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1 Ga-DOTATATE PET/CT can differentiate between intrapancreatic accessory spleens (IPAS) and pancreatic
2 the mechanisms responsible for the premature intrapancreatic activation of digestive enzyme zymogens.
3  provided compelling evidence that premature intrapancreatic activation of digestive proteases is cri
4 osphatidylinositol 3-kinase is necessary for intrapancreatic activation of trypsinogen and regulating
5 3-kinase inhibitor wortmannin can reduce the intrapancreatic activation of trypsinogen that occurs du
6 n thoracic spinal dorsal horn segments after intrapancreatic administration of proteinase-activated r
7 quantified following transplantation via the intrapancreatic and subrenal routes.
8                                              Intrapancreatic angiotensin II generation has been impli
9  Wistar rats, we observed that switching off intrapancreatic artery insulin infusions in vivo during
10                                    Increased intrapancreatic autoactivation of trypsinogen mutants ha
11 reatic duct cell, pancreatobiliary cell, and intrapancreatic bile duct cell homeostasis.
12 atic duct cells, pancreatobiliary cells, and intrapancreatic bile duct cells.
13 ion, biliary strictures were confined to the intrapancreatic bile duct in 51%; the proximal extrahepa
14  number of experimental studies suggest that intrapancreatic calcium concentrations play an important
15 sured simultaneously from 33 preparations of intrapancreatic canine ganglia and pancreatic parenchyma
16                          Here, we found that intrapancreatic CD8+ T cells progressively accumulate sp
17                               In particular, intrapancreatic CD8+ T cells specifically exhibit down-r
18 given cerulein, Ctrl-KO mice exhibited lower intrapancreatic chymotrypsin activation and a trend for
19                                              Intrapancreatic DCs acquired a distinct immune phenotype
20 ar cell injury, pancreatic inflammation, and intrapancreatic digestive enzyme (i.e., trypsinogen) act
21 cell differentiation from progenitors in the intrapancreatic duct (IPD).
22                          We examined whether intrapancreatic duct infusion of AAV containing an NF-ka
23 or of endocrine progenitors in the zebrafish intrapancreatic duct.
24 emical analyses support a model in which the intrapancreatic ductal system develops from progenitors
25 l (HPD) system connects the intrahepatic and intrapancreatic ducts to the intestine and ensures the a
26 tions of IgG1 isotype antibodies and reduced intrapancreatic expression of IFN-gamma, IL-6, and IL-17
27                                The amount of intrapancreatic fat (IPF) is increased in obese patients
28 dictors of type 2 diabetes mellitus included intrapancreatic fat percentage, pancreatic fractal dimen
29 lted in oil red O-positive areas, resembling intrapancreatic fat.
30 ed activation of parasympathetic cholinergic intrapancreatic ganglia and neurons doubled plasma-insul
31  circular muscle, and in fibers and cells in intrapancreatic ganglia.
32     These first simultaneous measurements of intrapancreatic ganglion activity and insulin secretion
33 reactivity was found in numerous enteric and intrapancreatic ganglion cells and in dense networks of
34 es in electrical activity in a population of intrapancreatic ganglion neurons.
35 nts with T1D exhibited pronounced inter- and intrapancreatic heterogeneity in signal intensity.
36  tumor angiogenesis and metastasis following intrapancreatic implantation with either PANC-1 or T3M4
37 d that genetic payloads can be delivered via intrapancreatic injection to target sites in efferent pa
38 e intestinal and pulmonary tracts were given intrapancreatic injections of Panc02 CEA(+) cells (expre
39                                              Intrapancreatic lavage catheter placement is essential t
40 te pancreatitis to account for nearly 15% of intrapancreatic leukocytes.
41 n of IFN-gamma, but not IL-4, was limited to intrapancreatic lymphocytes and was not detectable at ex
42 data distinguish macrophages within distinct intrapancreatic microenvironments and suggest how macrop
43                            In 8 patients, an intrapancreatic nasobiliary lavage catheter was placed i
44  the demonstration of the essential role for intrapancreatic nerves in mediating meal-induced respons
45         In addition, the endocrine pancreas, intrapancreatic nerves, and some extrapancreatic neural
46                 Perineural tumor invasion of intrapancreatic nerves, neurogenic inflammation, and tum
47 sults suggest that neurohormone release from intrapancreatic neurons could help synchronize islets in
48 sed from various cells within islets or from intrapancreatic neurons, are hypothesized to further adj
49  indirect and may be exerted at the level of intrapancreatic neurons.
50 ablished experimental evidence demonstrating intrapancreatic parasympathetic (cholinergic) ganglia an
51 tonin and the Rora agonist SR1078 attenuated intrapancreatic pathological changes in mouse models of
52                                              Intrapancreatic prostaglandin E(2) levels were reduced i
53         Terms such as pancreatic abscess and intrapancreatic pseudocyst have been abandoned.
54 action of cancer cells and their invasion of intrapancreatic sensory nerves.
55 a single clone with subsequent intramural or intrapancreatic spread.
56 inically optimize trypsin inhibitors towards intrapancreatic target inhibition.
57  was associated with increased percentage of intrapancreatic Tregs.
58 l type 1 (SPINK1) protects the pancreas from intrapancreatic trypsin activation that can lead to panc
59                                Inhibition of intrapancreatic trypsin activity is an attractive yet so
60 ancreatic oedema, acinar cell vacuolization, intrapancreatic trypsin activity, cell death signalling
61 m against pancreatitis through regulation of intrapancreatic trypsin activity.
62 eatitis by eliminating prematurely activated intrapancreatic trypsin.
63 these regulatory cleavages lead to increased intrapancreatic trypsinogen activation and cause heredit
64                The relative contributions of intrapancreatic trypsinogen activation and nuclear facto
65 epsin B is thought to play a central role in intrapancreatic trypsinogen activation and the onset of
66  is the dominant mechanism that can mitigate intrapancreatic trypsinogen activation.
67 in IAC and compare relapse rates in IAC with intrapancreatic vs proximal bile duct strictures.