ライフサイエンスコーパス: jaundice

1 severe bacterial infections, or pathological jaundice.

2 ears; mean age, 66.9 years) with obstructive jaundice.

3 rd clamping groups required phototherapy for jaundice.

4 ion- and drug-induced hemolysis and neonatal jaundice.

5 newborn children and leads to physiological jaundice.

6 jugated bilirubin in the blood, resulting in jaundice.

7 1-week history of abdominal pain, fever and jaundice.

8 onset of nausea, vomiting, malaise and deep jaundice.

9 ition to modulating the severity of neonatal jaundice.

10 6-week-old boy who presented with prolonged jaundice.

11 Most patients (43.4%) presented with jaundice.

12 for investigation of infants with persistent jaundice.

13 by high levels of aminotransferases and mild jaundice.

14 n effective method of palliating obstructive jaundice.

15 ients with preoperative biliary stent and/or jaundice.

16 e obstructive jaundice than in those without jaundice.

17 talloporphyrins in the treatment of neonatal jaundice.

18 omplications including bile leak, biloma, or jaundice.

19 therapeutic target for clinical treatment of jaundice.

20 humans as epigastric pain, weight loss, and jaundice.

21 ology, prognostic score, dyspnea, fever, and jaundice.

22 th encephalopathy, bleeding, and cholestatic jaundice.

23 hyrin is useful in the treatment of neonatal jaundice.

24 roved pharmaceutical treatments for neonatal jaundice.

25 reat neonatal, genetic, or acquired forms of jaundice.

26 y used in Asia to prevent and treat neonatal jaundice.

27 t of CAR activity may contribute to neonatal jaundice.

28 No patients had recurrent jaundice.

29 rase (ALT) elevations, and then symptoms and jaundice.

30 to accurately predict clinically significant jaundice.

31 inversin gene results in situs inversus and jaundice.

32 mporary relief for patients with obstructive jaundice.

33 atient with complaints of abdominal pain and jaundice.

34 design a simple clinical diagnostic tool for jaundice.

35 tients with clinically suspected obstructive jaundice.

36 HCV seroconversion illness, including 2 with jaundice.

37 13), which rose to 45.5% in the presence of jaundice.

38 day history of right upper quadrant pain and jaundice.

39 rval, 1.2-6.1) in pregnancies complicated by jaundice.

40 ed UGT1 (hUGT1) mice physiologically develop jaundice.

41 these infants, 6 of 15 (40%) presented with jaundice, 1 of whom also had petechiae.

42 reatitis (48% vs. 24%, P < 0.05) rather than jaundice (11% vs. 30%, P < 0.05) or cholangitis (0% vs.

43 learance (42.9%) compared with those without jaundice (13.7%).

44 cases typically presented with both clinical jaundice (254/318 [80%]) and severe anemia (hemoglobin l

45 20 days (range, 8-77 days); 26 patients had jaundice (27%) and 22 patients were hospitalized (23%) f

46 solid component (6.0 vs 1.4%, p = 0.010) or jaundice (3.6 vs 0.7%, p = 0.028) were associated with c

47 .001) and children were more likely to have jaundice (31.9% vs 11.6%; P < .001).

48 tal DILI (23% versus 4%, P = 0.001), but not jaundice (46% versus 35%, P = 0.2) or liver transplantat

49 equent symptoms predicted pancreatic cancer: jaundice (51 [49%] of 105 patients with pancreatic cance

50 28B rs12979860 C/C more frequently developed jaundice (53.2% vs 27.6%; P = .022) than carriers of the

51 istress, (4) initial low blood pressure, (5) jaundice, (6) rupture of liver abscess, (7) endophthalmi

52 learance (56.3%) compared with those without jaundice (60.6%).

53 included pain (67%), weight loss (65%), and jaundice (62%).

54 ic symptoms (23%), pain (12%), dyspnea (9%), jaundice (7%) or other symptoms (15%).

55 dominal pain (25%), fullness/mass (10%), and jaundice (7%); 47% were asymptomatic.

56 in the blood [15,19], as occurs in neonatal jaundice [7].

57 = 50) had significantly higher frequency of jaundice (74% versus 40%, P = 0.0001).

