ライフサイエンスコーパス: kainate

1 glutamate receptors (AMPA and NMDA, but not kainate).

2 of 2e and 2f shows a similar binding mode as kainate.

3 activity were evoked by local application of kainate.

4 highest sensitivity of all inner neurons to kainate.

5 omplex with 2i comparable to that induced by kainate.

6 arable to those of the distal carboxylate in kainate.

7 s containing glutamate receptor, ionotropic, kainate 2).

8 some 1 (GRIK3 (glutamate receptor ionotropic kainate 3)), chromosome 4 (KLHL2 (Kelch-like protein 2))

9 ogenetic modulation of gamma oscillations in kainate-activated hippocampal slices.

10 eversing behavioral effects of the selective kainate agonist ATPA.

11 ctive a dorsomedial subdivision; 3) distinct kainate, alpha2 , and muscarinic receptor densities that

12 Kainate and alpha-amino-3-hydroxy-5-methyl-4-isoxazolepr

13 he transient OFF bipolar cells, whereas both kainate and AMPA receptors contributed in the other cell

14 tabilities of homodimers and heterodimers of kainate and AMPA receptors using fluorescence-detected s

15 NA aptamers designed to individually inhibit kainate and AMPA receptors.

16 osing effects on 4.1N association with GluK2 kainate and GluA1 AMPA receptors.

17 nding-impaired subunits, as we show for both kainate and GluA2 AMPA receptors.

18 We therefore designed experiments to compare kainate and glutamate desensitization and efficacy in wi

19 The identification of AMPA, kainate and NMDA glutamate receptor subtypes by Watkins

20 p5I and CVNs were completely blocked by AMPA/kainate and NMDA glutamatergic receptor antagonists.

21 ynaptic pathways via modulation of both AMPA/kainate and NMDA receptors at different synapses in the

22 els of SE-induced epilepsy: intrahippocampal kainate and systemic administration of paraoxon.

23 in addition to the NMDA receptor, the AMPA, kainate and the metabotropic GluRs may be targets for th

24 ith an antagonist (CNQX), a partial agonist (kainate), and two full agonists (glutamate and quisquala

25 ntaining the GluN2A or GluN2B subunits AMPA, kainate, and GABA or glycine receptors or a variety of o

26 ic pathway, and mature N-methyl-d-aspartate, kainate, and GABA(A) receptors did not reach the synapse

27 inct clade from the well-characterized AMPA, kainate, and NMDA iGluR subtypes found in vertebrates.

28 that is notably absent from vertebrate AMPA, kainate, and NMDA iGluRs greatly increases the rate of r

29 Phylogenetic analysis reveals AMPA, kainate, and NMDA receptor families in insect genomes, s

30 iGluRs (AMPA, kainate, and NMDA receptor subtypes) are tetrameric asse

31 the proconvulsant activity of systemic AMPA, kainate, and pentylenetetrazol is not mediated by GluK1

32 aneo), and here we compare the properties of kainate- and carbachol-induced oscillatory activity gene

33 vey of iGluR gene expression revealed AMPA-, Kainate-, and NMDA-type subunits are expressed in zebraf

34 Area p32 contains higher kainate, benzodiazepine (BZ), and serotonin (5-HT)1A but

35 en historically defined by (3)H-radiolabeled kainate binding.

36 Notably, the dose of kainate capable of inducing an approximately 50% rate of

37 ull agonist glutamate or the partial agonist kainate, consistent with conductance state-dependent blo

38 ith stargazin was the result of both reduced kainate desensitization and increased kainate efficacy.

39 much less is known about factors that affect kainate desensitization.

40 A receptor-mediated responses to the agonist kainate differ from those of glutamate in two important

41 Kainate efficacy was affected by factors distinct from l

42 educed kainate desensitization and increased kainate efficacy.

43 ls of GluK2/3 in primary neurons and reduced kainate-evoked currents in CA1 pyramidal neurons in acut

44 s and largely rescue behavioural deficits in kainate-exposed animals.

45 luN1/GluN2B NMDA receptors, as well as AMPA, kainate, GABA, glycine, nicotinic, serotonin, and purine

46 e densities for glutamatergic AMPA, NMDA and kainate, GABAergic GABAA , muscarinic M1 , M2 and nicoti

47 ymorphism (rs2832407) in GRIK1, encoding the kainate GluK1 receptor subunit.

