ライフサイエンスコーパス: invasion

1 translocation to promote cell migration and invasion.

2 ential therapeutic target for inhibiting GBM invasion.

3 e gene regulatory network that promotes cell invasion.

4 lymph nodes, perineural invasion, and venous invasion.

5 stimulates SSA activity and promotes strand invasion.

6 iferation, induces apoptosis, and attenuates invasion.

7 -actinin 4 is required to promote tumor cell invasion.

8 activation that, in turn, promotes melanoma invasion.

9 ulator of genes important for red blood cell invasion.

10 pithelia (FAE) to limit entry points for STm invasion.

11 evented Akt2 polarization and Gln-driven CAF invasion.

12 ggesting a metabolically active state during invasion.

13 decreased survival, increased metastasis and invasion.

14 r microscopic tumor involvement and capsular invasion.

15 d leader cell collapse and halted collective invasion.

16 gen deprivation, anchorage independence, and invasion.

17 ional resistant alleles do not prevent drive invasion.

18 SRPs can suppress AR-antagonist-driven tumor invasion.

19 -DLL4 signaling in collective bladder cancer invasion.

20 correlated with a reduction in migration and invasion.

21 ta)-induced breast cancer cell migration and invasion.

22 ted in increased susceptibility to bacterial invasion.

23 ribosomal protein S6 kinase A1 drives tumor invasion.

24 A arising from genotoxic stress and pathogen invasion.

25 ion, MMP-2 transcription and, ultimately, BC invasion.

26 le for water channel protein function during invasion.

27 mpounds that suppress cancer cell spread and invasion.

28 (CAF) that facilitate epithelial tumor cell invasion.

29 bryonic development, wound repair, and tumor invasion.

30 tein and accumulates at the site of ookinete invasion.

31 and type II IFNs on S. aureus adherence and invasion.

32 s acidic tumor regions, for instance, during invasion.

33 ing cell proliferation FASN may also promote invasion.

34 and showed increased matrix degradation and invasion.

35 age, lymphatic invasion, and venous vascular invasion.

36 s HGF-mediated cell migration and reduces 3D invasion.

37 cacy and chemotactic superiority accelerates invasion.

38 to functional changes in cell migration and invasion.

39 bolic homeostasis and resistance to pathogen invasion.

40 macrophages (TAMs) promote tumor growth and invasion.

41 thelial cancers and strongly correlated with invasion.

42 ry and sufficient for RMS cell migration and invasion.

43 thway in leader cell function and collective invasion.

44 ropism, which results in frequent perineural invasion.

45 dynamics to promote tumor cell migration and invasion.

46 neuronal plasticity, development, and viral invasion.

47 frequency LFP before and after local seizure invasion.

48 as developed areas are more prone to species invasion.

49 that interact with and affect P. falciparum invasion.

50 suppressed both IMPAD1- and KDELR2-mediated invasion.

51 n with roles in neurogenesis and cancer cell invasion.

52 ponding confirmed histopathological level of invasion.

53 DBP, the key ligand involved in reticulocyte invasion.

54 gnal alerting the cell to imminent bacterial invasion.

55 oma has been shown to correlate with reduced invasion.

56 and improve our understanding of biological invasions.

57 t practices to proactively prevent microbial invasions.

58 educe potential future impacts of biological invasions.

59 ght, instead of being related to destructive invasions.

60 ay exacerbate negative impacts of biological invasions.

61 s mediated by regulating the process of cell invasion, adhesion and epithelial-mesenchymal transition

62 of cellular activities, including migration, invasion, adhesion and proliferation.

63 that both urban development and A. leptopus invasion affected community composition, albeit in diffe

64 t roles in cell polarization, migration, and invasion and are markedly upregulated in GBM and many ot

65 process to identify genes that regulate cell invasion and can be further investigated as potential ta

66 senchymal-like properties including cellular invasion and colony formation in vitro, as well as tumor

67 y as 72 tissues suggests that extrapulmonary invasion and damage is likely, which indeed has already

68 that PMX is a master modulator of merozoite invasion and direct maturation of proteins required for

69 am of Twist1 that is required for epithelial invasion and dissemination.