58 62 y) men (85%), presenting with obstructive jaundice (77%) associated with autoimmune pancreatitis (

59 The majority had ascites (91%), jaundice (88%), elevated prothrombin time (18 +/- 3 seco

60 in whom surgery was unsuccessful in clearing jaundice 9 (22%) died and 30 (73%) underwent liver trans

61 Newborns are at increased risk of jaundice, a condition in which excess bilirubin accumula

62 Various pathologic conditions result in jaundice, a yellowing of the skin due to a buildup of bi

63 Early clearance of jaundice after portoenterostomy was achieved in 81 of 14

64 When patients develop jaundice after transplant, the time to search for treata

65 y advanced disease) and later development of jaundice after transplantation predicted inferior surviv

66 y of diagnosis, multiple causes of symptoms, jaundice, an initial therapy algorithm, secondary therap

67 ed liver disease (REILD) has been defined as jaundice and ascites appearing 1 to 2 months after RE in

68 alcohol (age, 18-75 y) with recent onset of jaundice and biopsy-proven severe AH in our study, perfo

69 pe in particular is associated with neonatal jaundice and circulation of bilirubin in blood at high c

70 is characterized by an abrupt development of jaundice and complications related to liver insufficienc

71 vestigated the etiology and risk factors for jaundice and death.

72 nd encephalopathy 6 weeks after the onset of jaundice and fatigue.

73 iated cholangitis presented with obstructive jaundice and had increased serum IgG4 levels and IgG4-po

74 pigment responsible for the yellow color of jaundice and healing bruises.

75 variate analysis disclosed that preoperative jaundice and intraoperative blood transfusion were posit

76 Bilirubin, a key biomarker for the jaundice and its clinical diagnosis needs a better analy

77 , a well-known formulae for the treatment of jaundice and liver disorders, against the cholestasis us

78 Alcoholic hepatitis (AH) is a syndrome of jaundice and liver failure that occurs in a minority of

79 itis is a clinical syndrome characterized by jaundice and liver impairment that occurs in patients wi

80 All had jaundice and markedly abnormal results on liver function

81 Physical examination revealed jaundice and mild right upper quadrant tenderness.

82 A 65-year-old man with jaundice and peripheral blood eosinophilia.

83 th C3H/HeJ mouse strains was associated with jaundice and pulmonary hemorrhage, similar to the patien

84 ystitis subsequently developed waxing waning jaundice and recurrent episodes of upper gastrointestina

85 ed as clinically indicated for palliation of jaundice and to potentially facilitate neo-adjuvant chem

86 ded a higher level of monitoring for newborn jaundice and treatment of hyperbilirubinemia in an effor

87 sity score matching of open PD patients with jaundice and/or biliary stent confirmed a decrease in al

88 ype SSI with Broad-abx amongst patients with jaundice and/or biliary stent only, regardless of wound

89 She remained jaundiced and a liver biopsy revealed cirrhosis with reg

90 All were jaundiced and had low albumin levels, and most had coagu

91 one-half of bilirubin, the yellow pigment of jaundice) and its homologues with hexanoic and longer ac

92 normalization of pruritus, disappearance of jaundice, and alanine aminotransferase (ALT) levels <1.5

93 ediatric clinician-- immunizations, neonatal jaundice, and animal-induced injuries-are concisely revi

94 clinical pediatrics: immunizations, neonatal jaundice, and animal-induced injuries.

95 ticing pediatrician: immunizations, neonatal jaundice, and animal-induced injuries.

96 r damage, TG2(-/-) mice had more gallstones, jaundice, and ductal proliferation than wild-type mice.

97 nage a wide range of ailments such as edema, jaundice, and gonorrhea.

98 s, hepatitis, interstitial lung disease, and jaundice, and grade 4 cholestasis, and died on treatment

99 toxicity, thrombocytopenia, nausea, fatigue, jaundice, and muscle aches.

100 aim of improving hepatic function, relieving jaundice, and reducing adverse effects of obstruction.

