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1 he body (interoception) (e.g., visceral-cold-itch sensations).
2 h is critical for the maintenance of chronic itch sensation.
3 or exploring the mechanism underlying spinal itch sensation.
4 receptor-4 (PAR-4) was recently suggested in itch sensation.
5 nisms underlying the central transmission of itch sensation.
6 s required selectively for histamine-induced itch sensation.
7  to pruritic chemicals and playing a role in itch sensation.
8 erties to initiate and maintain long-lasting itch sensation.
9 nor has any such pathway been identified for itch sensation.
10 ists, playing a fundamental role in pain and itch sensation.
11 y discovered circuits also contribute to the itch sensation.
12 st cells, which leads to vasodilation and an itch sensation.
13 he molecules, cells, and circuits underlying itch sensation.
14 h a focus on the role of Mrgprs in mediating itch sensation.
15 g normal skin near a site of itch can elicit itch sensation, a phenomenon known as alloknesis.
16 urons in the central amygdala (CeA) for both itch sensation and associated aversion.
17 t progress has advanced our understanding of itch sensation and transmission, the neural mechanisms u
18 rin-releasing peptide, a peptide involved in itch sensation, and MrgprC11.
19  that hypersensitive subjects had a stronger itch sensation, and that the time to first itch sensatio
20 ortance, the underlying neural mechanisms of itch sensation are poorly understood.
21                                    Touch and itch sensations are crucial for evoking defensive and em
22 h, demonstrating a mechanistic difference in itch sensation between hairy and glabrous skin.
23 lation with cowhage induced a more intensive itch sensation compared with stimulation with other subs
24 fiber-dominated itch, (2) the time course of itch sensation differs between subjects with A-fiber- ve
25 t common side effect was a mild tingling and itching sensation during stimulation (100%) and temporar
26 nd unmyelinated fibers, (5) the time of peak itch sensation for subjects with A-fiber-dominated itch
27 myelinated fibers, and (6) the time for peak itch sensation for subjects with C-fiber-dominated itch
28 nown that algogens and cooling could inhibit itch sensation; however, the underlying molecular and ne
29 n of either BRAF or GRP signaling attenuated itch sensation in chronic itch mouse models.
30 ggest that broad-band UVB irradiation evokes itch sensation in mice by promoting TRPV1 channel functi
31  molecule that is dedicated to mediating the itch sensation in the dorsal horn of the spinal cord, an
32  (GRPR) plays an important part in mediating itch sensation in the dorsal spinal cord.
33 nt of local neuronal circuits for processing itch sensation in the spinal cord.
34 ns constitute a long-sought labeled line for itch sensation in the spinal cord.
35 strate that PLCbeta3 is required to mediate "itch" sensation in response to histamine acting on the h
36                                         This itch sensation is normally suppressed by inputs from mec
37       Despite the clinical importance of the itch sensation, its mechanism remains elusive.
38 nd light tactile touch may induce unpleasant itch sensations (mechanical itch or alloknesis).
39 ovide structural insights into understanding itch sensation, MRGPRX1 activation, and downstream G pro
40      Scratching significantly attenuated the itch sensation (P<0.001) and evoked an associated pleasu
41 ated G protein-coupled receptors (MRGPRs) in itch sensation promised a search for novel therapeutics
42                                              Itch sensation provokes the scratch reflex to protect us
43 rate that GRPR is required for mediating the itch sensation rather than pain, at the spinal level.
44                                              Itch sensation relies on dorsal root ganglion (DRG) sens
45                                          The itch sensation results from the excitation of primary se
46 rst sensation of itch, the strength of their itch sensation, the number of times mosquitoes attempted
47 s of this class of neurons parallel the pure itching sensation this stimulus elicits in humans, and m
48 ave shown a significant role of periostin in itch sensation through direct integrin-mediated stimulat
49                                              Itch sensation to histamine injection was lost in most s
50 r itch sensation, and that the time to first itch sensation was inversely correlated with wheal diame
51  tone via administration of 5-HT potentiates itch sensation, whereas mice lacking 5-HT or serotonergi
52 es, cells, and circuits known to mediate the itch sensation, which, coupled with advances in understa