48 We examined the AMPA and kainate glutamate receptor composition in each bipolar c

49 ious studies, stimulation of ionotropic AMPA/kainate glutamate receptors on cultured oligodendrocyte

50 pplication of a GABAA receptor antagonist or kainate, glutamate receptor agonist.

51 the relative efficacy of the partial agonist kainate (I(KA)/I(Glu) ratio 0.4) and a greater than five

52 nner retinal neuron populations responded to kainate in a concentration-dependent manner.

53 However, exposure of S940A mice to kainate induced lethality within 30 min of kainate injec

54 nction persists, indicated by alterations in kainate-induced gamma oscillations and impaired nest bui

55 ated deSUMOylation of PKC is involved in the kainate-induced GlyR endocytosis and thus plays an impor

56 2-expressing astrocytes reduces the power of kainate-induced hippocampal ex vivo gamma oscillation.

57 We found that 5-HT decreases the power of kainate-induced hippocampal gamma oscillations in both s

58 In a kainate-induced murine seizure model, administration of

59 f an M-channel inhibitor that did not affect kainate-induced seizure transiently eliminated the antic

60 ctly monitor the acute structural effects of kainate-induced seizures on cortical astrocytes.

61 at model of massive neuronal activation upon kainate-induced seizures we found that elevated neuronal

62 and SCaMC-3 KO mice are more susceptible to kainate-induced seizures, showing that early PARP-1-depe

63 major brain Nav channel, Nav1.2, after acute kainate-induced seizures.

64 ease, PRS rats were also highly resistant to kainate-induced seizures.

65 Lastly, kainate-induced status epilepticus causes GIRK1 and GIRK

66 irm that horizontal cell depolarization with kainate inhibits and horizontal cell hyperpolarization w

67 o kainate induced lethality within 30 min of kainate injection and subsequent entrance into SE.

68 When isoflurane was administered during kainate injection, duration and severity of SE were not

69 ts strongly desensitizing responses, whereas kainate is a partial agonist with responses that are oft

70 CRMP4 proteins with the GluK5 subunit of the kainate (KA) receptor (KAR) and investigated the role of

71 tivity in the rat hippocampus in response to kainate (KA)-induced epileptogenesis.

72 Using a combination of kainate (KA, 200 nm) and the cholinergic agonist carbach

73 campal astrocytes generated from control and kainate-lesioned rats.

74 rotransmission from AWC and through AMPA and kainate-like glutamate receptor subunits.

75 isoflurane reduced neuroinflammation in the kainate model.

76 able in the two species in layers III-Va for kainate, NMDA, gamma-aminobutyric acid (GABA)B , BZ, and

77 in all mouse OFF bipolar pathways depend on kainate, not AMPA, receptors.

78 d release acetylcholine when stimulated with kainate or a lowering in glucose concentration.

79 ny effect on other AMPA receptor subunits or kainate or NMDA receptors.

80 mplex in the presence of the partial agonist kainate or the full agonist quisqualate together with a

81 Isoflurane was either administered during (kainate) or after (paraoxon) induction of SE.

82 ligand-binding domain gating ring, enhancing kainate potency and diminishing the ensemble of desensit

83 Here, we show that kainate receptor (KAR) heteromerization and association

84 Phosphorylation or SUMOylation of the kainate receptor (KAR) subunit GluK2 have both individua

85 ole(s) of Rab17 in AMPA receptor (AMPAR) and kainate receptor (KAR) trafficking.

86 Neto) 1 and Neto2, the auxiliary subunits of kainate receptor (KARs), are phosphorylated by multiple

87 ed antidepressant-like effects required AMPA/kainate receptor activation, as evidenced by the ability

88 in GluK5 expression is sufficient to enhance kainate receptor activity by modulating receptor channel

89 at loss of synaptic AMPA receptors increased kainate receptor activity in cerebellar granule cells wi

90 acological inhibition or genetic ablation of kainate receptor activity reduces pain behaviors in a nu

91 ochemistry, and pharmacology to identify the kainate receptor and auxiliary subunits in ground squirr

92 on treatment of an acutely administered AMPA/kainate receptor antagonist and delayed transplantation

93 These findings show that a highly selective kainate receptor antagonist can affect the deficits in s

94 no-3-hydroxy-5-methyl-4-isoxazole propionate/kainate receptor antagonist CNQX (0, .01, .03, .1 mug/si

95 finding that intracore injection of the AMPA/kainate receptor antagonist CNQX attenuated the ability

