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1 get in each individual (i.e. the critical AT isthmus).
2  specificity for identifying the clinical VT isthmus.
3 med at the pulmonary vein and cavo-tricuspid isthmus.
4 ned vertebral phenotype featured by lamellar isthmus.
5 al of most midbrain/hindbrain cells near the isthmus.
6 71%) had an identifiable endocardial circuit isthmus.
7 tified to attempt to trace the course of the isthmus.
8 ding projections beyond the diencephalon and isthmus.
9 the incidence and microscopic anatomy of the isthmus.
10 t mapping overestimates the true size of the isthmus.
11 was consistent with entrainment criteria for isthmus.
12 s quiescent stem cells in the gastric corpus isthmus.
13 arrowest accessible slow-conducting critical isthmuses.
14 mic isthmuses containing VT re-entry circuit isthmuses.
15 +/-0.3 m/s) and nearly normal at the central isthmus (0.62+/-0.2 m/s).
16                                              Isthmus 1A had the greatest dimensions (mean length, 3.9
17                                              Isthmus 1A is associated with the largest morphological
18             For subjects>/=5 years at death, isthmuses 1A and 1B were present in 88%, isthmus 2 in 25
19 ength, 3.9+/-1.08; thickness, 1.5+/-0.3 cm), isthmus 1B intermediate dimensions (mean length, 2.4+/-0
20 mensions, whereas the nearby newly described isthmus 1B is significantly smaller.
21 th, isthmuses 1A and 1B were present in 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%
22 th, 2.4+/-0.8; thickness, 1.1+/-0.4 cm), and isthmuses 2, 3, and 4 the smallest dimensions (mean leng
23 ) was significantly shorter than the central isthmus (24 mm +/- 4.3; range, 12-43 mm) and the central
24 l isthmus was shorter than the inferolateral isthmus (27 mm +/- 4.8; range, 13-45 mm) (P < .001).
25 nd 1B were present in 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%.
26   Of isthmuses with the smallest dimensions, isthmus 3 is the most common.
27                          The incidence of an isthmus 3 mm from the apex was 89.3% and 100% in the MB
28 es (N=30) were less commonly observed within isthmuses (3.3%, P<0.001) or within 5 mm of the leading
29 ium roof (12 RF/22 PF), and 44 cavotricuspid isthmus (36 RF/8 PF) lines, with therapy duration times
30 88%, isthmus 2 in 25%, isthmus 3 in 94%, and isthmus 4 in 13%.
31  function and isthmus-dependent re-entry, VT isthmus ablation can be curative.
32  cava isolation in 6 patients, cavotricuspid isthmus ablation in 5 patients, and ablation of sites of
33                    Therefore, cavo-tricuspid isthmus ablation is appropriate during pulmonary vein is
34 block were thoroughly reviewed in 271 mitral isthmus ablation procedures undertaken among 236 patient
35                               Cavo-tricuspid isthmus ablation was performed in 28 of the 133 patients
36   Among the 105 patients who did not undergo isthmus ablation, 25 patients (24%) were documented to h
37 on was performed followed by roof and mitral isthmus ablation, before CFAE ablation in the CFAE arm.
38 ocal PFA catheter was used for cavotricuspid isthmus ablation.
39 0) with symptomatic atrial flutter underwent isthmus ablation.
40 timulation maneuvers and occasional complete isthmus ablation.
41 ibility of pulmonary vein and cavo-tricuspid isthmus ablation.
42               All patients had successful VT isthmus ablation.
43  posterior/inferior lines, and cavotricuspid isthmus ablation.
44              TTE cells were localized to the isthmus, above and distinct from TFF2 protein-expressing
45              TTE cells were localized in the isthmus adjacent to doublecortin CaM kinase-like-1(+) pu
46 ization and allows a better understanding of isthmus anatomy.
