ライフサイエンスコーパス: intragastric

1 eaction in ethanol-treated mice (5 g/kg/day, intragastric, 10 days + 24 hours), rat brain slice cultu

2 thanol decreased both pancreatic blood flow (intragastric, 40% decrease, p < 0.05; intravenous, 34% d

3 eeding using intravenous -[1-13C]leucine and intragastric -[5,5, 5- 2H3]leucine.

4 34% decrease, p < 0.05) and interstitial pH (intragastric, 7.24 +/- 0.04 to 7.08 +/- 0.04, p < 0.05;

5 Gastric injury was induced by either intragastric absolute ethanol (1.0 mL) or subcutaneous i

6 Injury-induced sensitization to intragastric acid administration is consistent with a po

7 In awake rats, visceromotor responses to intragastric acid are quantifiable, reliable, and reprod

8 lcers enhanced the visceromotor responses to intragastric acid.

9 cinogen N-nitrosomethylbenzylamine (NMBA) by intragastric administration and killed 12 weeks later.

10 jury in male Sprague-Dawley rats after 3-day intragastric administration of 100 mg/kg or 300 mg/kg Ge

11 In the rat mammary tumorigenesis study, intragastric administration of 2 or 4 mg of deguelin/kg

12 A single intragastric administration of 250 mg/kg BR931 (4-chloro

13 le s.c. injection of T3 (2 mg/kg) and single intragastric administration of 9-cis RA (40 mg/kg) or 4-

14 ingual administration of Ag/CTB conjugate or intragastric administration of a >100-fold higher dose o

15 In this report, we show that intragastric administration of a food allergen with a mu

16 Intraperitoneal injection of CCK or intragastric administration of a trypsin inhibitor to el

17 an agonistic monoclonal anti-Fas antibody or intragastric administration of alcohol is severely blunt

18 in rats in a model involving the continuous intragastric administration of an ethanol-containing, co

19 ous knockout mice were treated with a single intragastric administration of CCl(4).

20 etions were examined in conscious rats after intragastric administration of chopped rodent chow in th

21 In particular, intranasal but not intragastric administration of CNCM I-5314 increases the

22 and gut peptide secretion.We studied how the intragastric administration of DB affects interdigestive

23 The intragastric administration of DB decreased hunger (P =

24 Intragastric administration of either antagonist at 30 m

25 Intraperitoneal and intragastric administration of ENT at a dose of 120 nmol

26 Intragastric administration of ethanol (45%, 5 ml/kg) re

27 gdala of rats treated with a binge protocol (intragastric administration of ethanol 3 times daily for

28 Intragastric administration of ethanol to animals mainta

29 l and main veins) and urine of rats after an intragastric administration of fermented red beet juice

30 Notably, intragastric administration of HK-C60 significantly incr

31 We investigated the effects of the intragastric administration of leucine and isoleucine on

32 s of the solitary tract (NTS) in response to intragastric administration of lipid in DIO-P (C57Bl6) a

33 Subcutaneous immunization, followed by intragastric administration of MAP peptide entrapped or

34 We showed that, after intragastric administration of MSG, the MSG is preferent

35 A single intragastric administration of NAF resulted in a rapid i

36 nimals, in which oral tolerance was induced, intragastric administration of OVA did not result in a s

37 cific pathogen-free or germ-free conditions, intragastric administration of Pseudomonas aeruginosa, E

38 4% of pancreatic protein secretion evoked by intragastric administration of rodent chow.

39 Intragastric administration of soluble protein Ags resul

40 Intragastric administration of solutions prepared from o

41 this study we use a murine model of ethanol intragastric administration which has been widely used t

42 ion, given by voluntary (pair) feeding or by intragastric administration, affected the peptidase acti

43 and the recording continued for 2 hours with intragastric air infusion of 15 mL/min.

44 6-fold increase in TNFR1 knockout mice with intragastric alcohol exposure for 4 weeks.

45 ion to hyperhomocysteinemia and ER stress in intragastric alcohol fed mice.

46 ial respiration increased with both oral and intragastric alcohol feeding despite extensive N-acetyla

47 Intragastric alcohol feeding to mice resulted in 1) incr

48 , lower than the striking increase caused by intragastric alcohol feeding.

49 lic liver injury was studied in the model of intragastric alcohol-fed mice.

