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1 ton's disease, with the presence of neuronal intranuclear inclusions.
2                  Histology showed homogenous intranuclear inclusions.
3 the proteasome after it is incorporated into intranuclear inclusions.
4  occurred in a subset of huntingtin positive intranuclear inclusions.
5 loss and gliosis and the absence of neuronal intranuclear inclusions.
6 r neuron loss, with polyQ-AR accumulation in intranuclear inclusions.
7 l neurons contained ubiquitin-immunoreactive intranuclear inclusions.
8 both grey and white matter and also neuronal intranuclear inclusions.
9 icularly visceral neuropathies with neuronal intranuclear inclusions.
10 rs, including some characterized by neuronal intranuclear inclusions.
11 ith alterations in the formation of neuronal intranuclear inclusions.
12 eristic lentiform ub-immunoreactive neuronal intranuclear inclusions.
13 e, particularly in those cases with neuronal intranuclear inclusions.
14 gender-specific motor deficits, and neuronal intranuclear inclusions.
15 transglutaminase 2 and with huntingtin in HD intranuclear inclusions.
16 tologic disease with identifiable adenoviral intranuclear inclusions.
17 he nucleus, despite the presence of abundant intranuclear inclusions.
18  a dorsal to ventral gradient and occasional intranuclear inclusions.
19 t a common pathologic feature: ubiquitinated intranuclear inclusions affecting myocytes, osteoclasts,
20 ed extensively convoluted nuclear envelopes, intranuclear inclusions and chromatin clumps in cardiomy
21                                     Neuronal intranuclear inclusions and cytoplasmic aggregates compo
22                        Striatal and cortical intranuclear inclusions and cytoplasmic aggregates of mu
23 ve neuronal cytoplasmic inclusions, neuronal intranuclear inclusions and neurites were recorded and f
24 ese abnormalities, diffuse nuclear labeling, intranuclear inclusions and neuritic aggregates, all imm
25  N-terminal fragments are prone to form both intranuclear inclusions and neuritic aggregates.
26 containing protein underlies the presence of intranuclear inclusions and neurodegeneration in NIID.
27 hole mount immunohistochemistry, to identify intranuclear inclusions and quantify subsets of enteric
28                       Formation of fibrillar intranuclear inclusions and related neuropathologies of
29 nt of huntingtin has also been found to have intranuclear inclusions and this same fragment can aggre
30 nt huntingtin suppressed its ability to form intranuclear inclusions and to induce neurodegeneration.
31 axia, weight loss, premature death, neuronal intranuclear inclusions, and decreased tyrosine hydroxyl
32 ere distinct and temporally dissociated from intranuclear inclusions, and disappeared rapidly after c
33            Our results support the view that intranuclear inclusions are an integral part of the path
34                                     Neuronal intranuclear inclusions are found in the brains of patie
35                          These ubiquitinated intranuclear inclusions are morphologically similar to t
36         In contrast to other polyQ diseases, intranuclear inclusions are not prominent in SCA2.
37                                              Intranuclear inclusions are one of the ultrastructural h
38                                     Neuronal intranuclear inclusions are present in affected mice.
39                     We suggest that neuronal intranuclear inclusions are the common neuropathology fo
40 racterized by the presence of ubiquitinated, intranuclear inclusions associated with molecular chaper
41 hich is found in the neuronal and astrocytic intranuclear inclusions associated with the disorder.
42                                              Intranuclear inclusion bodies are the neuropathological
43 eliminated the formation of juxtanuclear and intranuclear inclusion bodies by HtEx1.
44 lly leading to deposition of cytoplasmic and intranuclear inclusion bodies containing htt.
45 pithelium and ballooning degeneration of and intranuclear inclusion bodies in epithelial cells, with
46 duct A or 1 (cp-A/1), form intracellular and intranuclear inclusion bodies in the brains of patients
47 lial cell (EC) damage and the presence of EC intranuclear inclusion bodies, demonstrating a direct vi
48 c of pan-cellular HD mouse models, including intranuclear inclusion bodies, motor impairment, and cha
49 as truncated fragments forming ubiquitinated intranuclear inclusion bodies.
50 vealed intranuclear inclusions suggestive of intranuclear inclusion body disease (NIID).