47 ed the intra-mural segment, 14 presented the isthmus and 15 presented the ampulla and fimbria segment
48            The low CF obtained at the mitral isthmus and anterior segments of the left pulmonary vein
49 oposed for gastric corpus stem cells in both isthmus and base regions.
50 e show that Gli3 regulates patterning of the isthmus and cerebellar anlage by confining Fgf8 expressi
51 and E11.0, Gli3 continues to be required for isthmus and cerebellum development, but primarily for de
52  Otx2 in the hindbrain, thereby allowing the isthmus and cerebellum to form.
53 or establishing a distinct posterior tectum, isthmus and cerebellum, but does not play a role in the
54 deling studies recapitulated DEEPs at the VT isthmus and demonstrated their role in VT initiation wit
55 on (CI) was performed incorporating putative isthmus and early exit site(s) based on standard criteri
56 ontractions of the muscles in the pharyngeal isthmus and function systemically to regulate an enhance
57  proliferation is reduced in both the normal isthmus and in the mutant anterior r1.
58  a linear lesion-successfully eliminated the isthmus and local abnormal voltage activities.
59  procedural success (transection of anatomic isthmus and noninducibility) and freedom of VT recurrenc
60  differences in the median dimensions of the isthmus and path length between fast and slow VTs and be
61 lation lines also were created in the mitral isthmus and posterior left atrium.
62 rtic sites terminated VT related to a septal isthmus and prevented reinduction.
63 rmation that forms ventromedially within the isthmus and r1.
64   Adult D673V homozygotes exhibit dysplastic isthmus and reduced bone volume of the dorsal vertebra r
65 overing that the IP develops strictly within isthmus and rhombomere 1.
66 d to be restricted to the Western end of the Isthmus and the "militaris" phenotypes restricted to sit
67        The mean dentin thickness between the isthmus and the distal root surface was <1 mm at a dista
68 s this, we conditionally ablated Fgf8 in the isthmus and uncovered that prolonged expression of Fgf8
69 ty of any VT and transection of the anatomic isthmus and was achieved in 25 (74%) patients.
70 fter PVI and linear lesions (roof and mitral isthmus), and biatrially after LA CFE ablation.
71 r anlage by confining Fgf8 expression to the isthmus, and attenuates growth of dorsal r1 (before E11.
72 enitors results in deletion of the midbrain, isthmus, and cerebellum.
73  secondary gustatory/visceral nucleus in the isthmus, and for distinguishing territories in the prima
74 ng for the loss of brain structures near the isthmus, and instead demonstrate that tissue transformat
75  regions such as the midbrain tectum, dorsal isthmus, and motor nuclei, ASP and GABA immunoreactivity
76 m body, anterior midbody, posterior midbody, isthmus, and splenium) and for overall CC size, with lef
77 pital fasciculus, internal capsule, callosal isthmus, and the corona radiata (p=0.04 for FIQ and p=0.
78  from the floor plate (DV) and Fgf8 from the isthmus (AP).
79 es that target both scar-related and classic isthmuses appear necessary to prevent long-term recurren
80  targeted deletion of PPARgamma in the bulge/isthmus area of the hair follicle epithelium generates a
81 iculum of the isthmus (PaSi), area 29 of the isthmus (area 29i) and area prostriata (Pro), which has
82 ial slowing during SR corresponded to the VT isthmus (area under curve=0.84 95% CI, 0.78-0.90) while
83 6 of the 19 patients, a total of 41 distinct isthmus areas of 41 distinct VTs were identified and suc
84 in comparison with controls, although aortic isthmus/arterial duct diameter ratio was lower in fetuse
85 8 signaling from the mid/hindbrain boundary (isthmus) as being responsible for induction of different
86 part of the cingulum and the cingulate gyrus isthmus, as well as the precuneal GM, may be distinctive
87 ngeal muscles and marginal cells forming the isthmus between the anterior and posterior pharyngeal bu
88 accharomyces cerevisiae they localize at the isthmus between the mother and the daughter cells, where
89        Linear radiofrequency ablation in the isthmus between the TA and IVC (TI isthmus) terminated t
90 sal r1 (before E11.0) and the dorsal mes and isthmus (beyond E11.0) through regulation of cell prolif
91     Short-term end points were bidirectional isthmus block and no inducible AFL.
92  radiofrequency catheter ablation similar to isthmus block for atrial flutter.
