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1 lity at 18 months among infants with hypoxic-ischemic encephalopathy.
2 died had a cause of death other than hypoxic-ischemic encephalopathy.
3 ity at age 18 months in infants with hypoxic-ischemic encephalopathy.
4 m recovery potential in patients with anoxic-ischemic encephalopathy.
5 ng the critical period for perinatal hypoxic-ischemic encephalopathy.
6 eonatal death and moderate-to-severe hypoxic-ischemic encephalopathy.
7 g children who did not have neonatal hypoxic-ischemic encephalopathy.
8 rapeutic target in infants suffering hypoxic-ischemic encephalopathy.
9 brain injury, and eight patients had hypoxic ischemic encephalopathy.
10 y in infants with moderate or severe hypoxic-ischemic encephalopathy.
11 ow of therapeutic opportunity during hypoxic-ischemic encephalopathy.
12 and received a clinical diagnosis of hypoxic-ischemic encephalopathy.
13 ge (IPH), extraaxial hemorrhage, and hypoxic-ischemic encephalopathy.
14 iac arrest could aggravate prolonged hypoxic ischemic encephalopathy.
15      One patient died as a result of hypoxic ischemic encephalopathy.
16 an brains in Alzheimer's disease and hypoxic-ischemic encephalopathy.
17 aumatic brain injury (62 [30%]), and hypoxic-ischemic encephalopathy (21 [10%]).
18 %), intracerebral hemorrhage (3.4%), hypoxic ischemic encephalopathy (3.6%), and spinal cord injury (
19 nce interval) were calculated: adult hypoxic-ischemic encephalopathy: absent 0% (0%-1%), abnormal 22%
20 justed HR, 46.4; 95% CI, 42.2-51.0), hypoxic ischemic encephalopathy (adjusted HR, 23.6; 95% CI, 20.6
21             They are often caused by hypoxic-ischemic encephalopathy and are frequently refractory to
22  and cerebral palsy in neonates with hypoxic ischemic encephalopathy and correlated well with qualita
23 were compared with severity of acute hypoxic-ischemic encephalopathy and long-term clinical outcome.
24 ebral sinovenous thrombosis, 11 with hypoxic ischemic encephalopathy, and 5 with neonatal arterial is
25 ontraumatic intracranial hemorrhage, hypoxic-ischemic encephalopathy, and neonatal mortality.
26 very room cardiorespiratory support, hypoxic-ischemic encephalopathy, and therapeutic hypothermia in
27 onates (>/=36 weeks' gestation) with hypoxic-ischemic encephalopathy at 18 US centers in the Eunice K
28 death or disability for infants with hypoxic-ischemic encephalopathy at 36 weeks' or later gestation.
29  to the NICU with moderate-to-severe hypoxic-ischemic encephalopathy at day 1 to 5 during hospitaliza
30  unit (NICU) with moderate-to-severe hypoxic-ischemic encephalopathy at day 1 to 5 during hospitaliza
31 s' gestation with moderate or severe hypoxic-ischemic encephalopathy at less than 6 hours after birth
32                      Upon unilateral hypoxic-ischemic encephalopathy at P7, monocular deprivation at
33 duce death or disability in neonatal hypoxic ischemic encephalopathy but data on seizures during rewa
34 roprotective effects in infants with hypoxic-ischemic encephalopathy, but its effects on neurodevelop
35 erm neonates with moderate or severe hypoxic-ischemic encephalopathy, cooling for longer than 72 hour
36 imaging following moderate or severe hypoxic-ischemic encephalopathy developed by the National Instit
37 dergoing therapeutic hypothermia for hypoxic-ischemic encephalopathy did not result in a lower risk o
38 er gestation with moderate or severe hypoxic-ischemic encephalopathy enrolled at 6 to 24 hours after
39 rth weight, very low birth weight, hypoxemic ischemic encephalopathy, extracorporeal membrane oxygena
40              Seven of these cases of hypoxic-ischemic encephalopathy followed uterine rupture (absolu
41 tment for term infants with moderate hypoxic-ischemic encephalopathy found reduced disability.
