ライフサイエンスコーパス: ionotropic

1 otor neurons express distinct populations of ionotropic acetylcholine receptors (iAChRs) requiring th

2 hibition results from the combined action of ionotropic acetylcholine receptors and associated calciu

3 is released to supplement influx through the ionotropic ACh receptor.

4 Ionotropic activation of NMDA receptors (NMDARs) require

5 icity-induced neuronal death through the non-ionotropic activity of GluN2ARs and the neuroprotective

6 ing NMDARs (GluN2ARs), suggesting that a non-ionotropic activity of GluN2ARs mediates glycine-induced

7 nexpected role of glycine in eliciting a non-ionotropic activity of GluN2ARs to confer neuroprotectio

8 amate receptor subunit B, glutamate receptor ionotropic AMPA 2 (GRIA2), modifies a codon, replacing t

9 begins with the binding of glutamate to the ionotropic AMPA receptors and metabotropic glutamate rec

10 long-lived changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie f

11 In previous studies, stimulation of ionotropic AMPA/kainate glutamate receptors on cultured

12 Ionotropic AMPARs have shorter time constants and have b

13 a local Ca(2+) rise, even in the presence of ionotropic and cell surface metabotropic receptor inhibi

14 and antagonist trials (verapamil) addressed ionotropic and chronotropic cell line-dependent features

15 ion, several sequences representing putative ionotropic and gustatory receptors were also identified.

16 Moreover, we demonstrate that these ionotropic and metabotropic 5-HT receptors have a synerg

17 We identify that both ionotropic and metabotropic 5-hydroxytryptamine (5-HT) r

18 e found that it is mediated via postsynaptic ionotropic and metabotropic GABA and metabotropic glutam

19 "MAG" PTLs for ionotropic and metabotropic glutamate receptors (GluRs)

20 oincided with transcriptionally dysregulated ionotropic and metabotropic glutamate receptors and glut

21 attenuated by antagonists targeting multiple ionotropic and metabotropic glutamate receptors, and int

22 ys, cytoskeletal elements, AD-related genes, ionotropic and metabotropic glutamate receptors, choline

23 citatory glutamatergic transmission, through ionotropic and metabotropic glutamate receptors, is nece

24 tes aberrant extrasynaptic signaling through ionotropic and metabotropic glutamate receptors, ultimat

25 MF-CA3 synapses, with implications for both ionotropic and metabotropic glutamatergic recruitment of

26 A similar loss of ionotropic and metabotropic KAR function was observed in

27 tive upregulation of gene sets implicated in ionotropic and metabotropic neurotransmission as well as

28 These ionotropic and metabotropic P2 purinergic receptors modu

29 ATP release triggers the activation of both ionotropic and metabotropic purinoceptors, with strong p

30 iguingly, a large number of genes coding for ionotropic and metabotropic receptors for various neurot

31 mate released from climbing fibers activates ionotropic and metabotropic receptors on Golgi cells thr

32 They express ionotropic and metabotropic receptors, and can release g

33 that glutamate and GABA signal through both ionotropic and metabotropic receptors.

34 movements therefore likely arise from fast (ionotropic) and slow (metabotropic) neural mechanisms, a

35 NMDA receptors are ionotropic calcium-permeable glutamate receptors with a

36 mechanism for the neuronal serotonin 5-HT3A ionotropic channel receptor, in which the role of routin

37 combines the activity of an unusual class of ionotropic cholinergic receptor with that of nearby calc

38 a novel and powerful means for probing GluD2 ionotropic contribution to neuronal physiology.

39 channels and is responsible for the positive ionotropic effect of adrenergic stimulation.

40 uingly, a recent report revealed a novel non-ionotropic function of the NMDAR in the regulation of sy

41 rstanding of disease-associated mutations in ionotropic GABA and glutamate receptor families, and dis

42 function of D-MNs is mediated by a specific ionotropic GABA receptor (UNC-49) in AVA, and depends on

43 Because we could not localize ionotropic GABA receptors on cone axon terminals using e

44 the expression of previously uncharacterized ionotropic GABA receptors.