51                            Striatal neuronal intranuclear inclusion burden was similar between HD kno
52 ation) CGG repeat, we have demonstrated that intranuclear inclusions can be formed in both primary ne
53 this patient showed renal tubular cells with intranuclear inclusions characteristic of PV infection,
54 ure death that are characterized by abnormal intranuclear inclusions composed of TDP-43 and fused in
55   Given that MSP is associated with neuronal intranuclear inclusions comprised of TDP-43 protein, we
56 essing highly expanded AR form ubiquitinated intranuclear inclusions containing amino-terminal epitop
57                                      Whether intranuclear inclusions containing DNA damage response (
58 odels have previously been unable to produce intranuclear inclusions containing only a portion of the
59  model mimics the formation of ubiquitinated intranuclear inclusions containing the amino-terminal po
60  SCA3/MJD brain, the proteasome localized to intranuclear inclusions containing the mutant protein, a
61                                     Neuronal intranuclear inclusion disease (NIID) is a multisystem n
62                                     Neuronal intranuclear inclusion disease (NIID) is a neurodegenera
63                                     Neuronal intranuclear inclusion disease (NIID) is a slowly progre
64  identified as the genetic cause of neuronal intranuclear inclusion disease (NIID).
65 -associated tremor/ataxia syndrome, neuronal intranuclear inclusion disease, oculopharyngeal myopathy
66 mmunolocalized to both nuclear membranes and intranuclear inclusions, fluorescent detection of NADPH-
67 , nuclear mutant huntingtin accumulation and intranuclear inclusion formation.
68  hallmark of FXTAS is the ubiquitin-positive intranuclear inclusion found in neurons and astrocytes i
69                           Ubiquitin-positive intranuclear inclusions have also been found in the neur
70                                              Intranuclear inclusions have also been reported in the n
71                                     Neuronal intranuclear inclusions have been found in the brain of
72                                      Because intranuclear inclusions have recently been shown to be a
73                        Characteristic smudgy intranuclear inclusions, immunohistochemistry for viral
74  and mSin3a, and CBP to localize to neuronal intranuclear inclusions in a transgenic mouse model of H
75 aining performed in 3 gene carriers revealed intranuclear inclusions in all 3 cases, including 1, wit
76 FTLD with ubiquitin-positive cytoplasmic and intranuclear inclusions in all PGRN mutation carriers.
77 ds to determine the pathological features of intranuclear inclusions in astroglia and neurons.
78          Histopathological findings included intranuclear inclusions in bile duct epithelium, lymphoi
79 ebral white matter and (iii) the presence of intranuclear inclusions in both brain and spinal cord.
80 the number of CGG repeats and the numbers of intranuclear inclusions in both neurons and astrocytes,
81                  This protein accumulates in intranuclear inclusions in cell and mouse models and in
82                                 1C2-positive intranuclear inclusions in cerebellar Purkinje and brain
83                            The morphology of intranuclear inclusions in CGG KI mice was compared to t
84 st time, de novo formation of ChHV5-positive intranuclear inclusions in cultured green turtle cells,
85 d a severe tubulointerstitial nephritis with intranuclear inclusions in epithelial cells.
86 complete striatal degeneration and rarity of intranuclear inclusions in HD, and the fact that mice ex
87          The discovery last year of neuronal intranuclear inclusions in Huntington's disease and othe
88 rectly relevant to the formation of neuronal intranuclear inclusions in Huntington's disease.
89 a syndrome typically have cerebellar ataxia, intranuclear inclusions in neurons and astrocytes, as we
90 allmark of the disease is ubiquitin-positive intranuclear inclusions in neurons and astrocytes.
91 isease is the presence of ubiquitin-positive intranuclear inclusions in neurons and in astrocytes.
92  FXTAS is the presence of ubiquitin-positive intranuclear inclusions in neurons and in astroglia.
93           The models exhibit the presence of intranuclear inclusions in Purkinje neurons, Purkinje ne
94 tion of FUS but facilitated the formation of intranuclear inclusions in rat hippocampal neurons with
95 logy and by the late development of neuronal intranuclear inclusions in spinal neurons.
96 rative disease characterized by eosinophilic intranuclear inclusions in the nervous system and multip
97 que pathological feature - appearance of the intranuclear inclusions in the neurons and astrocytes, i
98  weight loss, cerebral atrophy, and neuronal intranuclear inclusions in the R6/2 transgenic mouse mod
99 colytic fibres, containing a large number of intranuclear inclusions (INIs).