93 is study evaluates the long-term efficacy of isthmus block for treatment of re-entry VT in adults wit
94                              Complete mitral isthmus block predicted greater CFE reduction (P=0.02).
95  the focal PFA catheter, acute cavotricuspid isthmus block was achieved in 13 of 13 patients (median:
96      All the available assessments of mitral isthmus block were thoroughly reviewed in 271 mitral ist
97 uch-up lesions was necessary to complete the isthmus block with conventional fluoroscopy (median, thr
98 ictors for unsuccessful bidirectional mitral isthmus blockade were the need for epicardial ablation f
99                       Regions forming the VT isthmus borders had faster activation during SR while re
100 cardiomyopathy, obliteration of a conductive isthmus both anatomically and functionally and abolition
101 r versus parallel to the line of block along isthmus boundaries (19.3+/-7.1 versus 13.6+/-7.4 cm(2),
102 during SR correspond to sites forming the VT isthmus but not to bystander sites.
103 l in which Wnt regulates FGF activity at the isthmus by driving both FGF and Sprouty gene expression.
104 potent substrate for arrhythmogenesis at the isthmus/BZ of chronically infarcted hearts.
105                 Ventricular tachycardia (VT) isthmuses can be defined by fixed or functional block.
106                                           VT isthmuses can be identified and part of their course del
107 frequency ablation by targeting the critical isthmus (CI).
108      Atrophy in mesial and lateral temporal, isthmus cingulate, and orbitofrontal areas aided discrim
109 eral orbitofrontal cortex, and the bilateral isthmus cingulate.
110 , across Beringea, and across the Panamanian isthmus coincide in timing and location with multiple we
111 y AFL was ablated by achieving bidirectional isthmus conduction block.
112          Ablation aimed to transect anatomic isthmuses containing VT re-entry circuit isthmuses.
113                   Ablation targeted anatomic isthmuses containing VT re-entry circuits, which were id
114                                              Isthmus contraction occurred primarily in the posterior
115 ow-up, WM volume in the left cingulate gyrus isthmus correlated with clinical scores of anxiety (Spea
116 m of the study was to determine whether a VT isthmus could be identified and followed by pace mapping
117                           Of 89 ICM VTs, the isthmus could be identified by endocardial entrainment i
118 ined endocardial and epicardial mapping, the isthmus could be identified in 56 (63%) ICM VTs and 12 (
119        Achievement of complete cavotricuspid isthmus (CTI) conduction block reduces typical atrial fl
120 or scar-dependent (n = 15) and cavotricuspid isthmus (CTI)-dependent (n = 14) flutter were studied.
121 al fibrillation may experience cavotricuspid isthmus (CTI)-dependent atrial flutter during follow-up.
122  atrial flutter (AFL) from the cavotricuspid isthmus (CTI).
123                   However, dimensions of the isthmus defined by entrainment exceeded dimensions of th
124 cy lesions created a substrate for sustained isthmus-dependent AFL, confirmed by endocavity mapping.
125        The aim of this study was to separate isthmus-dependent atrial flutter (IDAFL) from non-isthmu
126 t atrial tachycardias included cavotricuspid isthmus-dependent atrial flutter (n=7), non-isthmus-depe
127 us-dependent atrial flutter (IDAFL) from non-isthmus-dependent atrial flutter (NIDAFL) from the elect
128 rial tachycardia was seen in 7 patients, and isthmus-dependent atrial flutter occurred in 14 patients
129 ring 66 ATs in 62 patients: 20 cavotricuspid isthmus-dependent ATs, 20 perimitral ATs, 13 focal ATs w
130 to delineate the precise mechanism of the TI isthmus-dependent clockwise right atrial flutters.
