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1 natures of hepatic steatosis and fibrosis in juvenile mice.
2 olectin injections increased femur length in juvenile mice.
3 imaging in acute slices from male and female juvenile mice.
4 ole of Iba1 in excitatory synaptic growth in juvenile mice.
5 tibody enhancement of signals in newborn and juvenile mice.
6 e cell synaptic connectivity is increased in juvenile mice.
7 ional programs in perinatal fentanyl exposed juvenile mice.
8 tions, bacterial burden, and inflammation in juvenile mice.
9 s lipid, cholesterol, and iron metabolism in juvenile mice.
10 These knockout experiments were performed on juvenile mice.
11 itors, which form the articular cartilage in juvenile mice.
12 se to characterize calsyntenin-1 function in juvenile mice.
13 chaffer collateral-CA1 synapses in adult and juvenile mice.
14 ic connections between the Purkinje cells of juvenile mice.
15 t was larger in neurons from adult mice than juvenile mice.
16 adult mice but not in the IC of experimental juvenile mice.
17 tween Hb9 INs in spinal cords of newborn and juvenile mice.
18 om layer I interneurons in brain slices from juvenile mice.
19  during the first wave of spermatogenesis in juvenile mice.
20 ses UCP levels and activity in hippocampi of juvenile mice.
21 l fibre synapses in the cerebellar cortex of juvenile mice.
22 e (nucleus) of growing oocytes isolated from juvenile mice.
23 nuated following intracranial inoculation of juvenile mice.
24 h the first wave of germ cell development in juvenile mice.
25 ed representational drift similar to that of juvenile mice.
26 erited Cdkn1c in the brains of embryonic and juvenile mice.
27 n of CR6 from infected dams occurred only in juvenile mice.
28  the visceral precursor cell pool in overfed juvenile mice.
29 ns and decreased hepatic bacterial burden in juvenile mice.
30 diomyocyte proliferation and regeneration in juvenile mice.
31 y MNTB-LSO synapses in brainstem slices from juvenile mice.
32                   The antibody prevalence in juvenile mice (14 g or less) was inversely proportional
33 a A virus is highly age-sensitive: robust in juvenile mice (4-6 wk old) but attenuated in mature mice
34    Conditional deletion of beta-cell Ezh2 in juvenile mice also reduced H3 trimethylation at the Ink4
35 nflammatory markers were already elevated in juvenile mice although at this age the number of dopamin
36 eterious effects of reducing EphA4 levels in juvenile mice and do not provide support for the hypothe
37 Vigorous pancreatic beta-cell replication in juvenile mice and humans declines with age, and elucidat
38 ids are incorporated into various tissues in juvenile mice and in a concentration dependent manner.
39 both in the first wave of spermatogenesis in juvenile mice and in ongoing spermatogenesis of adult mi
40                    Protein quantification in juvenile mice and in prophase mutants indicates that ear
41            VINP-28 enhanced early lesions in juvenile mice and resected human carotid artery plaques.
42 oth endogenous production of anti-GBS IgG in juvenile mice and vertical transfer of antibodies to off
43 aneous absence-like epileptiform activity in juvenile mice, and increased seizure susceptibility in r
44 Previously, we showed that some podocytes in juvenile mice are recruited from cells lining Bowman's c
45 al neurovasculature, it is not known whether juvenile mice are similarly impacted.
46 ory cues that enable specific recognition of juvenile mice are unknown.
47 anscriptome is significantly less complex in juvenile mice as compared to adult controls and, possibl
48 ific IgG production in exposed offspring and juvenile mice at age 12 and 14 days, respectively.
49 inister fractionated hindlimb irradiation to juvenile mice bearing implanted rhabdomyosarcoma (RMS) t
50 ntraepithelial lymphocytes), in suckling and juvenile mice before and after weaning, respectively.
51 e generation whereas activating Pdgfrbeta in juvenile mice blocks beige fat formation.
52 significant effect on synaptic plasticity in juvenile mice but impairs some forms of long-term potent
53 l numbers fall rapidly in the hippocampus of juvenile mice but stabilize during adulthood, ensuring l
54              NMDAR-dependent LTD prevails in juvenile mice, but a mechanistically different form of L
55 at Schaffer-collateral synapses monitored in juvenile mice, but again decreased NMDA-receptor mediate
56 ype 9 loss-of-function decreased survival in juvenile mice, but increased survival in adult mice with
57 d extends the cardiac regenerative window in juvenile mice by activating YAP-mediated transcriptional
58                                   Effects in juvenile mice can elucidate how erythropoietin (EPO) mig
59 suggest that isolated GnRH-EGFP neurons from juvenile mice can generate episodes of repetitive burst
60                       These effects of MI in juvenile mice closely resemble the effects of MD in adul
61 follicle development have been identified in juvenile mice deficient in heterologous oocyte-granulosa
62                                              Juvenile mice demonstrated alphaSma expression (indicati
63     The Abcb4(-/-) mice model of PFIC3, with juvenile mice developing progressive cholestatic liver i
64 elated differences in immunologic responses; juvenile mice displayed a sustained myeloid infiltrate (
65 ined influx of myeloid cells in the lungs of juvenile mice during acute RSV infection could potentiat
66 ry layer 2/3 neurons in the visual cortex of juvenile mice during the critical period, adult mice aft
67                                           In juvenile mice, early administration of IL-12/pulse IL-2
68           Administration of mFlt(1-3)-IgG to juvenile mice failed to induce apoptosis in liver endoth
69 , and fails to be efficiently transmitted to juvenile mice following birth.
