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1  RNA oligonucleotide abolished the effect of kallistatin.
2 llikrein is a specific target proteinase for kallistatin.
3 nhibitor, affected vasorelaxation induced by kallistatin.
4                         Infusion of purified kallistatin (0.07-1.42 nmol/kg) into cannulated rat jugu
5   Here, we report that circulation levels of kallistatin, a member of the serine proteinase inhibitor
6                          Renal expression of kallistatin, a potent vasodilator, was down-regulated in
7                                              Kallistatin, a serine proteinase inhibitor (serpin), is
8 d region between the H helix and C2 sheet of kallistatin acquired heparin-suppressed inhibitory activ
9                                              Kallistatin also caused dose-dependent vasodilation of t
10 8 (Keratin type II cytoskeletal 6A), P29622 [Kallistatin (also known as Serpin A4)], P17900 (Ganglios
11                                              Kallistatin, an endogenous plasma protein, exhibits plei
12 PTBN1 express much less of the Wnt inhibitor kallistatin and exhibit decreased beta-catenin phosphory
13  inhibition of Wnt/beta-catenin signaling by kallistatin and rescued the wound-healing deficiency in
14  Conversely, adenovirus-mediated transfer of kallistatin antisense cDNA into cultured VSMCs inhibited
15 rmore, local delivery of adenovirus carrying kallistatin antisense cDNA significantly downregulated k
16 stimulated A549 human lung epithelial cells, kallistatin attenuated apoptosis, down-regulated Fas/Fas
17                                              Kallistatin attenuated VEGF- or bFGF-induced capillary d
18              Sequence analysis revealed that kallistatin-binding protein is human Kruppel-like factor
19                                 To isolate a kallistatin-binding protein that mediates the vascular a
20          In summary, we identified KLF4 as a kallistatin-binding protein, which has a novel role in m
21                                     Specific kallistatin-binding sites were identified in rat aorta b
22 ular modeling of the reactive center loop of kallistatin bound to the reactive crevice of tissue kall
23 that an intravenous bolus injection of human kallistatin caused a rapid, potent, and transient reduct
24 s, we prepared adenovirus carrying the human kallistatin cDNA (Ad.HKBP) and evaluated the effect of k
25 esponse markers (HR6 model), including SYWC, kallistatin, complement C9, gelsolin, testican-2, and al
26                                     KLF4 and kallistatin complex formation was identified in endothel
27 e region between the H helix and C2 sheet of kallistatin, comprise a major heparin-binding site respo
28 The loop between the H helix and C2 sheet of kallistatin containing clusters of basic amino acid resi
29 hibitory characteristics similar to those of kallistatin demonstrates that the loop between the H hel
30 h regions were substituted to generate three kallistatin double mutants K187A/K188A (mutations in the
31 listatin in lung by gene transfer with human kallistatin-encoding plasmid ameliorated acute lung inju
32               Taken together, high levels of kallistatin exacerbate DN at least partly by inducing RA
33                  We observed an induction of kallistatin expression by platelet-derived growth factor
34                               Restoration of kallistatin expression in these cells reversed the obser
35 indings suggest the therapeutic potential of kallistatin for sepsis-related ALI/ARDS.
36 ion library by using an alkaline phosphatase-kallistatin fusion protein binding assay.
37 n cDNA (Ad.HKBP) and evaluated the effect of kallistatin gene delivery on spontaneous angiogenesis in
38                                        Local kallistatin gene delivery significantly reduced capillar
39 kallistatin levels, and decreased SPTBN1 and kallistatin gene expression is associated with decreased
40               Mouse survival was improved by kallistatin gene transfer or recombinant human kallistat
41      The aim of this study is to evaluate if kallistatin has a direct effect on the vasculature and o
42 in the SERPINA4 gene, whose protein product, kallistatin, has been linked to apoptosis in the kidney.
43 study, we investigated the potential role of kallistatin in angiogenesis in vitro and in vivo.
44           To further investigate the role of kallistatin in angiogenesis, we prepared adenovirus carr
45 -activated protein kinase (MAPK) activity by kallistatin in cultured VSMCs.
