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1 rane proteins and the formation of endosomal intraluminal vesicles.
2 reduces amyloid precursor protein sorting to intraluminal vesicles.
3 hat the EGFR complex is sequestered in these intraluminal vesicles.
4 mber of enlarged LE/MVBs densely packed with intraluminal vesicles.
5 h lysosomes, resulting in degradation of the intraluminal vesicles.
6 LV, targeted to a subpopulation of lysosomal intraluminal vesicles.
7 diverse conformations and frequently contain intraluminal vesicles.
8 escribe intermediate morphologies of nascent intraluminal vesicles.
9 ion of multivesicular-body membranes to form intraluminal vesicles(4,5).
10 ge, autophagosome-like organelles containing intraluminal vesicles and hemifused vesicles, structural
11 here it plays a key role in the formation of intraluminal vesicles and in lipid sorting.
12 al and chemical hallmarks of LROs, including intraluminal vesicles and metal deposits, similar to mel
13  in early and late endosomes associated with intraluminal vesicles and released from tumor cells in s
14  in that the organelles are enlarged and the intraluminal vesicles are almost completely absent and t
15 owed waste products to accumulate, including intraluminal vesicles, autophagy protein LC3, and choles
16         We also found increased formation of intraluminal vesicles coupled with enhanced release of e
17 s necessary for sorting surface receptors to intraluminal vesicles for signal silencing and lysosomal
18 nery that assists with cargo recruitment and intraluminal vesicle formation in MVBs.
19 nt biological processes, including endosomal intraluminal vesicle formation, HIV budding and cytokine
20 ions, such as membrane fusion, scission, and intraluminal vesicle formation, potentially overlooking
21 sates that associate with membranes to drive intraluminal vesicle formation.
22 d the ESCRT components that are required for intraluminal vesicle formation.
23              They are thought to derive from intraluminal vesicles formed in late endosomal multivesi
24               A mechanistic role for Vps4 in intraluminal vesicle (ILV) formation has been unclear.
25  orientations that lead to the production of intraluminal vesicles (ILVs) and recycling tubules.
26 ed for lysosomal degradation are sorted into intraluminal vesicles (ILVs) at endosomes by endosomal s
27 function at the endosome in the formation of intraluminal vesicles (ILVs) containing cargo proteins d
28     Many lysosome-related organelles contain intraluminal vesicles (ILVs) enriched in CD63 that are s
29  the limiting membrane of the MV-lEVs, their intraluminal vesicles (ILVs) escaped to the extracellula
30 NF7 and VPS4, and modulates the formation of intraluminal vesicles (ILVs) inside MVEs.
31                                          The intraluminal vesicles (ILVs) of endosomes mediate the de
32            This requires EGFR sorting to the intraluminal vesicles (ILVs) of multi-vesicular endosome
33 ble for sorting ubiquitinated receptors into intraluminal vesicles (ILVs) of multivesicular bodies (M
34  (ESCRT)-0, -I, -II, and -III complexes into intraluminal vesicles (ILVs) of multivesicular bodies (M
35 port (ESCRT) machinery, and then sorted into intraluminal vesicles (ILVs) of multivesicular bodies (M
36 ent, ESCRT/ALIX-dependent incorporation onto intraluminal vesicles (ILVs) of MVBs.
37 s of either protein blocks EGFR sorting into intraluminal vesicles (ILVs) of the multivesicular body.
38 nes caused APP redistribution from endosomal intraluminal vesicles (ILVs) to the endosomal limiting m
39 sg101 that are required for the formation of intraluminal vesicles (ILVs) within late endosomes.
40 ed, targeted to endosomes, internalized into intraluminal vesicles (ILVs), and excreted in exosomes.
41 vesicles (EVs) that originate from endosomal intraluminal vesicles (ILVs), have emerged as a new inte
42  plasma membrane results in the secretion of intraluminal vesicles (ILVs), or exosomes.
43 n the endosomal system, through formation of intraluminal vesicles (ILVs), which are subsequently rel
44 zed in punctate, dot-like structures, likely intraluminal vesicles (ILVs).
45 , endosomes are enlarged and fail to acquire intraluminal vesicles (ILVs).
46 osed EGFR before the receptor is sorted onto intraluminal vesicles (ILVs).
47 ecruited to sort the ubiquitinated Ypq1 into intraluminal vesicles (ILVs).
48 multaneously deform the membrane to generate intraluminal vesicles (ILVs).
49 d budding, which results in the formation of intraluminal vesicles (ILVs).
50  increases the formation of sphingosine-rich intraluminal vesicles (ILVs).
51 , such proteins are sorted at endosomes into intraluminal vesicles (ILVs).
52 PDH is required for the normal generation of intraluminal vesicles in endosomal compartments, and pro
53                                Cells release intraluminal vesicles in multivesicular bodies as exosom
54 K9 function leads to decreased number of the intraluminal vesicles in MVBs and diminished release of
55 mbrane that resulted in the release of their intraluminal vesicles in the extracellular space.
56 icular body-like organelle that releases its intraluminal vesicles in the vicinity of ingressing furr
57 rane remodeling and the release of endosomal intraluminal vesicles into multivesicular bodies.
58          In eukaryotic cells, the budding of intraluminal vesicles (IVLs) is mediated by the endosoma
59  sorting of amyloid precursor protein to the intraluminal vesicles of endosomes and enhances amyloid-
60 he inclusions, indicating their origins from intraluminal vesicles of late endosomes and of a lysosom
61 eveal that the localization of MHC-II on the intraluminal vesicles of multivesicular antigen processi
62  Here we show that SIMPLE resides within the intraluminal vesicles of multivesicular bodies (MVBs) an
63 e neck of caveolae, microvilli/filopodia and intraluminal vesicles of multivesicular bodies (MVBs).
64 ery controls the incorporation of cargo into intraluminal vesicles of multivesicular bodies.
65 ) are recognized, clustered, and sorted into intraluminal vesicles of multivesicular endosomes by end
66  imaging resolves Arc accumulates within the intraluminal vesicles of MVB.
67   When EGF.EGFR complexes accumulated in the intraluminal vesicles of the late endosome, phosphorylat
68 s that RAB7 is not required for formation of intraluminal vesicles of the LE/MVB, since RAB7-deficien
69 tetraspanin family member highly enriched in intraluminal vesicles tagged with GFP, to track changes
70 nto lysosomal membranes and the formation of intraluminal vesicles through microautophagy.
71 some secretion by preventing the delivery of intraluminal vesicles to lysosomes.
72                             The formation of intraluminal vesicles was not significantly detected in
73 gulfment of cytosolic proteins and RNAs into intraluminal vesicles which are then secreted as exosome
74 osomes are nano-sized endosome-derived small intraluminal vesicles, which are important facilitators
75                                      Loading intraluminal vesicles with specific miRNAs and releasing
76 c analysis suggests that, instead of forming intraluminal vesicles with the help of Vps4, ESCRT-III/S
77                        They are generated as intraluminal vesicles within endosomal compartments that
78                A major source of exosomes is intraluminal vesicles within multivesicular endosomes th
79 ult in the arrested accumulation of enlarged intraluminal vesicles within synaptic boutons.