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1 e proteins that are considered unique to the intraerythrocytic agent of Anaplasma marginale and the i
2 ller's sea cow Hb unresponsive to the potent intraerythrocytic allosteric effector 2,3-diphosphoglyce
3                                          The intraerythrocytic apicomplexan Babesia microti, the prim
4 er 37,000 Plasmodium falciparum cells across intraerythrocytic asexual and sexual development.
5 yprenol and dolichol profile both within the intraerythrocytic asexual cycle and between asexual and
6 Phenotype analysis of the KIC5 mutant during intraerythrocytic asexual development identified transcr
7  or Maurer's clefts (Pfsbp 1 or mAb LWL1) or intraerythrocytic asexual parasite proteins (PfEMP2 or H
8 mine N-methyltransferase that block parasite intraerythrocytic asexual replication and gametocyte dif
9 of dual activity antimalarials to block both intraerythrocytic asexual replication and gametocytogene
10 f severe and fatal malaria are caused by the intraerythrocytic asexual reproduction cycle of Plasmodi
11              A recent study reveals that the intraerythrocytic asexual reproduction cycle of Plasmodi
12  a malaria-like illness caused by tick-borne intraerythrocytic Babesia parasites of the Apicomplexa p
13 Babesiosis is a tick-borne disease caused by intraerythrocytic Babesia parasites.
14 tentially fatal tick-borne disease caused by intraerythrocytic Babesia parasites.
15                           A newly identified intraerythrocytic Babesia-like organism, WA1, and its re
16  facultative-intracellular pathogens causing intraerythrocytic bacteremia in their mammalian reservoi
17  type A tularemia, we showed the presence of intraerythrocytic bacteria by double-immunofluorescence
18                    This sexual-stage tubular intraerythrocytic compartment (STIC) is not recognized b
19                           At monthly visits, intraerythrocytic concentrations of TFV-DP and FTC-TP in
20                                  Some of the intraerythrocytic crystals in this syndrome are unusuall
21 lu-->Lys) present red blood cells (RBC) with intraerythrocytic crystals that form when hemoglobin (Hb
22 a mild clinical course, abundant circulating intraerythrocytic crystals, and increased folded red cel
23 RBC, which in turn enhanced the formation of intraerythrocytic crystals.
24  the time the parasites take to traverse the intraerythrocytic cycle (IEC).
25 vax genes occurs during short periods of the intraerythrocytic cycle and that this pattern of gene ex
26 sphoantigens were released at the end of the intraerythrocytic cycle at the time of parasite egress.
27 to promote var gene transcription during the intraerythrocytic cycle in vitro.
28 l response of P. falciparum to T4 during the intraerythrocytic cycle of this parasite.
29                                   During the intraerythrocytic cycle, malaria parasites metabolize la
30 ring nuclear division towards the end of the intraerythrocytic cycle.
31 molog of aminopeptidase P during its asexual intraerythrocytic cycle.
32                     We hypothesized elevated intraerythrocytic deoxyHb would limit resilience to oxid
33                            Polymerization of intraerythrocytic deoxyhemoglobin S (HbS) is the primary
34 acting via conversion to NO by reaction with intraerythrocytic deoxyhemoglobin.
35                                At the end of intraerythrocytic development (late schizogony), there w
36 viction of nucleosomes on strong TSSs during intraerythrocytic development and demonstrate that nucle
37 ological changes in the parasites during the intraerythrocytic development by applying the interpreta
38 exhibit a higher potency after two cycles of intraerythrocytic development compared to one.
39 s a widely used indicator for the Plasmodium intraerythrocytic development cycle (IDC), for example,
40                   Gene expression during the intraerythrocytic development cycle of the human malaria
41 ession through the first part of the asexual intraerythrocytic development cycle.
42                        Plasmodium falciparum intraerythrocytic development is a complex process.
43 rum revealed that fenpropimorph inhibits the intraerythrocytic development of both chloroquine- and p
44 one person to another relies on the complete intraerythrocytic development of non-pathogenic sexual g
45  matters over the alterations induced by the intraerythrocytic development of P. falciparum.
46 mosis) is a tick-borne disease caused by the intraerythrocytic development of protozoa parasites from
47  of potentially hundreds of genes during the intraerythrocytic development of this important human pa
48 lciparum structures or organelles across the intraerythrocytic development of this parasite and made
49 uctural analysis of P. falciparum during its intraerythrocytic development to date and sheds light on
50 er to analyse the expression of genes during intraerythrocytic development, DNA microarrays were cons
51                                 During their intraerythrocytic development, malaria parasites export
52                                       During intraerythrocytic development, Plasmodium falciparum exp
53 s and membrane biogenesis during the asexual intraerythrocytic development.