70 egulating tumour-initiating potential, local invasion and distant metastasis formation.

71 is an essential Ser/Thr kinase that controls invasion and egress by the protozoan parasite Toxoplasma

72 Indeed, inhibitors of invasion and egress offer hope for a desperately needed

73 ia receptor-mediated endocytosis, suppressed invasion and epithelial-mesenchymal transition of ovaria

74 he Tarp F-actin-binding domains in host cell invasion and for the Tarp effector as a bona fide C. tra

75 of proteins play a pivotal role in merozoite invasion and hence are important targets of immune respo

76 growth factor-beta (TGF-beta) promotes tumor invasion and metastasis by inducing epithelial-mesenchym

77 Plasticity of cancer invasion and metastasis depends on the ability of cancer

78 nal epithelial cells markedly enhances tumor invasion and metastasis in Trp53DeltaIEC mice (Trp53Delt

79 bition of this pathway prevents breast tumor invasion and metastasis in vivo.

80 ve metabolism with lipid utilization to fuel invasion and metastasis occurs.

81 Cancer cell invasion and metastasis rely on invadopodia, important e

82 ncluding primary tumor growth, angiogenesis, invasion and metastasis.

83 of the key cellular features altered during invasion and metastasis.

84 s robust, independent drivers of lung cancer invasion and metastasis.

85 ng that cellular subtypes cooperate to drive invasion and metastasis.

86 sults suggest that BCL11A sustains TNBC cell invasion and metastatic growth by repressing MBNL1-direc

87 out the underlying mechanisms that drive MCC invasion and metastatic progression.

88 MYCN-induced neuroblastoma, potentiates the invasion and migration of transformed sympathetic neurob

89 cellular level, Cj-P1 induced more C. jejuni invasion and neutrophil infiltration into the Il10(-/-)

90 species and lakes in Wisconsin following the invasion and proliferation of a novel predator (spiny wa

91 as suppressed CAV1-enhanced cell migration, invasion and Rac-1 activation in B16F10, metastatic colo

92 DRAIC downregulation increased cell invasion and soft agar colony formation; this was depend

93 LIMK2 significantly diminishes glioblastoma invasion and spread, suggesting the potential value of t

94 e of GBP2/Stat3/FN1 signaling cascade in GBM invasion and suggest GBP2 may serve as a potential thera

95 n an accurate diagnosis and assess for tumor invasion and the extent of the disease.

96 However, capsular invasion and tumor mass have a more aggressive course an

97 gest that gas migration occurs via capillary invasion and/or initiation and propagation of fractures

98 observation scale and climate in biological invasions and demonstrates that Darwin's two opposing hy

99 potential negative consequences of microbial invasions and describe a set of practices (Testing, Regu

100 initialization (landscape susceptibility to invasion) and expert-identified ecological uncertainties

101 motes angiogenesis, (3) facilitates vascular invasion, and (4) preserves the structural integrity and

102 ncluding proliferation, viability, migration/invasion, and also gene expression were not altered afte

103 necessity for pancreatic cancer (PC) growth, invasion, and chemotherapy resistance.

104 Effects on cell migration, invasion, and cytoskeletal organization in vitro were te

105 ociceptors protect against STm colonization, invasion, and dissemination from the gut.

106 ory organelle protein processing for egress, invasion, and effector export.

107 activation of AKT signaling, promoting cell invasion, and growth transformation induced by KSHV.

108 trong link between RBC tension and merozoite invasion, and identify a tension threshold above which i

109 nd protein kinase B phosphorylation, reduced invasion, and impaired melanoma cell-endothelial cell in

110 mor diameter, higher rate of extrapancreatic invasion, and lower rate of pathologic major response.

111 action, chromosome immunoprecipitation, cell invasion, and luciferase reporter assays; we measured ge

112 Caveolin-1 (CAV1) enhanced migration, invasion, and metastasis of cancer cells is inhibited by

113 esses in malignant cells including motility, invasion, and metastasis, thereby affecting tumor progre

114 sed epithelial to mesenchymal, migration and invasion, and metastasis.

