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1 change in iron status (serum ferritin, total iron binding capacity).
2 e effects of TF rs3811647 on transferrin and iron binding capacity.
3 ng women with higher concentrations of total-iron-binding capacity.
4 nsferrin level (1.77 +/- 0.08 g/L) and total iron-binding capacity (46.2 +/- 2.0 microM) were signifi
7 uble transferrin receptor, ferritin, unbound iron-binding capacity and full blood count were assayed.
9 erritin, and hepcidin were reduced and total iron-binding capacity and soluble transferrin receptor i
10 arameters (serum iron, serum ferritin, total iron-binding capacity and transferrin saturation), serum
11 in secondary structure, DNA binding ability, iron binding capacity, and the ability to form higher-or
12 ficant increases in serum iron levels, total iron-binding capacity, and transferrin saturation from b
14 95% CI: -16.3%, -2.6%) and lower unsaturated iron-binding capacity (by -7.8%; 95% CI: -13.4%, -1.7%).
15 combined analyses of studies involving total iron-binding capacity (combined risk ratio, 1.0; 95% CI,
17 centration, iron saturation, and unsaturated iron-binding capacity differed between the three investi
19 hange from baseline of serum ferritin, total-iron-binding capacity, erythrocyte protoporphyrin, or he
20 io 16.0, P < 0.0001), 24% higher unsaturated iron-binding capacity (F ratio 12.8, P < 0.0001) and 25%
22 g, serum ferritin level >15 ng/mL, and total iron-binding capacity <425 mug/dL at the 12-week visit),
25 owed the regulation of iron levels and total iron-binding capacity of beta cells by eugenol in strept
26 pendent transport system, which exploits the iron-binding capacity of citrate and its natural abundan
27 ence time did not affect the carbon specific iron-binding capacity of the humic substances which was
28 ver, in conditions of iron overload when the iron-binding capacity of transferrin is exceeded, non-tr
30 ve group 58.3+/-16.2%, P=0.05; minimum total iron-binding capacity: restrictive group 0.59+/-0.21%, n
31 itin, transferrin saturation (SAT) and total iron binding capacity (TIBC) in 2347 AAs participating i
32 ith available serum iron, ferritin and total iron binding capacity (TIBC) values were ultimately incl
33 s were change of hemoglobin, ferritin, total iron binding capacity (TIBC), and iron saturation (ISAT)
34 -wide association study of serum iron, total iron binding capacity (TIBC), transferrin saturation, an
37 rotein (CRP), cobalamin, folate, iron, total iron-binding capacity (TIBC), and 25-hydroxy (25-OH) vit
38 , vitamin B12, zinc, folate, ferritin, total iron-binding capacity (TIBC), and high sensitivity C-rea
39 stance index and increased serum iron, total iron binding capacity, transferrin saturation, and serum
40 decrease (P < 0.05) in serum ferritin, total-iron-binding capacity, transferrin saturation, the ratio
41 on mobilization (ferritin, serum iron, total-iron-binding-capacity, transferrin saturation) were sign
42 gly associated with transferrin, unsaturated iron-binding capacity (UIBC) and transferrin saturation
43 n (serum iron concentration divided by total iron binding capacity) was used as a measure of the amou
44 nalyses for transferrin saturation and total iron binding capacity, while the estimate for ferritin w