45 extrasynaptic effects being mediated by the ionotropic GABA type A receptors (GABA(A)Rs).

46 ow timescales depending on the activation of ionotropic GABA(A)-R or metabotropic GABA(B)-R.

47 electrophysiology to study the expression of ionotropic GABA, glutamate, and ATP receptors in oligode

48 the driving force for the chloride-permeable ionotropic GABAA receptor in mature neurons.

49 aptic inhibition in the brain is mediated by ionotropic GABAA receptors (GABAARs) and metabotropic GA

50 nsmission in the brain is mediated mostly by ionotropic GABAA receptors (GABAARs), but their essentia

51 e metabotropic GABAB receptor GBB-1, but not ionotropic GABAA receptors.

52 ers are, surprisingly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

53 The fast acting ionotropic Glu receptors (iGluRs) are ligand gated ion c

54 ective activation of glomerular mAChRs, with ionotropic GluRs and nAChRs blocked, increased IPSCs in

55 inases have been securely identified in many ionotropic glutamate (iGlu) receptor subunits, but which

56 nd wasps are potent open-channel blockers of ionotropic glutamate (iGlu) receptors.

57 ent but nonselective open-channel blocker of ionotropic glutamate (iGlu) receptors.

58 of angiotensin II receptor type 1, oxytocin, ionotropic glutamate and GABAA receptors.

59 Activation of ionotropic glutamate and/or GABA receptors along the GnR

60 Ionotropic glutamate delta receptors do not bind glutama

61 Kainate receptors (KARs) are ionotropic glutamate ion channels involved in synaptic p

62 ia angiotensin II type 1 receptor, oxytocin, ionotropic glutamate or GABAA receptors but instead invo

63 ptic depression is modulated by postsynaptic ionotropic glutamate receptor (iGluR) activity.

64 Exposure to ionotropic glutamate receptor (iGluR) agonists, kainic a

65 dria in astrocytic processes were blocked by ionotropic glutamate receptor (iGluR) antagonists, tetro

66 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

67 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

68 Ionotropic glutamate receptor (iGluR) channels control s

69 Ionotropic glutamate receptor (iGluR) family members are

70 as seen a revolution in our understanding of ionotropic glutamate receptor (iGluR) structure, startin

71 The GluD1 and GluD2 receptors form the GluD ionotropic glutamate receptor (iGluR) subfamily.

72 Loss of specific ionotropic glutamate receptor (iGluR) subunits during in

73 lated GluD1 are classified as members of the ionotropic glutamate receptor (iGluR) superfamily on the

74 trated that a member of the newly discovered ionotropic glutamate receptor (IR) family, IR76b, functi

75 excitatory mammalian ion channel light-gated ionotropic glutamate receptor (LiGluR) in retinal gangli

76 ype of positive allosteric modulators of the ionotropic glutamate receptor A2 (GluA2) are promising l

77 Using heterologous expression of excitatory ionotropic glutamate receptor AMPA subunits in Xenopus o

78 onents of neural signalling (specifically an ionotropic glutamate receptor and two regucalcins), and

79 structure-activity study of the broad-acting ionotropic glutamate receptor antagonist 1a.

80 y-NH2 , d(CH2 )5 [D-Tyr(2) ,Thr(4) ]OVT, the ionotropic glutamate receptor antagonist kynurenate or t

81 Ionotropic glutamate receptor antagonists are valuable t

82 Since ionotropic glutamate receptor antagonists can partially

83 ves, MC calcium transients were inhibited by ionotropic glutamate receptor antagonists, indicating th

84 ate and not 5-HT because it was abolished by ionotropic glutamate receptor antagonists.

85 We conclude that the two types of ionotropic glutamate receptor are built in different way

86 einstated by 4-aminopyridine, and blocked by ionotropic glutamate receptor blockers.

