ライフサイエンスコーパス: ionizing radiation

1 cytotoxic chemotherapy, ionizing radiation).

2 serious adverse events (SAE) and exposure to ionizing radiation.

3 bility, and cellular response to therapeutic ionizing radiation.

4 potential risks associated with exposure to ionizing radiation.

5 hromosome instability, which was enhanced by ionizing radiation.

6 roliferation, tumor growth and resistance to ionizing radiation.

7 vivo and enhanced the cytotoxic response to ionizing radiation.

8 erm cellular responses of human GBM cells to ionizing radiation.

9 pathogens and, as shown here, in response to ionizing radiation.

10 r resistance to cytotoxic chemotherapies and ionizing radiation.

11 ic species are known for their resistance to ionizing radiation.

12 e, significantly sensitizing cancer cells to ionizing radiation.

13 /CT-as it requires exposure to high doses of ionizing radiation.

14 ention of gammaH2AX and Rad51 foci following ionizing radiation.

15 effects arising from environmental gases and ionizing radiation.

16 ( 1.40 eV) on samples previously exposed to ionizing radiation.

17 nic RAS and reduced tissue inflammation upon ionizing radiation.

18 n measure bone properties without the use of ionizing radiation.

19 ection of the direct and indirect effects of ionizing radiation.

20 1 associates with gamma-H2A.X in response to ionizing radiation.

21 repair pathways and increases sensitivity to ionizing radiation.

22 tors of GSC quiescence following exposure to ionizing radiation.

23 y, with high diagnostic accuracy and without ionizing radiation.

24 hibitor hydroxyurea, but not the DSB inducer ionizing radiation.

25 hought to be responsible for the bulk of the ionizing radiation.

26 ds from plants exposed to elevated levels of ionizing radiation.

27 or to drive cell cycle progression following ionizing radiation.

28 ess and a major cause of oxidative stress is ionizing radiation.

29 ge compared to control in cells treated with ionizing radiation.

30 as well as increases sensitivity of cells to ionizing radiation.

31 s that occur after exposure to high doses of ionizing radiation.

32 l role in modulating biological responses to ionizing radiation.

33 ulated but later is strongly up-regulated by ionizing radiation.

34 ch recombination (CSR) and DSBs generated by ionizing radiation.

35 to STT-MRAM applications are robust against ionizing radiation.

36 are occupationally exposed to high doses of ionizing radiation.

37 of colonic epithelium is highly sensitive to ionizing radiation.

38 ely impact skeletal health: microgravity and ionizing radiation.

39 ve assessment of myocardial ischemia without ionizing radiation.

40 ildlife resulting from its field exposure to ionizing radiation.

41 ould be avoided whenever possible because of ionizing radiation.

42 rand breaks induced by either doxorubicin or ionizing radiation.

43 an disease and responses to stresses such as ionizing radiation.

44 sensitive to external toxic agents, such as ionizing radiation.

45 for dosimetry, including high sensitivity to ionizing radiation (20 times that of Al2O3:C, under the

46 We exposed male fish to sublethal low-dose ionizing radiation, a genotoxic stress affecting the som

47 Among these, ionizing radiation, a non-thermal process, has gained co

48 exposed to 11 genotoxins, including UV-B and ionizing radiation, alkylating compounds, aristolochic a

49 Our data show that ionizing radiation alters telomere homeostasis in wild a

50 ng the highest exposed to elevated levels of ionizing radiation, although their occupational doses ar

51 result of the indirect and direct effects of ionizing radiation, among other DNA damaging agents.

52 o exhibit significantly enhanced survival of ionizing radiation and bleomycin treatment, agents that

53 sponse to double-strand DNA breaks caused by ionizing radiation and chemotherapeutic agents.

54 caspase-3 in oral cancer cells treated with ionizing radiation and chemotherapeutic drug, paclitaxel

55 n multiple cancers and confers resistance to ionizing radiation and chemotherapeutic drugs.