50 pic dermatitis-like skin lesion, followed by intragastric allergen challenge to induce experimental f

51 l activation, splenocyte proliferations, and intragastric allergen challenge.

52 Interestingly, exposure of mice to oral or intragastric allergen does not promote eosinophilic esop

53 ow was already only 60% of normal flow, both intragastric and intravenous ethanol decreased both panc

54 n mutants were severely attenuated following intragastric and intravenous inoculation of mice.

55 hypothesized that both GALT-depleting diets (intragastric and intravenous TPN) would impair immunity

56 Repeated intragastric antigen challenge induced a significant inc

57 Repeated intragastric antigen challenge induced MMC9 development

58 OB at UBT, which is an indirect indicator of intragastric bacterial load.

59 Intragastric balloon (IGB) implantation is one of the me

60 Endoscopic procedures (ie, intragastric balloon and endoscopic sleeve gastroplasty)

61 tulated that orogastric contamination of the intragastric balloon may cause this phenomenon.

62 Over time, the intragastric balloon might influence the distribution an

63 Although conventional Intragastric Balloons (IGBs) have become an efficient an

64 Intragastric balloons are anatomy-preserving, minimally

65 trictive procedures, intestinal sleeves, and intragastric balloons have demonstrated short-term effic

66 Despite contaminating intragastric balloons with gastric aspirates, hyperinfla

67 Endoscopic bariatric techniques, such as intragastric balloons, have become an attractive alterna

68 In normal cats, intragastric, but not intravenous, ethanol reduced both

69 The results indicate that intragastric, but not voluntary, ethanol consumption dif

70 her high-fat control diet or ethanol diet by intragastric cannulation for 2 or 4 weeks.

71 ere fed ethanol or high-fat control diet via intragastric cannulation for 4 weeks.

72 automated infusion via surgically-implanted, intragastric catheters.

73 Twenty-one rats that received intragastric CCl(4) for 0-12 weeks were imaged with resp

74 LTB, or a combination of LT and LTB prior to intragastric challenge with H. pylori.

75 ve immunization with a CPB antibody prior to intragastric challenge.

76 Second, intragastric coinoculation with a wild-type Chlamydia re

77 longed to 7 days with the addition of K76 of intragastric CP at 5 mg/kg per day begun 1 day before op

78 d with intramuscular tacrolimus (TAC) and/or intragastric cyclophosphamide (CP) in rat recipients of

79 with 796 +/- 45 kcal; P = 0.08) in 20 women.Intragastric DB administration decreases both antral mot

80 0.04) plasma concentrations decreased after intragastric DB administration, whereas total and octano

81 In women (n = 10), intragastric DB switched the origin of phase III contrac

82 Strikingly, using intragastric delivery into fetal mouse intestine, preven

83 Direct intragastric delivery of a diet, nutrient or test substa

84 Both intranasal and intragastric delivery of EtxB were effective in preventi

85 The intrauterine but not the intragastric delivery of misoprostol significantly worse

86 eased bioavailability may limit the value of intragastric delivery of PPIs because of the high freque

87 20 mutant successfully targeted tumors after intragastric delivery, opening up the oral route as an o

88 ly visualize "H. heilmannii" types and their intragastric distribution.

89 re removal, the PED group was given a single intragastric dose of 1 mL saline, and the ALA and ALA+Vi

90 LA and ALA+Vit-C groups were treated with an intragastric dose of 50 mg/kg ALA and ALA+Vit-C for 15 d

91 cells were isolated 0 to 24 hours after one intragastric dose of ethanol daily, and intracellular Ca

92 ng animals in each group were given a single intragastric dose of NMBA at 2 mg/kg and sacrificed 12 w

93 Almost all of the mice given two intragastric doses also developed mucosal anti-H1 IgA an

94 After 6 wk, all rats received two intragastric doses of aspirin (1.4 mumol/kg body wt).

95 e rats (PD30-43) were given 10 intermittent, intragastric doses of ethanol (5.0 g/kg) or isovolumetri

96 ox+/- and Wwox+/+ mice were treated with six intragastric doses of N-nitrosomethylbenzylamine and obs

97 Our data show that multiple, intragastric doses of native OVA inhibited priming of CD

98 A and ZD+NMBA groups were treated with three intragastric doses of NMBA.

99 Intragastric emulsion instability was associated with a

100 sity-based tertiary care hospital, placed on intragastric enteral tube feeding through nasogastric or

101 However, intragastric ethanol administration caused a 35% to 40%

102 Therefore, in this study, a long-term intragastric ethanol feeding model was used to test the

103 alcohol-induced liver injury, the long-term intragastric ethanol feeding protocol developed by Tsuka

104 Therefore, a long-term intragastric ethanol feeding protocol was used here to t

105 lcoholic liver injury in the murine model of intragastric ethanol feeding.