87 Here, we design a light-controlled ionotropic glutamate receptor by genetically encoding a

88 We used RT-PCR to profile ionotropic glutamate receptor expression in cultured SCs

89 calcium elevations and Western blots reveal ionotropic glutamate receptor expression prior to immuno

90 s and MEF2C in the signal cascade leading to ionotropic glutamate receptor expression.

91 utamate delta (GluD) receptors belong to the ionotropic glutamate receptor family, yet they don't bin

92 We focus here on the control of ionotropic glutamate receptor function by GPCR signaling

93 ory synaptic transmission is mediated by the ionotropic glutamate receptor homolog cation channel, de

94 he responsible sensory receptor (the variant ionotropic glutamate receptor IR75b) and attraction-medi

95 y SNAG-mGluR2 and excitatory light-activated ionotropic glutamate receptor LiGluR yielded a distribut

96 -shifted PTL, L-MAG0460, for the light-gated ionotropic glutamate receptor LiGluR.

97 ations in melanoma and the significance that ionotropic glutamate receptor signaling has in malignant

98 onses, short-term dynamics and expression of ionotropic glutamate receptor subtypes.

99 nit of N-methyl-d-aspartate receptors in the ionotropic glutamate receptor superfamily have been targ

100 eptors (AMPARs) constitute a subclass of the ionotropic glutamate receptor superfamily, which functio

101 N2A subunit of the NMDA receptor (NMDAR), an ionotropic glutamate receptor that has important roles i

102 These results highlight the diversity of ionotropic glutamate receptor trafficking rules at a sin

103 methyl-4-isoxazolepropionic acid (AMPA)-type ionotropic glutamate receptors (AMPA receptors) predeter

104 orylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kinases and p

105 AMPA-subtype ionotropic glutamate receptors (AMPARs) mediate fast exc

106 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediate the majo

107 alian brain is largely mediated by AMPA-type ionotropic glutamate receptors (AMPARs), which are activ

108 GRIA3 encodes GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

109 eurotransmission is mediated by AMPA-subtype ionotropic glutamate receptors (AMPARs).

110 Ionotropic glutamate receptors (iGluRs) are key molecule

111 Ionotropic glutamate receptors (iGluRs) are ligand-gated

112 Ionotropic glutamate receptors (iGluRs) are responsible

113 Ionotropic glutamate receptors (iGluRs) are tetrameric c

114 Ionotropic glutamate receptors (iGluRs) are ubiquitous i

115 ation followed the arrival and clustering of ionotropic glutamate receptors (iGluRs) at NMJ synapses.

116 generated an extensive sequence alignment of ionotropic glutamate receptors (iGluRs) from diverse ani

117 hores have revealed the presence of numerous ionotropic glutamate receptors (iGluRs) in Mnemiopsis le

118 Ionotropic glutamate receptors (iGluRs) mediate most exc

119 Ionotropic glutamate receptors (iGluRs) mediate neurotra

120 Ionotropic glutamate receptors (iGluRs) mediate the majo

121 Presynaptic ionotropic glutamate receptors (iGluRs) play important r

122 is comb jelly encodes homologs of vertebrate ionotropic glutamate receptors (iGluRs) that are distant

123 NMDA receptors (NMDARs) are a class of ionotropic glutamate receptors (iGluRs) that are essenti

124 Kainate receptors (KARs) are ionotropic glutamate receptors (iGluRs) that modulate sy

125 Ionotropic glutamate receptors (iGluRs) transduce the ch

126 osstalk between Wnt receptors, activation of ionotropic glutamate receptors (iGluRs), and localized c

127 americ ion channels that together with other ionotropic glutamate receptors (iGluRs), the NMDA and ka

128 Here, we studied ionotropic glutamate receptors (iGluRs), which are ion c

129 stem is mediated by glutamate acting through ionotropic glutamate receptors (iGluRs).

130 neurons express either odorant receptors or ionotropic glutamate receptors (IRs).