56 essed in cancer cells, confers resistance to ionizing radiation and chemotherapy agents, and promotes

57 Rad18, and 53BP1, in cellular resistance to ionizing radiation and DNA repair.

58 the diversity of tumour cellular response to ionizing radiation and establish multiple lines of evide

59 diomycete fungus that is highly resistant to ionizing radiation and has been identified in highly rad

60 Thymidine radical cation (1) is produced by ionizing radiation and has been invoked as an intermedia

61 ession of abscopal tumours in the context of ionizing radiation and immune checkpoint blockade in viv

62 Defects in NHEJ result in sensitivity to ionizing radiation and loss of lymphocytes.

63 Telomeres can be damaged by ionizing radiation and oxidative stress, although the ef

64 hypersensitivity of BRCA1-depleted cells to ionizing radiation and PARP inhibition, highlighting the

65 letion leads to the increased sensitivity to ionizing radiation and poly (ADP-ribose) polymerase inhi

66 nd ATP production observed after exposure to ionizing radiation and reduced DNA repair.

67 Here we show that physical damage by ionizing radiation and ROS induces full-thickness membra

68 ool to map the amount of the body exposed to ionizing radiation and the location of exposure, which a

69 that radiation shielding reduces the flux of ionizing radiation and thereby increases the energy-rela

70 this patient showed increased sensitivity to ionizing radiations and phleomycin, attesting to a proba

71 ological tissue, as compared to UVB, UVC and ionizing radiation, and cause longlasting deep tissue da

72 es, including reactive oxygen species (ROS), ionizing radiation, and chemotherapies, activate acid sp

73 ection, increases HR and cell survival after ionizing radiation, and prevents cellular senescence.

74 IR imaging has relatively low cost, uses non-ionizing radiation, and provides minimally invasive spat

75 ization and activation in cells treated with ionizing radiation, and that loss of Mdm2 Ser394 phospho

76 n, as it does not involve the use of harmful ionizing radiation approaches.

77 ygen species generated by exposure to UV and ionizing radiation are characterized by clusters of lesi

78 adiation absorption of tumors, high doses of ionizing radiations are often needed during RT, leading

79 vant exposures to chemotherapeutic drugs and ionizing radiation, as well as BM recovery after drug-in

80 In response to ionizing radiation, ATM phosphorylates FBXW7 at serine 2

81 ized neuronal progenitors to apoptosis after ionizing radiation because of excessive DNA damage.

82 cores are known to emit very high levels of ionizing radiation, becoming visible over intergalactic

83 mbers of the Acidobacteria), or resistant to ionizing radiation (Brevundimonas and Truepera); elucida

84 of medical imaging which involve the use of ionizing radiation but not for ultrasonography.

85 hat could only be formed through exposure to ionizing radiation, but not by any other means of physic

86 induction by chemotherapeutic agents such as ionizing radiation caused SNP-dependent synergistic incr

87 Ionizing radiation causes acute radiation syndrome, whic

88 Following ionizing radiation, cells with disengaged BRCA1-PALB2 in

89 on, splicing, and translation in response to ionizing radiation. Changes at the transcriptional level

90 e knowledge and better inform the effects of ionizing radiation chronic exposure on nonhuman species,

91 Exposure to chronic ionizing radiation (CIR) from nuclear power plant accide

92 Ionizing radiation comes from a variety of sources, incl

93 lling and faster DNA repair upon exposure to ionizing radiations compared to vehicle-only treated cel

94 irectly from a human population, that MF non-ionizing radiation could have adverse biological impacts

95 to identify minimally invasive biomarkers of ionizing radiation damage to the CNS that are predictors

96 ted that melanin may also protect cells from ionizing radiation damage, against which C. neoformans i

97 s to stimulate intestinal regeneration after ionizing radiation damage.

98 rved to exhibit resistance to high levels of ionizing radiation despite sharing most DNA repair mecha

99 of material properties for high performance ionizing radiation detection based on direct detection m

100 ted increasing attention in recent years for ionizing radiation detection due to their strong stoppin

101 tion detectors with current state-of-the-art ionizing radiation detectors, we show the promising feat

102 ile opening the door to new possibilities in ionizing-radiation detectors.

103 al importance in understanding the effect of ionizing radiation, developing DNA biosensors and design

104 NEK1 is essential for the ionizing radiation DNA damage response and priming of th

105 ial and temporal resolution and reduction in ionizing radiation dose with new generation scanners.

106 es were subjected, for a week, to cumulative ionizing radiation doses, as used during cancer treatmen

107 opoietic dysfunction results from a range of ionizing radiation doses.

