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1 to accumulate with no detectable chloro- or iodotyrosine.
2 -labeled IAT derivative N-iodoacetyl-3-[125I]iodotyrosine ([125I]IAIT) covalently labels the immunopu
3 tments of dopamine and a pathway inhibitor 3-iodotyrosine (3-IT), implicating dopamine as a direct me
4 d Ser-Lys mimics 9 and 12 from (S)- or (R)-3-iodotyrosine and then grafting key enzyme recognition el
10 nimum structure required for activity in the iodotyrosine deiodinase subgroup of this superfamily, at
12 sponsible for iodide salvage in the thyroid, iodotyrosine deiodinase, was solubilized from porcine th
13 eveal that hormone formation is triggered by iodotyrosine deprotonation, facilitated by proximity to
14 ide salvage by reductive deiodination of the iodotyrosine derivatives formed as byproducts of thyroid
15 ed to form mono-iodotyrosine (MIT) and/or di-iodotyrosine (DIT); however, selective tyrosine residues
17 mmunotherapy has been the rapid diffusion of iodotyrosine from target cells after internalization and
19 metabolic profile (including the content of iodotyrosine, iodosalicylates and iodobenzoates) after t
20 e residues can become iodinated to form mono-iodotyrosine (MIT) and/or di-iodotyrosine (DIT); however
24 eceptor targeted proteins is the tendency of iodotyrosine to rapidly diffuse from cells following end