100 e, we present evidence that the formation of intranuclear inclusions is a key event in cataract forma
101                                     However, intranuclear inclusions may reflect a cellular mechanism
102                                     Neuronal intranuclear inclusion (NII) formation and cell loss is
103    Patient autopsy material reveals neuronal intranuclear inclusions (NII) in affected regions that c
104  mutant huntingtin was localized to neuronal intranuclear inclusions (NIIs) and dystrophic neurites (
105 d hastened both the presentation of neuronal intranuclear inclusions (NIIs) and the onset of behavior
106 arly as 9 weeks of age and striatal neuronal intranuclear inclusions (NIIs) by 20 weeks.
107                                     Neuronal intranuclear inclusions (NIIs) characteristically occur.
108 lar localization and development of neuronal intranuclear inclusions (NIIs) in cortex and striatum of
109 nsgenic mice described here develop neuronal intranuclear inclusions (NIIs), a hallmark of SBMA and t
110 other than widespread ubiquitinated neuronal intranuclear inclusions (NIIs).
111 demonstrates widespread ubiquinated neuronal intranuclear inclusions (NIIs).
112 l dysregulation and accumulation of neuronal intranuclear inclusions (NIIs).
113 opment of both disease symptoms and neuronal intranuclear inclusions (NIIs).
114 , and in some familial cases UBQ-ir neuronal intranuclear inclusions of a lentiform appearance.
115  is impaired, even prior to the formation of intranuclear inclusions of aggregated AR.
116 ive disease characterized by the presence of intranuclear inclusions of unknown origin.
117 ed syncytial cells with intracytoplasmic and intranuclear inclusions of viral nucleocapsids, consiste
118    All four patients with BKVAN demonstrated intranuclear inclusions on allograft biopsy and a progre
119 y it is characterised by rimmed vacuoles and intranuclear inclusions on muscle biopsy.
120 active gliosis and the formation of neuronal intranuclear inclusions predominating in the striatum.
121 he neuropathological hallmark of FXTAS is an intranuclear inclusion, present in both neurons and astr
122 owed that the formation of polyQ-expanded AR intranuclear inclusions promoted neurite retraction, whi
123 clei contained discreet PABP2 immunoreactive intranuclear inclusions, providing the first direct evid
124 rotein ataxin-3 accumulates in ubiquitinated intranuclear inclusions selectively in neurons of affect
125 ippocampus mirror the appearance of neuronal intranuclear inclusions, suggesting a relationship betwe
126 mination showed hypoplastic bone marrow with intranuclear inclusions suggestive of human Parvovirus.
127 omedullary junction and skin biopsy revealed intranuclear inclusions suggestive of intranuclear inclu
128 models of Huntington's disease with neuronal intranuclear inclusions, the identification of different
129 se carriers demonstrated the presence of the intranuclear inclusions throughout the cerebrum and brai
130                                              Intranuclear inclusions were absent from Purkinje cells,
131                                     When CMV intranuclear inclusions were absent within BAL cells of
132                           Ubiquitin-positive intranuclear inclusions were also present in protoplasmi
133 clear mutant huntingtin and the formation of intranuclear inclusions were fastest in the C57BL/6 back
134           In CGG KI mice, ubiquitin-positive intranuclear inclusions were found in neurons (e.g., pyr
135    In female CGG KI mice, ubiquitin-positive intranuclear inclusions were found in neurons and astrog
136 in sarcoma-positive neuronal cytoplasmic and intranuclear inclusions were found in the hippocampal gr
137                                              Intranuclear inclusions were not considered a feature in
138                                              Intranuclear inclusions were only seen when the polyQ re
139                                Eosinophilic, intranuclear inclusions were present in both neuronal an
140                  Neuropil aggregates, but no intranuclear inclusions, were observed in the LGP and SN
141  proteins colocalized with viral DNA to form intranuclear inclusions, whereas VP proteins formed holl
142 o induce the formation of ubiquitin-positive intranuclear inclusions, which also stain positive for t
143 ne tract that is misfolded and prone to form intranuclear inclusions, which are the hallmark of the d
144 its in all the areas examined and widespread intranuclear inclusions, which were particularly numerou

 
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