115 helial-to-mesenchymal transition (EMT), cell invasion, and metastasis.

116 l specimens: size, lymphovascular/perineural invasion, and microglandular adenosis.

117 pression profoundly repressed proliferation, invasion, and migration in thyroid tumor cell models.

118 nctions for MLC1 in promoting GSC growth and invasion, and suggest that targeting the Mlc1 protein or

119 ession, binding activity, cell migration and invasion, and transcriptional activity of transcription

120 rates of pathologic lymph nodes, perineural invasion, and venous invasion.

121 rt, which were pN stage, pT stage, lymphatic invasion, and venous vascular invasion.

122 ant regulator of cell migration, cancer cell invasion, and vesicular content release, we sought to el

123 -mesenchymal transition, cellular migration, invasion, angiogenesis, stemness, transcriptional activa

124 Wound healing and Matrigel invasion assay, sphere formation assay, and in vivo mice

125 onal modeling, and in vitro and in vivo cell invasion assays to investigate the function of Aquaporin

126 ur microfluidic model advances current tumor invasion assays towards a more physiologically realistic

127 And, using 2D invasion assays, we show that the inhibition of ESRPs ca

128 yden chamber and 3D hyaluronic acid spheroid invasion assays.

129 tion of lipogenesis; decreased expression of invasion associated proteins through AMPK activation; an

130 he aberrant cell survival, proliferation and invasion associated with TF during chronic diseases.

131 date-based approach to identify effectors of invasion-associated resistance, we targeted beta1 and al

132 s of source pools and dynamics of historical invasions, assuming a continuation of processes in the f

133 ing to identify genes that strongly regulate invasion (based on an in vitro assessment of their metas

134 P-AscH(-) inhibited invasion, basement membrane degradation, decreased matri

135 ressed cell proliferation, tumour growth and invasion, both in vitro and in vivo.

136 ables were significantly affected by species invasion, but not by urban development.

137 process rates typically increase after plant invasion, but the extent to which this is driven by (i)

138 -known to induce breast cancer migration and invasion, but the mechanism by which TGF-beta signaling

139 g during flooding, rehydration, and pathogen invasion-but little is known about the mechanisms by whi

140 ostasis but also in intestinal resistance to invasion by bacterial, viral, and parasitic pathogens.

141 Thus, RTK-mediated host invasion by chlamydia upregulated TGF-beta expression an

142 zed eBD2 and -3, promoting further host cell invasion by EHV1.

143 iated ER-endosome contact sites promote cell invasion by facilitating translocation of MT1-MMP-laden

144 Sensitivity to invasion by heterochromatin, surprisingly, is not depend

145 ough this study to correlate the detected-ON invasion by Magnetic resonance imaging (MRI) with the co

146 Red blood cell (RBC) invasion by malaria merozoites involves formation of a p

147 We delineate these modes of invasion by merging ancestral, topographic, and phenotyp

148 lites, and as a barrier that prevents tissue invasion by microorganisms and tempers inflammatory resp

149 issue block in cases where a post-laminar ON invasion by MRI is found but not confirmed histopatholog

150 CpEF1alpha plays a role in the parasite invasion by participating in the formation of electron-d

151 Moreover, BCL11A promotes cancer cell invasion by suppressing the expression of muscleblind-li

152 otein was used to evaluate its effect on the invasion by the parasite.

153 While this increased invasion can be modulated by additional inhibition of th

154 accine population composition and assess the invasion capacity of emerging lineages.

155 high CAFs decreases their ability to promote invasion, chemoresistance, and in vivo tumor growth, ind

156 yed accelerated prostate tumor formation and invasion compared with their littermates that expressed

157 l invasions, moderate (20%-30%) increases in invasions, compared to the current conditions, are expec

158 ty and negative predictive value of depth of invasion, currently the best predictor, were 0.95 and 0.