106 ulation of LPS enterally administered to the intragastric ethanol infusion model.

107 The rodent Tsukamoto-French model of intragastric ethanol infusion was used.

108 Ethanol dependence was induced by passive intragastric ethanol infusions in C57BL/6J (B6) and DBA/

109 Intragastric exposure of germ-free mice to Sphingomonas

110 Direct intragastric exposure to infected meat (A/Vietnam/1203/0

111 inated motivation deficits during flavorless intragastric feeding and increased oral intake of low-fa

112 es with the degree of liver pathology in the intragastric feeding model, which leads to the hypothesi

113 antly in Ad.lacZ-treated animals by using an intragastric feeding model.

114 ed either a control or ethanol diet using an intragastric feeding model.

115 lesterol and saturated fat and the rest from intragastric feeding of alcohol diet without or with wee

116 We used a mouse model of continuous intragastric feeding of alcohol or an isocaloric diet.

117 oto-French method (which involves continuous intragastric feeding of an isocaloric diet or alcohol) i

118 rench mouse model, which involves continuous intragastric feeding of isocaloric diet or alcohol for 3

119 Tolerance was induced by intragastric feeding of low-dose CII to DBA/1 mice durin

120 day-1) continuously for up to 4 weeks via an intragastric feeding protocol.

121 We used the intragastric feeding rat model for alcoholic liver disea

122 We used the intragastric feeding rat model to assess the relationshi

123 ministered to critically ill patients during intragastric feeding to augment small intestinal nutrien

124 Test meals were fed through an intragastric feeding tube on Sprague-Dawley male rats af

125 gimens have taken more invasive routes using intragastric feeding tubes to infuse alcohol directly in

126 x d[-1]) continuously for up to 4 weeks via intragastric feeding using an enteral feeding model.

127 2 g/kg/d) continuously for up to 4 weeks via intragastric feeding using an enteral model developed by

128 re placement at a dose of 75 or 150 mg/kg by intragastric feeding.

129 -transferase activity (feeding of 3 mg/mouse intragastric for 4 days), whereas the elevation of hepat

130 eproduces the stress dampening, whereas oral intragastric gavage of sucrose is without effect.

131 (54)Mn uptake after intragastric gavage was markedly reduced in ffe/+ mice (

132 c mice with a sealed anus were inoculated by intragastric gavage with type D isolates, 7 of 10 type D

133 When inoculated into BALB/c mice by intragastric gavage, 7 of 14 type C isolates were lethal

134 ombinant wildtype Cyp c 1 or carp extract by intragastric gavage.

135 n gravid and nongravid female mice following intragastric (gavage) inoculation, namely, (i) disease s

136 healthy, conscious baboons before and after intragastric glucose administration during infusions of

137 tion of GLP-1 impaired the disposition of an intragastric glucose meal and this was at least partly a

138 eference for a noncaloric flavor paired with intragastric glucose.

139 Swallow-induced intragastric hyper-pressurization was associated with in

140 colony housing (CSC) paradigm that repeated intragastric (i.g.) administrations of a heat-killed pre

141 ated lymphoid tissue and in the periphery to intragastric (i.g.) and i.p. administration of SEB.

142 An intragastric (I.G.) infusion of alcohol (2.5 g/kg b.wt)

143 susceptibilities of A/J and C57BL/6 mice to intragastric (i.g.) inoculation with L. monocytogenes.

144 Within hours after intragastric (i.g.) inoculation, virus appears in the ga

145 dministering M. vaccae ATCC 15483(T) via the intragastric (i.g.) route to female mice prior to mating

146 learn to prefer a taste that was paired with intragastric (IG) carbohydrate infusions during 22 hr/da

147 the water drinking response that follows an intragastric (ig) load of hypertonic NaCl.

