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1 in children with CBL syndrome and transient juvenile myelomonocytic leukemia.
2 cure a relevant proportion of children with juvenile myelomonocytic leukemia.
3 2AF35, and SRSF2 in 371 children with MDS or juvenile myelomonocytic leukemia.
4 A 3-year-old boy was treated for juvenile myelomonocytic leukemia.
5 alignancies, including an increased risk for juvenile myelomonocytic leukemia.
7 occurs in specific human cancers, including juvenile myelomonocytic leukemia, an aggressive myelopro
8 some mutations are rare in pediatric MDS and juvenile myelomonocytic leukemia and are unlikely to ope
9 suppressor gene is frequently inactivated in juvenile myelomonocytic leukemia, and Nf1 mutant mice mo
10 s associated with marked thrombocytosis, and juvenile myelomonocytic leukemia, are clonal hematologic
12 y contributing to the pathogenesis of NS and juvenile myelomonocytic leukemia caused by PTPN11 gain-o
13 e retrospectively analyzed 110 patients with juvenile myelomonocytic leukemia, given single-unit, unr
17 RAS family members are frequently mutated in juvenile myelomonocytic leukemia (JMML) and acute myeloi
18 boratory features that overlap with those of juvenile myelomonocytic leukemia (JMML) and chronic myel
19 t frequently occurring mutations in sporadic juvenile myelomonocytic leukemia (JMML) and JMML-like my
20 a (AML), acute lymphoblastic leukemia (ALL), juvenile myelomonocytic leukemia (JMML) and LEOPARD synd
22 human hematopoietic malignancies, including juvenile myelomonocytic leukemia (JMML) and T-cell lymph
23 Overactive RAS signaling is prevalent in juvenile myelomonocytic leukemia (JMML) and the myelopro
24 Chronic myelomonocytic leukemia (CMML) and juvenile myelomonocytic leukemia (JMML) are myelodysplas
25 r (GM-CSF) hypersensitivity is a hallmark of juvenile myelomonocytic leukemia (JMML) but has not been
26 re that individuals with Noonan syndrome and juvenile myelomonocytic leukemia (JMML) have germline mu
51 f mononuclear cells from five of seven (71%) juvenile myelomonocytic leukemia (JMML) patients and fro
52 dren with myelodysplastic syndrome (MDS) and juvenile myelomonocytic leukemia (JMML) treated in a uni
53 of validating the molecular underpinnings of juvenile myelomonocytic leukemia (JMML) with the generat
54 way is known to underlie the pathogenesis of juvenile myelomonocytic leukemia (JMML), a fatal childho
55 redisposes individuals to the development of juvenile myelomonocytic leukemia (JMML), a fatal myelopr
56 osphatase PTPN11 (SHP-2) are associated with juvenile myelomonocytic leukemia (JMML), a myeloprolifer
57 fibromatosis type 1 (NF1) are predisposed to juvenile myelomonocytic leukemia (JMML), an aggressive m
58 ype myelodysplastic syndrome (A-MDS), 60 had juvenile myelomonocytic leukemia (JMML), and 6 infants w
59 oodwin et al investigate the pathogenesis of juvenile myelomonocytic leukemia (JMML), demonstrating t
60 id in acute promyelocytic leukemia (APL) and juvenile myelomonocytic leukemia (JMML), we evaluated UA
61 in childhood acute leukemias, in addition to juvenile myelomonocytic leukemia (JMML), which is a myel
71 11 mutations occur in children with sporadic juvenile myelomonocytic leukemia, myelodysplasic syndrom
72 Indeed, some pediatric leukemias, such as juvenile myelomonocytic leukemia, myeloid leukemia of Do
76 identified in human leukemias, particularly juvenile myelomonocytic leukemia, which is characterized