159 BC surface, but, unexpectedly, inhibition of invasion does not correlate with specific RBC-parasite r

160 by regulating cell proliferation, migration, invasion, drug resistance, and epithelial-mesenchymal tr

161 Here we show that obstruction of vascular invasion during bone healing favours chondrogenic over o

162 plications for biodiversity conservation and invasion dynamics in freshwater ecosystems.

163 at such drives will have threshold-dependent invasion dynamics, spreading only when introduced above

164 We estimated invasion efficiency while accounting for red blood cell

165 ng domains are responsible for Tarp-mediated invasion efficiency.

166 Finally, in simulations of wound invasion, efficient collective migration is achieved wit

167 ative monitoring programs at early stages of invasion far outweigh the long-term costs associated wit

168 umor diameter, tumor pathology, and vascular invasion, female sex was associated with a 25% lower ris

169 sal fungi maintains homeostasis and prevents invasion from opportunistic commensals.

170 Invasion fronts in North America and China are projected

171 f V. cholerae c2-HDA significantly repressed invasion gene expression by Salmonella in the murine col

172 Notably, JQ1 induced upregulation of invasion genes through inhibition of Forkhead box protei

173 responsible for drug resistance, virulence, invasion, growth rate, and transmission.

174 While biological invasions have the potential for large negative impacts

175 results from one of the greatest biological invasions highlight how biogeographic processes and biot

176 N-stage (HR = 3.30 [2.09-5.21]), perineural invasion (HR = 1.50 [1.01-2.23]), and adjuvant chemother

177 ky tumors, variant histology, lymphovascular invasion, hydronephrosis and/or high-grade upper tract d

178 s (FIGO) stage IA1 with lymphovascular space invasion, IA2, or IB1 adenocarcinoma, squamous cell carc

179 e could exacerbate the impacts of biological invasions if climate change differentially affects invas

180 ments, required for invadopodia activity and invasion in a 3D collagen matrix.

181 exposure interrupted cell proliferation and invasion in a dose-dependent manner.

182 x and nucleotide synthesis, while decreasing invasion in cell line models and suppressing growth in t

183 her LIMK1 or LIMK2 only minimally influenced invasion in culture, simultaneous knockdown of both isof

184 ing pathways during replication, egress, and invasion in each of these stages.

185 2 dramatically promotes GBM tumor growth and invasion in mice and significantly reduces the survival

186 ional program, enhance motility, and promote invasion in PDA cells.

187 pathways of the mechanisms behind perineural invasion in PDAC and CCA.

188 at JQ1 has an unexpected effect of promoting invasion in prostate cancer.

189 signature commonly correlated with enhanced invasion in solid tumors.

190 %) with HRNMIBC, and two (10.0%) with muscle invasion in their specimen.

191 ncing furin reduced tumor cell migration and invasion in vitro and tumor growth and metastasis in viv

192 , in a number of 2D and 3D proliferation and invasion in vitro assays and an in vivo model.

193 O doses increase cancer cells' migration and invasion in vitro by up to 500%, whereas U1 over-express

194 gets tumor cell proliferation, migration and invasion in vitro in a broad spectrum of cancer types an

195 ignificantly promotes GBM cell migration and invasion in vitro, and GBP2 silencing by RNA interferenc

196 is critical to the management of biological invasions in a changing world, such as sea lamprey in th

197 nhibition reduces PC cell growth, migration, invasion, in vivo tumor growth and enhances GEM sensitiv

198 potential extent and impact of a buffelgrass invasion including size and frequency of fire events and

199 associated with primary melanoma growth and invasion, including oxidative stress.

200 Shallow invasion increases the risk of pregnancy complications,

201 eness in host populations following pathogen invasion into a vaccination zone.

202 nificantly decreased bacterial adherence and invasion into A549 cells and increased A549 cell viabili

203 Optic nerve (ON) invasion is an important high-risk feature, and an indic

204 nt risk factors, whereas depth of submucosal invasion is not an independent risk factor of LNM.

