ライフサイエンスコーパス: intramucosal

1 Those patients who died developed intramucosal acidosis of the sigmoid colon intraoperativ

2 hagectomy for high-grade dysplasia (HGD) and intramucosal adenocarcinoma (IMC) in light of recent adv

3 patients with high-grade dysplasia (HGD) or intramucosal adenocarcinoma (IMC) is well-established in

4 Intramucosal adenocarcinoma and high grade dysplasia hav

5 ing was found in villous adenomas with focal intramucosal adenocarcinoma but was negative in tubular

6 Survival with intramucosal adenocarcinoma or Barrett's with high-grade

7 ospective review of outcome in patients with intramucosal adenocarcinoma or high grade dysplasia that

8 is the preferred procedure for patients with intramucosal adenocarcinoma or high grade dysplasia.

9 l and en bloc esophagectomy in patients with intramucosal adenocarcinoma or high-grade dysplasia.

10 robability of having high-grade dysplasia or intramucosal adenocarcinoma was 0% (0-4).

11 robability of having high-grade dysplasia or intramucosal adenocarcinoma was 87% (73-95).

12 metastases are rare ( approximately 2%) with intramucosal adenocarcinoma, supporting the use of endos

13 tion with high-grade Barrett's esophagus and intramucosal adenocarcinoma.

14 dysplasia and 22 had high-grade dysplasia or intramucosal adenocarcinoma.

15 m 8 patients who underwent esophagectomy for intramucosal adenocarcinoma.

16 To prove this concept, trans-/intramucosal and lymph-node delivery of PLGA-PEG nanopar

17 define the risk of lymph node metastases for intramucosal and submucosal (T1) esophageal adenocarcino

18 These intramucosal B cells expressed AHNAK and CD43, suggestin

19 To validate the presence of intramucosal bacteria ex vivo, CLE was performed in pure

20 In addition, CLE allowed the detection of intramucosal bacteria in vivo.

21 jured intestinal epithelium and clearance of intramucosal bacteria require the presence of intact TLR

22 econd objective was to prove the presence of intramucosal bacteria using CLE.

23 sitivity and specificity for the presence of intramucosal bacteria were 100%.

24 In addition, intramucosal bacteria were visualized.

25 iopsies from the duodenal bulb showed higher intramucosal Brunner's gland scores and lower intraepith

26 .19; 95% confidence interval [CI]: 1, 1.41), intramucosal Brunner's glands with MPO (exp.

27 epletion, Paneth cell depletion, and reduced intramucosal Brunner's glands) were required to create a

28 of 39 patients) for high-grade dysplasia or intramucosal cancer and 44% (17 of 39) for any grade of

29 d by treatment of patients with dysplasia or intramucosal cancer costs an additional $240 (95% credib

30 The risk of high-grade dysplasia or intramucosal cancer in the high-risk group was 52% (68 o

31 tients found to have high-grade dysplasia or intramucosal cancer received endotherapy.

32 Patients with BE and HGD/intramucosal cancer were studied prospectively from 2002

33 d by treatment of patients with dysplasia or intramucosal cancer would reduce the number of cases of

34 ith BE and neoplasia (72% with HGD, 24% with intramucosal cancer, 4% with low-grade dysplasia [mean a

35 tive endoscopic treatments for dysplasia and intramucosal cancer, coupled with screening programs to

36 ectomy management of high-grade dysplasia or intramucosal cancer, screening by ultrathin nasal endosc

37 tients treated with ablative therapy for HGD/intramucosal cancer.

38 to accelerated (< or = 8 mo) development of intramucosal carcinoma (85%), with submucosal invasion (

39 gressed to high-grade dysplasia and invasive intramucosal carcinoma by 20 months.

40 Lesions consistent with in situ and intramucosal carcinoma were seen in H. pylori-infected m

41 newly diagnosed adenomas, carcinoma in situ, intramucosal carcinoma, and colorectal cancer), they fou

42 cinoma in situ, OR = 2.05, 95% CI 1.01-4.15; intramucosal carcinoma, OR = 1.30, 95% CI 0.61-2.77; and

43 d only WT(Hc) infection caused dysplasia and intramucosal carcinoma.

44 nclude that GCB is associated with selective intramucosal colonization by E. coli.

45 ) had baseline HGD, and 3 (33%) had baseline intramucosal EAC.

46 rred over esophagectomy for BE patients with intramucosal esophageal adenocarcinoma (T1a).

47 hial anastomosis after lung transplantation, intramucosal esophageal dissection, Boerhaave syndrome,

48 Intramucosal gram-negative coccobacilli were present in

49 Furthermore, an increase in gastric intramucosal hydrogen ion concentration was associated w

50 n = 5) collected before and 20 minutes after intramucosal injection of various food extracts in the e

51 These intramucosal nerve fibers may be a conduit by which intr

52 Intramucosal organisms harvested from sinonasal biopsies

53 Continuous monitoring of gastric intramucosal PCO2 and pH is feasible, has potential adva

54 l method for continuously monitoring gastric intramucosal PCO2 and pH was developed and tested.