131 iod of synaptic development, kainate-type of ionotropic glutamate receptors (KARs) are highly express

132 The ionotropic glutamate receptors (N-methyl-D-aspartate rec

133 and memory and a reduction in the amounts of ionotropic glutamate receptors (NMDA and AMPA receptors)

134 Ca(2+) entry through extrasynaptic NMDA-type ionotropic glutamate receptors (NMDARs).

135 Ionotropic glutamate receptors activate cell signaling i

136 Adenosine release required the activation of ionotropic glutamate receptors and could be evoked by lo

137 ynaptic neurexins and postsynaptic AMPA-type ionotropic glutamate receptors and induced the formation

138 These results identified ionotropic glutamate receptors and NMDA-Rs, specifically

139 or understanding gating across the family of ionotropic glutamate receptors and the role of AMPA rece

140 Ionotropic glutamate receptors are a family of tetrameri

141 Ionotropic glutamate receptors are important for estradi

142 Ionotropic glutamate receptors are ligand-gated ion chan

143 Ionotropic glutamate receptors are ligand-gated ion chan

144 Ionotropic glutamate receptors are postsynaptic tetramer

145 Positive allosteric modulators of ionotropic glutamate receptors are potential compounds f

146 The ionotropic glutamate receptors are primary mediators of

147 Ionotropic glutamate receptors are thought to play an es

148 Ionotropic glutamate receptors are widely distributed in

149 s NL1 isoform-specific cis-interactions with ionotropic glutamate receptors as a key mechanism for co

150 -93, and SAP97, are scaffolding proteins for ionotropic glutamate receptors at excitatory synapses.

151 al D1R/PKA/MEK1/2 pathway and independent of ionotropic glutamate receptors but blocked by antagonist

152 Ionotropic glutamate receptors comprise two conformation

153 The delta family of ionotropic glutamate receptors consists of glutamate del

154 The overstimulated ionotropic glutamate receptors exert their neurotoxic ef

155 n is necessary and sufficient to up-regulate ionotropic glutamate receptors from a pool of different

156 s been designed to enable optical control of ionotropic glutamate receptors in neurons via sensitized

157 Recently, several full-length structures of ionotropic glutamate receptors in putative desensitized

158 discovery of 20 genes encoding for putative ionotropic glutamate receptors in the Arabidopsis (Arabi

159 Blockade of ionotropic glutamate receptors in the DMH, or brain tran

160 e N-methyl-d-aspartate (NMDA) subtype of the ionotropic glutamate receptors is the primary mediator o

161 Zn(2+) inhibition of the NMDA subtype of the ionotropic glutamate receptors is well characterized, th

162 Therefore, our results suggest that ionotropic glutamate receptors may have been conserved t

163 The ionotropic glutamate receptors mediate excitatory neurot

164 N-methyl-d-aspartate (NMDA)-type ionotropic glutamate receptors mediate excitatory neurot

165 5-methyl-4-isoxazole propionic acid)-subtype ionotropic glutamate receptors mediate fast excitatory n

166 AMPA subtype ionotropic glutamate receptors mediate fast excitatory n

167 ced by systemic injections of antagonists of ionotropic glutamate receptors or metabotropic glutamate

168 ptors (IRs) are a large subfamily of variant ionotropic glutamate receptors present across Protostomi

169 enhanced by DEX (10 microM), and blockade of ionotropic glutamate receptors reduced the DEX effect on

170 Kainate receptors (KARs) are ionotropic glutamate receptors that also activate noncan

171 NMDA receptors (NMDARs) are ionotropic glutamate receptors that are crucial for neur

172 NMDA receptors are ionotropic glutamate receptors that function as heterote

173 ptors (NMDARs) are a subtype of postsynaptic ionotropic glutamate receptors that function as molecula

174 N-methyl-D-aspartate-receptors (NMDARs) are ionotropic glutamate receptors that function in synaptic

175 During apnea, induced by blockade of ionotropic glutamate receptors within the same region, m

176 Theories attribute an important role to ionotropic glutamate receptors, and it has been suggeste

177 contain beta-adrenergic receptors as well as ionotropic glutamate receptors, and retromer knockdown r