205 Contralateral invasion is suppressed by extracellular matrix (ECM) and

206 ding the likely future impacts of biological invasions is crucial yet highly challenging given the mu

207 External stability, or resistance to invasion, is thus an important but overlooked measure of

208 l mesothelial cells as well as migration and invasion, leading to colonization of peritoneal explants

209 evidence supporting a role of RBP2-P1 as an invasion ligand and its consideration as a vaccine targe

210 udy, the potential function of RBP2-P1 as an invasion ligand of P. vivax was evaluated.

211 ur findings regarding the role of submucosal invasion, LVI, and age.

212 erative index, localization, mesoappendiceal invasion, lymphovascular infiltration).

213 x to organize their secretory organelles and invasion machinery.

214 tion is urgently required to establish a FAW invasion management strategy.

215 nked to enhanced in vitro cell migration and invasion, mesenchymal markers (through the ERK-ZEB1-vime

216 rs and the TME, where they are implicated in invasion, metastasis, and angiogenesis.

217 promoting angiogenesis, immune suppression, invasion, migration, drug resistance and GBM recurrence.

218 d on responses from 36 experts in biological invasions, moderate (20%-30%) increases in invasions, co

219 validation, showing that LVI and perineural invasion, mucinous subtype, and low age constitute indep

220 Strand invasion occurs in uls1Delta cells with wild-type kineti

221 o reduce viability and inhibit migration and invasion of 4T1 murine breast cancer cells in vitro.

222 rget parasite invasion of hepatocytes or the invasion of and egress from erythrocytes.

223 million years(14), pointing to a substantial invasion of aquatic environments by dinosaurs.

224 ammation of bone, is most commonly caused by invasion of bacterial pathogens into the skeleton.

225 Here, we found that bacterial invasion of CRC cells and cocultured immune cells induce

226 Moreover, JAK3 suppresses the migration and invasion of cultured melanoma cells by modulating the ac

227 matter system; additionally, in humans, the invasion of dorsal tracts into the temporal lobe provide

228 d increases in proliferation, migration, and invasion of epithelial breast cancer cells, via non-cano

229 oteins required for parasite development and invasion of erythrocytes.

230 ize with other vaccines that target parasite invasion of hepatocytes or the invasion of and egress fr

231 formation by Nm, and impedes Nm adhesion and invasion of human airway cells.

232 cytoplasmic incompatibility, enabling rapid invasion of laboratory populations.

233 The vertebrate invasion of land may have been an important step in thei

234 We find that invasion of lncRNA transcription into the downstream gen

235 as neutral drift and can also cause downward invasion of mutant cells in the crypt.

236 The surrounding or invasion of nerves by cancer cells (perineural invasion)

237 early stages, supporting the hypothesis that invasion of nerves by cancer cells has a driving role in

238 id storage for subsequent utilization during invasion of pancreatic cancer cells, representing a pote

239 brain, but their roles in defending against invasion of pathogens into the CNS remain unclear.

240 ructures that form during transcription upon invasion of the DNA template by the nascent RNA.

241 to nutrient limitations and how this impacts invasion of the metapopulation as a whole.

242 ese structures, called appressoria, lead the invasion of the plant by the fungal hyphae.

243 at this is due to alterations in the initial invasion of the tissue.

244 The rates of R0 resection and pathologic invasion of venous and arterial walls were 52.4%, 74.2%,

245 terologous DNA and survive from the constant invasion of viruses, and they are widely used in biochem

246 No vascular invasion or cellular atypia were evident.

247 have shown that inhibition of monocyte brain invasion or systemic administration of an EP2 receptor a

248 d direct maturation of proteins required for invasion, parasite development, and egress.

249 nsidered within the management of biological invasions, particularly when complete eradication is not

250 btype, differentiation grade, lymphovascular invasion, perineural invasion, T-stage, N-stage, resecti

251 he extirpation of native species in the post-invasion period.

252 s not previously associated with erythrocyte invasion phenotypes, suggesting the possibility that mov

253 known to promote angiogenesis, migration and invasion, plays any role in metastasis of any tumor type

254 eport on the prognostic impact of perineural invasion (PNI) and desmoplasia on cutaneous squamous cel

255 otransferrin's effect on ovarian cancer cell invasion potential and proliferation by decreasing MMP1

256 with cells exhibiting enhanced migration and invasion potential in vitro.