108 allows for precise and instant detection of ionizing radiations down to the level of 10(-4) Gy, repr

109 f cell cycle checkpoint activation following ionizing radiation drives increased production of immuno

110 pecies, we investigated the transposition to ionizing radiation ecotoxicity of one method proposed fo

111 nvironments (e.g., elevated temperatures and ionizing radiation environments) found in many engineeri

112 This study uncovers mechanisms by which ionizing radiation, especially particle radiation, promo

113 n this article, we show that DSBs induced by ionizing radiation, etoposide, or bleomycin suppress Rag

114 on whether the linear no-threshold model of ionizing radiation exposure accurately predicts the subs

115 he chromatin factors H2AX and KAP1 following ionizing radiation exposure and drives local chromatin d

116 Purpose To assess the potential ionizing radiation exposure from CT scans for both scree

117 signatures of somatic mutation characterize ionizing radiation exposure irrespective of tumour type.

118 Little is known of the effects of ionizing radiation exposure on soil biota.

119 raphy may have harms resulting from low-dose ionizing radiation exposure or identification of extraco

120 Ionizing radiation exposure to the brain is common for p

121 )J recombination and sensitizes the cells to ionizing radiation exposure.

122 hat AIM2 may be a new therapeutic target for ionizing radiation exposure.

123 Nuclear medicine uses ionizing radiation for both in vivo diagnosis and therap

124 by accelerating cosmic rays that can deliver ionizing radiation for up to [Formula: see text] ky.

125 Here we describe a completely ionizing-radiation-free in vivo near-infrared (NIR) fluo

126 Here we provide evidence that ionizing radiation from environmental radioactive materi

127 imaging of both radionuclide- and beam-based ionizing radiation from high-energy photons and charged

128 Individuals are exposed to air pollution and ionizing radiation from natural sources through inhalati

129 Thus, ionizing radiation generates distinctive mutational sign

130 Ionizing radiation has been shown to produce negative ef

131 ugh transgenerational effects of exposure to ionizing radiation have long been a concern, human resea

132 human tissues following in vivo exposure to ionizing radiation have not been documented.

133 date novel epigenetic rheostats that promote ionizing radiation hypersensitivity in various normal st

134 As with other ionizing radiation imaging modalities, CBCT imaging shou

135 Here, we searched for signatures of ionizing radiation in 12 radiation-associated second mal

136 er, DYRK1A knock out conferred resistance to ionizing radiation in colony formation assays, suggestin

137 iciently high energy to produce ionization." Ionizing radiation in cosmic chemistry includes high-ene

138 ~100 mSv is lacking; however, concerns about ionizing radiation in medical imaging remain and can aff

139 linical decision-making regarding the use of ionizing radiation in medicine.

140 sms acting to buffer the negative effects of ionizing radiation in natural populations.

141 e for type I interferon signaling induced by ionizing radiation in the presence or absence of ATR inh

142 therapy to robustly sensitize NSCLC cells to ionizing radiation in vitro and in vivo.

143 with other DNA-damaging therapies, including ionizing radiation, in the ATRX-deficient context.

144 Herein, we show that ionizing radiation increases high mannose-type N-glycans

145 Exposure to ionizing radiation increases the risk of chronic metabol

146 ficient to repress the apoptotic response to ionizing radiation independent of developmental signalin

147 High doses of ionizing radiation induce acute damage to epithelial cel

148 induced DNA damage, and RAD51 recruitment to ionizing radiation induced foci (IRIF), which requires e

149 Moreover, ionizing radiation induced macrophage morphological alte

150 1 overexpression suppresses the formation of ionizing radiation-induced 53BP1 and BRCA1 but not RNF16

151 thosterol 5-desaturase, then UVR accelerates ionizing radiation-induced BCC carcinogenesis.

152 , chronic topical application of D3 inhibits ionizing radiation-induced BCC tumorigenesis.

153 ofluorescent detection of repair proteins at ionizing radiation-induced DNA damage foci that Wwox exp

154 lipid vesicles, and iii) ability to resolve ionizing radiation-induced DNA damage foci.

155 ese two proteins and variably contributes to ionizing radiation-induced DSB repair in human and chick

156 each colocalize with phosphorylated H2AX at ionizing radiation-induced DSBs but not with 53BP1.