42 peutic hypothermia (TH) for neonatal hypoxic-ischemic encephalopathy (HIE) (cases) compared to 42 con
43                                      Hypoxic ischemic encephalopathy (HIE) affects 2-3 per 1000 full-
44 le; 152 (72%) had moderate to severe hypoxic-ischemic encephalopathy (HIE) and 147 (84%) had electrog
45 xpression patterns in human neonatal hypoxic-ischemic encephalopathy (HIE) and MS as well as developm
46 Rationale: Asphyxiated neonates with hypoxic ischemic encephalopathy (HIE) are at risk of myocardial
47                       Outcomes after hypoxic-ischemic encephalopathy (HIE) are variable.
48 othermia, the standard treatment for hypoxic-ischemic encephalopathy (HIE) in high-income countries (
49 nd hypothermic neuroprotection after hypoxic-ischemic encephalopathy (HIE) in low- and middle-income
50                                      Hypoxic-ischemic encephalopathy (HIE) in neonates results in lon
51                                      Hypoxic-ischemic encephalopathy (HIE) is a critical cerebral eve
52                                      Hypoxic ischemic encephalopathy (HIE) is a major cause of neonat
53                            Perinatal hypoxic-ischemic encephalopathy (HIE) is a significant cause of
54                            Perinatal hypoxic-ischemic encephalopathy (HIE) is an important cause of m
55 ent (NDI) for infants diagnosed with hypoxic ischemic encephalopathy (HIE) is important for parental
56                                      Hypoxic-ischemic encephalopathy (HIE) occurs in 1 to 8 per 1000
57                                      Hypoxic-ischemic encephalopathy (HIE) remains an important contr
58                             Neonatal hypoxic-ischemic encephalopathy (HIE) still carries a high burde
59 ) and newborn brain injuries such as hypoxic ischemic encephalopathy (HIE) that cause cerebral palsy.
60                        Neonates with hypoxic-ischemic encephalopathy (HIE) undergoing therapeutic hyp
61 ected neonatal unit (NNU) admission, hypoxic ischemic encephalopathy (HIE), and perinatal mortality (
62 hermia is the standard treatment for hypoxic-ischemic encephalopathy (HIE), but despite its widesprea
63 a is widely used after mild neonatal hypoxic-ischemic encephalopathy (HIE), safety and efficacy have
64 ween simple T2h and complex T2h with hypoxic-ischemic encephalopathy (HIE).
65  imaging characteristics of neonatal hypoxic-ischemic encephalopathy (HIE).
66 th adverse neonatal outcomes such as hypoxic-ischemic encephalopathy (HIE).
67 ariation exists in the management of hypoxic-ischemic encephalopathy (HIE).
68 fants 33 to 35 weeks' gestation with hypoxic-ischemic encephalopathy, hypothermia at less than 6 hour
69              Among term infants with hypoxic-ischemic encephalopathy, hypothermia initiated at 6 to 2
70                    This model mimics hypoxic ischemic encephalopathy in humans and may be appropriate
71 n or equal to 17 mug/L argue against hypoxic-ischemic encephalopathy incompatible with reawakening.
72                             Neonatal hypoxic-ischemic encephalopathy is an important cause of death a
73 re full-term with moderate or severe hypoxic ischemic encephalopathy, longer cooling, deeper cooling,
74 rth (umbilical cord artery pH <7.0), hypoxic ischemic encephalopathy, neonatal seizures, neonatal res
75                                      Hypoxic-ischemic encephalopathy occurred in no infants whose mot
76 guidelines (adult cardiac arrest and hypoxic-ischemic encephalopathy of newborns) or intraoperative h
77 guidelines (adult cardiac arrest and hypoxic-ischemic encephalopathy of newborns).
78 ical ventilation, and risk of death, hypoxic ischemic encephalopathy or respiratory arrest.
79                   Among infants with hypoxic-ischemic encephalopathy, outcomes were similar between i
80 ely analyze the hypoxic component of hypoxic-ischemic encephalopathy, rats were prepared such that th
81  degrees C for 72 hours for neonatal hypoxic ischemic encephalopathy reduces death or disability to 4
82 hours at 33.5 degrees C for neonatal hypoxic-ischemic encephalopathy reduces death or disability, but
83 tic barbiturate therapy for neonatal hypoxic-ischemic encephalopathy showed no significant effect on
84  whole-body hypothermia for neonatal hypoxic-ischemic encephalopathy showing a significant reduction
85 of gestation with moderate or severe hypoxic-ischemic encephalopathy to receive erythropoietin or pla
86 elopmental outcomes in neonates with hypoxic-ischemic encephalopathy using MRI and basic clinical dat
87        Neuroprognostic approaches to hypoxic-ischemic encephalopathy vary among physicians and are of
88 s; age range, 2-12 days) with severe hypoxic-ischemic encephalopathy were examined during the first 1
89                      In infants with hypoxic-ischemic encephalopathy who have undergone therapeutic h
90 s stroke, traumatic brain injury, or hypoxic ischemic encephalopathy with a Glasgow Coma Scale score
91                  Adults in coma from hypoxic-ischemic encephalopathy with absent somatosensory evoked