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1 e-strand break is introduced next to a short inverted repeat sequence.
2 including a long spacer region containing an inverted repeat sequence.
3 at are repaired using multiple distinct long inverted repeat sequences.
4 involving homologous recombination of short inverted repeat sequences.
5 ten occur at short direct repeats that flank inverted repeat sequences.
6 bp in size and is bordered by 20-bp terminal inverted repeat sequences.
7 ity of target sites had no mismatches in the inverted repeat sequences.
8 t levels of homologous recombination between inverted repeat sequences.
9 help explain TMEJ repair events that include inverted repeat sequences.
10 ons that self-assemble from single identical inverted-repeat sequences.
11 abilizing this recombination intermediate in inverted-repeat sequences.
13 ylated PmrA bind to a 16-base pair imperfect inverted repeat sequence (5'-TTAAKTTCTTAAKGTT-3'), which
14 tified DNA-protein contact points at a novel inverted repeat sequence (AACCACTGGTT) and an Ets-like r
15 of replication between the arms of a GC-rich inverted repeat sequence adjacent to the binding site fo
16 n integrate both short oligonucleotides with inverted repeat sequences and a 2.8 kb excised mini-casp
18 -helix DNA-binding motif, recognizes a 19-bp inverted repeat sequence, and has a typical DNase I foot
19 owed in Saccharomyces cerevisiae that nearby inverted repeat sequences ( approximately 20-200 bp of h
20 taining many copies of the viral genome with inverted repeat sequences at the junctions between monom
21 coelicolor A3(2) were found to have 1.06 Mb inverted repeat sequences at their termini (i.e. long-te
22 " class of mutations that arise in imperfect inverted repeat sequences by DNA-strand misalignments du
23 of interest: an RNA/DNA hybrid domain and an inverted repeat sequence capable of forming a stem-loop
25 ent here the single-crystal structure of the inverted repeat sequence d(CCGGTACCGG) as a Holliday jun
26 Ser beta-sheet that binds specifically to an inverted repeat sequence distinct from those recognized
28 a cruciform DNA structure formed by a 60 bp inverted repeat sequence embedded in a negatively superc
30 ally to oriV DNA at several sites containing inverted repeat sequences (i.e., IR-1) and nonspecifical
32 sin occurred via binding directly to a short inverted repeat sequence in the intergenic region overla
34 otein in various genetic backgrounds, and an inverted repeat sequence in the promoter of hetP was nec
35 inal domain of AcrIIA13-AcrIIA15 binds to an inverted repeat sequence in the promoter of these Acr ge
36 R binds specifically and tightly to an 11-bp inverted repeat sequence in the promoter regions of the
37 lis OhrR binds cooperatively to two adjacent inverted repeat sequences in the ohrA control region and
38 pression of the operon by binding to several inverted repeat sequences in the promoter region, dnaKp.
39 spR confers repression by binding to several inverted repeat sequences in the promoter region, dnaKp.
41 rs and head-to-tail (h-t) concatemers within inverted repeat sequences (IRs) near probable origins of
42 One, O(C), which includes a 20-bp imperfect inverted-repeat sequence, is located between the two pro
46 he Tcr3 element contained imperfect terminal inverted repeat sequences of 56 bp and created a 2 bp ta
48 data suggest that ToxR is not recognizing an inverted repeat sequence per se in the activation of tox
50 nce is remarkably similar to a related 7-1-7 inverted repeat sequence recognized by PerR, a Fur paral
52 ly, deletion of a 26-bp region containing an inverted repeat sequence resulted in the loss of express
55 f the short transcript mapped adjacent to an inverted repeat sequence, suggesting that the sequence c
57 mRNA from the usd-spoIIID operon contains an inverted repeat sequence that is predicted to form a ste
58 for ohrA transcription identifies a perfect inverted repeat sequence that is required for OhrR-media
60 tprinting demonstrated binding of CouR to an inverted repeat sequence that overlaps the -10 region of
61 0-generated duplications are all symmetrical inverted repeat sequences that are apparently derived fr
62 d homologous recombination between the large inverted repeat sequences that exist in the genome, rath
67 ection" mechanism that can restore mutilated inverted-repeat sequences to a palindrome at the Ori of
68 posase binds specifically to ISY100 terminal inverted repeat sequences via an N-terminal DNA-binding
70 sses a single operon by binding to a perfect inverted repeat sequence, whereas HpNikR binds to promot
71 Upstream of each putative promoter is an inverted repeat sequence which resembles a similar eleme
72 he catA86 coding sequence contains a pair of inverted repeat sequences which cause sequestration of t
74 d oligonucleotide conjugates to hybridize to inverted repeat sequences within supercoiled double-stra