257 limited personnel and funding for biological invasion prevention.

258 ductive biology emphasise the impacts of the invasion process on multiple, interlinked aspects of org

259 ex toward the membrane in order to start the invasion process.

260 ity and genetic control of change during the invasion process.

261 ese topological changes repress stemness and invasion programs while inducing anti-tumor immunity gen

262 phagocytophilum adhesins A. phagocytophilum invasion protein A (AipA), A. phagocytophilum surface pr

263 and identify a tension threshold above which invasion rarely occurs, even in non-Dantu RBCs.

264 owed that TTCC blockers reduce migration and invasion rates because of autophagy blockade only in BRA

265 a: estrogen receptor (ER) status, lymph node invasion, recurrence free survival.

266 data show increased expression of particular invasion-related genes in switched parasites, as well as

267 Cx43), yet whether Cx43 regulates collective invasion remains unclear.

268 Yet, our understanding of biological invasions remains incomplete, partly due to the difficul

269 Tumor invasion requires efficient cell migration, which is ach

270 We conducted a gain-of-function invasion screen, which identified two separate hits, IMP

271 ication regarding the threat from biological invasions should be developed, and that conservation pra

272 t without neuronal evidence of local seizure invasion) showed stable waveforms.

273 ional analysis revealed increased migration, invasion, sphere formation, and tumor development in mic

274 matrix production (CsgD pathway), epithelial invasion (SPI1 invasion system), and, finally, chemical

275 sis, baseline mrTD/mrEMVI (extramural venous invasion) status was the only significant MRI factor for

276 ion in interspecific competition, may prompt invasion success, morphological evolution, speciation, a

277 by which ZNF750 regulates cell migration and invasion, suggesting a role in breast cancer metastasis.

278 ition of Forkhead box protein A1 (FOXA1), an invasion suppressor in prostate cancer.

279 on (CsgD pathway), epithelial invasion (SPI1 invasion system), and, finally, chemical efflux and anti

280 n grade, lymphovascular invasion, perineural invasion, T-stage, N-stage, resection margin, and adjuva

281 de are neuron-intrinsic hallmarks of seizure invasion that impede traditional spike sorting and could

282 Post-invasion, the population was increasingly dominated by i

283 LI1 transcriptional complex driving melanoma invasion through the induction of the sialyltransferase

284 tory effects on cell mobility (migration and invasion) through anti-glycolysis and pro-autophagy.

285 adication, TREE) based on the four stages of invasion to prevent microbial inoculants from becoming i

286 to apoptosis, angiogenesis, metastasis, and invasion to secondary sites.

287 system of three-dimensional (3D) collective invasion to study the behavior and importance of protrus

288 B knockout or inhibition decreased migration/invasion, tumor growth, and lung metastasis.

289 By following invasion using video microscopy, we find a strong link b

290 s suggest that ERalpha can suppress HCC cell invasion via altering the ERalpha/circRNA-SMG1.72/miR-14

291 On explant, vascular invasion was found in 23.7% of AC-DS versus 16.9% of UNO

292 vasion of nerves by cancer cells (perineural invasion) was detected in 12% of esophageal cancers and

293 model of Caenorhabditis elegans anchor cell invasion, we characterize the gene regulatory network th

294 vertical growth, the first step in melanoma invasion, we developed a zebrafish melanoma model in whi

295 Thyroid cancer development and local invasion were delayed in only the PVPV-Akt1 knock out (K

296 directly involved in leukocyte migration and invasion, were significantly upregulated at the intra an

297 water and ion channels enhance migration and invasion when upregulated in leading edges of certain cl

298 osure altered trophoblast cell proliferation/invasion which might be mediated by preeclampsia-related

299 amine deprivation promotes CAF migration and invasion, which in turn facilitates the movement of tumo

300 erozoites with RBCs leads to either abortive invasion with rapid RBC lysis, or successful entry but d