131  from the scar to another anatomic boundary (isthmus-dependent group).
132                                       In the isthmus-dependent group, three of seven (42%) patients w
133       Methods We studied 24 noncavotricuspid isthmus-dependent macroreentrant atypical atrial flutter
134 ease with preserved ventricular function and isthmus-dependent re-entry, VT isthmus ablation can be c
135  isthmus-dependent atrial flutter (n=7), non-isthmus-dependent right atrial reentry (n=7), and 1 foca
136 -macro-re-entrant (perimitral, cavotricuspid isthmus-dependent, and roof-dependent circuits) versus c
137                                      Central isthmus depth was classified as straight (3 mm), concave
138                                  Mean aortic isthmus diameter z scores measured either in sagittal (P
139 ate for the initial block at the mouth of an isthmus/diastolic channel leading to ventricular tachyca
140                             Consistencies in isthmus dimensions and histology are found among patient
141 ograms were indicative of spatially confined isthmus dimensions, confirmed by rapid termination of VT
142 est: the optic tectum, torus semicircularis, isthmus, dorsal and medial nuclei of the octavolateral a
143 size that HSPGs are necessary for pharyngeal isthmus elongation, and pyr-1 functions upstream of prot
144 t pyr-1(cu8) exhibiting defective pharyngeal isthmus elongation, cytoskeletal organization defects, a
145 ggest that volcanism also contributed to the Isthmus emergence in the Early Miocene.
146 ansion and the repolarizing cells within the isthmus enabled retrograde flow of depolarizing electrot
147 ere slowest at the inward curvature into the isthmus entrance (0.28+/-0.2 m/s), slightly faster at th
148 h gastric mucus neck cells located below the isthmus express trefoil factor family 2 (TFF2) protein,
149 ressing markers of junctional zone and upper isthmus follicular stem cells.
150 blation has been used to target the critical isthmuses for re-entrant monomorphic ventricular tachyca
151 amber of origin in all 10, and distinguished isthmus from nonisthmus dependent atrial flutter.
152 ing and the relationship to the protected VT isthmus identified by entrainment mapping is unknown.
153 sful ablation sites were localized within an isthmus identified by pace mapping in all of these 10 pa
154                         Of the 19 VT circuit isthmuses identified, 12 were associated with an endocar
155 ed RFCA for VTs dependent on septal anatomic isthmuses improves ablation outcome in repaired Tetralog
156                                 The critical isthmus in 115 of the 120 LA re-entrant ATs (96%) traver
157 lopment and becomes restricted mainly to the isthmus in adult glands, akin to its known localization
158                                The diastolic isthmus in ventricular tachycardia was mapped in 3 patie
159 ) in repaired Tetralogy of Fallot focuses on isthmuses in the right ventricle but may be hampered by
160 ing/block may aid identification of critical isthmuses in unmappable VTs.
161                         Anatomically defined isthmuses included (1A) ventriculotomy-to-tricuspid annu
162 molars, the likelihood of the presence of an isthmus increased.
163                                   The mitral isthmus is a critical part of perimitral reentrant tachy
164               These findings reveal that the isthmus is a key cortical zone connecting both the cingu
165 mical results presented here reveal that the isthmus is composed of four cortical areas.
166 with the addition of epicardial mapping, the isthmus is less commonly identified, possibly due to mid
167 timing of dispersal or vicariance across the Isthmus is not explained by the ecological factors teste
168 whereas the absence of Gly-ir neurons in the isthmus is shared by all these species, except for lampr
169                                   The mitral isthmus is the critical element of perimitral reentrant
170                                          The Isthmus is traditionally understood to have fully closed
171 e that the CV of the outer loop, rather than isthmus, is the principal determinant of the rate of VT.