70 t daily intranasal insulin administration to juvenile mice for 7 days prior to repeated isoflurane an
71                            Here we show that juvenile mice hemizygous for Cyfip1 have altered presyna
72                    Upon transplantation into juvenile mice, hPSC-derived cerebellar granule cells mig
73 f7 and, to lesser extent, E2f8 transgenes in juvenile mice impaired cell proliferation, caused replic
74 ability of CC17-GBS to cross into the CNS of juvenile mice in an in vivo model of meningitis.
75                 Knockdown of Ash1L in PFC of juvenile mice induces the downregulation of risk genes a
76                                   One of the juvenile mice initially tested negative for SNV RNA but
77                                       LTP in juvenile mice is resistant to the effects of Abeta oligo
78                                           In juvenile mice, LTD in NgR1(-/-), but not PirB(-/-), slic
79               Although visual deprivation in juvenile mice modifies the subunit composition and incre
80 as induced in the Y5Rs expressing neurons of juvenile mice (Npy1r(Y5R-/-) ).
81 n slices of the neocortex and hippocampus of juvenile mice of both sexes, using widefield and multiph
82 ccumulated in the calyx of Held terminals of juvenile mice of either sex during high-frequency spikin
83                                          All juvenile mice of the nonobese diabetic (NOD) strain deve
84 g, the cochlear nucleus, fusiform cells from juvenile mice (of either sex) generate robust 1-2 Hz osc
85  Here we examined the effect of HI injury in juvenile mice on retinal structure and function, in part
86 d that ablation of neuroligins in newborn or juvenile mice only modestly impaired basal synaptic func
87 ic morphology varies as a function of sex in juvenile mice or primary neuronal cell cultures is large
88                                           In juvenile mice, over-expression of E-Tmod is associated w
89 y, with widespread fMRI hyperconnectivity in juvenile mice reconfiguring to hippocampal hypoconnectiv
90 ocal malformation of cortical development in juvenile mice reduced neuronal cytomegaly and corrected
91                                 Treatment of juvenile mice restores the localization defect of RBM20
92 y of RDH10 in both Sertoli and germ cells in juvenile mice results in a blockage of spermatogonial di
93 tion of the first wave of spermatogenesis in juvenile mice results in agametic seminiferous tubules.
94 predominantly nonintegrating rAAV vectors to juvenile mice results in loss of persistent transgene ex
95 h-fat-low-fat diet regimen in adult, but not juvenile, mice results in an impoverished eVAT, but not
96 Patch-clamp recordings in layer 5 neurons of juvenile mice revealed increased intrinsic excitability.
97 te hippocampal slices and fixed tissues from juvenile mice revealed that Aif1(-/-) microglia display
98 climbing fiber/Purkinje cell connectivity in juvenile mice, showing the complexity of PCP action.
99 notypic preparations from the hippocampus of juvenile mice, stimulation of 5-HT(7)R/G(12) signaling p
100 re, we show that hippocampal astrocytes from juvenile mice subjected to social isolation exhibit incr
101 d a transient cardiac hypertrophy seen among juvenile mice that resolved with age.
102 elium led to a reversible delay in growth in juvenile mice that was associated with epithelial archit
103                                     Thus, in juvenile mice, the primary, but not exclusive, source of
104 wave of spermatogenesis in 12- to 28-day old juvenile mice to determine more precisely when HSP70-2 i
105 posure in childhood on LUT function by using juvenile mice treated with VCR (4 mg/kg) or saline and e
106        We next reassessed PEC recruitment in juvenile mice using a different reporter mouse and confi
107 t likely to be antibody positive (26.9%) and juvenile mice weighing between 13 and 14 g least likely
108  Serum metabolite profiles of adult lean and juvenile mice were comparable, while that of adult obese
109  doxorubicin-induced cardiotoxicity in which juvenile mice were exposed to doxorubicin, using a cumul
110                                              Juvenile mice were given partial brain irradiation of 10
111          Slow waves in intestinal muscles of juvenile mice were more sensitive to anoctamin-1 antagon
112 ns and levels of BDNF protein are highest in juvenile mice when adult motor patterns are shaped, whil
113 -term depression was selectively impaired in juvenile mice when NR2D overexpression was moderate.
114 ments for partner discrimination observed in juvenile mice which also show cognitive defects in adult
115       Notably, AAV-SYNGAP1 administration in juvenile mice, which corresponds to the typical age of d
116  distinct peripheral inflammatory profile in juvenile mice, which may impact the injury and repair re
117 ial defeat stress in males or females and in juvenile mice, which typically are excluded from classic
118                                              Juvenile mice with mutated c-Kit (c-Kit(Wv/+)) showed im
119 ved with systemic delivery of rAAV2/1 and in juvenile mice with rAAV2/9.

 
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