157 (TIRR) that specifically associates with the ionizing radiation-induced foci formation region of 53BP

158 ed ubiquitination as well as 53BP1 and BRCA1 ionizing radiation-induced foci formation.

159 tem cells exhibit reduced ATM activation and ionizing radiation-induced foci, they display apoptotic

160 onse kinase ATR can significantly potentiate ionizing radiation-induced innate immune responses.

161 nucleosome (50-500 bp) scale, obtained using ionizing radiation-induced spatially correlated cleavage

162 Acute exposure to ionizing radiation induces massive cell death and severe

163 Mechanistic understanding of how ionizing radiation induces type I interferon signaling a

164 itigates RIGS lethality in vivo after lethal ionizing radiation injury-induced intestinal epithelial

165 mutants demonstrate increased sensitivity to ionizing radiation (IR) and a decrease in DNA double-str

166 Ionizing radiation (IR) and chemotherapy are standard-of

167 rference sensitizes prostate cancer cells to ionizing radiation (IR) and etoposide treatment, as asse

168 we challenged Plk1-overexpressing mice with ionizing radiation (IR) and found that Plk1-overexpressi

169 -negative breast cancer cells in response to ionizing radiation (IR) and localizes to rDNA sequences

170 ivation and repression events in response to ionizing radiation (IR) and synthetic p53 activation.

171 s (GSCs) to survive exposure to low doses of ionizing radiation (IR) as a model of adult stem cell in

172 in these cells, we exposed pregnant mice to ionizing radiation (IR) at specific gestational time poi

173 Ionizing radiation (IR) can promote migration and invasi

174 sponse to a combination of PDI knockdown and ionizing radiation (IR) compared with either modality al

175 mal thyroid function is disturbed because of ionizing radiation (IR) exposure, deleterious effects ca

176 reported that cells exposed to low doses of ionizing radiation (IR) in the G(2)-phase of the cell cy

177 that the AR directly interacts with CHK2 and ionizing radiation (IR) increases this interaction.

178 In response to ionizing radiation (IR) induced DNA damage, cancer cells

179 Exposure of murine and human tissues to ionizing radiation (IR) induces the expression of p16(IN

180 Nonlethal exposure to ionizing radiation (IR) is a public concern due to its k

181 Ionizing radiation (IR) is commonly used in cancer thera

182 We studied the effect of ionizing radiation (IR) on continuous growth of seven hE

183 However, the effects of ionizing radiation (IR) on HKG expression is unclear.

184 Elderly cancer patients treated with ionizing radiation (IR) or chemotherapy experience more

185 l of cancer cells mediated by high levels of ionizing radiation (IR) reduces the effectiveness of rad

186 forts to understand the complex phenotype of ionizing radiation (IR) resistance, a genome sequence ca

187 melanogaster larvae irradiated with doses of ionizing radiation (IR) that kill about half of the cell

188 ak worker subjects occupationally exposed to ionizing radiation (IR) to investigate the contribution

189 Concern over potential exposures of ionizing radiation (IR) to large populations has emphasi

190 lved in sensitizing radioresistant tumors to ionizing radiation (IR) treatments while minimizing inju

191 ity and its combination with DDR inhibitors, ionizing radiation (IR), and chemotherapy can be exploit

192 h-MYCN/Trp53(KI/KI) tumors were resistant to ionizing radiation (IR), as expected.

193 In response to ionizing radiation (IR), cells activate a DNA damage res

194 ses of HuR in oral cancer cells treated with ionizing radiation (IR), determined that HuR cleavage pr

195 n growth under 36 Gy/h) and acute (10 kGy/h) ionizing radiation (IR), heavy metals (chromium, mercury

196 es mediating the response of glioblastoma to ionizing radiation (IR), we used polysome profiling to d

197 genomic instability and cell sensitivity to ionizing radiation (IR)-induced death.