178 Kainate receptors (KARs), a family of ionotropic glutamate receptors, are widely expressed in

179 eceptors (KARs), one of three subfamilies of ionotropic glutamate receptors, as well as the putative

180 lence of somatic mutations within one of the ionotropic glutamate receptors, GRIN2A, in malignant mel

181 al agent that helped unravel the key role of ionotropic glutamate receptors, including the kainate re

182 at central synapses depends on the number of ionotropic glutamate receptors, particularly the class g

183 structural basis of allosteric inhibition in ionotropic glutamate receptors, providing key insights i

184 inate receptors (KARs) consist of a class of ionotropic glutamate receptors, which exert diverse pre-

185 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors, which mediate most excit

186 Correspondingly, AMPA-subtype ionotropic glutamate receptors, which mediate the majori

187 osed to the hippocampus proper, also express ionotropic glutamate receptors, which might provide addi

188 ic transmission, modulating the postsynaptic ionotropic glutamate receptors.

189 ystemic elimination and the kidney expresses ionotropic glutamate receptors.

190 key neuronal signaling molecules, including ionotropic glutamate receptors.

191 provide target validation for this family of ionotropic glutamate receptors.

192 All three ionotropic glutamate subfamilies (i.e. AMPA-type, kainat

193 gle sub-psychotomimetic dose of ketamine, an ionotropic glutamatergic n-methyl-D-aspartate (NMDA) rec

194 nt clinical studies report that ketamine, an ionotropic glutamatergic N-methyl-D-aspartate (NMDA) rec

195 ngle subpsychotomimetic dose of ketamine, an ionotropic glutamatergic N-methyl-D-aspartate (NMDA) rec

196 ted by hypoxia was blunted after blockade of ionotropic glutamatergic receptors at the level of the p

197 signalling in PF-PC spines does not involve ionotropic glutamatergic receptors because postsynaptic

198 N-methyl-d-aspartate receptors (NMDARs) are ionotropic glutamatergic receptors that have been implic

199 hey show that GluD1, through a non-canonical ionotropic-independent mechanism, controls GABAergic syn

200 ovel analgesic strategy is to restore spinal ionotropic inhibition by enhancing KCC2-mediated chlorid

201 ng KCC2 may be a tenable method of restoring ionotropic inhibition not only in neuropathic pain but a

202 ps, it remains unclear whether and how these ionotropic inputs are amplified in olfactory receptor ne

203 s on chromosome 1 (GRIK3 (glutamate receptor ionotropic kainate 3)), chromosome 4 (KLHL2 (Kelch-like

204 ate receptors containing glutamate receptor, ionotropic, kainate 2).

205 Although a G-protein-dependent non-ionotropic mechanism has been suggested to underlie this

206 Spine shrinkage was initiated by non-ionotropic (metabotropic) signaling through NMDARs, and

207 Owing to the dual ionotropic/metabotropic nature of alpha7 receptors, sign

208 mplification) of the gene glutamate receptor ionotropic N-methyl D-aspertate as a potential new thera

209 based antidepressants by not only modulating ionotropic (N-methyl-D-aspartate and alpha-amino-3-hydro

210 d by UNC-3 also regulating the expression of ionotropic neurotransmitter receptors and putative stret

211 Ionotropic neurotransmitter receptors mediate fast synap

212 Ionotropic neurotransmitter receptors mediate fast synap

213 mental differences in the glutamate receptor ionotropic NMDA 2 (GluN2) subunit composition of NMDARs

214 ging and time-lapse imaging to show that non-ionotropic NMDAR signaling can drive shrinkage of dendri

215 Here, we tested whether non-ionotropic NMDAR signaling could also play a role in dri

216 ments, we identify key components of the non-ionotropic NMDAR signaling pathway driving dendritic spi

217 delineating the molecular mechanisms of non-ionotropic NMDAR signaling that can drive shrinkage and

218 MAPK is generally required downstream of non-ionotropic NMDAR signaling to drive both spine shrinkage

219 tivation of NMDARs, whereas costimulation of ionotropic non-NMDAR glutamate receptors transiently ant

220 lthough pharmacological activation of either ionotropic or cAMP-dependent pathways acted in synergy w

221 trains the high-power state because blocking ionotropic or metabotropic glutamate receptors results i

222 rtant functional roles mediated by ATP-gated ionotropic P2X receptors (P2XRs).