148 Intragastric (IG) TPN maintains antiviral defenses but o

149 fter 4 days of feeding chow, a complex diet, intragastric (IG)-PN, or PN.

150 persisted for up to 6 months, whereas after intragastric immunization, responsive T cells were not f

151 esenteric lymph nodes (MLN) and spleen after intragastric immunization.

152 The continuous intragastric in vivo enteral feeding model in the rat de

153 al sections taken from Peyer's patches after intragastric infection of mice showed that, in contrast

154 in rederived NF-kappaB-deficient mice using intragastric infection with Helicobacter hepaticus.

155 mal genes expressed in lymphoid tissue after intragastric infection.

156 This rodent intragastric infusion (iG) model has broad applications

157 libitum oral intake (half of the visits) and intragastric infusion (other half) of chocolate milkshak

158 were fed liquid control or ethanol diets by intragastric infusion for 4 weeks.

159 fed an ethanol-containing diet by continuous intragastric infusion for 42 days.

160 ssociated with ethanol hepatotoxicity in the intragastric infusion model of ethanol treatment.

161 ieber-DeCarli liquid diet feeding model, the intragastric infusion model, and the Gao-binge model.

162 learly dissociate the induction of CYP2E1 by intragastric infusion of ethanol from the generation of

163 lcohol-containing liquid diet and mice given intragastric infusion of ethanol).

164 Continuous intragastric infusion of ethanol-containing diets as par

165 We used an intragastric infusion of glucose and water to bypass the

166 e consumption (i.e., in both oral intake and intragastric infusion test sessions), whereas glucagon-l

167 urse experiment showed that after 2 weeks of intragastric infusion, a time point that results in isol

168 ebo during oral intake (P = 0.0005), but not intragastric infusion, of milkshake.

169 ated fat, palm oil, corn oil, or fish oil by intragastric infusion.

170 ning saturated fat, corn oil, or fish oil by intragastric infusion.

171 ich boluses daily or a continuous isocaloric intragastric infusion.

172 dematous children with PEM by using constant intragastric infusions of [2H3]leucine.

173 Specifically, intragastric infusions of glucose induced significantly

174 nses in accumbens and dorsal striatum during intragastric infusions of glucose or serine.

175 eferences for flavors paired with concurrent intragastric infusions of maltodextrin or corn oil and f

176 fer a flavor that was paired with concurrent intragastric infusions of maltodextrin.

177 g following either 22 h water deprivation or intragastric injection of hypertonic saline (1.5 M), att

178 Intragastric injection of S. typhimurium 14028 and SR-11

179 Adult male rats received intragastric injections of the vehicle or a moderately i

180 monoclonal anti-C5 i.p. on day 3 or 6 after intragastric inoculation and monitored for clinical sign

181 on in the liver and spleen of mice following intragastric inoculation and that mig-14 expression, whi

182 to colonize the gastrointestinal tract since intragastric inoculation did not rescue the mutants' col

183 ngolia/244/05, was able to infect mice after intragastric inoculation in liquid, whereas no evidence

184 (i) the noninfectious status developed after intragastric inoculation might be induced by a combinati

185 Here, we report that intragastric inoculation of a Shiga toxin 2 (Stx2)-produ

186 Intragastric inoculation of purified Stx2 also induced i

187 bacteria, whereas most control mice survived intragastric inoculation with 4 x 10(8) L. monocytogenes

188 Following intragastric inoculation with murine gamma-herpesvirus 6

189 Following an intragastric inoculation, the plasmid significantly impr

190 e of infection was observed in ferrets after intragastric inoculation.

191 ion genes during infection of mice following intragastric inoculation.

192 ng the gastrointestinal (GI) tract following intragastric inoculation.

193 ckling mouse GI tract at various times after intragastric inoculation.

194 We used a combination of intragastric, intrajejunal, and intracolon inoculations

195 ere exposed to ethanol (0 or 6 g/kg/day) via intragastric intubation from gestational day 8-20.

196 p to 27 mg/kg/d) in a high-fat diet using an intragastric intubation model for 28 days to male mice f

197 f ethanol (4.5g/kg body weight) delivered by intragastric intubation on PN4, PN4-6, or PN7-9.