198 ibed mechanism promoting cell survival after ionizing radiation (IR)-induced DNA damage involves frag

199 Ionizing radiation (IR)-induced DNA double-strand breaks

200 Ionizing radiation (IR)-induced DSBs lead to the same ou

201 Ionizing radiation (IR)-induced intestinal damage is cha

202 RC5A contributes significantly to preventing ionizing radiation (IR)-induced lung tumorigenesis.

203 scarce and the mutational processes defining ionizing radiation (IR)-induced mutagenesis in vivo are

204 In this study, we demonstrate that ionizing radiation (IR)-induces ATM-dependent phosphoryl

205 and NBS1, and renders mice hypersensitive to ionizing radiation (IR).

206 ry response elicited in mouse macrophages by ionizing radiation (IR).

207 emination in adjacent brain parenchyma after ionizing radiation (IR).

208 bility to repair DNA in the cells exposed to ionizing radiation (IR).

209 sence sensitizes the bacterium to killing by ionizing radiation (IR).

210 e hypersensitive to genotoxic stress such as ionizing radiation (IR).

211 tion to the severity of dystrophy induced by ionizing radiation (IR).

212 k, in cells treated with small molecules and ionizing radiation (IR).

213 human glomerular endothelial cells (GEnC) to ionizing radiation (IR).

214 drugs have been found to sensitize cells to ionizing radiation (IR).

215 s essential for optimal antitumor control by ionizing radiation (IR).

216 ver, the heart is an unintentional target of ionizing radiation (IR).

217 nes mutated in the other subunit, ARID1A, to ionizing radiation (IR).

218 anti-cancer therapeutics in combination with ionizing radiation (IR).

219 Using data generated with cells exposed to ionizing-radiation (IR) in G(2)-phase of the cell cycle,

220 Ionizing radiation is a common therapeutic modality and

221 Ionizing radiation is a potent carcinogen, inducing canc

222 Ionizing radiation is a well known human carcinogen.

223 ortance of nontargeted (systemic) effects of ionizing radiation is attracting increasing attention.

224 f competing risk methods, suggesting that if ionizing radiation is impacting the risk of death due to

225 Ionizing radiation is one of the most common cancer trea

226 Malignant glioma is rapidly fatal, and ionizing radiation is part of the current standard-of-ca

227 l method for imaging mineral content without ionizing radiation is proposed.

228 Magnetic field (MF) non-ionizing radiation is widespread and everyone is exposed

229 ren may be more vulnerable to the effects of ionizing radiation, it is necessary to develop methods t

230 vailability, its noninvasiveness and lack of ionizing radiation, its low cost, and its good reproduci

231 The burden of low-dose ionizing radiation (LDIR) exposure from medical procedur

232 e are increasingly being exposed to low-dose ionizing radiation (LDIR) from cardiac procedures.

233 re exposed to increasing amounts of low-dose ionizing radiation (LDIR) from cardiac procedures.

234 the effect of oxidative stress from low-dose ionizing radiation (LDIR) on wild-type and p53 mutant ce

235 The effect of ionizing radiation leads to an elevated quasiparticle de

236 eas around Chernobyl differing in background ionizing radiation levels and one control study site in

237 pathology is suspected, but the presence of ionizing radiation makes it harder to construct database

238 Eliminating exposure to imaging with ionizing radiation may enhance the safety of relapse sur

239 factor XLF result in extreme sensitivity for ionizing radiation, microcephaly, and growth retardation

240 the cellular response of tumours exposed to ionizing radiation, modelling the alteration of oxygen p

241 main clinical mainstays, optical readouts of ionizing radiation offer numerous benefits and complemen

242 nding that cancer chemotherapeutic drugs and ionizing radiation often promote autophagy has provided

243 tosis display similar therapeutic effects to ionizing radiation on cancer cell lines.

244 Particularly, the effect of ionizing radiation on macrophages, using therapeutically

245 The effects of ionizing radiation on materials often reduce to "bad new

246 , we test for the consequence of exposure to ionizing radiation on plant development.

247 d field data concerning potential effects of ionizing radiation on the abundance of mammals collected

248 We evaluated the effects of ionizing radiation on the MinION platform - including fl

249 However, studies on the effects of ionizing radiation on the STT-MRAM writing process are l

250 d that independently assessed the effects of ionizing radiation on transcription and post-transcripti

251 d cytotoxic chemotherapy as well as surgery, ionizing radiation or both.

252 ts normal cells from DNA damage induction by ionizing radiation or chemotherapeutics, whereas cancer

253 in combination with genotoxic agents such as ionizing radiation or chemotherapy.