96 NMDAR antagonist MK801, and also by the AMPA/kainate receptor antagonist CNQX.

97 The AMPA/kainate receptor antagonist NBQX was directly administer

98 havioral responses to kainic acid and that a kainate receptor antagonist normalized altered behaviors

99 We found that UBP-310, a kainate receptor antagonist with high selectivity for Gl

100 t work examined effects of another selective kainate receptor antagonist, (S)-1-(2-Amino-2-carboxyeth

101 A) receptor antagonist, but not an NMDA/AMPA/kainate receptor antagonist, suggesting that they were m

102 further support for the idea that selective kainate receptor antagonists could be novel therapeutic

103 Compounds 18i and (-)-4 were the most potent kainate receptor antagonists, and 18i was selective for

104 The association of kainate receptor ATD dimers is generally weaker than the

105 The free energy maps of AMPA and kainate receptor ATD dimers provide a framework for the

106 al/pharmacological data showed that CeA AMPA/kainate receptor blockade attenuates cisplatin-induced p

107 d knockdown, we determine that Tm5c uses the kainate receptor Clumsy to receive excitatory glutamate

108 However, many AMPA and kainate receptor complexes in vivo are heteromers compos

109 tly as auxiliary proteins that slowed GluK2a kainate receptor current kinetics without impacting rece

110 ) a dorsomedial region characterized by high kainate receptor density.

111 r, heterologous expression of the Drosophila kainate receptor DKaiR1D and the AMPA receptor DGluR1A r

112 However, the kainate receptor family consists of five different subun

113 The kainate receptor family of glutamate-gated ion channels

114 selectivity among the more distantly related kainate receptor family.

115 new roles for Neto1 and Neto2 in regulating kainate receptor function and identify domains within th

116 This review will cover kainate receptor function and participation in pain sign

117 ould underlie some of the unusual aspects of kainate receptor function in the mammalian CNS.

118 Here, by determining the structure of the kainate receptor GluK2 subtype in its desensitized state

119 entified some known SAP102 binding partners, kainate receptor GluR6/7 and inward rectifier potassium

120 Kainate receptor heteromerization and auxiliary subunits

121 he present study highlights the role of AMPA-kainate receptor in IVH-induced white matter injury and

122 pharmacogenetic findings also implicate the kainate receptor in the mechanism of topiramate's effect

123 AMPA-kainate receptor induced excitotoxicity contributes to o

124 Our data suggest that AMPA-kainate receptor inhibition alleviates OPC loss and IVH-

125 Here, we show that AMPA-kainate receptor inhibition by NBQX suppresses inflammat

126 via AMPA receptor activation, and that AMPA-kainate receptor inhibition suppresses inflammation and

127 normal LTP, as did an artificially expressed kainate receptor not normally found at these synapses.

128 The kainate receptor occupies a position distinct from that

129 utive mGluR7 recruitment and regulated GluK2 kainate receptor recruitment, revealing a mechanism that

130 The 7.6 A structure of a desensitized kainate receptor shows how these changes accommodate cha

131 oxy-5-methyl-4-isoxazole propionic acid) and kainate receptor subtypes in their major functional stat

132 resting and desensitized states of AMPA and kainate receptor subtypes, the ion channels are closed,

133 of a single gene coding for a high-affinity kainate receptor subunit (i.e., grik4) in a limited area

134 g that fusing the N-terminal 150 residues of kainate receptor subunit 2 (KA2) to the recently discove

135 The grik2 gene, coding for the kainate receptor subunit GluK2 (formerly GluR6), is asso

136 hippocampal neurons, we discovered that the kainate receptor subunit GluK2 and the auxiliary subunit

137 loop apex and the transmembrane M3 helix of kainate receptor subunit GluK2.

138 We conclude that GluK1 is the predominant kainate receptor subunit in cb1 and cb3 Off bipolar cell

139 reased expression of the GluK5 high-affinity kainate receptor subunit.

140 with homologous segments from the rat GluK2 kainate receptor subunit.

141 -expression of Neto1 and 2 with pore-forming kainate receptor subunits also increases the duration of

142 Here we report that the GluK1 and GluK2 kainate receptor subunits interact with the spectrin-act

143 ction with mice deficient in GluK1 and GluK2 kainate receptor subunits to assess the role of GluK1 ka

144 FB9s-b selectively inhibited GluK1 and GluK2 kainate receptor subunits, and also GluK1/GluK5 and GluK

145 ans in the disease state, including AMPA and kainate receptor subunits, glutamate transporters EAAT1

146 In particular, the slow decay of kainate receptor synaptic currents contrasts with the ra

147 Similarly, the most common phenotype for kainate receptor variants is intellectual disability.

148 onotropic glutamate receptors, including the kainate receptor, in the central nervous system.