55 In the animal experiments, gastric intramucosal PCO2 and pH were determined before and afte

56 Intramucosal PCO2 by both techniques remained significan

57 By 5 mins after inducing hemorrhage, intramucosal PCO2 by recirculating gas tonometry had inc

58 The technique is able to detect changes in intramucosal PCO2 in response to an induced insult over

59 he conventional method showed an increase in intramucosal PCO2 to 63.0 +/- 20.9 torr (8.4 kPa +/- 2.8

60 , arterial and mixed venous blood gases, and intramucosal PCO2 values were obtained by recirculating

61 gradient between arterial P(CO2) and gastric intramucosal Pco2 was 13+/-3 mm Hg (1.7+/-0.4 kPa) at a

62 Mean +/- SD baseline intramucosal PCO2 was 47.6 +/- 9.5 torr (6.3 +/- 1.3 kPa

63 Gastric intramucosal PCO2 was recorded continuously by capnometr

64 Significant increases in gastric intramucosal PCO2 were detectable by CRGT within 5 min o

65 rfusion by providing on-line measurements of intramucosal PCO2, which can also be used to derive intr

66 % confidence limits, -1.66, 1.72) or gastric intramucosal pH (mean difference, 0.015 pH units; 95% co

67 Hemodynamics, gastric intramucosal pH (pHi) urine volumes, urinary sodium excr

68 inactivation significantly improved gastric intramucosal pH after aortic occlusion and reperfusion (

69 There were significant correlations between, intramucosal pH and EndoCab antibodies, intramucosal pH

70 feedings for 2 hrs, after which the gastric intramucosal pH and gastric fluid pH measurements were r

71 The baseline fasting gastric intramucosal pH and gastric fluid pH were determined in

72 een, intramucosal pH and EndoCab antibodies, intramucosal pH and p55 TNF receptor, and p55 TNF recept

73 nsitive than arterial lactate and tonometric intramucosal pH and Pco2.

74 esulted in a significant decrease in gastric intramucosal pH as compared with sham-operated rabbits (

75 The mean +/- SD gastric intramucosal pH decreased from 7.40 +/- 0.08 to 7.33 +/-

76 After 30 mins of hemorrhage, intramucosal pH decreased to 6.88 +/- 0.14 (from 7.10 +/

77 Finally, we hypothesized that gastric intramucosal pH is influenced by xanthine oxidase, an ox

78 After induction of anesthesia, intramucosal pH of the sigmoid colon was measured using

79 ating the systemic markers to changes in the intramucosal pH of the sigmoid colon.

80 feeding results in a spuriously low gastric intramucosal pH reading.

81 The gastric intramucosal pH returned to the baseline value after 1 h

82 Gastric intramucosal pH values improved during reperfusion after

83 Gastric intramucosal pH was significantly decreased after hepato

84 1 +/- 1.2 vs. 5.3 +/- 0.7 kPa) and the ileal intramucosal pH was significantly lower (7.07 +/- .28 vs

85 nsumption, SMA blood flow, ileal Pco2, ileal intramucosal pH, and serum lactate levels of mixed venou

86 to arterial Pco2 gap (Pg-Paco2 gap), gastric intramucosal pH, arterial pH, arterial base excess, and

87 Gastric intramucosal pH, as determined by recirculating gas tono

88 nsfusion period for Pg-Paco2 gap and gastric intramucosal pH, respectively, was 0.56 kPa (95% confide

89 rtate aminotransferase and decreased gastric intramucosal pH, was significantly (p < 0.0167) attenuat

90 cosal hypercarbia, leading to improvement of intramucosal pH.

91 cosal PCO2, which can also be used to derive intramucosal pH.

92 for at least 1 hr when measuring the gastric intramucosal pH.

93 re was a concomitant significant decrease in intramucosal pH.

94 specimens were examined for the presence of intramucosal S. aureus and characterized to the strain l

95 The primary outcome was detection of intramucosal signet ring cell carcinoma foci.

96 total gastrectomy which revealed multifocal intramucosal signet ring cell carcinoma.

97 atients (85%) demonstrated 1 or more foci of intramucosal signet ring cell gastric cancer in the exam

98 oid excluding pathogenesis of tumorlets from intramucosal spread from carcinoid tumors.

99 e in interest in endoscopic therapy (ET) for intramucosal (T1a) or submucosal (T1b) esophageal carcin

100 Intramucosal terminals were not assessed.

101 Increase of intramucosal transforming growth factor alpha (TGFalpha)

102 in the given period, excluding patients with intramucosal tumors (n = 47), palliative resections (n =

103 Lymph node metastases were rare (1.3%) with intramucosal tumors but increased significantly with sub