254 specific environmental mutagen, specifically ionizing radiation or cigarette smoking.

255 HAEC) by replicative exhaustion, exposure to ionizing radiation or doxorubicin, and expression of the

256 ells to DNA damage under conditions, such as ionizing radiation or hydrogen peroxide treatment, that

257 inflammatory challenges, such as exposure to ionizing radiation or to bacterial lipopolysaccharides.

258 e induced by a variety of stimuli, including ionizing radiation, oxidative stress, and inflammation.

259 which are the likely sources of the elevated ionizing radiation powering the observed Lyman-alpha emi

260 molecular probes sensitive to byproducts of ionizing radiation (primarily reactive oxygen species, o

261 benefits of metastasis-directed therapy with ionizing radiation (primarily stereotactic ablative radi

262 Ionizing radiation produces clustered damage to DNA whic

263 exists in nature, where it provides superior ionizing radiation protection to organisms compared to k

264 ouse B-cell line, and dispensable for normal ionizing radiation resistance in both G1-arrested and cy

265 identification of mutations associated with ionizing radiation resistance.

266 adaptive laboratory evolution for increased ionizing radiation resistance.

267 dividuals with PPM1D truncating mutations to ionizing radiation resulted in normal p53 activation, su

268 e variation induced by chemical treatment or ionizing radiation results in higher mutation rates.

269 e greatest disadvantage of this technique is ionizing radiation risk to the patients.

270 od (only 10 minutes after injection) without ionizing radiation risk.

271 osis in dental clinics and surgeries without ionizing radiation risk.

272 r studying biological effects under low-dose ionizing radiation, safety control in medical radiation

273 MRI does not involve exposure to ionizing radiation, similar to echocardiography, which c

274 Furthermore, for ionizing radiation sources that cannot be imaged using t

275 of melanized fungi to cosmic and terrestrial ionizing radiation suggests that melanin also plays a pi

276 ace travelers are exposed to unique forms of ionizing radiation that pose potentially serious health

277 damage caused by exogenous sources, such as ionizing radiation, the tumour suppressor p53 mediates c

278 As it is altered by ionizing radiation, the vascular network is considered a

279 Increasing evidence suggests that ionizing radiation therapy (RT) in combination with chec

280 The abscopal effect following ionizing radiation therapy (RT) is considered to be a ra

281 nction and show that SNP309G cooperates with ionizing radiation to exacerbate tumor development.

282 as more recently been realized and relies on ionizing radiation to induce irreversible DNA damage, re

283 dy to E-cadherin, works synergistically with ionizing radiation to promote the epidermal damage.

284 therapy and precisely deliver high doses of ionizing radiation to small deep-seated target volumes.

285 Radiotherapy uses ionizing radiation to target and kill tumour tissue, but

286 t to conventional radiotherapy, TRT delivers ionizing radiation to tumors in a targeted manner, reduc

287 used to study clinically relevant markers of ionizing radiation toxicities to the brain.

288 We find that UVR exposure of ionizing radiation-treated Ptch1(+/-) mice accelerates B

289 ed the function of miR-24 in NPC cells after ionizing radiation treatment, resulting in increased apo

290 harmful biological effects from exposure to ionizing radiation, understanding the mechanisms by whic

291 ice and non-human primates (NHPs) exposed to ionizing radiation up to 8 days after radiation exposure

292 BSGI on the basis of the Biologic Effects of Ionizing Radiation VII report, the benefit-to-radiation

293 udy, stimulation by doxorubicin, hypoxia and ionizing radiation was used to induce MDR in HCC cells.

294 and dehydroascorbic acid (DHAA), induced by ionizing radiation, was investigated.

295 N109D) mutant RUNX1 conferred resistance to ionizing radiation when overexpressed in Ba/F3 cells und

296 , PDE12 or AKAP7 (which degrade 2-5A), or by ionizing radiation (which induces IFN-dependent signalin

297 qubits, reducing or mitigating the impact of ionizing radiation will be critical for realizing fault-

298 The galaxy is leaking ionizing radiation with an escape fraction of about 8 pe

299 There are well-known associations of ionizing radiation with female breast cancer, and emergi

300 The interaction of ionizing radiation with matter is of critical importance