54 2 genes are expressed in different stages of intraerythrocytic development.
55 ver before clinical representation caused by intraerythrocytic development.
56 s as the sole energy-yielding process during intraerythrocytic development.
57 l variants in the second and third cycles of intraerythrocytic development.
58 abesia duncani and Plasmodium falciparum for intraerythrocytic development.
59 rane and plays an essential role in parasite intraerythrocytic development.
60 characterizing the transcriptome of the 48 h intraerythrocytic developmental cycle (IDC) for two stra
61                                  The asexual intraerythrocytic developmental cycle (IDC) of Plasmodiu
62 stablished the P. vivax transcriptome of the Intraerythrocytic Developmental Cycle (IDC) of two clini
63 te Plasmodium falciparum undergoes a complex intraerythrocytic developmental cycle (IDC) that relies
64  transcriptional pattern during its ~48-hour intraerythrocytic developmental cycle (IDC) to publicly
65 istant parasite's capacity to decelerate its intraerythrocytic developmental cycle (IDC), which can c
66 d a significant reduction in growth over one intraerythrocytic developmental cycle (IDC).
67 onal activity over the course of the 48-hour intraerythrocytic developmental cycle (IDC); however, th
68 at V-type ATPase is expressed throughout the intraerythrocytic developmental cycle and is mainly loca
69 es with no known function during the asexual intraerythrocytic developmental cycle are enriched in Pf
70  is not only periodic throughout the asexual intraerythrocytic developmental cycle but is also highly
71 s in mining the malaria transcriptome of the intraerythrocytic developmental cycle of P. falciparum.
72 ta collected at seven time points during the intraerythrocytic developmental cycle, we (i) detect nov
73 in the cytoplasm of P. falciparum during the intraerythrocytic developmental cycle.
74                      Anaplasma marginale, an intraerythrocytic ehrlichial pathogen of cattle, establi
75 he extracellular Trypanosoma brucei, unusual intraerythrocytic Endotrypanum spp., phytoparasitic Phyt
76 of an in vitro screening assay targeting the intraerythrocytic form of the malaria parasite Plasmodiu
77  be secreted shortly after activation of the intraerythrocytic gametocyte, and during sporozoite migr
78                                  When mature intraerythrocytic gametocytes are taken up in a blood me
79 One cyclic biphenyl ether compound inhibited intraerythrocytic growth of P. falciparum with an IC50 o
80                                              Intraerythrocytic growth of the human malaria parasite P
81                                       During intraerythrocytic growth, the parasite expresses protein
82  is prolonged from one to two generations of intraerythrocytic growth, with AZ producing 50% inhibiti
83 gesting that PfMYST is essential for asexual intraerythrocytic growth.
84                            Here, we analyzed intraerythrocytic Hb, a protein that operates at the org
85 , we quantify the volume of the DV for live, intraerythrocytic HB3 (CQS), Dd2 (CQR via drug selection
86 our knowledge, to capture a temporal view of intraerythrocytic HbC phase separation, crystal formatio
87   The reaction rate between nitric oxide and intraerythrocytic hemoglobin plays a major role in nitri
88                 Babesia spp. are tick-borne, intraerythrocytic hemoparasites that use antigenic varia
89 een recognized to play a central role during intraerythrocytic infection by Plasmodium parasites, the
90 s is only part of the story, as the P. vivax intraerythrocytic life cycle is complex.
91                                   During the intraerythrocytic life cycle of P. falciparum, a subset
92 doplasmic reticulum expressed throughout the intraerythrocytic life cycle of the parasite but induced
93 ar structures throughout the parasite's 48-h intraerythrocytic life cycle.
94 e an atlas of parasite metabolism during its intraerythrocytic life cycle.
95 rrested parasite growth at all stages of the intraerythrocytic life cycle.
96 ose, and GDP-fucose in Plasmodium falciparum intraerythrocytic life stages.
97 ozoite egress near the end of the parasite's intraerythrocytic lifecycle.
98 etween the host erythrocyte membrane and the intraerythrocytic malaria parasite by demonstrating for
99                          We hypothesise that intraerythrocytic malaria parasite metabolism is not mer
100                                              Intraerythrocytic malaria parasites can obtain nearly th
101 t In(III) (R)-ENBPI metallo-complexes killed intraerythrocytic malaria parasites in a stage-specific
102                    Hemoglobin degradation in intraerythrocytic malaria parasites is a vast process th
103                                              Intraerythrocytic malaria parasites proliferate bounded
104                                              Intraerythrocytic malaria parasites replicate by the pro
105                                              Intraerythrocytic malaria parasites send hundreds of eff
106                                              Intraerythrocytic malaria parasites use host hemoglobin
107 e blood, including platelets, which can kill intraerythrocytic malaria parasites.