172 ovides an evaluation of the ET cartilage and isthmus level, which are small but important anatomical
173 the anterior region of r1 is converted to an isthmus-like tissue.
174                         The effect of mitral isthmus line (MIL) ablation on outcomes after SRI has no
175          Deployment of an endocardial mitral isthmus line (MIL) with the end point of bidirectional b
176 ary vein isolation in 50 (100%), left atrial isthmus line in 47 (94%), anterior line in 45 (90%), com
177 cing techniques in patients with left mitral isthmus linear ablation.
178 essment of bidirectional block across mitral isthmus linear lesion using differential coronary sinus
179 round the left septum primum with a critical isthmus located between the pulmonary veins posteriorly
180 tes (N=27) were more commonly located within isthmuses &lt;15 mm wide (67% versus 6.7%, P<0.00001; odds
181 on wavefront running perpendicular to the VT isthmus may increase sensitivity to detect arrhythmogeni
182               At middiastole, the paraseptal isthmus (mean length, 20 mm +/- 3.5; range, 11-34 mm) wa
183 ed by entrainment exceeded dimensions of the isthmus measured by activation mapping by 32+/-18%.
184                  All tachycardias crossed an isthmus (median, 0.52 mV, 13 mm) bordered by nonconducti
185               Achieving bidirectional mitral isthmus (MI) block using radiofrequency catheter ablatio
186 tion, the completion of the Central American Isthmus more extensively than any other.
187 n strategies, ablations of the cavotricuspid isthmus (n=4), fossa ovalis (n=4), and pulmonary veins (
188 ively cycling stem cells maintaining the pit-isthmus-neck region through a process of "punctuated" ne
189 hat rapidly cycling IsthSCs maintain the pit-isthmus-neck region.
190 had a sensitivity and specificity for the VT isthmus of 29+/-10% and 95+/-1%, respectively.
191                        Egypt, located on the isthmus of Africa, is an ideal region to study historica
192 d BZ channels identified 74% of the critical isthmus of clinical VTs and 50% of all the conducting ch
193 gene Axin2, is limited to the base and lower isthmus of gastric glands, where the stem cells reside.
194                         The stem cell in the isthmus of gastric units continually replenishes the epi
195 gh the activity of stem cells located in the isthmus of individual gland units.
196                             We show that the Isthmus of Kra represents a significant southern biogeog
197 h America starting with the emergence of the Isthmus of Panama 3-4 million years ago (Ma).
198 des occurred at 4.90 Ma, indicating that the Isthmus of Panama allowed genetic exchange until the Pli
199 the oldest lineage entered the basin via the isthmus of Panama and sequentially established aggregati
200  spatially-segregated populations within the Isthmus of Panama could reveal how genetic differences a
201 he linking of North and South America by the Isthmus of Panama had major impacts on global climate, o
202 South America and subsequent bridging of the Isthmus of Panama.
203 ectric fish Sternopygus dariensis across the Isthmus of Panama.
204 urred in situ, long after the closure of the Isthmus of Panama.
205  that are protected and part of the critical isthmus of post-infarction VT.
206  upstream (antidromic) capture of a confined isthmus of slow conduction can explain a dPPI <0.
207 reater connectivity in the posterior midbody/isthmus of the corpus callosum and that fractional aniso
208 ed that diffusion anisotropy in the body and isthmus of the corpus callosum was negatively correlated
209 on, and diffusion anisotropy in the body and isthmus of the corpus callosum was shown to mediate this
210 responsive Axin2(+)Lgr5(-) stem cells in the isthmus of the gastric gland and finally gastric gland h
211        Gastric stem cells are located in the isthmus of the gastric glands and give rise to epithelia
212                                 The critical isthmus of the re-entry circuit was identified by entrai
213 f the reentrant ventricular tachycardia (VT) isthmus or diastolic pathway.
