コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 utcome in patients with localization-related intractable epilepsy.
2 al malformations of cortical development and intractable epilepsy.
3 Forty individuals had intractable epilepsy.
4 D), a neurological disorder characterized by intractable epilepsy.
5 cal malformation of cortical development and intractable epilepsy.
6 3% accurate on a dataset of 16 patients with intractable epilepsy.
7 ely from somatic mosaic mutations, and cause intractable epilepsy.
8 developmental disorders, including childhood intractable epilepsy.
9 lepsy, or SUDEP, is a devastating outcome of intractable epilepsy.
10 liable treatment for patients suffering from intractable epilepsy.
11 y is a valuable option for pharmacologically intractable epilepsy.
12 most common cause of death in patients with intractable epilepsy.
13 haly, movement disorders, and/or early-onset intractable epilepsy.
14 injury (TBI) is one of the leading causes of intractable epilepsy.
15 on of dysplastic cortex for the treatment of intractable epilepsy.
16 pic cortex, and is generally associated with intractable epilepsy.
17 ng seizures in people with pharmacologically intractable epilepsy.
18 injury (TBI) and a common cause of medically-intractable epilepsy.
19 wer antiepileptic drugs for the treatment of intractable epilepsy.
20 es in 11 patients for surgical evaluation of intractable epilepsy.
21 brain development, cognitive disability, and intractable epilepsy.
22 of the corpus callosum for the treatment of intractable epilepsy.
23 sporadic, nonsyndromic patients with HH and intractable epilepsy.
24 re, benign congenital tumors associated with intractable epilepsy.
25 H tissue in 55 patients with sporadic HH and intractable epilepsy.
26 t support its use in children with treatment-intractable epilepsy.
27 ical management, particularly in the case of intractable epilepsy.
28 n patients undergoing surgical treatment for intractable epilepsy.
29 e frequency is reduced in some patients with intractable epilepsy.
30 an effective and safe therapy for medically intractable epilepsy.
31 and may be efficacious in treating medically intractable epilepsy.
32 tolerated treatment option for infants with intractable epilepsy.
33 vailable as adjunctive therapy for medically intractable epilepsy.
34 is a common and important cause of medically intractable epilepsy.
35 implantation for the surgical management of intractable epilepsy.
36 of lissencephaly are mental retardation and intractable epilepsy.
37 quency in animal models and in patients with intractable epilepsy.
38 r indications for the treatment of medically intractable epilepsy.
39 rted in ictal SPECT studies of patients with intractable epilepsy.
40 l temporal lobe resection in 25 patients for intractable epilepsy.
41 plied for the treatment of pharmacologically intractable epilepsy.
42 tion, may be important in the development of intractable epilepsy.
43 of surgical techniques for the treatment of intractable epilepsy.
44 ment (FMCDs) that are highly associated with intractable epilepsy.
45 (DGCs) harvested during hippocampectomy for intractable epilepsy.
46 various pathologies and are associated with intractable epilepsy.
47 language lateralization in 12 patients with intractable epilepsy.
48 ight or left temporal lobe for the relief of intractable epilepsy.
49 wake humans undergoing surgery for medically intractable epilepsy.
50 et (KD) is an effective treatment option for intractable epilepsy.
51 s from 11 patients undergoing resections for intractable epilepsy.
52 igration disorders are often associated with intractable epilepsy.
53 atients who underwent temporal lobectomy for intractable epilepsy.
54 cted deaths are more common in patients with intractable epilepsy.
55 be a viable therapeutic target for monogenic intractable epilepsies.
56 s a process that plays a significant role in intractable epilepsies.
57 aterial from patients surgically treated for intractable epilepsy (46/57), exhibited characteristics
59 r Dravet's Syndrome), which includes severe, intractable epilepsy and comorbidities of ataxia and cog
60 orized based on the effect of callosotomy on intractable epilepsy and dichotic listening research, re
62 N affecting a single patient presenting with intractable epilepsy and hemimegalencephaly that varied
63 ylphosphatidylinositol deficiency (IGD) with intractable epilepsy and intellectual developmental diso
64 erapy for potential neuropeptide delivery in intractable epilepsy and possibly other neurological dis
65 pe of severe neurological symptoms including intractable epilepsy and profound neurocognitive impairm
66 a 36-year-old woman with muscular dystrophy, intractable epilepsy, and bilateral temporo-occipital li
67 l EEG data from rare patients with medically intractable epilepsy, and found evidence for respiratory
68 order associated with mental retardation and intractable epilepsy, and Miller-Dieker syndrome (MDS) i
69 lepsy, one with focal cortical dysplasia and intractable epilepsy, and one dysmorphic term infant wit
70 he comorbidities due to decades of medically intractable epilepsy; and possible acceleration of commo
71 We recorded from patients with chronically intractable epilepsy as they performed a task that requi
72 This approach is of therapeutic interest for intractable epilepsy, as it spares cortical function bet
73 somatic SLC35A2 variants have been linked to intractable epilepsy associated with malformations of co
74 onsecutive temporal resections for medically intractable epilepsy associated with mesial temporal scl
76 n 136 pediatric patients who had surgery for intractable epilepsy at The Cleveland Clinic between Jan
77 rowth, cellular and architectural dysplasia, intractable epilepsy, autism and intellectual disability
78 (SUDEP) is the most common cause of death in intractable epilepsies, but physiological mechanisms tha
79 on (VNS) is a common treatment for medically intractable epilepsy, but response rates are highly vari
80 Dravet syndrome (DS) is an infantile-onset intractable epilepsy caused by heterozygous loss-of-func
81 racterized by abnormal brain development and intractable epilepsy, caused similar defects in Golgi lo
82 tissue resected from patients with medically intractable epilepsy demonstrated increased adenosine ki
83 phic (ECoG) data from 15 human patients with intractable epilepsy during a word completion task and e
86 tic resonance imaging (MRI) in children with intractable epilepsy has not been quantified in relation
88 ubjects were patients with pharmacologically intractable epilepsy implanted with depth electrodes to
90 rtical resection is effective in alleviating intractable epilepsy in children with tuberous sclerosis
92 ns in doublecortin (DCX) are associated with intractable epilepsy in humans, due to a severe disorgan
93 nate dimer SPECT studies in 43 patients with intractable epilepsy in whom seizure laterality could be
94 eservation of perisomatic GABAergic input in intractable epilepsies may be a key factor in the genera
100 e young patients (mean age, 10.8 years) with intractable epilepsy of neocortical origin underwent chr
101 s a new model for studying the mechanisms of intractable epilepsy of the complex partial seizure type
102 s gestation, one with hemimegalencephaly and intractable epilepsy, one with focal cortical dysplasia
103 related to duration of epilepsy, duration of intractable epilepsy, or continuation of medications.
105 encephalopathy (51%) were at greater risk of intractable epilepsy (P = 0.003), non-ambulance (P = 0.0
107 ype, including profound developmental delay, intractable epilepsy, progressive microcephaly, severe h
108 y; still, its clinical role in children with intractable epilepsy remains unclear, largely because of
110 Analysis of hippocampi from patients with intractable epilepsy revealed that Bim levels were signi
114 a cohort of 21 adult males and females with intractable epilepsy, we delivered 17,631 stimulation pu
115 tions from tissue removed during surgery for intractable epilepsy, we examined the human hippocampal
116 study a research participant with medically intractable epilepsy who had extensive bilateral frontot
117 lization in 10 children and adolescents with intractable epilepsy who sustained an early lesion in th
119 phy (iEEG) from a cohort of 35 patients with intractable epilepsy (with 71 contacts in amygdala, 31 i