosed with three intracranial aneurysms of the right and left middle cere

146 Surgery for intracranial aneurysm often results in postoperative neu

147 Deep learning (DL) models can help detect intracranial aneurysms on CTA, but high false positive (

148 lies with multiple members that had TAAD and intracranial aneurysms or TAAD and intracranial and abdo

149 nous thromboembolism (OR: 0.97; p = 0.63) or intracranial aneurysm (OR: 0.85; p = 0.15).

150 canalization after Endovascular Treatment of Intracranial Aneurysm, or ARETA, cohort.

151 lation, early angiogenesis, atherosclerosis, intracranial aneurysm, or interstitial flow.

152 iron and its related factors play a role in intracranial aneurysm pathophysiology and investigated i

153 d sex hormone drugs, providing insights into intracranial aneurysm pathophysiology.

154 ysmal subarachnoid haemorrhage or unruptured intracranial aneurysms, people with genetic or other dis

155 Thirty patients with 33 unruptured intracranial aneurysms prospectively underwent high-reso

156 In patients with ruptured intracranial aneurysms, rates of aneurysm obliteration w

157 Stent-assisted coiling (SAC) for ruptured intracranial aneurysms (RIAs) remains controversial due

158 inical risk factors, play important roles in intracranial aneurysm risk, and drive most of the geneti

159 types, including coronary artery disease and intracranial aneurysms (rs10757278 and rs1333040).

160 Risk of intracranial aneurysm rupture could be affected by geome

161 significantly and inversely associated with intracranial aneurysm rupture.

162 al analysis of consecutive attendances at an intracranial aneurysm screening clinic.

163 In clinical practice, people undergoing intracranial aneurysm screening had stronger family hist

164 Finally, CFD and immunofluorescence on human intracranial aneurysms showed a correlation similar to t

165 e: (1) The International Study of Unruptured Intracranial Aneurysms studied the natural history and t

166 Familial clustering of intracranial aneurysms suggests that genetic factors are

167 In patients with ruptured intracranial aneurysms suitable for both treatments, end

168 The only known risk factor was an intracranial aneurysm that was found on her grandmother'

169 itive, specific studies for the diagnosis of intracranial aneurysms that are of sufficient size to be

170 Out of 444 cases of intracranial aneurysms treated in our centre, 65 aneurys

171 ope, was performed to identify patients with intracranial aneurysms treated with the WEB device.

172 ular therapies, particularly in the field of intracranial aneurysm treatment, has been truly remarkab

173 for optimal reduction of artifacts following intracranial aneurysm treatment.

174 fold increased risk for having an unruptured intracranial aneurysm (UIA) in women aged between 30 and

175 Unruptured intracranial aneurysms (UIAs) are common incidental imag

176 Unruptured intracranial aneurysms (UIAs) are increasingly diagnosed

177 Small unruptured intracranial aneurysms (UIAs) are increasingly diagnosed

178 Background Unruptured intracranial aneurysms (UIAs) are relatively common and

179 Very small intracranial aneurysms (VSIAs) may cause many neurologic

180 go, the treatment of incidentally discovered intracranial aneurysms was straightforward with a good e

181 ew risk loci and the genetic architecture of intracranial aneurysms, we performed a cross-ancestry, g

182 d stenosis, carotid dissection, and extra or intracranial aneurysm were reviewed retrospectively.

183 2143 patients with ruptured intracranial aneurysms were enrolled between 1994 and 20

184 When intracranial aneurysms were identified, people were refe

185 Results A total of 683 intracranial aneurysms were treated using the WEB device

186 h ADPKD, and approximately 9% to 14% develop intracranial aneurysms, which have a rupture rate of 0.5

187 o play an important role in the formation of intracranial aneurysms, which is conditioned by the geom

188 All consecutive patients with intracranial aneurysms who were treated with the LVIS EV

189 2143 patients with ruptured intracranial aneurysms, who were admitted to 42 neurosur

190 um of 14 years after treatment of a ruptured intracranial aneurysm with either neurosurgical clipping

191 nd 9p that show significant association with intracranial aneurysm with odds ratios 1.24-1.36.

192 y continues to evolve which now implies that intracranial aneurysms with complex vascular architectur

193 Conclusion Screening for intracranial aneurysms with MR angiography in patients w

194 ness and safety of endovascular treatment of intracranial aneurysms with the use hydrogel coils.

195 punctures and higher detection of unruptured intracranial aneurysms, with no significant change in mi