101 On multivariate analysis, hyperparasitemia, jaundice, and shock were all associated independently wi

102 examination revealed the absence of fever or jaundice, and the laboratory tests, including that for p

103 ophysiology of breastfeeding and breast milk jaundice, and the realization that Gilbert's syndrome ma

104 enterostomy, may restore bile flow and clear jaundice, and, if successful, achieve a 10-year survival

105 evere distress, with unstable vital signs, a jaundiced appearance, and substantial pain in her chest

106 percentage of subjects with GS, episodes of jaundice are associated with other symptoms and nutritio

107 trics treatment recommendations for neonatal jaundice are based on age-specific total serum bilirubin

108 The most common causes of cholestatic jaundice are biliary atresia and idiopathic neonatal hep

109 itis, biphasic fever, flaccid paralysis, and jaundice are typical manifestations of diseases in human

110 r treatable causes is early in the course of jaundice, as the risk of mortality rises steeply with sm

111 Hepatitis E virus (HEV) causes outbreaks of jaundice associated with maternal mortality.

112 GPRs is a promising strategy for alleviating jaundice-associated itch.

113 sure total bilirubin in neonates at risk for jaundice at Queen Elizabeth Central Hospital in Blantyre

114 During jaundice attacks, subjects with GS had significant diffe

115 The duration of jaundice before onset of encephalopathy ranged from 4 to

116 al examination highlighted fever, increasing jaundice, bilateral laterocervical lymph nodes, erythema

117 We assessed clearance of jaundice (bilirubin <20 micromol/L) as an early outcome

118 with exaggerated physiologic and pathologic jaundice but adapts it to the microfluidic level for the

119 clamping is not apparently a risk factor for jaundice but warrants more study.

120 loss of IFN-gamma did not alter the onset of jaundice, but it remarkably suppressed the tissue-specif

121 A Chinese herbal remedy for jaundice called Yin Zhi Huang is now shown to activate a

122 Left untreated, jaundice can lead to neurological impairment and death.

123 performed to minimize risks of pancreatitis, jaundice, cholangitis, and stenosis.

124 transplantation, all four patients developed jaundice, cholestatic elevation of liver enzymes, and hi

125 r appearance at KPE, and early postoperative jaundice clearance are significant predictors of transpl

126 o thrive and had progressive cholestasis and jaundice, coagulation disorders, bilateral ureterostomie

127 Patients with jaundice commonly report experiencing an intense non-his

128 Preoperative obstructive jaundice considerably increases perioperative risk.

129 The management strategy for neonatal jaundice continues to focus on screening and prevention.

130 as admitted in the emergency department with jaundice, dark urine and pale stools.

131 patients with right upper quadrant pain and jaundice.Detailed imaging by MRI/MRCP should be done.

132 Preoperative jaundice did not protect against biliary stricture forma

133 Rates of gastrointestinal disorders, jaundice, dry skin, and photosensitivity were increased

134 Although rare, obstructive jaundice due to external bile duct compression or ruptur

135 Jaundice during acute infection was more common among pa

136 In C/C patients, jaundice during acute infection was not associated with

137 risk of perinatal mortality associated with jaundice during pregnancy.

138 with HH were screened for the development of jaundice during the course of HH.

139 rated an increase in hepatic enzymes without jaundice during the pregnancy.

140 f HCC resulted in complete resolution of his jaundice, enabling further treatment with nivolumab, whi

141 that ajmaline may induce severe cholestatic jaundice even after a single dose administration.

142 ed the observation that acute hepatocellular jaundice from a drug is associated with death or the nee

143 Patients who developed jaundice (group 1) needed vasopressor treatment (P < 0.0

144 ine (P < 0.05), compared to patients without jaundice (group 2).

145 is tissue specific, resulting in progressive jaundice, growth failure, and greater than 90% mortality

146 Double heterozygous mice exhibit jaundice, growth retardation, impaired differentiation o

147 Jaundiced Gunn rat pups (jjs) exhibit similar BAEP abnor

148 In summary, biliverdin administration in jaundiced Gunn rat pups produces BAEP abnormalities cons

149 oked potential (BAEP) abnormalities occur in jaundiced Gunn rats given sulfadimethoxine to displace b

150 UDP-glucuronosyltransferase (BUGT)-deficient jaundiced Gunn rats with a recombinant adenovirus (5 x 1

151 tal vein catheter in bilirubin-UGT-deficient jaundiced Gunn rats, mean serum bilirubin concentrations

152 the C/T or T/T genotype who did not develop jaundice had a lower chance of spontaneous clearance of

153 Although obstructive jaundice has been associated with a predisposition towar

154 Bilirubin, an abundant pigment that causes jaundice, has long lacked any clear physiologic role.

155 Symptomatic patients with jaundice have a higher likelihood of spontaneous viral c

156 Total diagnostic intervals were shorter when jaundice (hazard ratio [HR] 1.38, 95% CI 1.07-1.78; p=0.

157 surgery, liver transplantation, obstructive jaundice, hepatitis C antiviral treatment) does not impr