223 Actions of ATP are mediated through both ionotropic P2X receptors and metabotropic P2Y receptors.

224 P2Y receptors are G-protein-coupled, whereas ionotropic P2X receptors are ATP-gated ion channels.

225 osphate (ATP) induces pain via activation of ionotropic P2X receptors while adenosine mediates analge

226 is usually attributed to conduction through ionotropic P2X receptors.

227 This response depends on both ionotropic (P2X) and metabotropic (P2Y) purinergic recep

228 nel/pannexin1 (Panx1) channel and purinergic ionotropic P2X7 receptor (P2X7R) blockers.

229 The ATP-gated ionotropic P2X7 receptor (P2X7R) modulates glial activat

230 Here we show that the ATP-gated ionotropic P2X7 receptor restricts the expansion of aber

231 compatible with activation of the ATP-gated ionotropic P2X7 receptor.

232 mechanism involving autocrine activation of ionotropic P2X7 receptors (P2X7R) by ATP.

233 The lymphocytic ionotropic purinergic P2X receptors (P2X1R-P2X7R, or P2X

234 ing cell damage/activation, is sensed by the ionotropic purinergic receptor P2X7 (P2X7R) on lymphocyt

235 s extracellular stimulation of two prominent ionotropic purinergic receptors, P2X4 and P2X7, with the

236 Taste buds release ATP to activate ionotropic purinoceptors composed of P2X2 and P2X3 subun

237 n the hippocampal CA1 region is dependent on ionotropic, rather than metabotropic, NMDAR signaling.

238 ials required several members of the variant ionotropic receptor (IR) family (IR25a, IR62a, and IR76b

239 express IR92a, a member of the chemosensory ionotropic receptor (IR) family and project to a pair of

240 All sour GRNs prominently express two Ionotropic Receptor (IR) genes, IR76b and IR25a, and we

241 The Ionotropic Receptor (IR) superfamily is best known for i

242 odor receptor (Or), gustatory receptor (Gr), ionotropic receptor (IR), Pickpocket (Ppk), and Trp fami

243 One gene, Ionotropic receptor (IR)52a, is coexpressed in neurons o

244 Here we show that Drosophila Ionotropic Receptor 25a (IR25a) is required for behaviou

245 and behavioral analyses, we demonstrate that ionotropic receptor 8a (IR8a) is essential for acid-medi

246 A new study shows that the ionotropic receptor 8a (IR8a) plays a primary and non-re

247 glutamate-induced up-regulation of glutamate ionotropic receptor alpha-amino-3-hydroxy-5-methyl-4-iso

248 NMDA-type subunit 2B (GRIN2B) and glutamate ionotropic receptor AMPA-type subunit 2 (GRIA2).

249 rs and demonstrate that they form functional ionotropic receptor channels.

250 acts in mechanosensory neurons by modulating ionotropic receptor currents, the initiating step of cel

251 enriched in female antennae, thus making the ionotropic receptor family the largest of antennae-rich

252 These findings identify TRPV1 as an ionotropic receptor for retinoids and provide cellular a

253 y monocytes but does not stimulate canonical ionotropic receptor functions.