214 any critical component (defined as entrance, isthmus or exit) could be identified in 76 (85%) ICM VTs
215 a before the first humans crossed the Bering Isthmus or the onset of climate changes during the termi
216 cy results in reduced Fgf8 expression in the isthmus organiser (IsO), an embryonic signalling centre
217 and after (0.80+/-1.59 mV; 1+/-0.73 m/s) the isthmus (P<0.001 for all).
218                In 11 of 13 of the identified isthmus parts, the QRS morphology of the pace map matche
219  of the isthmus (PrSi), parasubiculum of the isthmus (PaSi), area 29 of the isthmus (area 29i) and ar
220 n at the isthmus requires the function of no isthmus/pax2.1, as well as Fgf signaling.
221 ria, activates overall feeding by activating isthmus peristalsis as well as pharyngeal pumping.
222 ing have no defects in pharyngeal pumping or isthmus peristalsis rates, but their growth defect depen
223 t of the two motions were distinct, but each isthmus peristalsis was coupled to the preceding pump.
224                               For activating isthmus peristalsis, SER-7 in the M4 (and possibly M2) m
225 inct feeding motions, pharyngeal pumping and isthmus peristalsis.
226 rly, and the PoS and PaS reach the cingulate isthmus posteriorly.
227        These include the presubiculum of the isthmus (PrSi), parasubiculum of the isthmus (PaSi), are
228 s analyzed in 546 adult COA patients, aortic isthmus ratio had the strongest correlation with e' (bet
229 indices stipulated in the guidelines, aortic isthmus ratio had the strongest correlation with LV dias
230 pper-to-lower-extremity SBP gradient, aortic isthmus ratio, presence of collaterals, and exercise-ind
231 rily in the posterior segment of the central isthmus (RCA to inferior vena cava distance).
232 phageal squamous epithelium, and in the neck/isthmus region of healthy stomach.
233                                          The isthmus region to be targeted was chosen based solely on
234              This suggests that although the isthmus regulates the size of the cerebellar anlage, the
235 n=7) revealed increased fibrosis in anatomic isthmuses relative to nonisthmus controls.
236 nce of lmx1b.1 and lmx1b.2 expression at the isthmus requires the function of no isthmus/pax2.1, as w
237 atrial (LA) ablation targets were the mitral isthmus, roof, and septum.
238 cond ablation was performed either along the isthmus (scar-dependent group) or from the scar to anoth
239 nus rhythm, pace mapping near the exit of an isthmus should produce a QRS similar to that of VT.
240 encephalon, diencephalon, mesencephalon, and isthmus showed some deviation from the main scheme.
241 pping data were analyzed from the identified isthmus site and from sites at progressively increasing
242                                         A VT isthmus site was contained within a channel in only 11 o
243                                           An isthmus site was defined by entrainment and/or VT termin
244 ively increasing distances from this initial isthmus site.
245     Radiofrequency ablation was performed at isthmus sites as defined by pace-mapping (perfect pace-m
246 was to identify ventricular tachycardia (VT) isthmus sites by pace-mapping within scar tissue and to
247                        Overdrive pacing from isthmus sites determined by activation mapping was consi
248          Among all endocardial sites, the VT isthmus sites displayed the most delay and broadest EGMs
249 teristics that are helpful in identifying VT isthmus sites during sinus rhythm (SR).
250                This method could help target isthmus sites for ablation during stable sinus rhythm.
251                                              Isthmus sites of the VT re-entry circuits were identifie
252 se channels in predicting the location of VT isthmus sites was only 30%.
253                                              Isthmus sites were defined using activation and entrainm
254                     However, the identity of isthmus stem cells (IsthSCs) and the interaction between
255 ting of two wide regions connected by a thin isthmus.Structural heterogeneities provided a substrate
256  at inferior levels and the midbrain-pontine isthmus suggests a vulnerable region of passage for comp
257 on standard characterization of suspected VT isthmus surrogates thus limiting ablation target size.