108 -dimensional (3D) versus time data for live, intraerythrocytic malarial parasites.
109 mportant substrate for lipid biosynthesis of intraerythrocytic malarial parasites.
110 e of extracellular nutrient solutes, and (3) intraerythrocytic membranes transport a parasite-encoded
111 a target for antimalarial drug design as the intraerythrocytic merozoite lifestage of P. falciparum i
112 sed from an individual schizont, termed the "intraerythrocytic multiplication factor" (IMF).
113 at hemoglobinopathic erythrocytes reduce the intraerythrocytic multiplication of P. falciparum, poten
114  stem cell transplantation revealed numerous intraerythrocytic organisms typical of the genus Babesia
115 the characterization of the carbohydrates in intraerythrocytic P. falciparum proteins and provides an
116 cine is withdrawn from the culture medium of intraerythrocytic P. falciparum, the parasite slows its
117 n reductase is essential for the survival of intraerythrocytic P. falciparum.
118 ental stage-specific biosynthesis of GPIs by intraerythrocytic P. falciparum.
119 ed to the digestive vacuolar membrane of the intraerythrocytic parasite and may function as a transpo
120 tle is known about interactions between this intraerythrocytic parasite and the macrophages of its bo
121                        Babesia microti is an intraerythrocytic parasite and the primary causative age
122 tric assessment and mathematical modeling of intraerythrocytic parasite development revealed an unexp
123 ted beta-hematin formation in vitro, delayed intraerythrocytic parasite development with apparent inh
124  analyzed its function and expression during intraerythrocytic parasite development.
125                                          The intraerythrocytic parasite itself exerts tight control o
126 identified ion channel on the surface of the intraerythrocytic parasite may provide direct access to
127                            Babesia bovis, an intraerythrocytic parasite of cattle, is sequestered in
128             Infection with this organism, an intraerythrocytic parasite of the phylum Apicomplexa, ca
129                 Babesia microti, a tickborne intraerythrocytic parasite that can be transmitted by me
130            Babesiosis, usually caused by the intraerythrocytic parasite, Babesia microti and transmit
131                  Babesia microti, a zoonotic intraerythrocytic parasite, is the primary etiological a
132 c function essential to the survival of this intraerythrocytic parasite.
133  inositol-polyphosphate production in intact intraerythrocytic parasite.
134 on in response to the metabolic needs of the intraerythrocytic parasite.
135  Merozoite invasion and subsequent egress of intraerythrocytic parasites are essential for this eryth
136 pies for the treatment of diseases caused by intraerythrocytic parasites are impeded by the rapid eme
137  of mechanisms, light microscopy showed that intraerythrocytic parasites develop slowly in HbF erythr
138 nificantly different from those expressed by intraerythrocytic parasites from the mammalian host.
139 eir relative contribution to the survival of intraerythrocytic parasites might be.
140 ific requirements and molecular functions in intraerythrocytic parasites remain poorly understood.
141                                 Accordingly, intraerythrocytic parasites scavenged radiolabelled lipo
142                     Using live, synchronized intraerythrocytic parasites under continuous perfusion,
143 s widespread protein and lipid damage inside intraerythrocytic parasites, necessitating macromolecule
144 tentially life-threatening disease caused by intraerythrocytic parasites, which usually are tickborne
145 nd in the digestive vacuole (DV) membrane of intraerythrocytic parasites.
146 the biology, pathogenicity, and virulence of intraerythrocytic parasites.
147 t in two subcellular compartments in asexual intraerythrocytic parasites; that is, the food vacuole,
148 he importance of understanding the impact of intraerythrocytic parasitic infections of SCD RBCs, espe
149 both the trophozoite and merozoite stages of intraerythrocytic parasitism.
150 asite Plasmodium falciparum replicates in an intraerythrocytic parasitophorous vacuole (PV).
151    The malaria parasite replicates within an intraerythrocytic parasitophorous vacuole (PV).
152 e Plasmodium falciparum replicates within an intraerythrocytic parasitophorous vacuole (PV).
153               The parasite divides within an intraerythrocytic parasitophorous vacuole until rupture
154 protozoan parasite that replicates within an intraerythrocytic parasitophorous vacuole.
155                   Our results reveal a novel intraerythrocytic phase during F. tularensis infection.
156                                   During the intraerythrocytic phase of the life cycle, malaria paras
157 s by glycosylphosphatidylinositols (GPIs) of intraerythrocytic Plasmodium falciparum is believed to c
158 aging of hemozoin within live, synchronized, intraerythrocytic Plasmodium falciparum malarial parasit
159 cteria, two mammalian cancer cell lines, and intraerythrocytic Plasmodium falciparum, which they were
160 me released during hemoglobin proteolysis in intraerythrocytic Plasmodium falciparum.