198 g/kg of alcohol or an isocaloric solution by intragastric intubation once a day from gestational day

199 fected with candidate bacteria by oral gauge intragastric intubation.

200 ine or vehicle from gestational days 8-22 by intragastric intubation.

201 r dose of lipopolysaccharide plus continuous intragastric KIC, PYR, or HCO3.

202 However, combining upper endoscopy with intragastric laparoscopic surgery offers advantages of b

203 Intragastric lipid administration activated neurons in t

204 Intragastric lipid administration to CD36 mice released

205 Assessing intragastric meal distribution (IMD) during gastric empt

206 rulent type C strain CN3685 avirulent in the intragastric model and nearly nonlethal in the intraduod

207 Here, a sublethal intragastric mouse infection model using wild-type and c

208 pseudotuberculosis to cause mortality in an intragastric mouse model of infection, and a strain lack

209 d metatranscriptomics in ethanol-feeding and intragastric mouse models to investigate the metabolic i

210 After 4 weeks, animals were given a single intragastric NMBA dose and were sacrificed 25 and 77 day

211 lick microstructure analysis indicated that intragastric non-essential AAs and HON optostimulation e

212 eks later, the mice were randomized to chow, intragastric Nutren (Clintec, Chicago, IL), intravenous

213 We show that intragastric nutrient infusion rapidly and durably inhib

214 ke peptide-1 levels before and for 4 h after intragastric nutrients during a control study and on 2 d

215 onally-relevant stimuli including food cues, intragastric nutrients, cholecystokinin and ghrelin.

216 ive absorption) and 5 h (steady state) after intragastric olive oil (70% triolein).

217 se liver or spleen at 2 and 4 days following intragastric or intraperitoneal infection.

218 nt strains from livers and spleens following intragastric or intraperitoneal infection.

219 markably, however, within 20 min after acute intragastric or intravenous glucose delivery (with blood

220 ro on epithelial cells and in vivo following intragastric or intravenous infusion in parenterally fed

221 In anesthetized mice, after intragastric or intravenous saline delivery, pancreatic

222 we immunized female mice by the intranasal, intragastric, or intraperitoneal route with purified OMV

223 ethyl alcohol was administered by the oral, intragastric, or intravenous route.

224 Eritoran administration via intracolonic, intragastric, or intravenous routes significantly reduce

225 l vaccination regimens involving intranasal, intragastric, or rectal routes of immunization and found

226 well as OVA-induced diarrhea occurrences on intragastric OVA challenges.

227 VA/alum-sensitized mice to repeated doses of intragastric OVA induced genetically restricted, dose-de

228 characterized steatohepatitis (SH) caused by intragastric overfeeding in mice.

229 Intragastric overfeeding reveals insights into the homeo

230 ted by THC during both oral (P = 0.0002) and intragastric (P = 0.0055) administration.

231 in vitro as well as against anaphylaxis upon intragastric peanut challenge in a peanut allergy mouse

232 Results for percentage of time with intragastric pH <3 were similar.

233 rion was 24-h percent of time post dose with intragastric pH above 3; secondary criteria were percent

234 ation occurred faster to produce homogeneous intragastric pH and deactivated amylase.

235 Acid-suppressive regimens that elevate the intragastric pH and maintain the pH over time have the p

236 Intragastric pH and serum gastrin were monitored 1 hour

237 pump inhibitors can elevate and maintain the intragastric pH at >6.

238 H 5 to mimic the fasted-to-fed transition in intragastric pH in rats.

239 data suggested that agents that elevate the intragastric pH may increase the susceptibility of patie

240 However, the resulting increase of intragastric pH may predispose to gastric cancer by allo

241 Twelve volunteers underwent overnight intragastric pH monitoring after 7 days of treatment wit

242 bedtime reduced the percentage of time with intragastric pH of <4 from 48% to 31% (P < 0.005) compar

243 through the mucus may be appropriate at the intragastric pH of the fasted, but not fed, animal.