258 preoptic area, ventral hypothalamus, nucleus isthmus, tectum mesencephali, inferior colliculus, and h
259 on in the isthmus between the TA and IVC (TI isthmus) terminated the tachycardia in all patients.
260        The substrate often includes anatomic isthmuses that can be transected by radiofrequency cathe
261                      Formation of the Panama Isthmus, that had global oceanographic and biotic effect
262 halamus, the habenula, the optic tectum, the isthmus, the cranial motor nuclei, and the spinal motor
263                     Thus, in addition to the isthmus, the roof plate represents an important signalin
264 i at approximately the middle portion of the isthmus; the latter lacking the CB+ neuropil.
265 tionship of the ventricular tachycardia (VT) isthmus to channels of preserved voltage on an electroan
266 structures found progressively closer to the isthmus to form.
267 the incidence and microscopic anatomy of the isthmus to provide more precise anatomical information a
268 nt, this CD44(+) population expands from the isthmus toward the base of the unit.
269 ft side, and (3) left-sided RFCA resulted in isthmus transection and prevention of VT induction.
270 d rostrocaudal restriction in their origins (isthmus versus anterior or posterior r1 regions).
271 riate analysis, PDAC primary tumor location (isthmus vs head: hazard ratio [HR], 2.06; 95% CI, 1.09-3
272                                  The reentry isthmus was characterized by a relatively small volume o
273 cepted date of 3 million years ago (Ma), the Isthmus was effectively complete by the middle Miocene,
274              The 16 patients in whom > or =1 isthmus was identified and ablated were free of arrhythm
275 hermore, 22 of the 81 VTs (27%) for which no isthmus was identified became noninducible after ablatio
276           The monkey cingulo-parahippocampal isthmus was identified recently in the depths and latera
277                                       The VT isthmus was identified using entrainment mapping.
278 mm +/- 4.3; range, 12-43 mm) and the central isthmus was shorter than the inferolateral isthmus (27 m
279                  At middiastole, the central isthmus was straight in 8% of patients, concave in 47% o
280 e posterior left atrium and along the mitral isthmus, was performed under the guidance of an electroa
281            Median CF at the ridge and mitral isthmus were 5.1g and 6.9g, respectively.
282 on during SR while regions forming the inner isthmus were activated faster during VT.
283      Conduction velocities within the shared isthmus were dependent on the activation vector, consist
284 hologies with opposite axes sharing the same isthmus were mapped.
285                                              Isthmuses were 16.4+/-7.2 mm long and 7.4+/-2.8 mm wide.
286 liminated, and if VT was tolerated, critical isthmuses were also approached.
287            In all these regions, parts of VT isthmuses were identified by pace mapping.
288 ber passage occurred at the midbrain-pontine isthmus where all of the fiber bundles overlapped.
289 , undifferentiated cells in the gastric unit isthmus where stem cells are known to reside.
290 t mating or insemination reaches the oviduct isthmus, where sperm are retained and thereby form a res
291  in positioning the mid/hindbrain organizer (isthmus), which regulates midbrain and cerebellar develo
292 nd (2) targeted illumination of the critical isthmus, which was identified via analysis of simulated
293 hibchan speakers on both sides of the Panama isthmus, who have ancestry from both North and South Ame
294 age channels with ILPs harbored the clinical isthmus with a sensitivity and specificity of 78% and 85
295 PPI <0 are markers of limited width critical isthmuses with slower conduction velocity, whereas sites
296                                           Of isthmuses with the smallest dimensions, isthmus 3 is the
297 ines in the posterior left atrium and mitral isthmus, with an 8-mm-tip catheter.
298 ally complex structures, particularly at the isthmus, with substrate for multiple VT morphologies aft
299 2 minutes) were formed at the cavo-tricuspid isthmus, with the end point of bidirectional block.
300  WM bilaterally and the left cingulate gyrus isthmus WM, as well as the right precuneal GM, showed si

 
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