158 e of severe nonspherocytic hemolytic anemia, jaundice, hepatosplenomegaly, and marked erythroblastosi

159 Patients exhibit prolonged neonatal jaundice, hepatosplenomegaly, and progressive neurodegen

160 high death rate, particularly in those with jaundice; however, children and human immunodeficiency v

161 measuring unbound unconjugated bilirubin in jaundiced human newborns or animal models of kernicterus

162 Both patients presented with jaundice, hyperbilirubinemia, and mild-to-moderate eleva

163 le duct destruction and effect resolution of jaundice if given early.

164 was manifested as weight gain, ascites, and jaundice in 7 patients.

165 st common clinical condition associated with jaundice in adults is Gilbert's syndrome, which is chara

166 valuate the incidence and clinical effect of jaundice in critically ill patients with HH.

167 resia is the commonest cause of pathological jaundice in infants and the leading indication for liver

168 the association between breast feeding with jaundice in mice.

169 ase to identify studies on the management of jaundice in patients undergoing PD or liver resection.

170 No clinical data exist about new onset of jaundice in patients with HH.

171 ts (CDC) are important causes of obstructive jaundice in pediatric patients.

172 this study, we examined the pathogenesis of jaundice in the inv mouse, a transgenic mouse in which a

173 The management of jaundice in the newborn infant is an area of clinical pr

174 c hepatitis, as indicated by recent onset of jaundice in the prior 3 months and a Maddrey score of at

175 ppropriate solutions to diagnose and monitor jaundice in these settings.

176 Risk factors for clinical jaundice included general anesthesia, pregnancy, fasting

177 utside hospital with symptoms of obstructive jaundice, including abdominal pain and yellowing of the

178 erations and functional impairment caused by jaundice increase the risk of surgery; therefore, preope

179 llular drug-induced liver injury (DILI) with jaundice indicates a serious reaction, is used widely to

180 3) ITx and was characterized by intermittent jaundice, intractable pruritus, increased serum bile aci

181 Jaundice is a common finding during the course of HH.

182 Obstructive jaundice is an uncommon presentation for patients with H

183 Obstructive jaundice is associated with immunologic derangements and

184 Although neonatal jaundice is mostly benign, excessively high levels of se

185 The extreme jaundice is present as a phenotype in skin color after 8

186 ignificant hepatic dysfunction with clinical jaundice is rare in KD without associated gall bladder h

187 only help confirm that the story of neonatal jaundice is still unfolding.

188 h a one-month history of epigastric pain and jaundice, itching, flushing, cough and wheezing.

189 m birth, hypoglycemia, respiratory distress, jaundice, large for gestational age, and hospitalization

190 nsferase >3 times the upper limit of normal, jaundice, liver failure, liver transplantation, or fatal

191 nonresponders and exhibited earlier onset of jaundice (<9 months), neonatal cholestasis, and higher A

192 sent with biliary strictures and obstructive jaundice, making cholangiocarcinoma (CCA) an important d

193 bilirubin concentration (Bf), would improve jaundice management as it better predicts bilirubin neur

194 have found that phototherapy and/or neonatal jaundice may be associated with asthma.

195 The resulting jaundice may be managed with phototherapy to isomerize t

196 Although transcutaneous quantification of jaundice may help discern which patients warrant further

197 ng Bf and BT into the management of neonatal jaundice may help move clinical practice from its tradit

198 d that the immunosuppression associated with jaundice may result from the functional impairment of li

199 Bodybuilding HDS caused prolonged jaundice (median, 91 days) in young men, but did not res

200 toimmune hepatitis (n = 10), and obstructive jaundice (n = 12).

201 cluded parasitemia >100 000/microL (n = 18), jaundice (n = 20), respiratory distress (n = 14), hypote

202 ntation was commonly clinical or biochemical jaundice, n = 30 (28%) each.