161 s is important for hemoglobin degradation in intraerythrocytic Plasmodium parasites.
162 ays an important role in the pathogenesis of intraerythrocytic Plasmodium parasites.
163                                   Rationale: Intraerythrocytic polymerization of Hb S promotes hemoly
164                        In patients with SCD, intraerythrocytic polymerization of mutant hemoglobin pr
165 itro by riboflavin starvation, inhibited the intraerythrocytic proliferation of Plasmodium falciparum
166 Because hemoglobin (Hb) is the most abundant intraerythrocytic protein and reacts rapidly with NO, th
167 osis, a tickborne zoonotic disease caused by intraerythrocytic protozoa of the genus babesia, is char
168 alth problem in humans, is caused by related intraerythrocytic protozoa with a similar pathogenesis a
169 ldwide emerging tick-borne disease caused by intraerythrocytic protozoa.
170                                 B. bovis, an intraerythrocytic protozoal parasite, establishes chroni
171         Babesia microti, a tick-transmitted, intraerythrocytic protozoan parasite circulating mainly
172       Therefore, tight immune control of the intraerythrocytic replication of the parasite is essenti
173 mental program that alternates non-canonical intraerythrocytic replication with dissemination to new
174            Bovine anaplasmosis caused by the intraerythrocytic rickettsial pathogen Anaplasma margina
175                    Anaplasma marginale is an intraerythrocytic rickettsial pathogen of cattle in whic
176 e ART's efficacy, small proportions of young intraerythrocytic ring stage parasites can survive the d
177 re the activity of existing antimalarials on intraerythrocytic sexual stage gametocytes and identify
178 xamined the expression of STEVOR proteins in intraerythrocytic sexual stages, gametocytes, and extrac
179     Here we show that (1) both cell-free and intraerythrocytic SNO-Hb (SNO-RBC) inhibit platelet aggr
180 ere a transcriptome-wide characterization of intraerythrocytic splicing events, as captured by RNA-Se
181                                   During the intraerythrocytic stage of infection, the malaria parasi
182                                   During the intraerythrocytic stage of Plasmodium falciparum's lifec
183  required for both a prolonged period of the intraerythrocytic stage of Plasmodium infection, as well
184 sub-micromolar in vitro activity against the intraerythrocytic stage of the parasite as well as littl
185 ia parasite Plasmodium falciparum during the intraerythrocytic stage of the parasite's lifecycle.
186 t the major carbohydrate modification in the intraerythrocytic stage P. falciparum proteins; 2) in co
187  anchors are crucial for the survival of the intraerythrocytic stage Plasmodium falciparum because of
188 e and extent of carbohydrate modification in intraerythrocytic stage Plasmodium falciparum proteins h
189 tols (GPIs) are the major glycoconjugates in intraerythrocytic stage Plasmodium falciparum.
190 lobin (Hb) as a major nutrient source in the intraerythrocytic stage, during which heme is converted
191 ies of gene expression throughout the entire intraerythrocytic stage.
192 althy RBCs and RBCs parasitized to different intraerythrocytic stages by the malaria-inducing parasit
193 cytic Plasmodium stages rely on FAS, whereas intraerythrocytic stages depend on scavenging FA from th
194                                              Intraerythrocytic stages efficiently trigger Vgamma9Vdel
195  1o is fast acting and highly active against intraerythrocytic stages of chloroquine-sensitive and re
196 he growth of the parasite during the asexual intraerythrocytic stages of infection.
197 t pathway may contribute to the virulence of intraerythrocytic stages of malarial infection.
198 d synthase and lipoic acid protein ligase in intraerythrocytic stages of P. falciparum.
199         Our systematic experiments cover all intraerythrocytic stages of parasite development under p
200  Plasmodium-infected erythrocytes during all intraerythrocytic stages of parasite development.
201                Northern analysis of RNA from intraerythrocytic stages of the parasite demonstrates th
202                                          The intraerythrocytic stages of the protozoan parasite Plasm
203 s expressed as a long and a short version in intraerythrocytic stages.
204  of multiple protein complexes implicated in intraerythrocytic survival of the parasite.
205 ipoate scavenging is critical for Plasmodium intraerythrocytic survival.
206                                        Using intraerythrocytic tenofovir-diphosphate data from the em
207                   Plasma tenofovir (TFV) and intraerythrocytic TFV-diphosphate (TFV-DP) concentration
208                  Plasma CAB, plasma TFV, and intraerythrocytic TFV-diphosphate concentrations were de

 
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