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1 dent mitotic delay and also cause defects in karyokinesis.
2 M II in maintaining mitotic stability during karyokinesis.
3 nd of daughter buds toward the completion of karyokinesis.
4 ntractility and in processes such as mitotic karyokinesis.
5 /B-C- cardiac myocytes show major defects in karyokinesis.
6 e states without carrying out cytokinesis or karyokinesis.
7 hase-anaphase transition leading to abnormal karyokinesis.
8 ton, increased cell spreading, and increased karyokinesis.
9  suggesting that byr4 is required for proper karyokinesis.
10 e to aberrant nuclear division, or defective karyokinesis.
11 of the centromere marker, CENH3 and impaired karyokinesis.
12 he diatoms permanently by controlling diatom karyokinesis.
13 ear lamina filament supporting cardiomyocyte karyokinesis, also facilitates cell division and cardiac
14 2 did not rescue the Wee1 inhibition-induced karyokinesis and cytokinesis defects.
15 hology and to highly enlarged cells in which karyokinesis and cytokinesis frequently are uncoupled.
16 ndle assembly as well as the coordination of karyokinesis and cytokinesis in mouse oocytes.
17 eate parasite can establish a state in which karyokinesis and cytokinesis occur in phase with the hos
18  centrosome that segregates the functions of karyokinesis and cytokinesis provides an explanation for
19 otypes consistent with spatial uncoupling of karyokinesis and cytokinesis suggesting that GEMINI POLL
20  were complemented with the demonstration of karyokinesis and cytokinesis to provide structural evide
21 ate breeding, implying distinct functions in karyokinesis and cytokinesis.
22 progression of all mitotic phases leading to karyokinesis and cytokinesis.
23  of growth of adjacent cells, and defects in karyokinesis and cytokinesis.
24  did not result in any inhibitory effects on karyokinesis and early stages of cytokinesis.
25 uman iPS cell-derived cardiomyocytes reduced karyokinesis and increased formation of polyploid nuclei
26 bitor of glucosylceramide synthesis, blocked karyokinesis and reduced cyst production in culture.
27 nd chromosome segregation defects, defective karyokinesis, and a failure to complete cytokinesis.
28 ssociated with rampant aneuploidy, defective karyokinesis, and consequently, a failure of cytokinesis
29 ence of mitotic spindles, contractile rings, karyokinesis, and cytokinesis was also recorded.
30 pindles, the formation of contractile rings, karyokinesis, and cytokinesis--were identified; these fe
31 furrow initiation, mitotic spindle function, karyokinesis, and partitioning of intrinsic components a
32 i composed of cells in which cytokinesis and karyokinesis are uncoupled.
33           Thus, CHO1 may not be required for karyokinesis, but it is essential for the proper midzone
34            CHO1 was originally implicated in karyokinesis, but the invertebrate homologues of CHO1 we
35 II is involved in regulating cardiac myocyte karyokinesis by affecting microtubule dynamics.
36 , in spite of the inhibition of cytokinesis, karyokinesis continued, with the result that cells conta
37 ans mutations that cause the same intestinal karyokinesis defect followed by genome sequencing of the
38 cherichia coli can cause the same intestinal karyokinesis defects in WT C. elegans Supporting this mo
39                                          The karyokinesis defects were restricted to intestinal cells
40  with donut-shaped nuclei exhibit defects in karyokinesis, develop aneuploidy, and are often binuclea
41 pression of a non-cleavable SCC1 resulted in karyokinesis failure.
42 onclusion, Lmnb2 expression is essential for karyokinesis in mammalian cardiomyocytes and heart regen
43 ation at E12.5, reflecting the occurrence of karyokinesis in the absence of cytokinesis.
44                This requirement for NM II in karyokinesis is further demonstrated in the HL-1 cell li
45 e rigid matrix facilitated nuclear division (karyokinesis) leading to binucleation, while compliant m
46 , and its depletion causes severe defects in karyokinesis, loss of individual chromosomes, and gross
47         Additionally, we propose that diatom karyokinesis might be controlled by a dual mechanism via
48 dicating that inaccurate endoreplication and karyokinesis occur during MK polyploidization.
49 onse to mutants that perturb cytokinesis and karyokinesis, suggesting interactions between byr4 and t
50 e segregation occur within 8 min followed by karyokinesis to generate haploid gametes.
51 f root-knot nematodes are formed by repeated karyokinesis uncoupled from cytokinesis, whereas the syn
52 s that replicated DNA but failed to complete karyokinesis were found to be CDC20(low)SPG20(low).