254 GRN activation requires the function of the Ionotropic Receptor genes IR25a, IR76b and IR56d.

255 Most odorant and ionotropic receptor genes seemed to be expressed in all

256 A neuron responding to moist air and its ionotropic receptor have been identified in Drosophila m

257 rosophila melanogaster to identify Hodor, an ionotropic receptor in enterocytes that sustains larval

258 Here, we identify ionotropic receptor IR21a, a receptor conserved througho

259 Neurons expressing the ionotropic receptor IR40a have been implicated in the se

260 regarding its mode of action: activation of ionotropic receptor IR40a vs. odorant receptor(s).

261 The ionotropic receptor IR76b has been shown to be expressed

262 y protein subunits) or NMDARs [via glutamate ionotropic receptor NMDA-type subunit 2B (GluN2B) subuni

263 s in the expression of its targets glutamate ionotropic receptor NMDA-type subunit 2B (GRIN2B) and gl

264 dependent on the subunit composition of the ionotropic receptor or channel as well as the GPCR subty

265 The P2X7 receptor is an ionotropic receptor predominantly expressed on the surfa

266 RNA levels for glycinergic and glutamatergic ionotropic receptor subunits, confirming a switch from G

267 ct activation of an odorant receptor, not an ionotropic receptor, is necessary for DEET reception and

268 h the synaptic activation of the fast-acting ionotropic receptor, LGC-55, and extrasynaptic activatio

269 t volatile molecules using olfactory (OR) or ionotropic receptors (IR) and in some cases gustatory re

270 Ionotropic Receptors (IRs) are a large subfamily of vari

271 ceptors (ORs), gustatory receptors (GRs) and ionotropic receptors (IRs) function to interface the ins

272 at Drosophila hygrosensation relies on three Ionotropic Receptors (IRs) required for dry cell functio

273 e show that DOCC cool-sensing is mediated by Ionotropic Receptors (IRs), a family of sensory receptor

274 olfactory receptors that remain, such as the ionotropic receptors (IRs), could play a significant rol

275 Instead, three Ionotropic Receptors (IRs), IR21a, IR25a, and IR93a, spe

276 ne in Drosophila a group of approximately 35 ionotropic receptors (IRs), the IR20a clade, about which

277 the odorant receptors (ORs) and the variant ionotropic receptors (IRs).

278 (Rs) constitute a subclass of ATP-sensitive ionotropic receptors (P2X1-P2X7).

279 lear hair cells via alpha9alpha10-containing ionotropic receptors and associated calcium-activated (S

280 Axons can be depolarized by ionotropic receptors and transmit subthreshold depolariz

281 Ion channels and ionotropic receptors can also be reconstituted from memb

282 a simple behavioral assay, we find that the ionotropic receptors IR40a, IR93a, and IR25a are all req

283 Up-regulation of these ionotropic receptors is independent of signaling by meta

284 ethyl d-aspartate receptors are ligand-gated ionotropic receptors mediating a slow, calcium-permeable

285 Ionotropic receptors of gamma-aminobutyric acid (GABAAR)

286 of intracellular Ca(2+) via metabotropic and ionotropic receptors or direct UV-uncaging.

287 Identification of ionotropic receptors required for hygrosensation in Dros

288 tion of most representative Ca(2+)-permeable ionotropic receptors similarly regulate T-type current p

289 of additional lipids and the trafficking of ionotropic receptors to plasma membranes.

290 Transcripts for as many as 43 ionotropic receptors were enriched in female antennae, t

291 w' (G-protein-coupled receptors) and 'fast' (ionotropic receptors) neurotransmission converging on th

292 y with regard to targets on metabotropic and ionotropic receptors.

293 noamine transporters should be considered as ionotropic receptors.

294 ions in the subunit composition of glutamate ionotropic receptors.

295 luding the feature and surface expression of ionotropic receptors.

296 annel in vitro, identifying it as a probable ionotropic sensory receptor.

297 The ionotropic serotonin receptor, 5-HT3 , is expressed by m

298 x through the NMDAR normally overcomes a non-ionotropic shrinkage signal to drive spine growth.

299 g an amplification mechanism compatible with ionotropic signaling.

300 Thus, to explore the role of ionotropic versus metabotropic NMDAR signaling in LTD, w