149 nabling efficient 2P activation of the GluK2 kainate receptor, LiGluR.

150 The augmentation of kainate receptor-mediated currents in the absence of AMP

151 ction of both NMDA receptor (NMDAR) and AMPA/kainate receptor-mediated evoked excitatory postsynaptic

152 e activity of MSNs via a glutamatergic, AMPA/kainate receptor-mediated mechanism, indicated by increa

153 Incorporation of NETO proteins into kainate receptor-signaling complexes therefore extends t

154 Like classical LTP, kainate-receptor-dependent LTP recruits recycling endoso

155 Kainate receptors (KARs) are a subfamily of ionotropic g

156 Kainate receptors (KARs) are found ubiquitously in the C

157 Kainate receptors (KARs) are important modulators of syn

158 Kainate receptors (KARs) are ionotropic glutamate ion ch

159 Kainate receptors (KARs) are ionotropic glutamate recept

160 Kainate receptors (KARs) are ionotropic glutamate recept

161 mit high temporal frequency signals, whereas kainate receptors (KARs) are presumed to encode lower te

162 Heteromeric kainate receptors (KARs) can be assembled from varying c

163 Here we show that activation of kainate receptors (KARs) causes GlyR endocytosis in a ca

164 Kainate receptors (KARs) consist of a class of ionotropi

165 Presynaptic kainate receptors (KARs) exert a modulatory action on tr

166 e classes of ionotropic glutamate receptors, kainate receptors (KARs) have a unique brain distributio

167 Kainate receptors (KARs) have been implicated in a numbe

168 Interneuronal kainate receptors (KARs) regulate GABAergic transmission

169 coassemble with NMDA receptors (NMDARs) and kainate receptors (KARs) to modulate their function.

170 Kainate receptors (KARs), a family of ionotropic glutama

171 ons of the dorsal root ganglia (DRG) express kainate receptors (KARs), a subfamily of glutamate recep

172 urons in the neonatal DRG express functional kainate receptors (KARs), one of three subfamilies of io

173 NETO1 and NETO2 are auxiliary subunits of kainate receptors (KARs).

174 ; a presynaptic form (pre-LTP) that requires kainate receptors and a postsynaptic form (post-LTP) tha

175 pathway requires the metabotropic action of kainate receptors and activation of G protein, protein k

176 ke-2 (Neto2) as a novel accessory subunit of kainate receptors and showed that Neto2 modulates the ga

177 l-4-isoxazolepropionic acid, and heteromeric kainate receptors are all downstream targets of GPCR sig

178 Kainate receptors are also expressed throughout central

179 Kainate receptors are glutamate-gated cation-selective c

180 AMPA and kainate receptors are glutamate-gated ion channels whose

181 Kainate receptors are less well understood than other gl

182 n performed on homomeric GluK2 but, in vivo, kainate receptors are likely heterotetramers.

183 uK2 knock-out mice, demonstrating that GluK1 kainate receptors are not required for epileptogenesis o

184 properties and kinetic behaviour of AMPA and kainate receptors at the level of single receptor molecu

185 -5-methylisoxazol-4-yl)propanoate (AMPA) and kainate receptors bound to full and partial agonists.

186 bipolar cells was blocked by antagonists to kainate receptors but not AMPA receptors.

187 In neurons, kainate receptors can associate with the auxiliary subun

188 Kainate receptors can be subject to voltage-dependent bl

189 While some AMPA and kainate receptors can form functional homomeric ion chan

190 ycosylation and cell surface localization of kainate receptors composed of GluK2/3 subunits.

191 tropic glutamate receptor 7) and GluK2-KARs (kainate receptors containing glutamate receptor, ionotro

192 We conclude that selective activation of kainate receptors containing the GluK1 subunit can trigg

193 Kainate receptors containing the GluK1 subunit have an i

194 than other glutamate receptors, and synaptic kainate receptors display properties that differ from re

195 ptic Ca(2+) influx through calcium-permeable kainate receptors during its induction.

