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1  not known, however, whether a population of intratubular adult progenitor cells are responsible for
2 mechanisms responsible for the regulation of intratubular AngII concentrations remain to be determine
3  flow-dependent K(+) secretion and increased intratubular availability of Na(+) that can be reabsorbe
4 eas no significant change is observed in the intratubular axial or equatorial dimensions.
5 thy (CN), a disease that is characterized by intratubular bile casts and tubular injury.
6 ated with rapamycin and tacrolimus developed intratubular cast formation indistinguishable from myelo
7  to determine the incidence of ATN, ATN with intratubular casts, and casts with the classic myoglobin
8 bular CD8+ and CD103+ cells and the ratio of intratubular CD103+ to CD8+ cells increased significantl
9 ells expressed IL-15 constitutively, whereas intratubular CD8 T cells expressed IL-15 receptor alpha.
10                               The numbers of intratubular CD8+ and CD103+ cells and the ratio of intr
11                              CD8+ and CD103+ intratubular cells were enumerated as mean numbers per t
12  that circulating gonadotropin can reach the intratubular compartment.
13  recent findings of remarkably high proximal intratubular concentrations of AngII (in the range of 10
14                                     The high intratubular concentrations of AngII, as well as angiote
15                      In the stenotic kidney, intratubular contrast content has decreased in all nephr
16 age inflicted on tubular epithelial cells by intratubular cytotoxic T lymphocytes.
17 ed occluded sites with mineral deposition of intratubular dentin.
18  OPN-deficient mice demonstrated significant intratubular deposits of CaOx crystals, whereas wild-typ
19 w, tubular dilation, water reabsorption, and intratubular flow all play important roles in diffusion-
20 ci were detected in three patients, two with intratubular germ cell neoplasia and one with sex cord t
21  confirmed to be benign; however, a focus of intratubular germ cell neoplasia was found in a Sertoli
22 lting in germ cell depletion, hypofertility, intratubular germ cell neoplasias, and seminoma developm
23  do not produce such plaque but instead form intratubular hydroxyapatite crystals in collecting ducts
24 nal production is a limiting step in shaping intratubular lesions and tumor expansion both in a mouse
25  testis, including ectopic Sertoli cells and intratubular Leydig cells (ITLCs).
26 ells in all specimens were biased towards an intratubular localization.
27 mokines can influence the composition of the intratubular lymphocyte population.
28 on and function of CD103 observed in situ on intratubular lymphocytes.
29 Histopathologic analysis at autopsy revealed intratubular microlithiasis without the calcification of
30 es were unaltered in transparent dentin, and intratubular mineral had values between those of normal
31 torted tubules without peritubular dentin or intratubular mineral.
32                  All of these mice developed intratubular neoplasia by 3 months, which progressed to
33        Neutrophilic tubulitis accompanied by intratubular neutrophil clusters in the renal allograft
34          This mechanism links proteinuria to intratubular proinflammatory signaling.
35    These findings provide no evidence for an intratubular stem-cell population, but rather indicate t
36 ature of renal allograft rejection with many intratubular T cells expressing CD8 and CD103 (the alpha
37 on of the alphaEbeta7-integrin by activated, intratubular T cells in acute renal allograft rejection
38 the renal allograft, a similar proportion of intratubular T cells was observed to proliferate during
39 in (IL)-15Ralpha, to enumerate proliferating intratubular T cells, and to quantify IL-15 expression w