46 3-P2-P1 residues (residues 386-388) of human kallistatin in determining inhibitory specificity toward
47 udy introduces the potential significance of kallistatin in directly regulating blood pressure to red
48 ride (LPS)-treated mice, expression of human kallistatin in lung by gene transfer with human kallista
49    These results demonstrate a novel role of kallistatin in the inhibition of angiogenesis and tumor
50                    The potential function of kallistatin in vascular biology was investigated by stud
51                                              Kallistatin increased endothelial nitric-oxide synthase
52                                    Exogenous kallistatin induced a >2-fold increase of VSMC prolifera
53                               We showed that kallistatin inhibits tumor necrosis factor-alpha-induced
54                                              Kallistatin is a heparin-binding serine proteinase inhib
55                                              Kallistatin is a plasma protein that exhibits pleiotropi
56                  These results indicate that kallistatin is a potent vasodilator which may function d
57                                              Kallistatin is a serine proteinase inhibitor (serpin) th
58                                              Kallistatin is a serine proteinase inhibitor which binds
59                                              Kallistatin is a serpin with a unique P1 Phe, which conf
60                                              Kallistatin is a unique serine proteinase inhibitor (ser
61                   The inhibitory activity of kallistatin is abolished upon heparin binding.
62                                              Kallistatin is an inhibitor of tissue kallikrein and als
63                   The inhibitory activity of kallistatin is blocked upon its binding to heparin.
64 the loop between the H helix and C2 sheet of kallistatin is crucial in tissue kallikrein inhibition,
65          Moreover, a newly described serpin, kallistatin (KAL, gene symbol PI4), was also mapped with
66                                        Human kallistatin (KAL; gene SERPINA4) is a serine proteinase
67 eet of kallistatin was shown to suppress the kallistatin-kallikrein interaction through competition f
68  demonstrated a positive correlation between kallistatin levels and DN, suggesting a potential biomar
69         To test the hypothesis that elevated kallistatin levels could contribute to a wound-healing d
70                    Here, we demonstrate that kallistatin levels were positively correlated with the c
71                  Here we reported that serum kallistatin levels were significantly increased in diabe
72                           In addition, renal kallistatin levels were significantly upregulated in mou
73 is positively correlated with E-cadherin and kallistatin levels, and decreased SPTBN1 and kallistatin
74 ells of human blood vessels, suggesting that kallistatin may be involved in the regulation of vascula
75                  These results indicate that kallistatin may play an important role in mediating PDGF
76       In balloon-injured vessels, endogenous kallistatin mRNA and protein levels increased up to 10-f
77 n antisense cDNA significantly downregulated kallistatin mRNA levels and attenuated neointima formati
78                          Intense staining of kallistatin mRNA was identified in the proliferating VSM
79                                              Kallistatin mutants containing double Ala substitutions
80                                        Human kallistatin mutants with 19 different amino acid substit
81                     To unveil the effects of kallistatin on DN and its underlying mechanism, we cross
82 S inhibitor abolished the blocking effect of kallistatin on vascular cell adhesion molecule-1 and mon
83                                              Kallistatin overexpression exacerbated albuminuria, rena
84 n between the H helix and C2 sheet), using a kallistatin P1Arg variant as a scaffold.
85 t of vasodilation mediated by the kallikrein-kallistatin pathway and vascular hypertrophy.
86               Our previous study showed that kallistatin plays a role in neointima hyperplasia.
87 t of the other chimera, which contained only kallistatin's P3-P2' sequence, and 2300-fold higher than
88           The combination of I-309, SYWC and kallistatin showed the most promising results to discern
89                               Purified human kallistatin significantly inhibited vascular endothelial
90                                         Like kallistatin, the binding activity of K187A/K188A to tiss
91                               Lower ratio of kallistatin to total protein in BALF showed a significan
92  distinguishes the inhibitory specificity of kallistatin toward kallikrein versus chymotrypsin.
93  of Wnt/beta-catenin signaling, we generated kallistatin-transgenic (KS-TG) mice.
94 d a decreased number of blood vessels in the kallistatin-treated group as compared to the control.
95 llistatin gene transfer or recombinant human kallistatin treatment after LPS challenge.
96 empted to locate the heparin-binding site of kallistatin using synthetic peptides derived from its su
97 n mediating the anti-inflammatory actions of kallistatin via increasing eNOS expression in endothelia
98       Relaxation of isolated aortic rings by kallistatin was observed in the presence (ED50 of 3.4 x
99 sequence between the H helix and C2 sheet of kallistatin was shown to suppress the kallistatin-kallik
100 rotein that mediates the vascular actions of kallistatin, we screened and identified a positive clone
101 ion encompassing the H helix and C2 sheet of kallistatin, were used to assess their heparin binding a
102 n, human serum factor H-related protein, and kallistatin, which exhibited large abundance shifts betw
103 m, we crossed transgenic mice overexpressing kallistatin with OVE26 mice (KS-tg/OVE).
104 , had no effect on the hypotensive effect of kallistatin yet it abolished the blood pressure-lowering

 
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