196 Kainate receptors exhibit a highly compartmentalized dis

197 nglion cells confirmed the essential role of kainate receptors for signaling in both transient and su

198 n of Neto subunits with recombinant GluK2(Q) kainate receptors greatly reduced inward rectification w

199 good evidence that both heteromeric AMPA and kainate receptors have a 2:2 subunit stoichiometry and a

200 sed 4.1N interaction with GluK2/3-containing kainate receptors in acute brain slices, an effect that

201 d with combined activation of muscarinic and kainate receptors in different subregions of the medial

202 Neuronal kainate receptors in dorsal root ganglia were sensitive

203 The presence of GluK2 kainate receptors in L2/3 enhances synaptic transmission

204 eceptor subunits to assess the role of GluK1 kainate receptors in provoking seizures and in kindling

205 previously unsuspected role for postsynaptic kainate receptors in the induction of functional and str

206 ceptors was mediated exclusively through the kainate receptors in the transient OFF bipolar cells, wh

207 Postsynaptic kainate receptors mediate excitatory synaptic transmissi

208 Kainate receptors mediate fast, excitatory synaptic tran

209 Kainate receptors mediated approximately 80% of the syna

210 Kainate receptors mediated responses to contrast modulat

211 Although the kainate receptors mediated the light responses in the su

212 Kainate receptors of astrocytes act as sensors of extrac

213 identified glutamate receptors, for example, kainate receptors on which NMDA acts as a competitive an

214 NMDAR antagonists, but not blockers of AMPA/kainate receptors or voltage-gated sodium channels, prev

215 r results have revealed a mechanism by which kainate receptors regulate KCC2 expression in the hippoc

216 Kainate receptors require the presence of external ions

217 AMPA and kainate receptors share a high degree of sequence and st

218 1a/b cells were mediated by GluK1-containing kainate receptors that behaved differently from the rece

219 id (ATPA), a potent and selective agonist of kainate receptors that include the GluK1 subunit, in con

220 However, allocation of functional kainate receptors to known cell types and their sensitiv

221 mately 21 A resolution, of full-length GluK2 kainate receptors trapped in antagonist-bound resting an

222 at an aberrant readout of synaptic inputs by kainate receptors triggered a long-lasting impairment of

223 erologous systems, the temporal responses of kainate receptors vary when different channel-forming an

224 Galectin modulation of GluK2 kainate receptors was dependent upon complex oligosaccha

225 also GluK1/GluK5 and GluK2/GluK5 heteromeric kainate receptors with equal potency.

226 Pharmacological blockade of AMPA-kainate receptors with systemic NBQX, or selective AMPA

227 of glutamate receptors (i.e. AMPA, NMDA, and kainate receptors) at the synapse.

228 ropic receptor with metabotropic properties (kainate receptors) failed to prevent depolarization-indu

229 nals at synapses that contain either AMPA or kainate receptors, all with different temporal propertie

230 AMPA and kainate receptors, along with NMDA receptors, represent

231 d pentylenetetrazol is not mediated by GluK1 kainate receptors, and deletion of these receptors does

232 g the different temporal signalling roles of kainate receptors, as cones release glutamate over a ran

233 -methyl-4-isoxazolepropionic acid (AMPA) and kainate receptors, but only oscillations in the DP were

234 systemic ATPA, acting specifically via GluK1 kainate receptors, causes locomotor arrest and forelimb

235 just through the NMDA but also mGlu and AMPA/Kainate receptors, completely reversed the cell death ph

236 Although annotated as glutamate and kainate receptors, crystal structures of the ML032222a a

237 ntagonists of these receptors, especially of kainate receptors, is useful for developing potential tr

238 c glutamate receptors (iGluRs), the NMDA and kainate receptors, mediate a majority of excitatory neur

239 Relative to the homologous AMPA and kainate receptors, NMDA receptors have additional inters

240 ndly slows the desensitization rate of GluK1 kainate receptors, promotes plasma membrane localization

241 ubunits that combined segments from NMDA and kainate receptors, subtypes with distinct pharmacologica

242 In contrast, the lesser-studied kainate receptors, which are often present at both pre-

243 promoting the insertion and stabilization of kainate receptors, which may be important for tuning neu

244 -hydroxy-5-methyl-4-isoxazolepropionic acid)/kainate receptors.

245 e granule cells and operate via postsynaptic kainate receptors.

246 found in all vertebrate NMDA but not AMPA or kainate receptors.

247 xcitability through their actions on AMPA or kainate receptors.

248 roxy-5-methyl-4-isoxazolepropionic acid] and kainate receptors.

