ライフサイエンスコーパス: junction potential

1 ative SDK channels, and nitrergic inhibitory junction potentials.

2 eased the amplitude of purinergic excitatory junction potentials.

3 cation of longitudinal stretch inhibited all junction potentials.

4 he amplitude of slow waves and of inhibitory junction potentials.

5 bition of Ca(2+) -transients with inhibitory junction potentials.

6 ntials followed by more sustained inhibitory junction potentials.

7 microM) affected the amplitude of excitatory junction potentials (2 +/- 5 % and -3 +/- 10 %) or NCTs

8 etected through measurement of the change in junction potential across the bipolar electrode.

9 Inhibitory junction potentials and responses to exogenous beta-NAD,

10 e, and cyclopiazonic acid, reduce inhibitory junction potentials and responses to sodium nitroprussid

11 temporally associated with rapid excitatory junction potentials and the inhibition of Ca(2+) -transi

12 ctivity (oral excitatory and anal inhibitory junction potentials) and Ca(2+) waves in LM and CM.

13 eference electrode (RE), the introduction of junction potentials, and the possibility of sample conta

14 Responses to beta-NAD and inhibitory junction potentials are blocked by the P2Y1-selective an

15 junction, and the current-voltage curves and junction potentials are strongly and self-consistently m

16 servations cannot be explained by the liquid junction potential between two mutually miscible electro

17 equation is usually used to calculate liquid-junction potentials between miscible electrolyte solutio

18 rfacial potential with an additive diffusion/junction potential due to the increase in ionization fro

19 represents a discreet cholinergic excitatory junction potential (EJP) that involves the synaptic acti

20 (5) Evoked excitatory junction potentials (EJP) were partially blocked by 4-DA

21 e cells to examine excitatory and inhibitory junction potentials (EJPs and IJPs).

22 microm), an ongoing discharge of excitatory junction potentials (EJPs) and inhibitory junction poten

23 ns evoked an ongoing discharge of excitatory junction potentials (EJPs) at the oral recording site (r

24 T potently facilitated B38-evoked excitatory junction potentials (EJPs) but had only a small effect o

25 CgTX GVIA) irreversibly abolished excitatory junction potentials (EJPs) evoked by trains of < or = fi

26 ormed intracellular recordings of excitatory junction potentials (EJPs) in the muscle fibers to deter

27 tion potentials (IJPs) anally, or excitatory junction potentials (EJPs) orally.

28 ding electrodes elicited compound excitatory junction potentials (EJPs) synchronously in both muscles

29 Supramaximal stimuli evoked excitatory junction potentials (EJPs) which could be divided into t

30 temporally associated with rapid excitatory junction potentials followed by more sustained inhibitor

31 time-locked with the onset of an inhibitory junction potential (IJP) at an anal recording electrode,

32 Inhibitory junction potentials (IJP) of circular smooth muscle of g

33 ls showed an ongoing discharge of inhibitory junction potentials (IJPs) anally, or excitatory junctio

34 Inhibitory junction potentials (IJPs) and relaxations evoked in res

35 bitory nerve stimulation elicited inhibitory junction potentials (IJPs) and relaxations in wild-type

36 ssion resulting in a reduction in inhibitory junction potentials (IJPs) in colonic circular muscle.

37 eld stimulation evoked purinergic inhibitory junction potentials (IJPs) in CSMCs.

38 of OFQ on muscle contractions and inhibitory junction potentials (IJPs) in rat colon.

39 EVS evoked inhibitory junction potentials (IJPs) in wild type muscles that wer

40 in PDGFRalpha(+) cells by EFS and inhibitory junction potentials (IJPs) recorded with intracellular m

41 between colonic MMCs, spontaneous inhibitory junction potentials (IJPs) were always present.

42 ry junction potentials (EJPs) and inhibitory junction potentials (IJPs) were recorded simultaneously

43 sed in relation to the idea that spontaneous junction potentials in colonic CM are not monoquantal ev

44 y of membrane potential following inhibitory junction potentials in gastrointestinal smooth muscle or

45 ongoing oral excitatory and anal inhibitory junction potentials in the CM.

46 al field stimulation yielded fast excitatory junction potentials in the smooth muscle that were block

47 her enzymes capable of directly altering the junction potential of an electrode surface.

48 ngs from CM cells revealed either no ongoing junction potentials, or alternatively, small potentials

49 ulting from changes to the working electrode junction potential (phi) were observed as glucose and la

50 The amplitude of NO-mediated slow inhibitory junction potentials (S-IJPs) evoked by electric field st

51 Spontaneous excitatory junction potentials (SEJPs) were also recorded from most

52 y junction potentials (sIJPs) and excitatory junction potentials (sEJPs) were recorded from all anima

53 gesting that they are spontaneous excitatory junction potentials (sEJPs).

54 Spontaneous inhibitory junction potentials (sIJPs) and excitatory junction pote

55 ime with those in CM, spontaneous inhibitory junction potentials (sIJPs) were found to occur synchron

56 region of smooth muscle at which spontaneous junction potentials (sJPs) were coordinated in both spac

57 reduction in evoked and miniature excitatory junction potentials, suggesting a neuronal deficit.

58 nt for salinity-induced variations in liquid junction potentials that, if not taken into account, wou

59 light resulted in excitatory and inhibitory junction potentials, the electrical events underlying co

60 This change in junction potential was caused by reduction of amino reac

61 The liquid junction potentials were estimated in the framework of I

62 Slow wave amplitude and nitrergic inhibitory junction potentials were reduced while solid emptying wa