244 he United States, is as effective in raising intragastric pH on the first day as a continuous infusio

245 enting with overt bleeding due to ulcers had intragastric pH probes placed after endoscopy and baseli

246 Intragastric pH studies have demonstrated that, whereas

247 proton pump inhibitors to raise and maintain intragastric pH suggest that, although both can raise th

248 Acid breakthrough, or decrease in intragastric pH to <4 for an hour or longer, occurs in t

249 volunteers to document regional variation in intragastric pH under both fasting and postprandial cond

250 Intragastric pH was > 6 for > 60% of the study period in

251 Intragastric pH was > 6 for 67.8% of the study period wi

252 Intragastric pH was maintained at 4.0 by in vivo intraga

253 th sucralfate, an agent that does not affect intragastric pH.

254 Multi-channel pH-monitoring assessed intragastric pH.

255 tion and colonization over the wide range of intragastric pHs experienced by the organism.

256 ring the first 4 hours after intravenous and intragastric Pi loading in rats.

257 This was accomplished by monitoring intragastric pressure (gastric tone) and contractility o

258 timulated gastric contractions and increased intragastric pressure (IGP).

259 ndex (BMI) and waist circumference (WC) with intragastric pressure (inspiration, BMI [r = 0.57], WC [

260 The belt increased intragastric pressure by a median of 6.9 mmHg during fas

261 Without the belt, intragastric pressure correlated with waist circumferenc

262 LES relaxation pressure, and intraesophageal-intragastric pressure gradient.

263 tric distension caused a smaller increase in intragastric pressure in response to gastric distension

264 stric distension (6 ml) evoked a much larger intragastric pressure in the denervated, vascularly isol

265 ) evoked an increase of 9.0 +/- 1.0 cmH2O of intragastric pressure in vivo.

266 The intragastric pressure rise inducing this effect is well

267 In addition, intragastric pressure was lower in the prone position (p

268 ate of 100 mL/min, with pauses for measuring intragastric pressure, until no further distension was t

269 ition of gastric motility and an increase in intragastric pressure.

270 sally, and simultaneous intra-esophageal and intragastric pressures were measured over 6-8 respirator

271 ong strains; often correlates with different intragastric regions and evolves during chronic infectio

272 tect BALB/c mice against intraperitoneal and intragastric ricin challenges.

273 S. typhimurium for infection of mice by the intragastric route but not the intraperitoneal route, an

274 e to be an important virulence factor by the intragastric route of infection in mice.

275 re immunized by either the intranasal or the intragastric route with a single dose of 10(9) or 10(10)

276 ation, and lethality in mice infected by the intragastric route.

277 the prototype arenavirus, implicate oral and intragastric routes as natural routes of infection.

278 f the current study was to establish whether intragastric self-administration of fat emulsions, that

279 aintenance, extinction, and reinstatement of intragastric self-administration of lipid emulsions to d

280 linical models of acute toxin-associated and intragastric, spore-induced colonic disease.

281 We aimed to visualize the influence of the intragastric stability of fat emulsions on their dynamic

282 Intragastric stimulation of dopamine release seemed to d

283 reases significantly after administration of intragastric sucrose, and deletion of the NMDA receptor

284 and provide insight into mechanisms used for intragastric survival.

285 agastric pH was maintained at 4.0 by in vivo intragastric titration with 0.3N sodium bicarbonate for

286 Both intravenous and intragastric total parenteral nutrition (TPN) produce GA

287 e complex enteral diet group (n = 5) and the intragastric TPN group (n = 5) after 30 minutes of gut i

288 r 30 minutes of gut ischemia, the IV-TPN and intragastric TPN groups showed a higher death rate than

289 in leak between the complex enteral diet and intragastric TPN groups.

290 emia (30 minutes) eliminated any benefits of intragastric TPN on survival.

291 Intragastric TPN partially preserved this respiratory im

292 trition (TPN) produce GALT atrophy, but only intragastric TPN preserves established antiviral immunit

293 Mice were randomized to chow, IV-TPN, intragastric TPN, or complex enteral diet for 5 days' fe

294 e randomized to chow, complex enteral diets, intragastric TPN, or intravenous TPN.

295 (Clintec, Chicago, IL), intravenous TPN, or intragastric TPN.

296 In contrast, intragastric treatment evoked a brief, modest elevation

297 Mice fed alcohol by intragastric tube were assayed for serum alanine aminotr

298 ion via different routes (intraperitoneal vs intragastric), we noted a difference in the time course

299 response that was protective against lethal intragastric Y. pseudotuberculosis challenge.

300 nc-replenished esophagi after treatment with intragastric zinc compared with zinc-sufficient esophagi