249 ontaining NMDA receptors as well as AMPA and kainate receptors.

250 combinant homomeric and heteromeric AMPA and kainate receptors.

251 discharges in BLA slices in vitro via GluK1 kainate receptors.

252 rgic ACs do not appear to express functional kainate receptors.

253 e involvement of Ca(2+)/calcineurin and AMPA/kainate receptors.

254 ties by having subnanomolar IC(50) values at kainate receptors.

255 tion, desensitization, and ion modulation in kainate receptors.

256 st that Neto2 modulates the function of most kainate receptors.

257 ferent from those of the homologous AMPA and kainate receptors.

258 ivity for effectively blocking both AMPA and kainate receptors.

259 s that can potentially inhibit both AMPA and kainate receptors.

260 es that contained GluK1, GluK1/5, or GluK2/5 kainate receptors.

261 c currents in neurons expressing recombinant kainate receptors.

262 tly the activity of both the GluK1 and GluK2 kainate receptors.

263 on structure currently exists of heteromeric kainate receptors.

264 hyl-4-isoxazolepropionic acid receptors, and kainate receptors.

265 rter version (55 nt) inhibited both AMPA and kainate receptors.

266 F bipolar cells were exclusively mediated by kainate receptors.

267 densities for glutamatergic AMPA, NMDA, and kainate receptors; GABAA receptors; muscarinic M1 , M2 a

268 no-terminal domain tetramer in AMPA, but not kainate, receptors with a two-fold to four-fold symmetry

269 The efficacy of kainate relative to glutamate has previously been shown

270 and a greater than fivefold potentiation of kainate responses by cyclothiazide-suggest AMPAR associa

271 The increase in kainate responses for AMPA receptors coexpressed with st

272 acrine (ACs) and ganglion cells (GCs) showed kainate responses with different sensitivities between m

273 In wild-type mice, SE induced by kainate resulted in dephosphorylation of S940 and KCC2 i

274 Brief treatment with NMDA and kainate results in loss of IHC-SGN synapses and degenera

275 Kainate seizures cause an immediate, but transient, vacu

276 that type 2 bipolar cells were part of this kainate-sensitive population.

277 uanidobutane agmatine (AGB), we investigated kainate sensitivity of neurochemically identified cell p

278 On GluA2(A621G), the partial agonist kainate showed efficacy similar to a full agonist, and c

279 er, hyperactive shifts in neuronal activity (kainate status epilepticus and CaMKIIa Gq DREADD activat

280 a selective impairment of depolarization- or kainate-stimulated glutamate and [(3)H]d-aspartate relea

281 -methyl-3-hydroxy-4-isoxazole propionate and kainate subtypes of the ionotropic glutamate receptors.

282 non-NMDA glutamate receptors (both AMPA and kainate subtypes) are modulated by the association of th

283 as studied and compared between controls and kainate-treated rats, a model of temporal lobe epilepsy,

284 the blood-brain barrier between control and kainate-treated rats, and the effect of tariquidar treat

285 B(max) was decreased in kainate-treated rats, compared with controls, but no alt

286 B(max) was decreased by 12% in kainate-treated rats, compared with controls.

287 f episodic-like memory in rats, we show that kainate-treated TLE rats exhibit a selective impairment

288 We further evaluated the effects of kainate treatment on the total number, density, and soma

289 ding for a glutamate receptor subunit of the kainate type.

290 d since the first report of the cloning of a kainate-type glutamate receptor (KAR) subunit.

291 a mutant allele of glr-3 gene that encodes a kainate-type glutamate receptor.

292 Presynaptic kainate-type glutamate receptors (KARs) regulate glutama

293 Postsynaptic kainate-type glutamate receptors (KARs) regulate synapti

294 mammals, the selective expression of AMPA or kainate-type glutamate receptors in the dendrites of dif

295 by the differential expression of AMPA- and kainate-type glutamate receptors, respectively.

296 ecently identified as auxiliary subunits for kainate-type glutamate receptors.

297 ssembly occurs for homomeric and heteromeric kainate-type glutamate receptors.

298 s restricted period of synaptic development, kainate-type of ionotropic glutamate receptors (KARs) ar

299 ropic glutamate subfamilies (i.e. AMPA-type, kainate-type, and NMDA-type) assemble as tetramers of fo

300 ed cholinergic ACs were highly responsive to kainate, whereas dopaminergic ACs do not appear to expre