203 gy Clinic for urgent evaluation of new onset jaundice, nausea and fatigue associated with a >40-fold

204 ist, gastroenterologist, and radiologist) of jaundiced neoplastic patients should be performed before

205 ion by the combined method in plasma from 20 jaundiced newborns was significantly greater than and po

206 In summary, significant jaundice occurred in 26% of patients and was predominant

207 New onset of jaundice occurred in 63 of 175 patients with HH (36%).

208 Four deaths among pregnant women with jaundice occurred in an urban community near Dhaka, Bang

209 nfidence interval, 6.39-490; P < .0001), and jaundice (odds ratio, 6.16; 95% confidence interval, 1.0

210 ve to antibiotics and related to cholestatic jaundice, oedema or erythema of the extremity associated

211 tres with an early success rate for clearing jaundice of 55% overall.

212 port the possibility that the phenomenon of "jaundice of sepsis" represents an adaptive physiological

213 syndrome may play a greater role in neonatal jaundice, only help confirm that the story of neonatal j

214 (95% CI 51,000-380,000) cases with fever and jaundice or haemorrhage including 78,000 (95% CI 19,000-

215 by hepatic complications such as cholestatic jaundice or hepatomegaly.

216 n-NICCD, infants with idiopathic cholestatic jaundice or INH were enrolled.

217 Individuals with preexisting jaundice or liver cirrhosis at the time of admission (n

218 R = 2.167, 95% CI: 2.104-13.150, p = 0.003), jaundice (OR = 1.9, 95% CI: 1.246-3.297, p = 0.008), rup

219 6.661 [95% CI, 2.126-20.876], P = .001) and jaundice (OR, 5.701 [95% CI, 1.776-18.306], P = .003) we

220 cant differences with gender, apneic spells, jaundice, or phototherapy.

221 ilatation of more than 5 mm (P < 0.001), and jaundice (P < 0.001) were statistically significant vari

222 (p =0.007), low blood pressure (p = 0.024), jaundice (p = < 0.001), rupture of liver abscess (p < 0.

223 T) > 3 x upper limit of normal (P = 0.10) or jaundice (P = 0.16).

224 were more likely to be white (P =.004), have jaundice (P =.03), and have lower peak viral titer (P =.

225 on cohort (n = 681) with symptomatic tumors (jaundice, pain, bleeding), tumors >2 cm, Ki67 >3%, and l

226 ominal pain and cholestasis with progressive jaundice, particularly in subjects without evidence of b

227 can quantify GCDC acid serum on obstructive jaundice patients and can be used to support its pharmac

228 ntify the serum level of GCDC in obstructive jaundice patients.

229 ne sequences, obtained from serum samples of jaundiced patients from Laos.

230 Treatment-naive, jaundiced patients presenting to our tertiary unit betwe

231 Jaundiced patients represent a major challenge for surge

232 Papers considering palliative drainage in jaundiced patients were excluded.

233 the indications for preoperative drainage in jaundiced patients who are candidates for pancreaticoduo

234 e and to report on the current management of jaundiced patients with periampullary or proximal bile d

235 bilirubin level of more than 7 mg/dL or, in jaundiced patients, an increasing bilirubin level on day

236 She decompensated with jaundice, peripheral edema, ascites, encephalopathy, coa

237 cted into newborn mice causes an obstructive jaundice phenotype with lower mortality rates.

238 The jaundice-pollution relationship is not affected by top-o

239 ternal smoking during pregnancy, or neonatal jaundice predict islet autoimmunity in children at genet

240 article, we review recent research regarding jaundice predischarge risk assessment, current expert re

241 cluding antifibrinolytic agents, obstructive jaundice, prostaglandin inhibitors, cyclosporine A, radi

242 there were no clinical signs of cholestatic jaundice, pruritis, or liver dysfunction.

243 mary sclerosing cholangitis include fatigue, jaundice, pruritus, or steatorrhoea.

244 First, clinical assessment of jaundice remains critically important as "early discharg

245 eductions were also seen in the frequency of jaundice, renal insufficiency, mechanical ventilation, h

246 Jaundice resolved in all NICCD and in 87.5% of non-NICCD

247 As a consequence, jaundice resolved, and long-term survival improved to gr

248 Mild gastrointestinal disorders, jaundice resulting from isolated unconjugated hyperbilir

249 Jaundice resulting from unconjugated hyperbilirubinemia

250 bilirubin levels, the indicator of neonatal jaundice risk, by 0.076 (95% CI: 0.027-0.125), 0.029 (0.

251 ay therefore be key to lowering the neonatal jaundice risk.

252 erm birth (RR = 1.06; 95% CI, 0.93-1.21), or jaundice (RR = 0.96; 95% CI, 0.48-1.91).

253 6%) who underwent PD developed postoperative jaundice secondary to a stricture of the biliary-enteric

254 e role of preoperative ERCP in patients with jaundice secondary to pancreatic cancer was raised in a

255 Thus, obstructive jaundice selectively expands liver myeloid DCs that are

256 N: A 50 year male presented with acute onset jaundice, significant weight loss and elevated liver enz

257 d abrupt onset of fever, abdominal pain, and jaundice, sometimes with the presence of ascites even in

258 al recent series with up to 20% mortality in jaundiced subjects.

259 Among patients who were deeply jaundiced, survival was related to the absence of multio

260 auma, eight vs three; sepsis, six vs 13; and jaundice, ten vs 12 after vaginal delivery and caesarean

261 ly in patients with preoperative obstructive jaundice than in those without jaundice.

262 ms could contribute to pruritus and painless jaundice that occur during cholestatic liver diseases.

263 ale) had other documented causes of neonatal jaundice; the mean ages at US assessment were 48.5 and 5

264 age <24 months (GRAFT), and time from onset jaundice to encephalopathy <7 days (PATIENT).

265 cCrCl <60 mL/min/1.73m, and time from onset jaundice to encephalopathy <7 days.

266 disseminated histoplasmosis with cholestatic jaundice to highlight histoplasmosis involvement in the

267 is a standard treatment for severe neonatal jaundice to remove toxic bilirubin from the blood.

268 male infant with type I biliary atresia with jaundice (total serum bilirubin, 22.2 mg/dL), hypoalbumi

269 testing the hypothesis that the duration of jaundice up to a given point in time provides more progn

270 ian time to stricture formation resulting in jaundice was 13 months (range, 1-106 months) and was sim

271 ed the acute event of HH, median duration of jaundice was 6 days (interquartile range, 3-8).

272 In contrast, in non-C/C patients, jaundice was associated with a higher likelihood of spon

273 presentation in 47 (56.627%) GS subjects and jaundice was associated with abdominal pain, dyspepsia o

274 Occurrence of jaundice was associated with an increased frequency of c

275 (adjusted HR, 2.43; 2.21-2.66), and neonatal jaundice was associated with more than a 50% increased r

276 The major cause for obstructive jaundice was choledocholithiasis.

277 Jaundice was diagnosed in patients with plasma total bil

278 Jaundice was investigated and infections, autoimmune dis

279 Jaundice was more frequent in patients with cancer (12.5

280 Recurrent jaundice was the only presentation in 47 (56.627%) GS su

281 bin, the yellow-orange neurotoxic pigment of jaundice, was synthesized following Friedel-Crafts acyla

282 low pH, hyperparasitemia, severe anemia, and jaundice were statistically significant indicators of de

283 ce suggests increased incidences of neonatal jaundice when air quality worsens, yet no studies have q

284 acute BCS had a significantly higher rate of jaundice whereas a lower rate of abdominal and chest var

285 ater, he presented with painless cholestatic jaundice which peaked in severity at eleven weeks after

286 nusual clinical manifestation of obstructive jaundice (which has not been reported so far) along with

287 on has been suggested as a cause of neonatal jaundice, which can further cause permanent brain damage

288 pparently healthy and persons suffering from jaundice, which correlated well with a standard colorime

289 Jaundice, which is caused by accumulation of bilirubin,

290 Defects in this process result in jaundice, which is particularly common in neonates.

291 nient and efficient method to treat neonatal jaundice while allowing continuous breastfeeding.

292 resented with encephalopathy and cholestatic jaundice with a Hemoglobin S (HbS) level of 69.6%.

293 The patient was found to have obstructive jaundice with multiple mass lesions in the liver, spleen

294 nt accuracy in the evaluation of obstructive jaundice with regards to the level and cause of obstruct

295 ole of MDCT in the evaluation of obstructive jaundice with respect to the cause and level of the obst

296 She became clinically jaundiced with evidence of hepatic artery narrowing on u

297 ks of gestation by cesarean section, and was jaundiced, with low birth weight and height.

298 th hypoalbuminemia, cholangitis or long-term jaundice; with an FLR < 30% or 40%) given the high risk

299 astrointestinal bleeding, encephalopathy, or jaundice) without esophageal varices was included, and 5

300 astrointestinal bleeding, encephalopathy, or jaundice) without esophageal varices was included, and 5